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1 on meal (3.9 g; 6% kcal from fat) at 8 hours postprandially.
2  isolated at regular time intervals over 8 h postprandially.
3 025 g/mL) before and at 7 serial time points postprandially.
4 opy) were measured after fasting and 4 hours postprandially.
5 d adhesion molecules were assessed 2 and 4 h postprandially.
6 not change other lipoproteins or glucose 3 h postprandially.
7  increased significantly from fasting to 6 h postprandially.
8  SMLPL were done in the fasted state and 6 h postprandially.
9  to active serine protease for several hours postprandially.
10 A mobilization upon cAMP stimulation as seen postprandially.
11              A subgroup (n = 17) was studied postprandially after consumption of 2 meals (containing
12 d levels of PP were noted only at 60 minutes postprandially after LSG.
13      Plasma and urine samples were collected postprandially and analyzed.
14            Cholecystokinin (CCK) is released postprandially and elicits satiety signals.
15 ma glucose concentrations during fasting and postprandially and glycosylated hemoglobin values were m
16       apoA-IV is secreted from the intestine postprandially and has demonstrated benefits for both gl
17 se tissue to trap and store free fatty acids postprandially and the presence of very low circulating
18          A second FMD scan was performed 2 h postprandially, and adhesion molecules were assessed 2 a
19 injected at 0800 after an overnight fast and postprandially at 1300, 3 h after ingestion of a fat-enr
20                        Plasma IL-6 increased postprandially, but IL-8, TNF-alpha, and E-selectin decr
21 e nucleus bound to GKRP, and it is activated postprandially by portal hyperglycemia and fructose thro
22                (Leptin levels did not change postprandially.) Euglycemic hyperinsulinemia did not alt
23 in levels peak during acute fasting, whereas postprandially ghrelin is at a nadir, and islet SST conc
24 l, triglyceride levels measured 2 to 4 hours postprandially had the strongest association with cardio
25 an in men with the RR genotype but increased postprandially in both groups with no evidence of a geno
26  is released from the gastrointestinal tract postprandially in proportion to the calorie content of a
27 cose (cumulative percentage cleared over 6 h postprandially) increased after RYGB (from 10% +/- 8% be
28 echanism by which monocytes become activated postprandially is by exposure to triglyceride-rich lipop
29  been reported that prouroguanylin, secreted postprandially, is converted to uroguanylin in the brain
30  GIP infusion further worsened hyperglycemia postprandially, most likely through its suppressive effe
31 function, assessed in fasting conditions and postprandially, on liver injury, glucose and lipoprotein
32 ndex (r = 0.326, P = 0.02) and was shortened postprandially (P < 0.00001) independent of the fat cont
33 tments significantly increased apo B and LpB postprandially (P < 0.03 for both), but the magnitude of
34 hmias or failure of signal power to increase postprandially; rhythm abnormalities may be independent
35           Fructose intakes of <50 g/d had no postprandially significant effect on triacylglycerol and
36                                              Postprandially, the liver experiences an extensive metab
37 nd retinyl palmitate were measured over 12 h postprandially via high-performance liquid chromatograph
38 onal uptake (Ki) between time 90 and 120 min postprandially was also higher in IGT versus control sub
39     Blood samples drawn -15, 30, and 120 min postprandially were analyzed by untargeted liquid chroma
40 did not change, but marked changes were seen postprandially with 1.7-fold increased peak concentratio

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