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1 y, yet few cases are documented in mammalian postnatal life.
2 observed during the critical period in early postnatal life.
3 essential functions throughout gestation and postnatal life.
4 ully replace fetal chondrocytes during early postnatal life.
5 to the maintenance of neuronal viability in postnatal life.
6 ctional maintenance of the epithelium during postnatal life.
7 progenitor cells in embryogenesis and during postnatal life.
8 iods of heightened brain plasticity in early postnatal life.
9 s many as half in a specific period of early postnatal life.
10 velopment, growth and metabolism in pre- and postnatal life.
11 etal development which are reversed in early postnatal life.
12 capacity to renew cardiomyocytes throughout postnatal life.
13 well as in growth and tissue homeostasis in postnatal life.
14 aintenance of the blood-brain barrier during postnatal life.
15 f LCPUFA during the critical first months of postnatal life.
16 n is compatible with development to term and postnatal life.
17 e absence of PIPKIgamma is incompatible with postnatal life.
18 fects brain development during both pre- and postnatal life.
19 daptation that occur throughout neonatal and postnatal life.
20 and angiogenesis during embryonic and early postnatal life.
21 is become mature and functional during early postnatal life.
22 ibuting to the gammadelta lineage throughout postnatal life.
23 fate in many tissues during development and postnatal life.
24 vival until birth, but it is dispensable for postnatal life.
25 loss of photoreceptors if performed early in postnatal life.
26 ogenesis and during hair follicle cycling in postnatal life.
27 ady committed, either prenatally or early in postnatal life.
28 retina from fetal development through early postnatal life.
29 in the removal of axon branches during early postnatal life.
30 s after injection or on day 15, 30, or 60 of postnatal life.
31 for bone formation during embryogenesis and postnatal life.
32 that myogenin has separate functions during postnatal life.
33 l circuits are shaped by experience in early postnatal life.
34 n from multiple to single innervation during postnatal life.
35 hermogenesis develops during the 1st week of postnatal life.
36 ate brain development during fetal and early postnatal life.
37 or maintaining tissue homeostasis throughout postnatal life.
38 ic stages and through the first few weeks of postnatal life.
39 during development, both in utero and early postnatal life.
40 stereotyped behaviors that manifest in early postnatal life.
41 t regulates cell fate during development and postnatal life.
42 ponse during the transition from prenatal to postnatal life.
43 embryonic cusp remodeling and continues into postnatal life.
44 for fetal survival but is incompatible with postnatal life.
45 and function are critical in development and postnatal life.
46 e role of the myogenic regulator myogenin in postnatal life.
47 ich is not manifest until the second week of postnatal life.
48 m late embryonic stages through to day 13 of postnatal life.
49 during the later part of gestation and early postnatal life.
50 mental factors during intrauterine and early postnatal life.
51 ic decrease in their expression during early postnatal life.
52 iss-Webster mice during the first 2 weeks of postnatal life.
53 normally during embryogenesis and survive to postnatal life.
54 oral manner during embryonic development and postnatal life.
55 ontinuously produces male gametes throughout postnatal life.
56 is guided by visual experience during early postnatal life.
57 e formation was significantly reduced during postnatal life.
58 d changes in the transition from prenatal to postnatal life.
59 embryos but closes during the first 48 h of postnatal life.
60 ecomes vulnerable to ethanol insult in early postnatal life.
61 nic development and low bone mass throughout postnatal life.
62 epigenetic patterning during development and postnatal life.
63 tion at sites of injury and tumorigenesis in postnatal life.
64 ontribute significantly to vasculogenesis in postnatal life.
65 t (FMR1-KO) mice during the first 5 weeks of postnatal life.
66 y and physiology over the first few weeks of postnatal life.
67 temporarily deprived of vision during early postnatal life.
68 ory may influence cardiovascular function in postnatal life.
69 and tissues during embryonic development and postnatal life.
70 mber may be developmentally regulated during postnatal life.
71 occurring during a critical period in early postnatal life.
72 of this gene in maintaining heart rhythm in postnatal life.
73 endocytosed OVA for delayed presentation in postnatal life.
74 hylation nor influenced cell survival during postnatal life.
75 ing, postmitotic neurons and persisting into postnatal life.
76 cisions throughout embryonic development and postnatal life.
77 was normal during embryogenesis and in early postnatal life.
78 ted quickly from the brain within 3 weeks of postnatal life.
79 ng one eye during a critical period in early postnatal life.
80 lian gerbils throughout the first 3 weeks of postnatal life.
81 t to marginal contribution in late fetal and postnatal life.
82 al at birth and remained elevated throughout postnatal life.
83 gan development and function during pre- and postnatal life.
84 trioventricular (AV) conduction block during postnatal life.
85 s essential for growth and survival in early postnatal life.
86 nal role Trk-mediated signaling plays during postnatal life.
87 This type was relatively constant throughout postnatal life.
88 white matter and cortical layer VI well into postnatal life.
89 lian kidney to the physiological stresses of postnatal life.
90 ular integrity during development as well as postnatal life.
91 defect is manifest within the first week of postnatal life.
92 plasticity in sensory neocortex during early postnatal life.
93 skeletal neuromuscular junction during early postnatal life.
94 neurons were found during the first 10 d of postnatal life.
95 many of these first-born cells die early in postnatal life.
96 nactive" versus "active" GnRH neurons during postnatal life.
97 response to hypoxia typically seen in early postnatal life.
98 predominant expression of c-Krox in skin in postnatal life.
99 efore they complete their differentiation in postnatal life.
100 g-term control of carbohydrate metabolism in postnatal life.
101 rvival and maturation of DA neurons in early postnatal life.
102 (telogen), and regeneration (anagen) during postnatal life.
103 normally assemble during the first 2-3 y of postnatal life.
104 sis and for cardiac contractility to support postnatal life.
105 de mammalian heart regeneration during early postnatal life.
106 igh fat diet during early development and in postnatal life.
107 of large quantities of embryonic globins in postnatal life.
108 rcuitry is becoming established during early postnatal life.
109 ts observed during the second week of murine postnatal life.
110 normal airway smooth muscle (SM) function in postnatal life.
111 tions with nonmotor brain regions throughout postnatal life.
112 inuously generated from NG2 cells throughout postnatal life.
113 d, cell-specific ablation of SCs in pre- and postnatal life.
114 enewal of oocyte-containing follicles during postnatal life.
115 s induced during late embryogenesis or early postnatal life.
116 ting the amygdala during the first 3 days of postnatal life.
117 re critical for neurotransmission throughout postnatal life.
118 s and contribute to organ homeostasis during postnatal life.
119 e internodes than OLs generated during early postnatal life.
120 during development but rapidly decreases in postnatal life.
121 em is critical for embryonic development and postnatal life.
122 ration units, the nephrons, is essential for postnatal life.
123 ioral milestones, to the third year of human postnatal life.
124 vailability of energy-dense foods throughout postnatal life.
125 f hematopoietic progenitor and stem cells in postnatal life.
126 l-2-associated X)-dependent apoptosis during postnatal life.
127 es and limit such reactive seizures to early postnatal life?
128 Here, we show that, at the beginning of postnatal life, 0- to 3-d-old neonates reacted to a simu
129 icted period of sensory deprivation in early postnatal life (1) impairs intracortical relay of depriv
130 come in not having experienced adjustment to postnatal life, a potentially important determinant of o
131 elopment is synapse elimination during early postnatal life, a process known to depend on neuronal ac
132 L-type Ca2+ channels and occur during early postnatal life, a time when retinogeniculate connections
133 an neuromuscular junction occur during early postnatal life: acetylcholine receptors (AChRs) disappea
135 dicate that the quality of dietary FA during postnatal life affects the development of the central re
136 ystem to coevolve with the microbiota during postnatal life allows the host and microbiota to coexist
137 elial precursors and vascular development in postnatal life and a possible novel therapeutic target.
138 he developing lung during prenatal and early postnatal life and adult respiratory morbidity or reduce
139 ific cell lineages from NSCs in vivo, during postnatal life and adulthood, as well as in regenerative
140 F in embryonic life to dependence on GDNF in postnatal life and are likely regulated by GDNF in matur
141 rain growth in different periods of pre- and postnatal life and cognitive function in 221 9-year-old
142 irth in these infants persists through their postnatal life and contributes to adverse pulmonary outc
143 these processes are rudimentary during early postnatal life and develop only gradually thereafter.
145 y circuits are shaped by experience in early postnatal life and in many brain areas late maturation o
146 cortical neurons was first evident in early postnatal life and increased during subsequent developme
148 an survive through embryonic development and postnatal life and MEF cells from the Ppp5-deficient mic
150 sults clarify the complex roles for RIPK1 in postnatal life and provide insights into the regulation
151 essential for the survival of animals during postnatal life and that its ablation leads to distinct b
152 l muscle defects in Myog-deleted mice during postnatal life and the efficient differentiation of cult
153 bryonic day 12.5, which persisted throughout postnatal life and was not complemented by the Zfy genes
154 tress of maternal separation examined during postnatal life and young adulthood exhibited enhanced hi
155 at midgestation but was evident by day 2 of postnatal life, and by 42 days, hearts exhibited multifo
156 ural stem/progenitor cells, divide late into postnatal life, and can generate both neurons and astroc
157 ogenesis was extended into the third week of postnatal life, and ectopic neurons and progenitors coll
158 es undergoes pronounced alterations in early postnatal life, and environmental cues may be responsibl
159 is the major carrier for circulating IGFs in postnatal life, and has been shown to have IGF-independe
160 VSMCs, is activated from late fetal to early postnatal life, and is required to maintain the morpholo
161 of pulmonary vascular function during early postnatal life, and its production in the pulmonary vasc
162 on in the cerebellum during intrauterine and postnatal life, and that normal ErbB-NRG signaling in th
163 n a thermocline, as early as the 1st week of postnatal life, and these pups can also produce heat met
164 o suggest that interactions with V1 in early postnatal life are critical for establishing stimulus se
165 d differentiation during embryonic and early postnatal life are prerequisites for selective synaptic
167 iculus neurons emerge gradually during early postnatal life as a consequence of experience with cross
168 Gata6 alters atrioventricular conduction in postnatal life as assessed by surface and invasive elect
169 ained in these cells into the second week of postnatal life, at which time Prox1 is dynamically down
170 systematically interrelated at the start of postnatal life, before acquisition of language and cultu
171 ents increased considerably throughout early postnatal life beginning shortly after the peak of the c
174 These differences were apparent in early postnatal life but decreased during progression into adu
175 infants demonstrated growth deficit early in postnatal life but had greater percentage body fat at te
176 ation during beta-cell development and early postnatal life but not for maintenance of adult mass.
177 decreases slightly during the first week of postnatal life, but increases between 1 and 6 weeks of a
178 cardiomyocyte proliferation during the early postnatal life, but it is largely ineffective in driving
179 th occurs throughout embryogenesis and early postnatal life, but its foundation is laid in the primit
180 ontinue to arise from Sox9(+) cells in early postnatal life, but no endocrine or acinar cell neogenes
181 whole length from their time of formation to postnatal life, but some have more complex age-dependent
182 ctional maturation during the first month of postnatal life by developing faster response kinetics, h
183 ol of catabolic and anabolic states early in postnatal life by modulating the growth hormone-insulin-
184 tory cortex (A1) is easily degraded in early postnatal life by raising rat pups in the presence of pu
186 deposition by differentiated osteoblasts in postnatal life, called hereafter bone formation, are unk
187 tical periods of embryonic, fetal, and early postnatal life can affect the development of body weight
188 thymocytes during human late fetal and early postnatal life can be reproduced in humanized mice, and
189 view that environmental events during early postnatal life can influence the formation of neural cir
191 ows that Abeta overexpression, even early in postnatal life, can perturb plasticity in cerebral corte
193 development of FC during the first months of postnatal life compared with wild-type animals, resultin
196 Monocular deprivation (MD) imposed early in postnatal life elicits profound structural and functiona
198 ctroencephalography in the first 72 hours of postnatal life, focusing on the electrical burst activit
199 samples collected during the first 30 mo of postnatal life from eight pairs of mono- and dizygotic M
200 nvironment during a critical period in early postnatal life has lifelong effects on the structure and
201 , a complete absence of both proteins during postnatal life has little or no effect on the survival a
205 itional status during intrauterine and early postnatal life impacts the risk of chronic diseases; how
206 on, chronic administration of ghrelin during postnatal life impaired the normal development of ARH pr
207 dergo activity-dependent maturation in early postnatal life in a manner analogous to sensory systems.
208 essive loss of enteric neurons occurs during postnatal life in Hipk2(-/-) mutant mice that preferenti
211 They also underscore the importance of early postnatal life in the rat, which corresponds to late ges
213 ession of myelin-related proteins throughout postnatal life in the somatosensory (areas 3b/3a/1/2), m
215 feration during development and in the early postnatal life; in adult zebrafish, reactivation of this
216 ternal obesity during intrauterine and early postnatal life increases the risk of low bone mass and f
217 mouse brain during embryonic development and postnatal life indicate that it may be involved in the r
218 Rs1 was expressed after the first 4 weeks of postnatal life, indicating an exquisite temporal sensiti
219 ility to anoxic stress at a specific time in postnatal life induced by a partial defect in raphe func
220 e tissue expansion progresses rapidly during postnatal life, influenced by both prenatal maternal fac
221 indings imply that growth in both foetal and postnatal life influences cognitive performance, little
222 any parts of the nervous system during early postnatal life is a competitive process called 'synapse
223 xpenditure in females and suggest that early postnatal life is a critical period during which nutriti
225 homeostasis during both gestation and early postnatal life is crucial for the development of the fet
226 cation of neuronal architecture during early postnatal life is essential for refining the precision o
228 GUS transport into brain parenchyma in early postnatal life is mediated by the mannose 6-phosphate/in
230 ts that nutrition during pregnancy and early postnatal life is one of the most important environmenta
231 ific regions of the dorsal horn during early postnatal life is shown to have multiple, deep-rooted un
232 e genome level, the transition from fetal to postnatal life is typified by a reversal of direction, f
233 espite its important role in development and postnatal life, little is known about the signaling path
236 adrenal (HPA) axis during prenatal and early postnatal life may explain, in part, the association bet
237 as expression of therapeutic proteins during postnatal life may limit the pathologic consequences and
238 or hypertension; moreover, in utero or early postnatal life may represent a possible therapeutic wind
242 he developing embryo but also throughout the postnatal life of mammals from neuronal precursor cells
243 how that a dietary modification in the early postnatal life of the 1-HC female rat sets up a vicious
246 Exposure to infectious agents during early postnatal life often alters glucocorticoid responses to
247 l fatty acid deficiency during gestation and postnatal life on the development of visual function in
248 rmacological inhibition of S6K1 during early postnatal life or by reducing the activity of mPFC-BLA c
251 ensive brain development occurs during early postnatal life-particularly within the prefrontal cortex
252 stem cell function in utero and during early postnatal life, PAX3/FOXO1A and PAX7/FOXO1A may subvert
254 lly to 0, 25 or 50 mg Mn/kg/day during early postnatal life (PND 1-21) or throughout life from PND 1
255 rance toward alloantigens encountered during postnatal life pointing to the existence of a process fo
256 omoting greater neuronal plasticity early in postnatal life preceded the divergence of the human and
257 rt learning and generalization in very early postnatal life, providing evidence that PFC and the fron
258 AEP-derived population is eliminated during postnatal life, raising questions about the nature and p
261 on from the in utero maternal environment to postnatal life requires the activation of thermogenesis
262 kappaB activation in astrocytes during early postnatal life results in hydrocephalus formation and ad
263 situ hybridization, we found that from early postnatal life retinoschisin mRNA is present only in the
265 ing and grooming (low LG offspring) in early postnatal life show reduced long term potentiation (LTP)
266 mice, mice fed a LOW n-6 or HIGH n-3 during postnatal life showed significant reductions in the dens
267 hat is present in fetal life and persists in postnatal life, suggesting opportunities for early detec
268 hem, are particularly prominent during early postnatal life, suggesting that endogenous CRH may contr
269 in is strongly influenced by events in early postnatal life that eliminate approximately half of the
270 sine-resistant accessory pathways induced in postnatal life that may rarely disappear later in life.
273 at develop throughout embryogenesis and into postnatal life, the generation of differentiated cells t
274 during gametogenesis and through fetal/early-postnatal life, the resultant phenotype is likely the ag
275 These results suggest that, during early postnatal life, there is exuberant outgrowth of local CA
276 turation of cortical inhibition during early postnatal life, thereby regulating the critical period f
279 al touch and pain processing emerges in late postnatal life to allow flexible and context-dependent b
280 tic interaction in SW mice operates early in postnatal life to influence the expression of GABA(A)R a
281 mammalian heart undergoes maturation during postnatal life to meet the increased functional requirem
282 s switch from strong depression during early postnatal life, to weaker depression and in some cases f
283 examining gene function at selected times in postnatal life, under selected physiologic or pathophysi
284 s and interpeak intervals decreased early in postnatal life, values decreased over a delayed and prot
285 essive photoreceptor degeneration throughout postnatal life, via derepression of fetal expression of
287 ions of Bcl-xL related to neuron survival in postnatal life, we generated transgenic mice overexpress
288 t regulates human lymphoid commitment during postnatal life, we used RNA sequencing to assemble the l
289 e directly assess the importance of cilia in postnatal life, we utilized conditional alleles of two c
290 animals continue to proliferate at day 17 of postnatal life when cell division in wild-type littermat
292 Disruption of this pathway during early postnatal life, when olivocochlear axons are forming the
293 xpressed TrkA during embryogenesis and early postnatal life, whereas only 43% expressed TrkA at postn
294 Here, we identify a time window in early postnatal life wherein partial amputation culminates in
295 , in large part, during the first 2 weeks of postnatal life, while thyroid hormone levels are known t
297 cis-regulatory elements are primed in early postnatal life whose functions may be compromised in hum
298 n was restricted during both development and postnatal life, with high levels present only in skeleta
299 gamma-globin chain production diminishes in postnatal life, with transient production of HbF reticul
300 us system (CNS) germinal zones and, in early postnatal life, within numerous cells throughout the CNS
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