ライフサイエンスコーパス: postnatal

1 enotypes including WT at three and ten weeks postnatal.

2 , using an in vitro, brain slice approach in postnatal 15-30 day mice.

3 ith FAdE expression/activity retained in the postnatal 20alphaHSD-positive postnatal X-zone cells.

4 We demonstrate that following postnatal ablation of granule cell progenitors, Nestin-e

5 Consecutive in vivo studies showed that postnatal ablation of LPL in glial fibrillary acidic pro

6 fection, acquisition from maternal flora, or postnatal acquisition from the hospital or community.

7 s, gestational age at birth, infant sex, and postnatal age at magnetic resonance imaging scan.

8 or a higher-protein group at a median (IQR) postnatal age of 7 (6-8) days.

9 cy matched according to sex, gestational and postnatal age, and preimaging serum Cr levels with neona

10 of the mouse PFC, and found that, from early postnatal age, ChCs and BCs differ in laminar location.

11 fic cortical region of conscious mice of any postnatal age, including perinatal and neonatal stages,

12 Since the anomalous current declines with postnatal age, PIEZO2 may contribute to hair cell develo

13 ne anesthesia exposure conducted at an early postnatal age.

14 ates of cell death were seen at the earliest postnatal ages in most regions.

15 Postnatal analysis of male offspring at P63 showed signi

16 Postnatal and adult brain organization dictates strict a

17 persed along the rostral migratory stream in postnatal and adult brains.

18 distribution of cells expressing nNOS in the postnatal and adult female mouse hypothalamus using immu

19 ls and inactivated more slowly than it is in postnatal and adult neurons.

20 pulation by comparing the responses of early postnatal and mature entheses to injury.

21 Coinciding with postnatal ASM maturation, there was a critical time wind

22 men's groups on key antenatal, delivery, and postnatal behaviours in order to understand pathways to

23 Ablation of these postnatal beige adipocytes inhibited cold-induced beige

24 ient sensing may be an essential trigger for postnatal beta-cell maturation and islet development.

25 ial for longitudinal and appositional murine postnatal bone development, which involves proper timing

26 establishing the transcriptional program of postnatal cardiomyocytes during heart development has be

27 were more likely to rate their delivery and postnatal care as good or very good.

28 ormalities causing subsequent disruptions in postnatal cerebellar foliation and lamination.

29 Whether new neurons are added in the postnatal cerebral cortex is still debated.

30 Postnatal circadian disruption was associated with reduc

31 of the nervous system found on imaging with postnatal clinical deficits.

32 Its role during later postnatal CNS development or in response to demyelinatio

33 for detectable regenerative potential in the postnatal cochlea beyond 1 week of age.

34 how all these changes are coordinated during postnatal colonisation, or after the introduction of mic

35 in the spinal cord, which were normalized by postnatal colonization with microbiota from conventional

36 Postnatal contagious yawning and laughing indicate that

37 Our results indicate that the effect of postnatal CRISPR/Cas9-based cardiac gene editing using a

38 hest, and to emphasize factors that optimize postnatal CT imaging in infants.

39 nic neurons is lethal in mice, but mice with postnatal CTCF depletion are less well studied.

40 n [born on embryonic day (E) 29; examined on postnatal day (D) 3 and D7] and term-born (born on E32;

41 ncRNA expression profiles in mouse hearts at postnatal day (P) 1, P7 and P28 via microarray.

42 amidal cortical neurons from male and female postnatal day (P)28 C57BL/6J mice.

43 We explanted testis tissue at postnatal day (P)5.5 and cultured it until P11.5.

44 Six female C57BL/6 mice at postnatal day (PND) 10 were administered a single gavage

45 In the present study, we fed postnatal day (PND) 24 weanling female rats an SPI diet

46 b transgenic (Scnn1b-Tg(+)) mice to SHS from postnatal day (PND) 3-21 and lung phenotypes were examin

47 tic minipumps from gestational day 8 through postnatal day (PND)16.

48 uscularis of the small intestine of newborn (postnatal day 0) wild-type C57BL/6 mice as well as from

49 rly VV Prox1(hi) endothelial organization at postnatal day 0, and this likely underlies the VV defect

50 -cre;ROSA:YFP mice at embryonic day 12.5 and postnatal day 0, representing opposite ends of the notoc

51 nization") of Prox1(hi) endothelial cells by postnatal day 0.

52 hen treated during a critical period between postnatal day 1 and 5, respectively.

53 ried out RNA-seq on 20,424 single cells from postnatal day 1 mouse kidneys, comparing the results of

54 applied chronically, or as a single bolus at postnatal day 1, markedly worsened AAA outcomes in XY in

55 creased within 10 min after oral delivery in postnatal day 1-7 mice.

56 inctive laminar distribution was observed by postnatal day 12 (P12), when we first identified ChCs by

57 mined that the critical period closes around postnatal day 14.

58 pression of both NEEP21/Nsg1 and P19/Nsg2 at postnatal day 16 as well as in the CA1-3 regions of the

59 On postnatal day 17, eyeballs were enucleated.

60 ckout (KO) mice develop hair cell defects by postnatal day 2 (P2) and are deaf by P21-P25.

61 By postnatal day 2 (P2), SCN oscillators displayed the dail

62 arbofuran exposure from gestational day 7 to postnatal day 21 altered expression of genes and transcr

63 ontrolled cortical impact or sham surgery at postnatal day 21, approximating a toddler-aged child.

64 ictable stress (CUS) for 12 days starting at postnatal day 28 (PND28).

65 loses its neurotrophic potential at or near postnatal day 28.

66 tion End Products (RAGE) knockout mice after postnatal day 3, an identical OT increase was not observ

67 t observed at the onset of alveologenesis at postnatal day 3.

68 bryonic day 15.5 fetuses, and persists until postnatal day 30 in cerebellar Purkinje neurons.

69 In cerebellar Purkinje neurons from postnatal day 30 Snord116p-/m+ mice the reduction in neu

70 pre- and perinatally, but is not detected at postnatal day 35 (P35).

71 iet (0.35 mg retinol/kg diet) and treated on postnatal day 4 with an oral dose of either VA (6 mug re

72 upted by thoracic spinal cord transection at postnatal day 5 (P5TX).

73 n of these circuits is abnormal after early (postnatal day 5) removal of descending systems, inducing

74 rocnemius and tibialis anterior in mice from postnatal day 55 to 100 and the results obtained were as

75 ally formed normally, then degenerated after postnatal day 5; large numbers of vesicles invaded the c

76 In mouse (ret)Arl13b(-/-) central retina at postnatal day 6 (P6) and older, outer segments were abse

77 tly developed model of status epilepticus in postnatal day 7 rat pups that results in widespread neur

78 o osteoblasts expressing Col1 and BSP during postnatal day 7-10, when serum levels of thyroid hormone

79 of dividing cells peaks in the CC lining on postnatal day 8 (P8), with division occurring in 19.2% +

80 he region of mouse first mandibular molar at postnatal day 8 (PN8) induced AI-like pathologies when t

81 c nerve from one hind limb was transected at postnatal day 8 to cause paralysis to that limb.

82 Outer segments appeared rapidly at postnatal day six and their appearance coincided with a

83 ed with profound ocular anomalies evident by postnatal days 1-4, including severe cryptophthalmos, mi

84 outcomes between control and PNE rat pups at postnatal days 11-14: 1) the cardiorespiratory responses

85 imulation in young Fmr1 knock-out (KO) mice (postnatal days 14-16), a model of FXS.

86 ue, and/or if urine output was <1 mL/kg/h on postnatal days 2 to 7.

87 us-dependent spatial memory was evaluated on postnatal days 49-60.

88 Inhaled nitric oxide, initiated at 20 ppm on postnatal days 5 to 14 to high-risk preterm infants and

89 nally, we identify a developmental window at postnatal Days 6 to 9 when Muller arbors first colonize

90 pendent endocytosis occurred at calyces from postnatal days 8-15, suggesting its existence before and

91 land and EDEN (Etude des determinants pre et postnatals de la sante et du developpement de l'enfant)

92 as severe chronic airway disease leading to postnatal death.

93 wed maturation defects associated with early postnatal death.

94 Here, we show that postnatal deletion of Cdh1, a cofactor of the anaphase-p

95 Inducible postnatal deletion of Dsp under the transcriptional cont

96 Moreover, premature postnatal deletion of Pofut1 in skeletal myofibers can i

97 ccination strategies (antenatal delivery vs. postnatal delivery) and the benefit of revaccination ove

98 t include embryonic cells and populations of postnatal dental pulp cells; however, these cells are un

99 he number of neural stem cells (NSCs) in the postnatal dentate gyrus (DG), drastically increased peri

100 Finally, we show that postnatal depletion of GLP-1R in the PVN increases food

101 Antenatal and postnatal depression are known to be common and associat

102 n symptoms were measured using the Edinburgh Postnatal Depression Scale and the 9-item Patient Health

103 remodeling of the organ of Corti throughout postnatal development and associated loss of non-sensory

104 ssed in healthy cerebellar tissue throughout postnatal development and in primary cerebellar medullob

105 , little is known about what regulates early postnatal development and maturation of islets.

106 These effects are observed early in postnatal development and progress as animals age.

107 ual rod bipolar cells in mouse retina during postnatal development and quantified the number of dendr

108 TEMENT Acquisition of mature behavior during postnatal development correlates with the arrival and ma

109 Here the authors show that during early postnatal development in mice, NMDAR signaling via activ

110 ut rapidly relocated into the nucleus during postnatal development in mice.

111 N-methyl-D-aspartate (NMDA) receptors during postnatal development leads to epigenetic repression of

112 ABSTRACT: The early postnatal development of functional corticospinal connec

113 rence to lumbar spinal segment L4 during the postnatal development of rats.

114 hondrocytes within the growth plate regulate postnatal development of the long bones.

115 eal a novel role for ACKR2 in regulating the postnatal development of the mammary gland.

116 a role for chemokines and their receptors in postnatal development processes.

117 tion of ERK signaling during early phases of postnatal development, but not in the adult state, resul

118 During postnatal development, crypts multiply via fission, gene

119 During early postnatal development, NFIA labels astrocytes on the day

120 sitive approach to monitoring the IVD during postnatal development.

121 ia at birth, and kyphosis progression during postnatal development.

122 impact of alternative splicing during muscle postnatal development.

123 from in utero development to first months of postnatal development.

124 in significant body weight (BW) during early postnatal development.

125 t the primary cilia to sustain embryonic and postnatal development.

126 tion, as did deletion from astrocytes during postnatal development.

127 ted at different times of day and throughout postnatal development.

128 l circuits in vivo This work describes early postnatal developmental abnormalities in visual and olfa

129 uenced by both prenatal maternal factors and postnatal developmental cues.

130 It is also required for postnatal differentiation of articular chondrocytes and

131 TLs may mediate the function of GWAS loci on postnatal disease susceptibility.

132 We satellite tracked postnatal dispersal in African penguins (Spheniscus deme

133 s while suppressing dopaminergic fate in the postnatal dorsal lineage.

134 owing in vitro expansion, and at ratios >1:3 postnatal:embryonic cells, they inhibit the ability of e

135 Single-cell RNA-seq analysis of pre- and postnatal endolymphatic sacs demonstrates two types of d

136 ht to identify genetic determinants of early postnatal eNO levels and subsequent respiratory symptoms

137 Identifying genetic determinants of postnatal eNO levels might aid in unraveling the role of

138 ts of infant eNO levels might implicate that postnatal eNO metabolism in healthy infants before first

139 The postnatal environment, on the other hand, is characteriz

140 rather continue to mature in response to the postnatal environment.

141 g behavioral adaptation to their anticipated postnatal environment.

142 In this review, we outline the postnatal establishment of the microbiota and maturation

143 The current study suggests that postnatal experience is not required for this preference

144 and preclinical studies indicate that early postnatal exposure to anesthetics can lead to lasting de

145 Postnatal exposure to perfluorinated alkylate substances

146 Prenatal and early postnatal exposures to environmental factors are conside

147 In wild-type mice, there was persistent postnatal expression of embryonic myosin in the small mu

148 AdE expression and delayed regression of the postnatal fetal cortex (X-zone) were detected in both th

149 pression and the resultant regression of the postnatal fetal cortex (X-zone).

150 questions of how the important embryonic and postnatal functions of VEGF can be reconciled with an ap

151 the germline in zygotes, its application in postnatal gene editing remains incompletely characterize

152 ably, when only mesenchyme cells surrounding postnatal GPs were killed, left bone growth was neverthe

153 eases in the gene expression in bone, normal postnatal growth and body weight acquisition compared to

154 However, they retard significantly in postnatal growth and development and all die of multi-or

155 nter prospective telemedicine study) and the Postnatal Growth and Retinopathy of Prematurity (G-ROP)

156 appears to be under matriline influence, and postnatal growth appears to be under patriline intergene

157 role for the lncRNA Haglr in the control of postnatal growth could not be corroborated.

158 essive disorder principally characterized by postnatal growth failure and progressive neurological dy

159 espite unprecedented levels of intervention, postnatal growth faltering persisted, leading to poor nu

160 We noted secular improvements in all postnatal growth parameters (except weight-for-length),

161 Postnatal growth restriction occurred in pups in UPI/RA,

162 aniofacial dysmorphism, cardiac defects, and postnatal growth retardation.

163 Four of the 5 patients also had postnatal growth retardation.

164 There was no substantive deviation in postnatal growth trajectory or adult body morphometry af

165 nsable step for skeletal muscle development, postnatal growth, and regeneration.

166 abundant in the mouse pancreas during early postnatal growth, pregnancy, and in mouse models of panc

167 replication and underlies intra-uterine (and postnatal) growth retardation, chronic neutropenia, and

168 racting oxygen-induced stress and regulating postnatal heart development.

169 amined the effects of LEDGF/p75 depletion in postnatal hematopoiesis and the initiation of MLL leukem

170 euronal PAS domain protein 3 (NPAS3) impairs postnatal hippocampal neurogenesis, while loss of the re

171 multivariable odds ratios for mortality and postnatal HIV transmission for each biomarker using logi

172 tial for bone formation, skeletal growth and postnatal homeostasis.

173 ammary buds and normalizes the embryonic and postnatal hyperbranching phenotype of Eda overexpressing

174 malities in signalling pathways required for postnatal hypertrophic growth were also observed in skel

175 etic stem cells/precursor cells (HSC/PC) and postnatal infections for human-like pB-ALL.

176 pollution exposure indicates less buffer for postnatal influences of factors decreasing telomere leng

177 a posterior SATB2+ domain in developing and postnatal intestinal epithelium.

178 MYD2 deficiency delayed renal cyst growth in postnatal kidneys from Pkd1 mutant mice.

179 Interposed between these two postnatal layers is a hybrid zone, which is composed of

180 ) is more potent at causing brain damage and postnatal lethality than MEX1-44.

181 tor behavior, reduced weight gain, and early postnatal lethality.

182 ules leads to early-onset cyst formation and postnatal lethality.

183 lly to 0, 25 or 50 mg Mn/kg/day during early postnatal life (PND 1-21) or throughout life from PND 1

184 irth in these infants persists through their postnatal life and contributes to adverse pulmonary outc

185 Monocular deprivation (MD) imposed early in postnatal life elicits profound structural and functiona

186 Early postnatal life is a key time for development of the immu

187 mammalian heart undergoes maturation during postnatal life to meet the increased functional requirem

188 Here, we identify a time window in early postnatal life wherein partial amputation culminates in

189 e tissue expansion progresses rapidly during postnatal life, influenced by both prenatal maternal fac

190 ration units, the nephrons, is essential for postnatal life.

191 observed during the critical period in early postnatal life.

192 y, yet few cases are documented in mammalian postnatal life.

193 g-term control of carbohydrate metabolism in postnatal life.

194 Efforts to mitigate postnatal linear growth faltering in low-income and midd

195 These findings show that the early postnatal mammalian intestine functions as an environmen

196 Early postnatal mammals, including human babies, can perform o

197 isolated orofacial movements in utero to the postnatal mastery of suckling at 4 months after birth; a

198 Throughout postnatal maturation of the mouse inner ear, cochlear ha

199 accessibility around cell cycle genes during postnatal maturation.

200 2-3 weeks after birth, suggesting a link to postnatal maturation.

201 Positive associations between postnatal MEP and summation operatorDEHP concentrations

202 discordant for ASD to test whether fetal and postnatal metal dysregulation increases ASD risk.

203 ctional assay with morphological analyses in postnatal mice and investigated the dendritic differenti

204 li2 (DeltaNGli2) in the adipocyte lineage of postnatal mice, we show that targeted activation of Hh s

205 ue for inner ear transgene delivery in early postnatal mice.

206 Cas9-based cardiac genome editing in vivo in postnatal mice.

207 al disability (10/10), speech delay (10/10), postnatal microcephaly (7/9), and dysmorphic features (9

208 et epilepsy, severe intellectual disability, postnatal microcephaly, and movement disorders.

209 Early postnatal Mn exposure caused lasting attentional dysfunc

210 al period that, for cats, peaks at about one postnatal month and declines thereafter so that by about

211 indicator of fat accrual during the first 5 postnatal months.

212 ey cellular and molecular mechanisms driving postnatal motor circuit reorganization and the resulting

213 elrhodopsin, were isolated from the fetal or postnatal mouse bowel and transplanted into the distal c

214 d proteomic profiling was performed on early postnatal mouse corpus callosi, for which limited eviden

215 nsplanted at 8 d of differentiation into the postnatal mouse cortex, are functional and establish lon

216 howing a direct role for this pathway during postnatal mouse development, and it suggests that change

217 caspase-3 (AC3) to quantify apoptosis in the postnatal mouse ventral forebrain and hypothalamus, and

218 re definitive diagnosis made by antenatal or postnatal MRI.

219 S18-SSX expression in the embryonic, but not postnatal, Myf5 lineage share an anatomic location that

220 tant structural and functional properties of postnatal myocardium, including: (1) rod-shaped cardiomy

221 at a similar level as observed in bona fide postnatal myocardium; (3) a positive force-frequency res

222 e to demonstrate a central role of Ptpn11 in postnatal myogenesis of mice.

223 ontrol of cell cycle withdrawal in fetal and postnatal myogenic stem cells, and assign to Ptpn11 sign

224 We lowered the fetal and postnatal n-6/n-3 PUFA ratio exposure in wild-type offsp

225 During postnatal nephrogenesis, Troy(+) cells are present in th

226 ein has a crucial role in various aspects of postnatal neural development.

227 urons and displays dynamic expression during postnatal neurodevelopment.

228 l age, but the effects of CTCF deficiency in postnatal neurons are less well studied.

229 Our findings confirm that Ctcf KO in postnatal neurons causes a neurobehavioral phenotype in

230 our findings reflect the role of paternal or postnatal nicotine exposure, as opposed to maternal or i

231 ns controlling RGP-mediated glia genesis and postnatal NSC behavior.

232 s in turn diminished the embryonic origin of postnatal NSCs, resulting in loss of adult NSCs and defe

233 eage tracing to define a detailed map of the postnatal olfactory epithelium, revealing cell fate pote

234 ighly penetrant syndromic feature with early postnatal onset, we would gain new insights into the mec

235 birth, and these features persist throughout postnatal ontogeny.

236 To determine whether early postnatal oral Mn exposure causes lasting attentional an

237 Teeth undergo postnatal organogenesis relatively late in life and only

238 s on the day of birth, AII amacrine cells at postnatal (P) day 5, and Muller glia by P10, when horizo

239 Studies suggest that a postnatal parental intervention may reduce the incidence

240 he same interneuron subtype over an extended postnatal period and across a range of distances, and pr

241 ight on the critical importance of the early postnatal period for life-long immune homeostasis.

242 s model in mice wherein stress at a specific postnatal period increases susceptibility to adult socia

243 tion of macrophage function during the early postnatal period is indispensable for preventing the dev

244 ng newborn infants at birth and in the early postnatal period underscores the limitations of tasking

245 ile before (P5) and after (P30) the critical postnatal period when mice acquire mature visual functio

246 number of neurons that develop in the early postnatal period, which are thus vulnerable to perturbat

247 ensity of apoptotic neurons during the early postnatal period.

248 s develop in the mouse meninges during early postnatal periods and display remarkable plasticity in a

249 applied to determine the association between postnatal PFAS exposures and antibody concentrations.

250 ompetences, originating already in the early postnatal phase.

251 o developmental cues, in response to diverse postnatal physiologic conditions, and in disease states

252 fter HFD and/or OVX, independent of previous postnatal programming, yet only HFD evoked this effect i

253 Postnatal pulp cells, however, lose all tooth-inducing a

254 histological analysis of the FT NSC niche in postnatal rats and humans.

255 mice, which are characterized of accelerated postnatal retinal angiogenesis.

256 ricytes promote endothelial sprouting in the postnatal retinal vasculature.

257 d [Ca(2+) ]i signals in adult, but not early postnatal, RGCs.

258 underlie autosomal dominant SCT, identify a postnatal role for embryonic myosin and suggest that alt

259 velopment, beta cells further adapt to their postnatal role through functional maturation.

260 genetically defined mouse models, studies of postnatal sequelae associated with CLP have been hampere

261 dicating lagged unfavorable effects of early postnatal SHS exposure in later life.

262 Postnatal size changes in most vertebrates are unidirect

263 cle satellite cells (PSCs) are important for postnatal skeletal muscle growth, and Notch1 signaling p

264 Mice lacking Klhl31 exhibited stunted postnatal skeletal muscle growth, centronuclear myopathy

265 pair myogenesis but that it is necessary for postnatal skeletal muscle growth.

266 ata reveals a programmed cell fate change in postnatal skeleton and unravels a regulatory mechanism u

267 However, specific features of postnatal spinal circuit development remain poorly under

268 We focused on the postnatal splicing transitions of the three calcineurin

269 elective loss of VTA DA neurons in the early postnatal stage.

270 Pcdh19 and Ncdh in limbic structures at four postnatal stages of C57BL/6J mice by using double-label

271 rotein, in the thalamus and the cortex, from postnatal stages to adult stages, and in the cerebellum

272 cells and extends cortical neurogenesis into postnatal stages.

273 We show that MDM cells contribute to the postnatal stromal cells at the dorsal aspect of the pros

274 rents (21.5% men, 78.5% women) completed the postnatal survey.

275 ndents answering individual questions on the postnatal surveys, 10958 mothers (91.0%) and 2950 father

276 on in the atrophied thymus by utilizing both postnatal TEC-defective (resulting from FoxN1-floxed con

277 levels of mTORC1 signaling is essential for postnatal tendon development and maturation.

278 nsively, the molecular mechanisms regulating postnatal tendon development are not well understood.

279 e we examine the role of mTORC1 signaling in postnatal tendon development using mouse genetic approac

280 ess, indicating that mTORC1 is necessary for postnatal tendon development.

281 xp2-null mutants also show a loss of ITCs at postnatal time points, suggesting that Foxp2 may functio

282 dSPNs of Sox8-EGFP BAC mice is maintained at postnatal timepoints.

283 uptly in the skin during a defined window of postnatal tissue development.

284 iferation and differentiation is crucial for postnatal tissue homoeostasis and organogenesis.

285 longitudinal decline of function from early postnatal to adult ages in female Mecp2 heterozygotes of

286 Here, we report that prenatal to early postnatal treatment with a ciliary neurotrophic factor (

287 morbidity (defined as severe brain injury on postnatal ultrasonography, severe retinopathy of prematu

288 of the uterus and is a critical regulator of postnatal uterine gland differentiation in mice.

289 62) to 315 per 100,000 (IQR, 260-370), while postnatal vaccination produced a minimal reduction, with

290 eural-specific deletion of paxillin are also postnatal viable, but show evidence of a cortical neuron

291 12 and is essential for multiple aspects of postnatal visual system development.

292 ecruited at birth from the neonatal unit and postnatal wards of the Royal Women's Hospital, Melbourne

293 xis or treatment (prenatal, intrapartum, and postnatal) was 22.4% in 2002-2005 and 31.8% in 2010-2013

294 es of BG microglia emerged during the second postnatal week and were re-established following genetic

295 discrete cell clusters emerge over the first postnatal week, yielding an identifiable modular network

296 ring development, until the end of the third postnatal week.

297 ciceptive spinal activity in the first three postnatal weeks.

298 elphia Retinopathy of Prematurity (CHOP-ROP) postnatal weight gain predictive model are 2 approaches

299 robial dynamics during critical prenatal and postnatal windows, we used high-resolution 16S rRNA mark

300 etained in the postnatal 20alphaHSD-positive postnatal X-zone cells.