ライフサイエンスコーパス: postnatal development

1 tion, as did deletion from astrocytes during postnatal development.

2 lion size, and Let-7 expression during early postnatal development.

3 ation of gene-expression programs throughout postnatal development.

4 om the prospective white matter (PWM) during postnatal development.

5 l crest cells causes craniosynostosis during postnatal development.

6 in the diversity of functional nAChRs during postnatal development.

7 sistent with its important role during early postnatal development.

8 ollowed their distributions during fetal and postnatal development.

9 microbiota undergoes a definable program of postnatal development.

10 lycine remains the main coagonist throughout postnatal development.

11 ia at birth, and kyphosis progression during postnatal development.

12 ndrogenesis occurs during the first weeks of postnatal development.

13 ferentially expressed in mouse hearts during postnatal development.

14 ed in neocortical layers II/III and V during postnatal development.

15 in the cortex and white matter tracts during postnatal development.

16 s pattern of expression during embryonic and postnatal development.

17 icit becoming evident during stages of early postnatal development.

18 impact of alternative splicing during muscle postnatal development.

19 Aergic control of GFAP(+) cells during early postnatal development.

20 and SK3) in the rat CNS during embryonic and postnatal development.

21 eplenish IBCs/IPhCs effectively during early postnatal development.

22 stablishment of MF connectivity during mouse postnatal development.

23 OB and that its expression increases during postnatal development.

24 le as to whether klotho levels change across postnatal development.

25 from in utero development to first months of postnatal development.

26 have changes in growth and body size during postnatal development.

27 sively homogenous racial experience in early postnatal development.

28 nstrate that klotho levels vary during early postnatal development.

29 in significant body weight (BW) during early postnatal development.

30 ntal potential of pericytes during fetal and postnatal development.

31 of proliferating cells in dentate gyrus over postnatal development.

32 ARs alpha1, alpha2, and alpha3) during early postnatal development.

33 neurons gradually increased during pre- and postnatal development.

34 f cardiomyocytes (heart muscle cells) during postnatal development.

35 t the primary cilia to sustain embryonic and postnatal development.

36 36 (Cx36), transiently increase during early postnatal development.

37 sed in the Purkinje cells (PCs) during early postnatal development.

38 F-1 and the CSF-1R were maximal during early postnatal development.

39 on of initial neuronal networks during early postnatal development.

40 ody weight that began from the first week of postnatal development.

41 eripheral and central nervous systems during postnatal development.

42 orticospinal (CS) system damage during early postnatal development.

43 s the perfect nourishment, crucial for their postnatal development.

44 r neuron or astrocyte numbers changed during postnatal development.

45 ion of cerebellar granule cells during early postnatal development.

46 e regulation of synaptic connectivity during postnatal development.

47 e climbing fibre collateral pathway in early postnatal development.

48 al hyperplasia during late embryogenesis and postnatal development.

49 structural reorganization mechanisms during postnatal development.

50 nsively refined by sensory experience during postnatal development.

51 as protein function, cancer progression, and postnatal development.

52 crease in spermatogonia proliferation during postnatal development.

53 id muscles that remain immobile during early postnatal development.

54 eurons and for regulating gliogenesis during postnatal development.

55 ted at different times of day and throughout postnatal development.

56 -dependent and independent mechanisms during postnatal development.

57 on of mutant DISC1 in astrocytes during late postnatal development.

58 t2 and Gad mRNA in POMC neurons during early postnatal development.

59 oes significant developmental changes during postnatal development.

60 low levels of intraspecific variation during postnatal development.

61 like molecular profiles surprisingly late in postnatal development.

62 mice, C4 mediated synapse elimination during postnatal development.

63 lls, which deteriorated progressively during postnatal development.

64 GluN2A-dominated subunit composition during postnatal development.

65 s synaptic pruning by microglia during early postnatal development.

66 Thus, PKM2 is not required for embryonic or postnatal development.

67 sitive approach to monitoring the IVD during postnatal development.

68 (BMD) and content (BMC) first evident during postnatal development.

69 voltage-gated calcium channels during early postnatal development.

70 s diversified into five adult classes during postnatal development.

71 xpected dynamic developmental changes during postnatal development.

72 indices provide a microbial measure of human postnatal development, a way of classifying malnourished

73 Postnatal development analysis revealed that in cilia-de

74 ls with an oligodendroglial phenotype during postnatal development and adulthood in the SC of intact

75 at these cells arose in the CC lining during postnatal development and adulthood.

76 the lining of the central canal (CC) during postnatal development and adulthood.

77 in rodents, detecting OSN generation during postnatal development and aging-associated neurodegenera

78 kinases is required throughout embryonic and postnatal development and also regulates multiple homeos

79 remodeling of the organ of Corti throughout postnatal development and associated loss of non-sensory

80 imary olfactory neurons during embryonic and postnatal development and axons of the degraded neurons

81 lar Cl(-) accumulation, such as during early postnatal development and brain injury.

82 ent gene regulation changes substantially in postnatal development and can be strongly affected by fa

83 rCS3 dynamic expression during embryonic and postnatal development and compare the expression pattern

84 nitor morphological changes of myelin during postnatal development and degeneration.

85 genes were consistently up-regulated during postnatal development and down-regulated in aging, displ

86 synapses that are maintained throughout late postnatal development and early adulthood.

87 ch displays a decreased proliferation during postnatal development and even ceased at the adult stage

88 y, human hearts from both physiologic (i.e., postnatal development and exercise) and pathologic (i.e.

89 of multiple cardiac cell populations during postnatal development and following injury, which enable

90 e results define a role for microglia during postnatal development and identify underlying mechanisms

91 neurons during a critical window of time in postnatal development and in adult neurons in response t

92 This requirement for Bdnf exists both during postnatal development and in adulthood, suggesting that

93 hanisms control OB neurogenesis during early postnatal development and in adulthood.

94 5 subunit knockout (alpha5(-/-)) mice during postnatal development and in adulthood.

95 ssed in healthy cerebellar tissue throughout postnatal development and in primary cerebellar medullob

96 all pancreatic endocrine cells during mouse postnatal development and in the adult islet.

97 However, during postnatal development and in the adult, a switch in the

98 ng subplate cells during embryonic and early postnatal development and in the adult.

99 ed within these regions during embryonic and postnatal development and in the adult.

100 ed the effects of antenatal IL-1 exposure on postnatal development and investigated two IL-1 receptor

101 e primary visual cortex (V1) declines during postnatal development and is absent beyond postnatal day

102 differentiation and Th2 inflammation during postnatal development and is required for goblet cell me

103 notype of Syngap1 mice decreased slowly over postnatal development and mapped onto the developmental

104 , little is known about what regulates early postnatal development and maturation of islets.

105 l role in the refinement of circuitry during postnatal development and may be disrupted in conditions

106 riods of time to mice at different stages of postnatal development and monitored the rate of uptake o

107 ve lipidome of rat synaptic membranes during postnatal development and observe dramatic developmental

108 These effects are observed early in postnatal development and progress as animals age.

109 ual rod bipolar cells in mouse retina during postnatal development and quantified the number of dendr

110 ing between cortex and striatum during early postnatal development and suggest a potential common cir

111 ty to autoresuscitate at critical periods in postnatal development and that baseline indices of breat

112 Oligodendrocytes form myelin during postnatal development and then maintain a functional mye

113 lation of beta cell mass and function during postnatal development and throughout adulthood is incomp

114 NMDAR supercomplexes are assembled late in postnatal development and triggered by synapse maturatio

115 le in restricting synaptic plasticity during postnatal development, and are altered in several models

116 peared most sensitive to TTA exposure during postnatal development, and doxycycline treatment during

117 and excitatory currents in adulthood, across postnatal development, and following the early stress of

118 ns of the BLA undergo vast change throughout postnatal development, and studies of emotional developm

119 ssion in the lung gradually increases during postnatal development, and that mice and humans with NEC

120 f their upstream Janus kinases (JAKs) during postnatal development are less well defined.

121 sion patterns of RGS14 in mouse brain during postnatal development are unknown.

122 signaling critically regulates embryonic and postnatal development as well as adult tissue homeostasi

123 urnover is an integral part of embryonic and postnatal development, as well as routine tissue homeost

124 ion may be the outcome of competition during postnatal development based on intrinsic neuronal differ

125 ercomes the defects in OL development during postnatal development but also OL regeneration during CN

126 SC1 is not downregulated in postnatal development, but its expression shifts to dist

127 xit from the cell cycle during pre- or early postnatal development, but little is known about epigene

128 tion of ERK signaling during early phases of postnatal development, but not in the adult state, resul

129 ns coexpress calbindin through embryonic and postnatal development, but only a small proportion coexp

130 ik3c3 did not disturb embryogenesis or early postnatal development, but resulted in progressive disea

131 deprived of retinal input at early stages of postnatal development by binocular enucleation, a signif

132 ctable and stable environment of the uterus, postnatal development can be affected by a multitude of

133 r expressed in most cell types mainly during postnatal development, caused a negative shift in E(GABA

134 three family members were upregulated during postnatal development coinciding with synaptogenesis.

135 ependent learning emerges relatively late in postnatal development compared with simple associative l

136 in mouse hearts and emerge in humans during postnatal development, concomitant with increased demand

137 TEMENT Acquisition of mature behavior during postnatal development correlates with the arrival and ma

138 During postnatal development, Cripto-1 regulates the branching

139 During postnatal development, crypts multiply via fission, gene

140 fication of planar polarity deficits through postnatal development demonstrates the activity of a Van

141 icroscopy (ssEM) revealed that, during early postnatal development, dendritic spines often receive mu

142 is its high capacity for plasticity in early postnatal development during a time commonly referred to

143 t, neurons express maternal Ube3a throughout postnatal development, during which time localization of

144 During embryonic and postnatal development, EMILIN-3 showed a peculiar and dy

145 mGluR5 signaling during a critical period of postnatal development establishes the biochemical condit

146 During early postnatal development, F-actin-enriched Schmidt-Lanterma

147 1A)(-)/(-)) encoded by CACNA1a suffer during postnatal development from increasing breathing disturba

148 with transcriptome data across embryonic and postnatal development from two standard mouse strains, C

149 In postnatal development, GluN2B-containing NMDARs are crit

150 itability of F-type motoneurons during early postnatal development has long been hypothesized to cont

151 t their origins and cellular dynamics during postnatal development have not been well characterized.

152 totic activity observed in the lining during postnatal development have often been contradictory, and

153 everal stages during embryogenesis and early postnatal development; however, it is unknown how the su

154 ng in the rhesus monkey dentate gyrus across postnatal development identified a highly overlapping se

155 Hoxb7(+) fractions within the kidney across postnatal development, identifying a neonatal interstiti

156 nsient reduction of synaptic strength during postnatal development, impacting on the proper formation

157 tiple Kiss1r-positive cilia increases during postnatal development in a progression that correlates w

158 l profiling over the course of embryonic and postnatal development in animal models and humans.

159 proaches to examine CF/PC innervation during postnatal development in ATXN1[30Q]-D776 and ATXN1[82Q]

160 fovea of the marmoset undergoes a more rapid postnatal development in comparison with the Macaca monk

161 natal day 1), but failed to match subsequent postnatal development in control littermates.

162 igenetically silenced in most neurons during postnatal development in humans and mice; hence, loss of

163 Blocking Alk1 signaling during postnatal development in mice leads to retinal hypervasc

164 Here the authors show that during early postnatal development in mice, NMDAR signaling via activ

165 fically blocking ghrelin action during early postnatal development in mice.

166 ut rapidly relocated into the nucleus during postnatal development in mice.

167 further, we genetically deleted Mef2c during postnatal development in mice.

168 d of olfactory sensory axon targeting during postnatal development in mouse.

169 OPCs and mature oligodendrocytes throughout postnatal development in Olig1Cre(+/-) x ILK(fl/fl) mice

170 Here the authors show that during postnatal development in rats, a subpopulation of pre-my

171 crease in cell proliferation activity during postnatal development in rats, mice, gerbils, and ferret

172 mTOR signalling is dysregulated during early postnatal development in the cerebral cortex of germ-lin

173 d that conditional removal of the Y1R during postnatal development in the forebrain excitatory neuron

174 that corresponds to late embryonic and early postnatal development in the human.

175 d the density of myelinated axons throughout postnatal development in the inferior colliculus (IC), m

176 of genes with an essential role in pre- and postnatal development in the mouse [essential genes (EGs

177 During early postnatal development in the rodent, leptin promotes axo

178 ap of NLGN and NRXN expression patterns over postnatal development in WT and FMR1-KO mice.

179 duced during a critical time period of early postnatal development, in which IKK/NF-kappaB-induced ne

180 of moderate or severe undernutrition during postnatal development increases type 2 diabetes risk in

181 of WT and Slitrk6-deficient mouse retinas in postnatal development indicated a delay in synaptogenesi

182 rosourea (ENU) during late prenatal or early postnatal development induces a high incidence of malign

183 interneurons in the mouse brain during early postnatal development is sufficient to trigger several b

184 sensory experience during distinct phases of postnatal development known as critical periods.

185 N-methyl-D-aspartate (NMDA) receptors during postnatal development leads to epigenetic repression of

186 These results suggest that during early postnatal development light stimuli control granule cell

187 forms, whereas a separate role essential for postnatal development, likely in neuronal wiring, requir

188 ater in life and provides support that early postnatal development may represent a sensitive period f

189 nctions in smooth muscle cells (SMCs) during postnatal development, mice harboring a SMC-restricted c

190 together, these data demonstrate that during postnatal development, myocardin plays a unique, and imp

191 recent evidence indicates that during early postnatal development neuronal genomes also accumulate u

192 During postnatal development, neuronal activity controls the re

193 During early postnatal development, neuronal networks successively pr

194 During early postnatal development, NFIA labels astrocytes on the day

195 We show here that, during postnatal development, Ng is also expressed by OB neuron

196 rked overexpression of fast-twitch genes and postnatal development of a fatal dilated cardiomyopathy.

197 e immune responses participate in the normal postnatal development of a non-lymphoid epithelial tissu

198 or tyrosine kinase is critical for embryonic/postnatal development of a specific region of the DG kno

199 ogation of both genes in mice leads to rapid postnatal development of AML.

200 cerebellar granule neurons (CGNs) during the postnatal development of cerebellum.

201 d-to-expected FLV ratio for association with postnatal development of CLD.

202 During further postnatal development of Cyp26B1 mice, an anomalous DM(h

203 We identified different periods of postnatal development of distinct amygdala nuclei and ce

204 ge populations of DGCs, we characterized the postnatal development of firing properties of DG neurons

205 ABSTRACT: The early postnatal development of functional corticospinal connec

206 KEY POINTS: The early postnatal development of functional corticospinal connec

207 ation of ectopic Merkel cells, and defective postnatal development of hair follicles.

208 LRIG1 regulates the postnatal development of ICC-DMP and ICC-SMP from smooth

209 Here, we characterize postnatal development of key spinal microcircuits in the

210 ng deep sequencing of small RNAs and CAGE of postnatal development of mouse brain, we identified piRN

211 regulated and Nasp was down-regulated during postnatal development of mouse epididymis, heart, liver,

212 splicing factors that are essential for the postnatal development of multiple tissues, and the inhib

213 ant revealed no significant effects on early postnatal development of myelin.

214 n essential step in synaptic pruning and the postnatal development of neural circuits.

215 This work demonstrates that the postnatal development of neuronal connectivity is accomp

216 in a time frame similar to that seen during postnatal development of normal Merkel cells.

217 vide further understanding of the functional postnatal development of pain perception.

218 ata suggest that mucolipin-1 plays a role in postnatal development of photoreceptors and provides a s

219 In addition, we quantified the normal postnatal development of PNNs in the human PFC.

220 Finally, we found that postnatal development of PNNs on CA2 pyramidal neurons i

221 miR-29a was dramatically up-regulated during postnatal development of rat epididymis.

222 29a, was significantly down-regulated during postnatal development of rat epididymis.

223 rence to lumbar spinal segment L4 during the postnatal development of rats.

224 Postnatal development of skeletal muscle is a highly dyn

225 oliferation often decreases gradually during postnatal development of some organs.

226 The protracted postnatal development of sparse, selective firing proper

227 The neural mechanisms that support the late postnatal development of spatial navigation are currentl

228 analysis), we were able to characterize the postnatal development of substantia nigra dopaminergic n

229 However, the postnatal development of the amygdala is not easily expl

230 This study focuses on postnatal development of the area postrema, a crucial AN

231 ing SK channel subunits in the embryonic and postnatal development of the central nervous system (CNS

232 During early postnatal development of the CNS, neuronal networks are

233 nd this trend likely reflects the protracted postnatal development of the cortex.

234 lobal knock-out of cpg15 results in abnormal postnatal development of the excitatory network in visua

235 ght to play a particularly important role in postnatal development of the gastrointestinal, metabolic

236 cal importance to global brain function, the postnatal development of the human pons remains poorly u

237 hondrocytes within the growth plate regulate postnatal development of the long bones.

238 eal a novel role for ACKR2 in regulating the postnatal development of the mammary gland.

239 f Ca(2+) influx among the AZs of IHCs-during postnatal development of the mouse cochlea.

240 in ON and OFF RGCs during the second week of postnatal development of the mouse.

241 neage NMDARs are neither required for timely postnatal development of the oligodendroglial lineage, n

242 mate receptors, is necessary for the correct postnatal development of the Pv(+) GABAergic network.

243 l established, but little is known about the postnatal development of the raphe where serotonin is ma

244 This study examines the role of EYA in the postnatal development of the retinal vasculature and und

245 ell (EC) migration contributes to a delay in postnatal development of the retinal vasculature when Ey

246 , articulations that close during the normal postnatal development of the skull have also lower relia

247 Electrical synapses in the TRN precede the postnatal development of TRN-to-VB inhibition.

248 Here we investigate the postnatal development of two PNN markers (Wisteria flori

249 tional insults applied at various periods of postnatal development on maturation and long-term integr

250 hat pharmacologic MAOA blockade during early postnatal development (P2-P21) but not during peri-adole

251 mouse thalamic slices revealed that early in postnatal development (P7-P8), the mIPSC duration is pro

252 mouse thalamic slices revealed that early in postnatal development (P7-P8), the mIPSC duration is pro

253 ar coupling may be altered, including during postnatal development, pathological states, and aging, n

254 mate transmission during critical periods of postnatal development plays an important role in the ref

255 a role for chemokines and their receptors in postnatal development processes.

256 deprivation (MD) during a critical period of postnatal development produces significant changes in th

257 , or the decline in number of these cells as postnatal development progresses parallels the loss of s

258 ow this circuit reorganization occurs during postnatal development remains poorly understood.

259 However, its neuronal function during postnatal development remains unknown.

260 Hh-responsive cells during the first week of postnatal development resulted in a loss of mineralized

261 c complex, but we now show that during early postnatal development Sox14/Otx2-expressing precursor ce

262 ype Fam65b is expressed during embryonic and postnatal development stages in murine cochlea, and that

263 During embryonic and early postnatal development subplate neurons integrate into th

264 During postnatal development, sympathetic axons express TNFalph

265 to develop new concepts about our evolution, postnatal development, systems physiology, individuality

266 Here, we reveal that throughout postnatal development, thalamic, and entorhinal cortical

267 expression during critical periods of early postnatal development that could contribute to observed

268 During postnatal development the heart undergoes a rapid and dr

269 s glutamic acid decarboxylase (GAD) in early postnatal development, the functional role of GABA synth

270 During the first three weeks of postnatal development, the glial cell population, which

271 During postnatal development, the number of Bassoon puncta incr

272 During postnatal development, the replacement of GluN2B- by Glu

273 During postnatal development there is a switch from predominant

274 During postnatal development, there was a protracted, progressi

275 bellum express GABArho subunits during early postnatal development, thereby conferring peculiar pharm

276 During perinatal and postnatal development, these TrkA lineage neurons are gl

277 also promoted button formation during early postnatal development through a direct effect involving

278 ICDs are formed and matured during postnatal development through a profound redistribution

279 e number of PNNs in the PFC increased during postnatal development through the peripubertal period un

280 taining the morphology they achieve in early postnatal development throughout most of life.

281 e Nav1.2-driven action potentials throughout postnatal development to early adulthood.

282 s widely known to increase in the brain from postnatal development to sexual maturity and to aging, y

283 Glutamatergic synapses in early postnatal development transiently express calcium-permea

284 and cross-fostering (CF) rodent pups during postnatal development triggers changes in maternal behav

285 umber of PNNs progressively increases during postnatal development until plateauing around the period

286 n of the cortical blood flow response during postnatal development using exposed-cortex multispectral

287 n slices from male mice during perinatal and postnatal development using fast-scan cyclic voltammetry

288 feration during late embryogenesis and early postnatal development was reduced.

289 eavage following glutamate activation during postnatal development, we also explored ICAM-5 expressio

290 tial functions of prostasin in embryonic and postnatal development were compensated for by loss of HA

291 During a period of postnatal development when activity plays a large role i

292 ons by a p75(NTR)-dependent mechanism during postnatal development when the axons of these neurons ar

293 cently identified a brief time period during postnatal development when the mammalian heart retains s

294 stigated CD200/CD200R at different stages of postnatal development, when microglial maturation takes

295 L/6 mice at two different time points: early postnatal development, when the brain is growing at its

296 de of AMPA-EPSCs at a critical time point of postnatal development, whereas the NMDA component is spa

297 gdala nuclei exhibited different patterns of postnatal development, which were largely similar to tho

298 rotein and mRNA are upregulated during early postnatal development, with protein first detected at P7

299 eath of retinal neurons during embryonic and postnatal development without affecting their proliferat

300 ation of and reduction in rhodopsin early in postnatal development without loss of photoreceptors.