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1 cutaneously (s.c.) at 2nd,4th, 6th, 8th,10th postnatal day.
2 for NaV1.4 did not survive beyond the second postnatal day.
3 n extremely preterm infants during the first postnatal days.
4 e for more than 48 hours during the first 28 postnatal days.
5 and 1,525,739 drug exposures in the first 28 postnatal days.
6 Here, we show that tau haploinsufficiency in postnatal day 0 (P0) heterozygous (Mapt(+/-)) pups, but
7  hippocampal CA1 region pyramidal neurons at postnatal day 0 (P0) or day 21 (P21) and measured synapt
8 ive dose for 2 hrs, and pups were studied at postnatal day 0 (P0) or P30.
9 OC and cochlear nucleus at various ages from postnatal day 0 (P0) to P90 in control hearing and neona
10  and D2R expression at the promoter level in postnatal day 0 (PD0) Drd1a-tdTomato/Drd2-GFP BAC transg
11     Plethysmographic recordings in vivo from Postnatal Day 0 rats demonstrated that CX1739 (10 mg/kg)
12                                  Older NPCs (postnatal day 0) exit the progenitor niche at a higher r
13 uscularis of the small intestine of newborn (postnatal day 0) wild-type C57BL/6 mice as well as from
14  global homozygous knockout (KO) mice die at postnatal day 0, and conditional deletion of Snrk in car
15 rly VV Prox1(hi) endothelial organization at postnatal day 0, and this likely underlies the VV defect
16 -cre;ROSA:YFP mice at embryonic day 12.5 and postnatal day 0, representing opposite ends of the notoc
17                  In wild-type neonatal mice (postnatal day 0-1), we show that intravenous injection o
18   In experiments on CA3 pyramidal neurons on postnatal day 0-6 rat hippocampal slices, we detected ro
19 nization") of Prox1(hi) endothelial cells by postnatal day 0.
20 evels of apoptotic proteins were elevated at postnatal day 0.
21     Prox1DTA mice injected with tamoxifen at postnatal days 0 (P0) and P1 show significant DC and out
22 exposed to >90% hyperoxia or room air during postnatal days 0 to 7, and then all pups were returned t
23 volume postnatally; growth is fastest during postnatal days 0-4 (P0-P4), preceding most myelination.
24                                Specifically, postnatal days 0-7 in mice show no significant change in
25 ion of a neutralizing anti-BMP10 antibody at postnatal day 1 (P1) and P3 in these pups exacerbated th
26 ce at diverse postnatal stages, ranging from postnatal day 1 (P1) to 17 months.
27              Motor deficits were observed on postnatal day 1 (P1) when WM tracts were unmyelinated.
28 tically tested, including intraperitoneal at postnatal day 1 (P1), intramuscular at P12, and retro-or
29 , apical and high-frequency basal neurons at postnatal day 1 (P1)-P3 were predominantly slowly accomm
30  during gestation (GLP) or pups suckled from postnatal day 1 (PN1) to PN11 (E-UN), or PN11 to PN22 (L
31 hen treated during a critical period between postnatal day 1 and 5, respectively.
32 ried out RNA-seq on 20,424 single cells from postnatal day 1 mouse kidneys, comparing the results of
33 somal repeat length increases from 190 bp at postnatal day 1 to 206 bp in the adult retina.
34 )) mice were exposed to hyperoxia (75%) from postnatal day 1 to 7.
35  cord was normal in presymptomatic SMA mice (postnatal day 1), but failed to match subsequent postnat
36                   We focused our analyses on postnatal day 1, a late stage of cortical neurogenesis w
37 applied chronically, or as a single bolus at postnatal day 1, markedly worsened AAA outcomes in XY in
38 creased within 10 min after oral delivery in postnatal day 1-7 mice.
39 after 15 days of treatment that started from postnatal day 1.
40 ) mice develop normally, but fail to survive postnatal day 1.
41 Carotid arteries were isolated from newborn (postnatal day 1; P1), postnatal day 7 (P7) and postnatal
42 pression and AS that occur primarily between postnatal days 1 and 28.
43 ed with profound ocular anomalies evident by postnatal days 1-4, including severe cryptophthalmos, mi
44 vels in their oligodendrocytes, beginning at postnatal day 10 (P10) and persisting through adulthood.
45                            Ultrastructure of postnatal day 10 (P10) central (ret)Arl13b(-/-) photorec
46 of approximately 30 dB was induced either at postnatal day 10 or after sexual maturation.
47  results in mice displaying rapid tremors at postnatal day 10, followed by death at postnatal week 3.
48 postnatal day 30 and compared the results to postnatal day 10-21 animals.
49 ed in cell-fate determined photoreceptors at postnatal day 10.
50 re of the root to elongate, were observed by postnatal day 10.
51   The striata were isolated from the pups at postnatal days 10 (P10) and P21.
52 sed the occurrence of distal necrosis beyond postnatal day 100, and induced more complete phenotypic
53  reared with bilateral earplugs, either from postnatal day 11 (P11) to postnatal day 23 (P23) (a mani
54 r to identify the miRNAs in adult and young (postnatal day 11/12) Muller glia of the neural retina, w
55 cific decreases in spine density starting at postnatal day 11; however, a compensatory enhancement of
56  As previously reported, early inactivation (postnatal days 11 and 12) of Pkd1 resulted in PKD develo
57 outcomes between control and PNE rat pups at postnatal days 11-14: 1) the cardiorespiratory responses
58 g postnatal development and is absent beyond postnatal day 110 if mice are raised in standard cages (
59 ression of glucocorticoid receptors (GRs) at postnatal day 12 (P12) and P60, finding a significantly
60 inctive laminar distribution was observed by postnatal day 12 (P12), when we first identified ChCs by
61 ongly upregulated in the cochlea from around postnatal day 12 (P12), which corresponds to the onset o
62 IHCs, which normally occurs at approximately postnatal day 12, was partially prevented.
63 ion of IHCs, which normally occurs at around postnatal day 12, was partially prevented.
64 I/III pyramidal cells of the mouse juvenile (postnatal day 12-27) visual cortex.
65 espiratory development occurs in rats around postnatal days 12-13, when enhanced inhibition dominates
66 yer 5 of the developing mouse visual cortex (postnatal days 12-21).
67 ed from mice following the onset of hearing (postnatal days 12-21).
68                          We demonstrate that postnatal-day-12-13 rats (equivalent to human newborns)
69 y postnatal period in the mouse; however, at postnatal days 13-15, leptin causes membrane depolarizat
70 p2(ZFN/+ )rats were significantly reduced by postnatal day 14 and 21, respectively.
71 s milder in mice given doxycycline after age postnatal day 14 and did not develop after postnatal day
72 dition, disturbed myelination was evident in postnatal Day 14 HdhQ250 mouse brain, including reduced
73                    Intranasal inoculation in postnatal day 14 mice with VSVDeltaG-CHIKV or VLV evoked
74 ly 70-fold increase in retrotransposition in postnatal day 14 Mov10l1(-/-) germ cells compared with t
75 a critical period from embryonic day 15.5 to postnatal day 14 was accompanied by respiratory distress
76 a non-linear trajectory reaching maturity by postnatal day 14, with two developmental transitions occ
77 -71 from gestation day 6 through sampling on postnatal day 14.
78 mined that the critical period closes around postnatal day 14.
79                                      Between postnatal days 14 and 18, synapse density significantly
80 imulation in young Fmr1 knock-out (KO) mice (postnatal days 14-16), a model of FXS.
81 ess this question, we analyzed synapses from postnatal day 15 (P15) and adult rat hippocampus that ha
82 e activates the IGF-1/IGF-1-R/Akt pathway on postnatal day 15 and initiates a brief but intense proli
83      Also, the response to cardiac injury at postnatal day 15 is intermediate between that observed a
84  development and synaptic transmission after postnatal day 15.
85 st of cardiac myocyte cell cycle activity at postnatal day 15.
86  been shown to appear adult-like as early as postnatal day 16 (P16) in the rat pup, just after eye op
87 pression of both NEEP21/Nsg1 and P19/Nsg2 at postnatal day 16 as well as in the CA1-3 regions of the
88                                          All postnatal day 16 mice given intranasal VSV-12'GFP surviv
89                                  However, by postnatal day 16, mouse MG lose neurogenic capacity, des
90 poor left ventricle contraction and death by postnatal day 16, suggesting that CaM inhibition of RyR2
91  impact (CCI) animal model of pediatric TBI (postnatal day 16-17) it was demonstrated that injury res
92 ide of their nest for the first time, around postnatal day 16-18 (P16-P18).
93 helial cells of the proximal epididymis from postnatal day 17 (P17) onward.
94 n increased number of neovessels, peaking at postnatal day 17 (P17; P=0.001).
95 ectron microscopic examination of retinas at postnatal day 17 revealed the presence of vacuolar struc
96                                           On postnatal day 17, eyeballs were enucleated.
97 Insensitivity to low SMN emerged abruptly at postnatal day 17, which coincided with establishment of
98 scular endothelial cells (ECs) in retinas at postnatal day 18 (p18), when pathological angiogenesis i
99 ospinal populations in Fischer 344 rats from postnatal day 18 through 75 using retrograde tracer inje
100 (NG2(+)) progenitors lagged in maturation by postnatal day 18, when the mouse null mutation was letha
101                                           At postnatal day 180 (P180), FF and S NMJs of SOD1 already
102 ckout (KO) mice develop hair cell defects by postnatal day 2 (P2) and are deaf by P21-P25.
103     Here we identify the period ranging from postnatal day 2 (P2) to P11 as 5-HT sensitive, with 5-HT
104                                           By postnatal day 2 (P2), SCN oscillators displayed the dail
105 r weight beta1-catenin increased 2-3-fold in postnatal day 2 lenses of DKO lenses compared with WT le
106  15 is intermediate between that observed at postnatal days 2 and 21, further suggesting persistence
107 ue, and/or if urine output was <1 mL/kg/h on postnatal days 2 to 7.
108       Pups were injected with terbutaline on postnatal days 2-5.
109 discrete window in development, beginning at postnatal day 20 in mice.
110 dence was found for GA treatment (started on postnatal day 20).
111  Synaptic transmission in the LSO matures by postnatal day 20, with EPSCs and IPSCs having fast kinet
112 embryonic day 9.5 (primordial heart tube) to postnatal day 21 (mature heart).
113 stnatal day 1; P1), postnatal day 7 (P7) and postnatal day 21 (P21) mice and assessed in a pressure m
114                      During development from postnatal day 21 (P21) to P35, GABAA receptor function c
115  are evident in arg(-/-) animals as early as postnatal day 21 (P21), a time that precedes any observa
116 d and matured at an accelerated pace through postnatal day 21 (P21).
117 arbofuran exposure from gestational day 7 to postnatal day 21 altered expression of genes and transcr
118 SC frequency compared with wild-type SPNs in postnatal day 21 mice.
119 ontrolled cortical impact or sham surgery at postnatal day 21, approximating a toddler-aged child.
120 phaly and corpus callosum deficiency, and by postnatal Day 21, microcephaly; the mice died at an earl
121             Upon tamoxifen administration at postnatal day 21, the floxed mHtt-exon1 was removed and
122 /y)) were noticeably symptomatic as early as postnatal day 21, with most dying by postnatal day 55, w
123 cope, a total of 685,673 cells in 56 mice at postnatal day 21.
124 orated all morphological defects in liver by postnatal day 21.
125 elid opening in mice, remaining stable after postnatal day 21.
126 ined around CA2 until reaching adult form by postnatal day 21.
127 seizures, muscle stiffness, and morbidity by postnatal day 21.
128  -0.382 c/d, but 0.037 c/d in T(-/-) mice at postnatal day 210 (P210).
129 plugs, either from postnatal day 11 (P11) to postnatal day 23 (P23) (a manipulation previously found
130 that starts prenatally and continues through postnatal day 25 has a major impact on the structure of
131 infant nerve injured mice reach adolescence (postnatal day 25-30), the dorsal horn immune profile swi
132  was significantly reduced in all regions at postnatal day 25.
133 ing within weeks, whereas late inactivation (postnatal days 25-28) resulted in PKD developing in mont
134 ictable stress (CUS) for 12 days starting at postnatal day 28 (PND28).
135 /l) or water for 28 days during adolescence (postnatal day 28-55; P28-P55) or adulthood (P67-P94).
136  loses its neurotrophic potential at or near postnatal day 28.
137 lescent intermittent ethanol (AIE) exposure (postnatal days 28-42) by vapor inhalation on different a
138 thymocytes of both the mother and fetuses on postnatal day 3 (gestation day 17) were studied.
139       Cortical fibers began to enter dLGN at postnatal day 3 (P3) to P4, a time when retinogeniculate
140 defect of CH in cDKO first became evident on postnatal day 3 (P3), a time point when CH forms and gro
141 s extracellular multielectrode recordings in postnatal day 3 (P3)-P5 rat primary somatosensory cortex
142 lls showed signs of degeneration as early as postnatal Day 3 (P3); thereafter, blebs devoid of actin
143 tion End Products (RAGE) knockout mice after postnatal day 3, an identical OT increase was not observ
144 t observed at the onset of alveologenesis at postnatal day 3.
145 ative effects on ultrasonic vocalizations at postnatal day 3.
146 the S-phase marker bromodeoxyuridine between postnatal days 3 and 14 showed that much of this populat
147  developmental transitions occurring between postnatal days 3-5 and 9-11.
148                                           On postnatal day 30 (P30) mutant mice had lower SGN density
149 rdings for RVM neurons in animals older than postnatal day 30 and compared the results to postnatal d
150 bryonic day 15.5 fetuses, and persists until postnatal day 30 in cerebellar Purkinje neurons.
151          In cerebellar Purkinje neurons from postnatal day 30 Snord116p-/m+ mice the reduction in neu
152 hed C57BL/6J (wild-type, WT) control mice at postnatal day 30, 4 months, and 16 months of age.
153 stem are linked with high ABP in both young (postnatal day 30-58) and adult SHRs (4-6 months).
154  the high ABP are measureable in young SHRs (postnatal day 30-58) and become greater in adult SHRs.
155 ng acuity in treated animals is unaltered at postnatal day 30.
156 f saline or the NMDAR antagonist MK-801 from postnatal day 35 (P35) to P40.
157 /kg, bi-daily, for 15 d) to male adolescent [postnatal day 35 (P35) to P49] C57BL/6 mice.
158 pre- and perinatally, but is not detected at postnatal day 35 (P35).
159 pamine projections do not mature until after postnatal day 35 in the rat.
160 e postnatal day 14 and did not develop after postnatal day 35.
161 as a retrograde axonal tracer in adolescent (postnatal day 39) and early adult (8-9-week-old) rats.
162                         Serotonin neurons at postnatal day 4 (P4) were hyperexcitable.
163 mouse forebrain on the day of birth (P0), on postnatal day 4 (P4), and on P21.
164 iet (0.35 mg retinol/kg diet) and treated on postnatal day 4 with an oral dose of either VA (6 mug re
165 ed that PCP4 immunostaining first appears at postnatal day 4-5 and becomes successively more refined
166 endymal cilia, resulting in hydrocephalus by postnatal day 4.
167 ressed in preodontoblast and odontoblasts at postnatal day 4.
168 la circuitry and anxiety-related behavior by postnatal day 45 (P45), when AEA levels begin to decreas
169 us-dependent spatial memory was evaluated on postnatal days 49-60.
170  onset of the critical period of plasticity [postnatal day 5 (P5)], at its peak (P26), and at the mat
171 upted by thoracic spinal cord transection at postnatal day 5 (P5TX).
172          A second administration of PMO25 at postnatal day 5 (PND5) demonstrated an additive effect o
173 nt with dantrolene in 4L;C* mice starting at postnatal day 5 delayed neurological pathology and prolo
174  volume were reduced in all mutant mice from postnatal day 5 onward.
175 n of these circuits is abnormal after early (postnatal day 5) removal of descending systems, inducing
176 e in an autosomal recessive manner, dying by postnatal day 5-6.
177 o C57BL/6J dams from gestational day 11.5 to postnatal day 5.
178 ally formed normally, then degenerated after postnatal day 5; large numbers of vesicles invaded the c
179 supporting presensory spiking formed between postnatal days 5 (P5) and P7, including Ankyrin-G, NaV1.
180 ing positive pressure respiratory support on postnatal days 5 to 14 improves the rate of survival wit
181 Inhaled nitric oxide, initiated at 20 ppm on postnatal days 5 to 14 to high-risk preterm infants and
182  or positive pressure respiratory support on postnatal days 5 to 14.
183 rocnemius and tibialis anterior in mice from postnatal day 55 to 100 and the results obtained were as
184 arly as postnatal day 21, with most dying by postnatal day 55, while females lacking one copy of Mecp
185  In mouse (ret)Arl13b(-/-) central retina at postnatal day 6 (P6) and older, outer segments were abse
186 ebroventricularly in the right hemisphere on postnatal day 6 at 30min prior to the first dose of Dex
187  h of systemic kainic acid administration at postnatal day 6, mRNA levels of Fgf9, Fgf10, Fgfr2c, and
188 X2(+) cell numbers and retinal morphology at postnatal day 6.
189 er numbers than when cells were generated at postnatal day 6.
190 nally, we identify a developmental window at postnatal Days 6 to 9 when Muller arbors first colonize
191                             Surprisingly, by postnatal day 60 it also caused CA1 histological disorga
192 DE10A, PDE11A4 expression begins very low at postnatal day 7 (P7) and dramatically increases until P2
193 isolated from newborn (postnatal day 1; P1), postnatal day 7 (P7) and postnatal day 21 (P21) mice and
194 , using NMDARs in dopaminergic neurones from postnatal day 7 (P7) rats as a model system, we characte
195 erving neurovascular integrity, we subjected postnatal day 7 (P7) rats depleted of microglial cells,
196 d norepinephrine (NE) were dysregulated from Postnatal day 7 (P7) to P21 in En2-KO, though NE exhibit
197  the mouse lateral superior olive (LSO) from postnatal day 7 (P7) to P96 using voltage-clamp and audi
198 d in the rat STN throughout development [age postnatal day 7 (P7)-P60] and in the adult (age P120).
199 to-parahippocampal projections is present by postnatal day 7 (P7).
200                                      Between postnatal day 7 and 21, VGCCs formed variable sized clus
201    Phytosterols in the LEs and in plasma (on postnatal day 7 and day 14) were measured by gas chromat
202     Here we show that rats exposed to GAs at postnatal day 7 display a lasting reduction in inhibitor
203   The human transgenic tau was detected from postnatal day 7 onward in motoneurons, axons in the scia
204            Perturbing axon projection beyond postnatal day 7 permanently disrupts targeting specifici
205 tly developed model of status epilepticus in postnatal day 7 rat pups that results in widespread neur
206 the present study showed that inoculation on postnatal day 7 with the porcine reproductive and respir
207                                           At postnatal day 7, when litters showed clear FAA, pups fro
208 o osteoblasts expressing Col1 and BSP during postnatal day 7-10, when serum levels of thyroid hormone
209 l progenitor cells from embryonic day 9.5 to postnatal day 7.
210 lles that exhibits peak expression levels at postnatal day 7.
211 nel beta subunit exhibited NAFLD as early as postnatal day 7.
212 d intake, as well as growth velocity between postnatal days 7 and 28 (adjusted for GA and birth weigh
213 ced NMDAR hypofunction in infant mice during postnatal days 7-11, followed by testing fear memory spe
214                                 In contrast, postnatal days 7-20 show a significant increase in volum
215  regions remained stable across development (postnatal days 7-23), there was a developmental emergenc
216  or Scx(flx/flx)] littermates were killed at postnatal days 7-56 (P7-P56).
217 nied by overt astrogliosis (at approximately postnatal days 70-80).
218                                           At postnatal day 8 (P8), theta is expressed as brief bursts
219  of dividing cells peaks in the CC lining on postnatal day 8 (P8), with division occurring in 19.2% +
220 he region of mouse first mandibular molar at postnatal day 8 (PN8) induced AI-like pathologies when t
221  activity occur between embryonic day 18 and postnatal day 8 and originate in pacemaker circuits in t
222  (phosphatase and tensin homolog) protein at postnatal day 8 in mice harboring Nf1 haploinsufficiency
223 c nerve from one hind limb was transected at postnatal day 8 to cause paralysis to that limb.
224 en different ages (embryonic days 15 and 18, postnatal day 8, and adult), we reveal the dynamic and c
225 bistratified morphology was first visible at postnatal day 8, reaching the adult shape at P13, around
226    However, the pups develop overt ataxia by postnatal day 8-10 and die shortly thereafter.
227 pendent endocytosis occurred at calyces from postnatal days 8-15, suggesting its existence before and
228 isingly, the Cre(+) mice became cachectic by postnatal day 80 due to bilateral GCT.
229 ed for two cycles before being euthanized at postnatal day 9.
230 ), which simulates a bacterial infection, on postnatal day 9.
231 ng natural interactions with their mother on postnatal days approximately 12-19.
232 n [born on embryonic day (E) 29; examined on postnatal day (D) 3 and D7] and term-born (born on E32;
233 profiled the F2 serum metabolome at weaning [postnatal day (d)21; n = 76] and adulthood (d160; n = 12
234     Cataract development was seen at various postnatal days in the majority of mice expressing active
235 tal lipidomic remodeling during the first 60 postnatal days, including progressive accumulation of ch
236 intraventricular hemorrhage during the first postnatal days is possible from bedside measures of brai
237 a single dose of 80 mg ENU/kg body weight on postnatal day one.
238 ber of Drd1-positive cells retaining BrdU in postnatal day (P) 0 Rarb(-/-) striatum.
239  tracts of myelin-deficient shiverer mice on postnatal day (P) 0, P7, and P21.
240 ed 4-hydroxytamoxifen (4OHT) to back skin at postnatal day (P) 1 and P2.
241 s in the brains of male and female mice from postnatal day (P) 1 to P11.
242 ncRNA expression profiles in mouse hearts at postnatal day (P) 1, P7 and P28 via microarray.
243  perinatal ages from embryonic day (E) 17 to postnatal day (P) 11 and found that cell death peaks jus
244 ) declines during aging and is absent beyond postnatal day (P) 110 when mice are raised in standard c
245 es from individual developing astroglia from postnatal day (P) 14 to P26, when astroglia undergo dram
246 enesis, increasing NG2 progenitor cells from postnatal day (P) 15, then O4+ and CC1+ cells at P30 and
247  subretinally injected in rpe65(-/-) mice at postnatal day (P) 16 and evaluated at 15 months PI.
248  buffered Ca(2+) diffusion and SV release in postnatal day (P) 16-19 mouse calyces, as their release
249 from hippocampal and neocortical slices from postnatal day (P) 2-P15 mice, photostimulation caused de
250 ild-type mice; these changes were present at postnatal day (P) 20 for PV neurons and P40 for WFA/PNN+
251    Apical dendritic regression occurred from postnatal day (P) 28, dendritic spine loss from P21, and
252                              When induced at postnatal day (P) 28, Sox2-CreER activity was exclusive
253 ical activity patterns of mice enucleated at postnatal day (P) 45, 90, or 120.
254 ryptophan hydroxylase 2 (Tph2)(+) neurons by postnatal day (P) 5 through P12: approximately one-third
255 at the end of the first postnatal week, from postnatal day (P) 5 to P9, after the end of neurogenesis
256                                           At postnatal day (P) 56, the population of parvalbumin-posi
257 aKO kidneys were hypoplastic and not cystic, postnatal day (P) 7 mutants had proximal tubular-derived
258                                           At postnatal day (P) 7, immunoelectron microscopy revealed
259 n GABAA signalling matures, as occurs beyond postnatal day (P) 7.
260 OZ) causes a neuropathy at 90 d after birth [postnatal day (P) 90], with a subsequent spontaneous reg
261    We used cultures of cortical neurons from postnatal day (P)0-P2 golden Syrian hamsters (Mesocricet
262 ry periphery in H and CF pups manipulated at postnatal day (P)1, P5, or P9.
263 ent before eye-opening, earliest detected in postnatal day (P)11 animals.
264              In acute cerebellar slices from postnatal day (P)12-14 mice, light-evoked EPSCs were lar
265 emistry for ICAM-5 in mouse visual cortex at postnatal day (P)14, a period of intense synaptogenesis,
266 derwent MD during the pre-CP [eye-opening to postnatal day (p)17] or CP (p22-p25), and FS-->SP synapt
267 termined in two distinct age groups of rats: postnatal day (P)25 to P40 and P50 to P80.
268 amidal cortical neurons from male and female postnatal day (P)28 C57BL/6J mice.
269                We explanted testis tissue at postnatal day (P)5.5 and cultured it until P11.5.
270 ndoplasmic reticulum was evident as early as postnatal day (P)6.
271 xhibited a mild decrease in vascular loss at postnatal day (P)8 compared with age- and strain-matched
272  Upregulation of GLT-1 was completed only at postnatal days (P) P20-25 and was thus delayed by about
273 excitability in offspring during infancy (at postnatal days (P)10, 14, 17 and 21), preadolescence (da
274 S1) neurons (E15.5), and calretinin neurons (postnatal day [P]0).
275 oid body underwent a rapid cell loss between postnatal days P0 and P4, shortly after circuit formatio
276 that a history of WIN exposure during early (postnatal days - P35-40) or mid-(P40-45) adolescence, bu
277             During the first postnatal week (postnatal days P4-P7), V1 was not visually responsive an
278            Slices were obtained from rats at postnatal days (P7-P14) and maintained in organotypic cu
279 radigm, was conducted every thirty days from postnatal day (PD) 30 to PD 180.
280             On day one of the CPFE paradigm, postnatal day (PD) 31 rats (+/-1) were pre-exposed to Co
281  binge-like ethanol treatment of rat pups on postnatal days (PD) 4-9, we found that maturation of GAB
282                        Adolescent male rats (postnatal day [PD] 35) received two ketamine (0, 5, 10,
283  consecutive days either during adolescence (postnatal days [PD] 35-49) or adulthood (PD 65-79).
284 y acid (HIGH n-3) compared with the CTR from postnatal day (PN) 2 to 42].
285                   Six female C57BL/6 mice at postnatal day (PND) 10 were administered a single gavage
286                                           By postnatal day (PND) 12, communities separated based on e
287          Transmission electron microscopy of postnatal day (PND) 14 Rho(P23H/+) mouse retina revealed
288 nitiated orally from embryonic day (ED) 6 to postnatal day (PND) 15.
289                 In the present study, we fed postnatal day (PND) 24 weanling female rats an SPI diet
290 or was studied using the forced-swim test on postnatal day (PND) 25 in rats either weaned on PND 21,
291 b transgenic (Scnn1b-Tg(+)) mice to SHS from postnatal day (PND) 3-21 and lung phenotypes were examin
292 estation day 0 of the parental generation to postnatal day (PND) 6 of the F2 generation to a realisti
293 varies were removed from the F1 offspring on postnatal day (PND)-1 and various analyses were performe
294 tic minipumps from gestational day 8 through postnatal day (PND)16.
295 fly exposed to 0.1 to 5,000 mug BPA/kg BW on postnatal days (PND) 1, 3, and 5.
296 mbient Particle System or to filtered air on postnatal days (PNDs) 4-7 and 10-13, and the animals wer
297                                           At postnatal-day seven, rat pups underwent moderate or seve
298           Outer segments appeared rapidly at postnatal day six and their appearance coincided with a
299 sis of the effect of feeding in the first 14 postnatal days with own mother's milk, with or without h
300  at three developmental ages (15, 30, and 45 postnatal days) with array tomography three-dimensional

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