ライフサイエンスコーパス: postreplication repair

1 eplication soon after UVC irradiation (i.e., postreplication repair).

2 protein ligase that has crucial functions in postreplication repair.

3 illess H2A/H3 mutant, indicating a defect in postreplication repair.

4 on serves as a molecular mark to orchestrate postreplication repair.

5 into recombination-dependent and error-free postreplication repair.

6 n the RAD18 epistasis group, which regulates postreplication repair.

7 -cycle response to UV is to provide time for postreplication repair.

8 DNA damage signaling and (iii) facilitating postreplication repair.

9 on in the RING finger motif are defective in postreplication repair.

10 multiple DNA damaging agents and a defect in postreplication repair.

11 er eukaryotes, Rad18 plays a crucial role in postreplication repair.

12 to maintenance of genomic stability through postreplication repair.

13 s that perhaps determine the fidelity of DNA postreplication repair.

14 s 10 and 13 lie in or near genes involved in postreplication repair, a DNA damage-tolerance pathway.

15 is necessary to restore normal survival and postreplication repair after ultraviolet irradiation in

16 kpoints, in homologous recombination, and in postreplication repair after UV damage.

17 itin-conjugating enzyme, and a key player in postreplication repair and induced mutagenesis in the ye

18 ecombination and nucleotide excision repair, postreplication repair, and checkpoints in the repair an

19 rough homologous recombinational repair, not postreplication repair, and confirm findings of a G1 che

20 chromatid cohesion, the spindle checkpoint, postreplication repair, and telomere maintenance.

21 induced by ultraviolet light, and defective postreplication repair, but normal nucleotide excision r

22 ion synthesis DNA polymerase, the Mms2-Ubc13 postreplication repair complex, downstream DNA-damage ch

23 include DNA replication, DNA damage repair, postreplication repair, damage checkpoint activation, ch

24 are homologous recombination and error-free postreplication repair (EF-PRR).

25 nhomologous end joining, excision repair, or postreplication repair enhanced sensitivity to Top2 targ

26 ite damaged DNA during replication, and this postreplication repair function depends on its HIRAN dom

27 tolerance process in eukaryotes (also called postreplication repair) has existed for more than two de

28 odification central to DNA damage bypass and postreplication repair in both yeast and vertebrates.

29 poorly defined RAD6-RAD18-RAD5 mechanism of postreplication repair in eukaryotes occurs by an analog

30 combination with rad52, possibly implicating postreplication repair in the removal of unrepaired 3'-t

31 ide excision repair, recombinational repair, postreplication repair including translesional synthesis

32 ranslesion synthesis, one of the pathways of postreplication repair, is thought to account for some r

33 or-free translesion synthesis by Poleta, and postreplication repair of discontinuities by a Rad5-depe

34 in-conjugating enzymes that are required for postreplication repair of DNA and damage-induced mutagen

35 ated as a participant in the recombinational postreplication repair of these replication products.

36 h belong to the RAD6 epistasis group, to the postreplication repair of UV-damaged DNA.

37 umor suppressor gene that might operate in a postreplication repair or a cell cycle checkpoint functi

38 encodes a DNA helicase that is active in the postreplication repair pathway and homologous recombinat

39 ons to promote recombination through a novel postreplication repair pathway and the structure-specifi

40 provides a distinctive opportunity to study postreplication repair processes.

41 in nucleotide excision repair (NER) (rad14), postreplication repair (PRR) (rad6, rad18, mms2, and rad

42 therefore arising through homology-mediated postreplication repair (PRR) events.

43 NA polymerases eta or zeta or Rad5-dependent postreplication repair (PRR) in which error-free replica

44 in conjugating enzymes that are required for postreplication repair (PRR) of DNA and damage-induced m

45 In Saccharomyces cerevisiae, postreplication repair (PRR) of UV-damaged DNA occurs by

46 First, we show that genes in the error-free postreplication repair (PRR) pathway prevent fusion of i

47 ts parallel to the PCNA/Ubc13/Mms2-dependent postreplication repair (PRR) pathway.

48 e damage whereas the recombination (REC) and postreplication repair (PRR) pathways bypass the damage,

49 f1 and different gene products implicated in postreplication repair (PRR) pathways in the suppression

50 Postreplication repair (PRR) pathways play important rol

51 Cells employ error-free or error-prone postreplication repair (PRR) processes to tolerate DNA d

52 This suppression requires the postreplication repair (PRR) protein Rhp18 and the struc

53 have evolved lesion bypass pathways such as postreplication repair (PRR) to resolve these arrested f

54 in conjunction with the error-free branch of postreplication repair (PRR).

55 that, in addition to its established role in postreplication repair, RAD18 is also required for the o

56 HR, resolution of branched HR intermediates, postreplication repair, sumoylation in response to DNA d

57 at, in addition to homologous recombination, postreplication repair, the Fanconi anemia pathway, poly

58 ecognized as an essential step in initiating postreplication repair, the mechanistic relevance of thi

59 In addition to its role in postreplication repair, the MMR machinery serves to poli

60 repair, homologous recombination repair, or postreplication repair when compared with platin control

61 lesion, and homology-dependent repair (HDR; postreplication repair), which is based on the homologou