ライフサイエンスコーパス: postreplication

1 ng mitosis and accurate repair of DNA damage postreplication.

2 nd single nucleotide insertions or deletions postreplication.

3 These observations support a model in which postreplication DNA gaps with unrepaired UV lesions in t

4 ned from mice with targeted inactivations of postreplication DNA mismatch repair (MMR) genes have bee

5 Postreplication DNA mismatch repair is essential for mai

6 Postreplication DNA mismatch repair is initiated by the

7 Here, we identify HLTF, a postreplication DNA repair helicase, as a common target

8 s distinctive interfaces to recruit multiple postreplication DNA repair proteins to the CRL4-DCAF1 E3

9 and Csm3, but decreased in the knockouts of postreplication DNA repair proteins, Rad6 and Rad5, and

10 for integration of S phase progression with postreplication DNA repair to maintain genome stability.

11 egulated by the RAD6 pathway, which mediates postreplication DNA repair, have remained elusive.

12 he role of UBZ domain-containing proteins in postreplication DNA repair.

13 cO, and RecR proteins in the early stages of postreplication DNA repair.

14 In eukaryotes, the recognition of the DNA postreplication errors and initiation of the mismatch re

15 recombination efficiency and fidelity during postreplication gap repair.

16 on, which are uncorrected due to a defective postreplication mismatch repair (MMR) system.

17 Base selectivity, proofreading, and postreplication mismatch repair are important for replic

18 suggesting that they participate directly in postreplication mismatch repair as well as in other DNA

19 Ab V genes in mice deficient for the postreplication mismatch repair factor MutS homolog (MSH

20 Long-patch postreplication mismatch repair was measured for the fir

21 DNA synthesis and postreplication mismatch repair were measured in vitro u

22 fluent and high-density SY5Y cells catalyzed postreplication mismatch repair with efficiencies compar

23 ontrolled by DNA polymerase proofreading and postreplication mismatch repair.

24 replicate the very end of the telomeres) and postreplication processing.

25 This postreplication recruitment of cohesin is not functional

26 are homologous recombination and error-free postreplication repair (EF-PRR).

27 in nucleotide excision repair (NER) (rad14), postreplication repair (PRR) (rad6, rad18, mms2, and rad

28 therefore arising through homology-mediated postreplication repair (PRR) events.

29 NA polymerases eta or zeta or Rad5-dependent postreplication repair (PRR) in which error-free replica

30 in conjugating enzymes that are required for postreplication repair (PRR) of DNA and damage-induced m

31 In Saccharomyces cerevisiae, postreplication repair (PRR) of UV-damaged DNA occurs by

32 First, we show that genes in the error-free postreplication repair (PRR) pathway prevent fusion of i

33 ts parallel to the PCNA/Ubc13/Mms2-dependent postreplication repair (PRR) pathway.

34 e damage whereas the recombination (REC) and postreplication repair (PRR) pathways bypass the damage,

35 f1 and different gene products implicated in postreplication repair (PRR) pathways in the suppression

36 Postreplication repair (PRR) pathways play important rol

37 Cells employ error-free or error-prone postreplication repair (PRR) processes to tolerate DNA d

38 This suppression requires the postreplication repair (PRR) protein Rhp18 and the struc

39 have evolved lesion bypass pathways such as postreplication repair (PRR) to resolve these arrested f

40 in conjunction with the error-free branch of postreplication repair (PRR).

41 is necessary to restore normal survival and postreplication repair after ultraviolet irradiation in

42 kpoints, in homologous recombination, and in postreplication repair after UV damage.

43 itin-conjugating enzyme, and a key player in postreplication repair and induced mutagenesis in the ye

44 ion synthesis DNA polymerase, the Mms2-Ubc13 postreplication repair complex, downstream DNA-damage ch

45 nhomologous end joining, excision repair, or postreplication repair enhanced sensitivity to Top2 targ

46 ite damaged DNA during replication, and this postreplication repair function depends on its HIRAN dom

47 odification central to DNA damage bypass and postreplication repair in both yeast and vertebrates.

48 poorly defined RAD6-RAD18-RAD5 mechanism of postreplication repair in eukaryotes occurs by an analog

49 combination with rad52, possibly implicating postreplication repair in the removal of unrepaired 3'-t

50 ide excision repair, recombinational repair, postreplication repair including translesional synthesis

51 or-free translesion synthesis by Poleta, and postreplication repair of discontinuities by a Rad5-depe

52 in-conjugating enzymes that are required for postreplication repair of DNA and damage-induced mutagen

53 ated as a participant in the recombinational postreplication repair of these replication products.

54 h belong to the RAD6 epistasis group, to the postreplication repair of UV-damaged DNA.

55 umor suppressor gene that might operate in a postreplication repair or a cell cycle checkpoint functi

56 encodes a DNA helicase that is active in the postreplication repair pathway and homologous recombinat

57 ons to promote recombination through a novel postreplication repair pathway and the structure-specifi

58 provides a distinctive opportunity to study postreplication repair processes.

59 repair, homologous recombination repair, or postreplication repair when compared with platin control

60 tolerance process in eukaryotes (also called postreplication repair) has existed for more than two de

61 lesion, and homology-dependent repair (HDR; postreplication repair), which is based on the homologou

62 eplication soon after UVC irradiation (i.e., postreplication repair).

63 s 10 and 13 lie in or near genes involved in postreplication repair, a DNA damage-tolerance pathway.

64 ecombination and nucleotide excision repair, postreplication repair, and checkpoints in the repair an

65 rough homologous recombinational repair, not postreplication repair, and confirm findings of a G1 che

66 chromatid cohesion, the spindle checkpoint, postreplication repair, and telomere maintenance.

67 induced by ultraviolet light, and defective postreplication repair, but normal nucleotide excision r

68 include DNA replication, DNA damage repair, postreplication repair, damage checkpoint activation, ch

69 ranslesion synthesis, one of the pathways of postreplication repair, is thought to account for some r

70 that, in addition to its established role in postreplication repair, RAD18 is also required for the o

71 HR, resolution of branched HR intermediates, postreplication repair, sumoylation in response to DNA d

72 at, in addition to homologous recombination, postreplication repair, the Fanconi anemia pathway, poly

73 ecognized as an essential step in initiating postreplication repair, the mechanistic relevance of thi

74 In addition to its role in postreplication repair, the MMR machinery serves to poli

75 protein ligase that has crucial functions in postreplication repair.

76 on serves as a molecular mark to orchestrate postreplication repair.

77 into recombination-dependent and error-free postreplication repair.

78 illess H2A/H3 mutant, indicating a defect in postreplication repair.

79 n the RAD18 epistasis group, which regulates postreplication repair.

80 -cycle response to UV is to provide time for postreplication repair.

81 DNA damage signaling and (iii) facilitating postreplication repair.

82 on in the RING finger motif are defective in postreplication repair.

83 multiple DNA damaging agents and a defect in postreplication repair.

84 er eukaryotes, Rad18 plays a crucial role in postreplication repair.

85 to maintenance of genomic stability through postreplication repair.

86 s that perhaps determine the fidelity of DNA postreplication repair.

87 others have also reported NS4B's function in postreplication steps.

88 sults indicate an essential role for PPL2 in postreplication tolerance of endogenous DNA damage, thus