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1 cutaneously (s.c.) at 2nd,4th, 6th, 8th,10th postnatal day.
2 for NaV1.4 did not survive beyond the second postnatal day.
3 n extremely preterm infants during the first postnatal days.
4 e for more than 48 hours during the first 28 postnatal days.
5 and 1,525,739 drug exposures in the first 28 postnatal days.
6 Here, we show that tau haploinsufficiency in postnatal day 0 (P0) heterozygous (Mapt(+/-)) pups, but
7 hippocampal CA1 region pyramidal neurons at postnatal day 0 (P0) or day 21 (P21) and measured synapt
9 OC and cochlear nucleus at various ages from postnatal day 0 (P0) to P90 in control hearing and neona
10 and D2R expression at the promoter level in postnatal day 0 (PD0) Drd1a-tdTomato/Drd2-GFP BAC transg
11 Plethysmographic recordings in vivo from Postnatal Day 0 rats demonstrated that CX1739 (10 mg/kg)
13 uscularis of the small intestine of newborn (postnatal day 0) wild-type C57BL/6 mice as well as from
14 global homozygous knockout (KO) mice die at postnatal day 0, and conditional deletion of Snrk in car
15 rly VV Prox1(hi) endothelial organization at postnatal day 0, and this likely underlies the VV defect
16 -cre;ROSA:YFP mice at embryonic day 12.5 and postnatal day 0, representing opposite ends of the notoc
18 In experiments on CA3 pyramidal neurons on postnatal day 0-6 rat hippocampal slices, we detected ro
21 Prox1DTA mice injected with tamoxifen at postnatal days 0 (P0) and P1 show significant DC and out
22 exposed to >90% hyperoxia or room air during postnatal days 0 to 7, and then all pups were returned t
23 volume postnatally; growth is fastest during postnatal days 0-4 (P0-P4), preceding most myelination.
25 ion of a neutralizing anti-BMP10 antibody at postnatal day 1 (P1) and P3 in these pups exacerbated th
28 tically tested, including intraperitoneal at postnatal day 1 (P1), intramuscular at P12, and retro-or
29 , apical and high-frequency basal neurons at postnatal day 1 (P1)-P3 were predominantly slowly accomm
30 during gestation (GLP) or pups suckled from postnatal day 1 (PN1) to PN11 (E-UN), or PN11 to PN22 (L
32 ried out RNA-seq on 20,424 single cells from postnatal day 1 mouse kidneys, comparing the results of
35 cord was normal in presymptomatic SMA mice (postnatal day 1), but failed to match subsequent postnat
37 applied chronically, or as a single bolus at postnatal day 1, markedly worsened AAA outcomes in XY in
41 Carotid arteries were isolated from newborn (postnatal day 1; P1), postnatal day 7 (P7) and postnatal
43 ed with profound ocular anomalies evident by postnatal days 1-4, including severe cryptophthalmos, mi
44 vels in their oligodendrocytes, beginning at postnatal day 10 (P10) and persisting through adulthood.
47 results in mice displaying rapid tremors at postnatal day 10, followed by death at postnatal week 3.
52 sed the occurrence of distal necrosis beyond postnatal day 100, and induced more complete phenotypic
53 reared with bilateral earplugs, either from postnatal day 11 (P11) to postnatal day 23 (P23) (a mani
54 r to identify the miRNAs in adult and young (postnatal day 11/12) Muller glia of the neural retina, w
55 cific decreases in spine density starting at postnatal day 11; however, a compensatory enhancement of
56 As previously reported, early inactivation (postnatal days 11 and 12) of Pkd1 resulted in PKD develo
57 outcomes between control and PNE rat pups at postnatal days 11-14: 1) the cardiorespiratory responses
58 g postnatal development and is absent beyond postnatal day 110 if mice are raised in standard cages (
59 ression of glucocorticoid receptors (GRs) at postnatal day 12 (P12) and P60, finding a significantly
60 inctive laminar distribution was observed by postnatal day 12 (P12), when we first identified ChCs by
61 ongly upregulated in the cochlea from around postnatal day 12 (P12), which corresponds to the onset o
65 espiratory development occurs in rats around postnatal days 12-13, when enhanced inhibition dominates
69 y postnatal period in the mouse; however, at postnatal days 13-15, leptin causes membrane depolarizat
71 s milder in mice given doxycycline after age postnatal day 14 and did not develop after postnatal day
72 dition, disturbed myelination was evident in postnatal Day 14 HdhQ250 mouse brain, including reduced
74 ly 70-fold increase in retrotransposition in postnatal day 14 Mov10l1(-/-) germ cells compared with t
75 a critical period from embryonic day 15.5 to postnatal day 14 was accompanied by respiratory distress
76 a non-linear trajectory reaching maturity by postnatal day 14, with two developmental transitions occ
81 ess this question, we analyzed synapses from postnatal day 15 (P15) and adult rat hippocampus that ha
82 e activates the IGF-1/IGF-1-R/Akt pathway on postnatal day 15 and initiates a brief but intense proli
86 been shown to appear adult-like as early as postnatal day 16 (P16) in the rat pup, just after eye op
87 pression of both NEEP21/Nsg1 and P19/Nsg2 at postnatal day 16 as well as in the CA1-3 regions of the
90 poor left ventricle contraction and death by postnatal day 16, suggesting that CaM inhibition of RyR2
91 impact (CCI) animal model of pediatric TBI (postnatal day 16-17) it was demonstrated that injury res
95 ectron microscopic examination of retinas at postnatal day 17 revealed the presence of vacuolar struc
97 Insensitivity to low SMN emerged abruptly at postnatal day 17, which coincided with establishment of
98 scular endothelial cells (ECs) in retinas at postnatal day 18 (p18), when pathological angiogenesis i
99 ospinal populations in Fischer 344 rats from postnatal day 18 through 75 using retrograde tracer inje
100 (NG2(+)) progenitors lagged in maturation by postnatal day 18, when the mouse null mutation was letha
103 Here we identify the period ranging from postnatal day 2 (P2) to P11 as 5-HT sensitive, with 5-HT
105 r weight beta1-catenin increased 2-3-fold in postnatal day 2 lenses of DKO lenses compared with WT le
106 15 is intermediate between that observed at postnatal days 2 and 21, further suggesting persistence
111 Synaptic transmission in the LSO matures by postnatal day 20, with EPSCs and IPSCs having fast kinet
113 stnatal day 1; P1), postnatal day 7 (P7) and postnatal day 21 (P21) mice and assessed in a pressure m
115 are evident in arg(-/-) animals as early as postnatal day 21 (P21), a time that precedes any observa
117 arbofuran exposure from gestational day 7 to postnatal day 21 altered expression of genes and transcr
119 ontrolled cortical impact or sham surgery at postnatal day 21, approximating a toddler-aged child.
120 phaly and corpus callosum deficiency, and by postnatal Day 21, microcephaly; the mice died at an earl
122 /y)) were noticeably symptomatic as early as postnatal day 21, with most dying by postnatal day 55, w
129 plugs, either from postnatal day 11 (P11) to postnatal day 23 (P23) (a manipulation previously found
130 that starts prenatally and continues through postnatal day 25 has a major impact on the structure of
131 infant nerve injured mice reach adolescence (postnatal day 25-30), the dorsal horn immune profile swi
133 ing within weeks, whereas late inactivation (postnatal days 25-28) resulted in PKD developing in mont
135 /l) or water for 28 days during adolescence (postnatal day 28-55; P28-P55) or adulthood (P67-P94).
137 lescent intermittent ethanol (AIE) exposure (postnatal days 28-42) by vapor inhalation on different a
140 defect of CH in cDKO first became evident on postnatal day 3 (P3), a time point when CH forms and gro
141 s extracellular multielectrode recordings in postnatal day 3 (P3)-P5 rat primary somatosensory cortex
142 lls showed signs of degeneration as early as postnatal Day 3 (P3); thereafter, blebs devoid of actin
143 tion End Products (RAGE) knockout mice after postnatal day 3, an identical OT increase was not observ
146 the S-phase marker bromodeoxyuridine between postnatal days 3 and 14 showed that much of this populat
149 rdings for RVM neurons in animals older than postnatal day 30 and compared the results to postnatal d
154 the high ABP are measureable in young SHRs (postnatal day 30-58) and become greater in adult SHRs.
161 as a retrograde axonal tracer in adolescent (postnatal day 39) and early adult (8-9-week-old) rats.
164 iet (0.35 mg retinol/kg diet) and treated on postnatal day 4 with an oral dose of either VA (6 mug re
165 ed that PCP4 immunostaining first appears at postnatal day 4-5 and becomes successively more refined
168 la circuitry and anxiety-related behavior by postnatal day 45 (P45), when AEA levels begin to decreas
170 onset of the critical period of plasticity [postnatal day 5 (P5)], at its peak (P26), and at the mat
173 nt with dantrolene in 4L;C* mice starting at postnatal day 5 delayed neurological pathology and prolo
175 n of these circuits is abnormal after early (postnatal day 5) removal of descending systems, inducing
178 ally formed normally, then degenerated after postnatal day 5; large numbers of vesicles invaded the c
179 supporting presensory spiking formed between postnatal days 5 (P5) and P7, including Ankyrin-G, NaV1.
180 ing positive pressure respiratory support on postnatal days 5 to 14 improves the rate of survival wit
181 Inhaled nitric oxide, initiated at 20 ppm on postnatal days 5 to 14 to high-risk preterm infants and
183 rocnemius and tibialis anterior in mice from postnatal day 55 to 100 and the results obtained were as
184 arly as postnatal day 21, with most dying by postnatal day 55, while females lacking one copy of Mecp
185 In mouse (ret)Arl13b(-/-) central retina at postnatal day 6 (P6) and older, outer segments were abse
186 ebroventricularly in the right hemisphere on postnatal day 6 at 30min prior to the first dose of Dex
187 h of systemic kainic acid administration at postnatal day 6, mRNA levels of Fgf9, Fgf10, Fgfr2c, and
190 nally, we identify a developmental window at postnatal Days 6 to 9 when Muller arbors first colonize
192 DE10A, PDE11A4 expression begins very low at postnatal day 7 (P7) and dramatically increases until P2
193 isolated from newborn (postnatal day 1; P1), postnatal day 7 (P7) and postnatal day 21 (P21) mice and
194 , using NMDARs in dopaminergic neurones from postnatal day 7 (P7) rats as a model system, we characte
195 erving neurovascular integrity, we subjected postnatal day 7 (P7) rats depleted of microglial cells,
196 d norepinephrine (NE) were dysregulated from Postnatal day 7 (P7) to P21 in En2-KO, though NE exhibit
197 the mouse lateral superior olive (LSO) from postnatal day 7 (P7) to P96 using voltage-clamp and audi
198 d in the rat STN throughout development [age postnatal day 7 (P7)-P60] and in the adult (age P120).
201 Phytosterols in the LEs and in plasma (on postnatal day 7 and day 14) were measured by gas chromat
202 Here we show that rats exposed to GAs at postnatal day 7 display a lasting reduction in inhibitor
203 The human transgenic tau was detected from postnatal day 7 onward in motoneurons, axons in the scia
205 tly developed model of status epilepticus in postnatal day 7 rat pups that results in widespread neur
206 the present study showed that inoculation on postnatal day 7 with the porcine reproductive and respir
208 o osteoblasts expressing Col1 and BSP during postnatal day 7-10, when serum levels of thyroid hormone
212 d intake, as well as growth velocity between postnatal days 7 and 28 (adjusted for GA and birth weigh
213 ced NMDAR hypofunction in infant mice during postnatal days 7-11, followed by testing fear memory spe
215 regions remained stable across development (postnatal days 7-23), there was a developmental emergenc
219 of dividing cells peaks in the CC lining on postnatal day 8 (P8), with division occurring in 19.2% +
220 he region of mouse first mandibular molar at postnatal day 8 (PN8) induced AI-like pathologies when t
221 activity occur between embryonic day 18 and postnatal day 8 and originate in pacemaker circuits in t
222 (phosphatase and tensin homolog) protein at postnatal day 8 in mice harboring Nf1 haploinsufficiency
224 en different ages (embryonic days 15 and 18, postnatal day 8, and adult), we reveal the dynamic and c
225 bistratified morphology was first visible at postnatal day 8, reaching the adult shape at P13, around
227 pendent endocytosis occurred at calyces from postnatal days 8-15, suggesting its existence before and
232 n [born on embryonic day (E) 29; examined on postnatal day (D) 3 and D7] and term-born (born on E32;
233 profiled the F2 serum metabolome at weaning [postnatal day (d)21; n = 76] and adulthood (d160; n = 12
234 Cataract development was seen at various postnatal days in the majority of mice expressing active
235 tal lipidomic remodeling during the first 60 postnatal days, including progressive accumulation of ch
236 intraventricular hemorrhage during the first postnatal days is possible from bedside measures of brai
243 perinatal ages from embryonic day (E) 17 to postnatal day (P) 11 and found that cell death peaks jus
244 ) declines during aging and is absent beyond postnatal day (P) 110 when mice are raised in standard c
245 es from individual developing astroglia from postnatal day (P) 14 to P26, when astroglia undergo dram
246 enesis, increasing NG2 progenitor cells from postnatal day (P) 15, then O4+ and CC1+ cells at P30 and
248 buffered Ca(2+) diffusion and SV release in postnatal day (P) 16-19 mouse calyces, as their release
249 from hippocampal and neocortical slices from postnatal day (P) 2-P15 mice, photostimulation caused de
250 ild-type mice; these changes were present at postnatal day (P) 20 for PV neurons and P40 for WFA/PNN+
251 Apical dendritic regression occurred from postnatal day (P) 28, dendritic spine loss from P21, and
254 ryptophan hydroxylase 2 (Tph2)(+) neurons by postnatal day (P) 5 through P12: approximately one-third
255 at the end of the first postnatal week, from postnatal day (P) 5 to P9, after the end of neurogenesis
257 aKO kidneys were hypoplastic and not cystic, postnatal day (P) 7 mutants had proximal tubular-derived
260 OZ) causes a neuropathy at 90 d after birth [postnatal day (P) 90], with a subsequent spontaneous reg
261 We used cultures of cortical neurons from postnatal day (P)0-P2 golden Syrian hamsters (Mesocricet
265 emistry for ICAM-5 in mouse visual cortex at postnatal day (P)14, a period of intense synaptogenesis,
266 derwent MD during the pre-CP [eye-opening to postnatal day (p)17] or CP (p22-p25), and FS-->SP synapt
271 xhibited a mild decrease in vascular loss at postnatal day (P)8 compared with age- and strain-matched
272 Upregulation of GLT-1 was completed only at postnatal days (P) P20-25 and was thus delayed by about
273 excitability in offspring during infancy (at postnatal days (P)10, 14, 17 and 21), preadolescence (da
275 oid body underwent a rapid cell loss between postnatal days P0 and P4, shortly after circuit formatio
276 that a history of WIN exposure during early (postnatal days - P35-40) or mid-(P40-45) adolescence, bu
281 binge-like ethanol treatment of rat pups on postnatal days (PD) 4-9, we found that maturation of GAB
290 or was studied using the forced-swim test on postnatal day (PND) 25 in rats either weaned on PND 21,
291 b transgenic (Scnn1b-Tg(+)) mice to SHS from postnatal day (PND) 3-21 and lung phenotypes were examin
292 estation day 0 of the parental generation to postnatal day (PND) 6 of the F2 generation to a realisti
293 varies were removed from the F1 offspring on postnatal day (PND)-1 and various analyses were performe
296 mbient Particle System or to filtered air on postnatal days (PNDs) 4-7 and 10-13, and the animals wer
299 sis of the effect of feeding in the first 14 postnatal days with own mother's milk, with or without h
300 at three developmental ages (15, 30, and 45 postnatal days) with array tomography three-dimensional
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