ライフサイエンスコーパス: postsynaptic

1 nducing the submembranous clustering and the postsynaptic accumulation of gephyrin, which is a scaffo

2 how manipulating the timing of expression of postsynaptic acetylcholine receptors (AChRs) impacts pre

3 tory mechanism in response to the absence of postsynaptic AChRs, may underlie symptoms of neuromuscul

4 en identified, of which a salient one is the postsynaptic actin stabilising protein Drebrin (DBN).

5 o prevented synaptic potentiation induced by postsynaptic action potential trains.

6 ity exploiting the time relationship between postsynaptic action potentials (APs) and EPSPs.

7 postsynaptic MNTB neurons and induced extra postsynaptic action potentials in response to presynapti

8 ind that ACh produces significant excitatory postsynaptic actions on young MGB neurons, probably medi

9 hat were neither causal nor close in time to postsynaptic activation.

10 e inputs exceed the threshold for dominating postsynaptic activity.

11 ibbon size is correlated with differences in postsynaptic activity.

12 Elevated potassium depolarizes the postsynaptic afferent by altering ion permeation through

13 Depolarization of the postsynaptic afferent could also elevate potassium in th

14 ebrafish that have enlarged ribbons, without postsynaptic alterations.

15 utamate release or by direct modification of postsynaptic AMPA (alpha-amino-3-hydroxy-5-methyl-4-isox

16 nd a decrease in the frequency of excitatory postsynaptic AMPA receptor currents in medium spiny proj

17 synaptic connections and is a key player in postsynaptic AMPA receptor endocytosis, providing multip

18 n can occur by the recruitment of additional postsynaptic AMPA receptors (AMPARs), sourced either fro

19 Our data suggest mGlu3-LTD is mediated by postsynaptic AMPAR internalization in PFC pyramidal cell

20 glutamatergic fibers is sufficient to induce postsynaptic AP firing in the absence of AMPA receptors.

21 ter systems are correctly partitioned onto a postsynaptic arbor, are incompletely understood.

22 scale distribution of GABAA receptors in the postsynaptic area is a crucial determinant for the expre

23 he direct physiological functions of PIP2 at postsynaptic as opposed to presynaptic sites.

24 eurotransmitter glutamate that activates the postsynaptic auditory fibers.

25 f long-term potentiation (LTP) that requires postsynaptic brain-derived neurotrophic factor (BDNF)/Tr

26 Brief (1 hr) periods of postsynaptic bursting selectively depressed AMPA recepto

27 specifically is required for LTP induced by postsynaptic Ca(2+)-elevations, a function which may con

28 results suggest presynaptic cadherin-9 binds postsynaptic cadherins-6 and -10 to regulate mushroom sp

29 Our findings reveal how unique postsynaptic CAMs work in concert to control synaptogene

30 zed "detonator" synapses that potently drive postsynaptic cell firing through their profound frequenc

31 y recruiting AMPA glutamate receptors to the postsynaptic cell surface.

32 Neuroligins are postsynaptic cell-adhesion molecules implicated in autis

33 Neuroligins are postsynaptic cell-adhesion molecules that bind to presyn

34 be mediated by interactions between pre- and postsynaptic cell-adhesion molecules.

35 tes to the collection of signals sent to the postsynaptic cell.

36 then discuss their alignment across pre- and postsynaptic cells at a nanometer scale.

37 Neurotransmitter receptors on postsynaptic cells change to match the identity of the n

38 homeostatic potentiation of inhibition onto postsynaptic cells that show increased levels of excitat

39 orchestrated communication between pre- and postsynaptic cells via coordinated trans-synaptic signal

40 ted to impose a transient temporal filter on postsynaptic cells.

41 e resting potentials of both presynaptic and postsynaptic cells.

42 and physiological properties of the pre- and postsynaptic cells.

43 lymerize directly into dendritic spines, the postsynaptic compartment of excitatory neurons in the CN

44 Structural defects observed in the postsynaptic compartment of mutant NMJs include reduced

45 Dendritic spines are small postsynaptic compartments of excitatory synapses in the

46 tion and extrusion of Ca(2+) in the pre- and postsynaptic compartments play a critical role in initia

47 theta frequency stimulation fails to elicit postsynaptic complex-spike bursting and does not induce

48 the induction of synaptic weakening is under postsynaptic control, as it can be prevented by correlat

49 mic cells and the intracellular potential of postsynaptic cortical cells in input layers of primary v

50 nnectivity and the receptive fields (RFs) of postsynaptic cortical neurons.

51 ceptor (AMPAR)-mediated miniature excitatory postsynaptic current (mEPSC) amplitudes due to postsynap

52 ant increases in both spontaneous excitatory postsynaptic current (spEPSC) amplitude and RRP size (es

53 They contribute to the excitatory postsynaptic current and to the detection of painful aci

54 efects, long-term potentiation and miniature postsynaptic current defects.

55 This steady-state postsynaptic current does not increase overall synaptic

56 ty, but induces a steady-state, asynchronous postsynaptic current during stimulus trains.

57 Inhibitory postsynaptic current frequency, measured on pyramidal ne

58 l fiber synapses, mGluR1-mediated excitatory postsynaptic currents (EPSCs) and associated calcium tra

59 ic gene Bax in stem cells reduced excitatory postsynaptic currents (EPSCs) and spine density in matur

60 OF increased depression of inhibitory postsynaptic currents (IPSCs) along IPSC trains evoked b

61 sure in vivo caused a decrease in inhibitory postsynaptic currents (IPSCs) and an increase in the AMP

62 entiated GABAA and GABAB-mediated inhibitory postsynaptic currents (IPSCs) in VTA dopamine neurons, a

63 hibition by morphine of GABAergic inhibitory postsynaptic currents (IPSCs) recorded from neurons in t

64 up of the amplitude of miniature excitatory postsynaptic currents (mEPSCs) and of synaptic levels of

65 Excitatory miniature postsynaptic currents (mEPSCs), but not miniature inhibi

66 rents (mEPSCs), but not miniature inhibitory postsynaptic currents (mIPSCs), increase in amplitude af

67 ency of spontaneous and miniature excitatory postsynaptic currents (s/mEPSCs).

68 currents (sIPSCs) and spontaneous excitatory postsynaptic currents (sEPSCs) three- and two-fold highe

69 sented frequencies of spontaneous inhibitory postsynaptic currents (sIPSCs) and spontaneous excitator

70 nock out (KO) mice display larger excitatory postsynaptic currents and increased spontaneous activity

71 ese ASIC-1as contribute to the generation of postsynaptic currents and, more relevant, to calcium inf

72 gnificant decrease in spontaneous excitatory postsynaptic currents at both two and twenty four hours,

73 n pyramidal neurons and supported inhibitory postsynaptic currents in distal dendrites better than GI

74 ty and the frequency of miniature excitatory postsynaptic currents in the mPFC were prevented by PKMz

75 ame visual stimulus is presented repeatedly, postsynaptic currents vary in amplitude.

76 uced, whereas the amplitude and frequency of postsynaptic currents were enhanced compared with contro

77 t in nanodomains stabilizes the amplitude of postsynaptic currents, indicating that, in addition to t

78 robust monosynaptic GABAergic and nicotinic postsynaptic currents.

79 gnal-to-noise ratio power ratio (SNR) of its postsynaptic currents.

80 tio (SNR) from its presynaptic arrays to its postsynaptic currents.

81 ncreased frequency of spontaneous inhibitory postsynaptic currents.

82 sue exhibit decreased spontaneous inhibitory postsynaptic currents.

83 , accounting for the disappearance of evoked postsynaptic currents.

84 tle consequences for nerve-evoked excitatory postsynaptic currents: vesicle heterogeneity, refractori

85 model that considers the microenvironment of postsynaptic D2 receptors and integrates association and

86 In this study, postsynaptic deletion of Lphn2 selectively decreased spi

87 napse, and the integrative properties of the postsynaptic dendrites.

88 e we show that REM sleep prunes newly formed postsynaptic dendritic spines of layer 5 pyramidal neuro

89 uble-stranded RNA, and longitudinally imaged postsynaptic dendritic spines of layer V pyramidal neuro

90 se sites of rods are apposed by one to three postsynaptic densities (PSDs).

91 labeled endings in the DCN formed asymmetric postsynaptic densities, a feature of excitatory neurotra

92 ), a K63-specific deubiquitinase enriched in postsynaptic densities, cleaves K63-chains from PSD-95.

93 The postsynaptic density (PSD) contains a collection of scaf

94 .2 L-type Ca(2+) channel mRNA and protein in postsynaptic density (PSD) fractions of the hippocampus,

95 ses overall synapse proteome complexity, the postsynaptic density (PSD) proteome of zebrafish has low

96 g, and receptor molecules concentrate at the postsynaptic density (PSD) to regulate synaptic strength

97 nd GEFs have been shown to be present at the postsynaptic density (PSD) within excitatory glutamaterg

98 uter and inner stratifying dendrites express postsynaptic density (PSD95) immunoreactive puncta sugge

99 e we demonstrate that OGT is enriched in the postsynaptic density of excitatory synapses.

100 mals and cultured slices, and an increase in postsynaptic density protein 95 (PSD-95) by overexpressi

101 Postsynaptic density protein 95 (PSD-95), a member of th

102 egradation of the synaptic scaffold protein, postsynaptic density protein 95 (PSD-95), a process that

103 found that the postsynaptic scaffold PSD-95 (postsynaptic density protein 95) undergoes K63 polyubiqu

104 which targets the synaptic scaffold protein, postsynaptic density protein 95, to enhance downstream s

105 Postsynaptic density protein-95 (PSD-95) localizes AMPA-

106 re immunopositive for glutamate receptor and postsynaptic density proteins (viz., GluR1, GluR4, NR1,

107 of Ptchd1-interacting proteins that include postsynaptic density proteins and the retromer complex,

108 ed, including the subsynaptic reticulum, the postsynaptic density, and the glutamate receptor cluster

109 Within the postsynaptic density, however, AMPA receptors coassemble

110 In the postsynaptic density, O-GlcNAcylation on multiple protei

111 tructural protein found predominantly at the postsynaptic density.

112 Its postsynaptic displacement is necessary for loss of AMPAR

113 on the nature of regulation between pre- and postsynaptic dopamine function in healthy adults, which

114 and highlight its role as a key regulator of postsynaptic efficacy.

115 Fourth, both presynaptic and postsynaptic entities are significantly smaller in the m

116 ugments unitary event amplitudes by reducing postsynaptic eukaryotic elongation factor 2 kinase (eEF2

117 perties and reducing frequency of excitatory postsynaptic events.

118 tibular loss triggers transient increases in postsynaptic excitability, occlusion of firing rate pote

119 sting total synaptic conductance is tuned to postsynaptic excitability.

120 s of miniature EPSCs indicative of excessive postsynaptic excitation.

121 the hippocampal CA1 region in mice, blocking postsynaptic expression of both synaptotagmin-1 (Syt1) a

122 Blocking postsynaptic expression of Syt1 and Syt7 did not impair

123 ulum, which could be rescued by pre- but not postsynaptic expression of unc-104.

124 dy provides in vivo indication of a role for postsynaptic factors in amphetamine-induced psychosis in

125 in vitro slice recordings, we have analysed postsynaptic function and pharmacology of neuronal nicot

126 sults indicate that all these differences in postsynaptic function are due to an enhanced activation

127 nd distribution contribute to alterations in postsynaptic function.

128 t various neuroligin isoforms perform unique postsynaptic functions in organizing synapses but are no

129 he presence of either D2L or D2S can support postsynaptic functions related to the control of motor a

130 mate receptors, which exert diverse pre- and postsynaptic functions through complex signaling regulat

131 e GABAergic LTP in mitral cells by enhancing postsynaptic GABA receptor responses.

132 ration (depolarizing vs. hyperpolarizing) of postsynaptic GABAA receptor actions.

133 t responding was best explained by increased postsynaptic gain in primary auditory cortex activity as

134 y and estimated slow fluctuations in neural (postsynaptic) gain.

135 genes encoding neuronal functions including postsynaptic gene ontology categories.

136 ce synaptic bouton enlargement, and increase postsynaptic glutamate receptor abundance.

137 transmission, possibly via the modulation of postsynaptic glutamate receptor functionality.

138 t in GABAergic or glutamatergic neurons) and postsynaptic glutamate receptors.

139 s or 'deleted-in-colorectal-cancer', and the postsynaptic glutamate-receptor-related proteins GluD1 a

140 al sclerosis, also contributes to defects in postsynaptic growth and ubiquitin homeostasis.

141 the Drosophila USP5 deubiquitinase, controls postsynaptic growth.

142 naptic proteins from accumulation to confine postsynaptic growth.

143 stsynaptic current (mEPSC) amplitudes due to postsynaptic homeostatic plasticity that was dependent o

144 GABABRs to suppress GABA release and induce postsynaptic hyperpolarization was unaffected.

145 ic trains of action potentials or by pairing postsynaptic hyperpolarization with activation of group

146 The temporal precision of postsynaptic input integration is known to vary with the

147 rons, Lphn2 maintained synapse numbers via a postsynaptic instead of a presynaptic mechanism, which w

148 NMDA receptors (NMDARs) are a subtype of postsynaptic ionotropic glutamate receptors that functio

149 However, their locus of expression-pre- or postsynaptic-is highly variable.

150 r knowledge, previously unsuspected role for postsynaptic kainate receptors in the induction of funct

151 esynaptic terminals was sharply reduced upon postsynaptic knockout of OGT.

152 lume targeted by TC afferents, the resulting postsynaptic LFP signals were found to be sharply attenu

153 ies via differential binding to multifarious postsynaptic ligands, such as neuroligins, cerebellin/Gl

154 presynaptic neurexins, which bind to diverse postsynaptic ligands.

155 We observed presynaptic and postsynaptic localization of all APP family members and

156 nd enhances the cell surface trafficking and postsynaptic localization of Cav1.2 L-type Ca(2+) channe

157 Presynaptic, but not postsynaptic, loss of vti1a and VAMP7 occludes NMDAR ant

158 synapse damage, revealed by loss of pre- and postsynaptic markers.

159 While postsynaptic maturation, terminal branching and neurotra

160 MB are consolidated into stable LTM at a few postsynaptic MBONs through sequential ORB-regulated loca

161 n the efficacy of GABAergic inhibition via a postsynaptic mechanism.

162 We therefore propose that both pre- and postsynaptic mechanisms at the IHC ribbon synapse contri

163 The molecular composition of the postsynaptic membrane is sculpted by synaptic activity.

164 establish these effects are mediated by both postsynaptic membrane polarization and afferent axon fib

165 itory postsynapses and thus the formation of postsynaptic membrane specializations.

166 n the gene ontologies 'cell projection' and 'postsynaptic membrane' in the gene lists derived from PD

167 ceptors (NMDARs) are stably expressed at the postsynaptic membrane, where they act via Ca(2+) to sign

168 MAP1B in modulating access of AMPARs to the postsynaptic membrane.

169 g of internalized AMPA receptors back to the postsynaptic membrane.

170 Immunoelectron microscopy identified postsynaptic mGluR2/3 in the spines, in addition to the

171 ission to rod ON-bipolar cells, and disrupts postsynaptic mGluR6 clustering.

172 el directing glutamate release (CaV1.4) with postsynaptic mGluR6 receptors.

173 nal transmission from presynaptic calyces to postsynaptic MNTB neurons and induced extra postsynaptic

174 reverses the cocaine-evoked presynaptic and postsynaptic modifications in PL-mPFC and causes long-la

175 e that ppk29 contributes specifically to the postsynaptic modulation of excitatory synaptic transmiss

176 as downstream misregulation of both pre- and postsynaptic molecular assembly.

177 ynapse, we find that Notum secreted from the postsynaptic muscle acts to strongly modulate synapse gr

178 findings indicate selective presynaptic and postsynaptic Nav expression in glutamatergic synapses of

179 d, where they detach from the nerve and form postsynaptic neuroendocrine chromaffin cells.

180 s able to communicate independently with the postsynaptic neuron and trigger downstream signaling cas

181 potassium concentration also depolarized the postsynaptic neuron by altering ion permeation through h

182 associative LTF at another input to the same postsynaptic neuron in an Aplysia sensorimotor preparati

183 rites between different input domains of one postsynaptic neuron without affecting total arbor size.

184 vity and the error signal projected onto the postsynaptic neuron.

185 rotein kinase M (PKM), putative PRPs, in the postsynaptic neuron.

186 ignal independently of evoked release to the postsynaptic neuron.

187 e cause of initial excitotoxic damage to the postsynaptic neuron.

188 e-dependent transgene expression in selected postsynaptic neuronal targets, thus allowing axonal trac

189 Barcodes in pre- and postsynaptic neurons are then associated through protein

190 mmetric, recent work has found that pre- and postsynaptic neurons can contribute different GJ-forming

191 ted channels that flux Na(+) and Ca(2+) into postsynaptic neurons during synaptic transmission.

192 Whether developing postsynaptic neurons establish connections with each pre

193 E STATEMENT The manuscript demonstrates that postsynaptic neurons of the medial nucleus of the trapez

194 DCS polarizes afferent axons and postsynaptic neurons, boosting cooperativity between syn

195 presynaptic partners shapes the responses of postsynaptic neurons.

196 negative constructs in either presynaptic or postsynaptic neurons.

197 e on the spiking activity of presynaptic and postsynaptic neurons.

198 n markers, presynaptic vesicular release and postsynaptic neurotransduction-related machinery is pres

199 m of NMJ plasticity in which perturbation to postsynaptic neurotransmitter receptors leads to a retro

200 ow that activation of presynaptic Nrxns with postsynaptic Nlgn1 or inhibition of ectodomain shedding

201 om L-type voltage-gated Ca(2+) channels, not postsynaptic NMDA receptors (NMDARs), and does not requi

202 ostsynaptic spiking activity, and depends on postsynaptic NMDA receptors and GSK3beta activity.

203 d contributes to potentiated presynaptic and postsynaptic NMDAR activity to elevate sympathetic vasom

204 amatergic synapses, where it associates with postsynaptic NMDARs to modify receptor gating.

205 synaptic proteins cause different aspects of postsynaptic overgrowth in leon mutants.

206 ons, thereby activating the pathway in their postsynaptic partners and providing genetic access to th

207 f individual basket cells on their different postsynaptic partners show high variability, the impact

208 g neurons reprograms the properties of their postsynaptic partners, the RMG hub interneurons.

209 radically differs from traditional models of postsynaptic plasticity.

210 duce stronger attenuation of visually evoked postsynaptic potentials (PSPs) than to auditory evoked P

211 in the inferior olive generate bidirectional postsynaptic potentials (PSPs), with a fast excitatory c

212 entials fully invade spines, that excitatory postsynaptic potentials are large in the spine head (mea

213 ly studied using intracellular recordings of postsynaptic potentials or currents evoked by presynapti

214 rtmentalize voltage, specifically excitatory postsynaptic potentials, albeit critical, remains contro

215 l dendritic interactions via backpropagating postsynaptic potentials.

216 2, and Nav 1.6 were selectively expressed in postsynaptic profiles.

217 K63-polyUb chains play a significant role in postsynaptic protein scaffolding and synaptic strength a

218 2A are enriched for coding variants altering postsynaptic protein-binding domains.

219 Importantly, accumulations of postsynaptic proteins cause different aspects of postsyn

220 meostasis and proper Ubqn levels, preventing postsynaptic proteins from accumulation to confine posts

221 aging and behavior, and to study the role of postsynaptic proteins in localization of brain functions

222 Here, we find that the postsynaptic proteins PSD-93, PSD-95, and SAP102 differe

223 The neocortical postsynaptic proteome data resource can be used to link

224 The postsynaptic proteome of excitatory synapses comprises

225 Here, we document the composition of postsynaptic proteomes in human neocortical regions and

226 dominant interactors play important roles in postsynaptic receptor recycling.

227 azide (CTZ) revealed that desensitization of postsynaptic receptors contributed to synaptic depressio

228 modulation of striatal inputs is mediated by postsynaptic receptors, and that of globus pallidus-evok

229 ubcellular colocalization at presynaptic and postsynaptic regions.

230 ion channels play a fundamental role in the postsynaptic regulation of excitatory neurotransmission.

231 petitive activity, with a large several fold postsynaptic response increase.

232 In the brain, the postsynaptic response of a neuron to time-varying inputs

233 tic and dendrodendritic input determines the postsynaptic response profile.

234 itory balance in which changes in inhibitory postsynaptic response statistics specifically target the

235 m plasticity performs an optimization of the postsynaptic response statistics toward a given mean wit

236 ptic depression results in a quite different postsynaptic response to a large population input depend

237 that not only the rate but the phase of the postsynaptic response to a rhythmic population input var

238 naptic depression, presumably truncating the postsynaptic response to afferent stimuli.

239 receptors that dominate the horizontal cell postsynaptic response.

240 se synapses in vitro evoked large excitatory postsynaptic responses in the majority of pyramidal cell

241 ls and mitral cells, respectively, tunes the postsynaptic responses to high frequency, naturalistic s

242 ential functions in setting the bandwidth of postsynaptic responses, sensitivity to mechanical/excito

243 omains stabilize the amplitude of inhibitory postsynaptic responses, thus identifying the nanoscale g

244 age-related loss in ACh efficacy in evoking postsynaptic responses.

245 a-aminobutyric acid release without altering postsynaptic responses.

246 e neurotransmitter release without affecting postsynaptic responsiveness.

247 n of ppk29 in muscle is necessary for normal postsynaptic responsivity to neurotransmitter release an

248 onserved transmembrane protein known for its postsynaptic roles, functions presynaptically as an orga

249 We found that the postsynaptic scaffold PSD-95 (postsynaptic density prote

250 The postsynaptic scaffolding protein SH3 and multiple ankyri

251 In addition, we observed marked loss of postsynaptic scaffolding proteins and reduced complexity

252 used immunostaining for PSD-95 and gephyrin postsynaptic scaffolding proteins as proxies for excitat

253 genes in these disorders are those encoding postsynaptic scaffolding proteins with roles in synaptic

254 functions asymmetrically, exclusively on the postsynaptic side of the synapse.

255 ate release (e.g., SV2A, synaptogyrin-1) and postsynaptic signaling (e.g., GluA1, PRRT2) with no chan

256 n proteins controlling glutamate release and postsynaptic signaling and discovered several proteins r

257 results reveal a mechanism through which the postsynaptic signaling scaffolds bridge the aroused brai

258 trolling presynaptic release of transmitter, postsynaptic signaling, and synaptic integration.

259 namically from either the presynaptic or the postsynaptic site.

260 ng approaches to identify pathways and their postsynaptic sites in the amygdala in rhesus monkeys, we

261 ory LTP, the increase of gephyrin density at postsynaptic sites is associated with the promoted forma

262 es AMPA-type glutamate receptors (AMPARs) to postsynaptic sites of glutamatergic synapses.

263 the positioning and clustering of AMPARs at postsynaptic sites.

264 llowing subthreshold electrical stimulation, postsynaptic sodium entry is almost entirely through AMP

265 rganization of neurotransmitter receptors in postsynaptic specializations and suggest that they do no

266 In leon mutants, postsynaptic specializations of neuromuscular junctions

267 Our models also capture the differences in postsynaptic spike responses to presynaptic spikes follo

268 iking as a function of the observed pre- and postsynaptic spikes and allow the connection strength (c

269 Pairing presynaptic and postsynaptic spikes at 6-10 Hz reliably induced STDP at

270 but instead sustains reliable generation of postsynaptic spikes that are precisely time locked to pr

271 ths based on the relative timing of pre- and postsynaptic spikes.

272 However, STP also affects the statistics of postsynaptic spikes.

273 ontrol, as it can be prevented by correlated postsynaptic spiking activity, and depends on postsynapt

274 generalized linear model (GLM) that predicts postsynaptic spiking as a function of the observed pre-

275 at LTPpre can be induced by pairing pre- and postsynaptic spiking in the absence of glutamate signall

276 synaptic efficacy as well as current-evoked postsynaptic spiking.

277 ound that Tomo-1 expression levels influence postsynaptic spine density.

278 hair cells (IHCs), and was lacking from the postsynaptic spiral ganglion neurons (SGNs).

279 nts reveal excess dendritic spines, pre- and postsynaptic structural defects, long-term potentiation

280 We show that changes occur in excitatory postsynaptic structure and function in the somatosensory

281 e assembly of the highly asymmetric pre- and postsynaptic structures are still poorly defined.

282 Our results point toward a postsynaptic target cell type-dependent regulation of Ca

283 measured intracellular responses of the main postsynaptic target cell types and with biologically pla

284 monosynaptic thalamic activation of cortical postsynaptic target cells, so called spike-trigger-avera

285 iculation between sensory neuron HOA and its postsynaptic target interneuron AVG: BAM-2/neurexin-rela

286 e axon has high release probability when its postsynaptic target is a parvalbumin-expressing IN.

287 Synapse assembly likely requires postsynaptic target recognition by incoming presynaptic

288 on the number of synapses it makes with its postsynaptic target, the strength of each individual syn

289 egion circuit by acting as a domain-specific postsynaptic target-recognition molecule.

290 tic functions in a stratification level- and postsynaptic target-specific manner, while preserving th

291 raxin then accumulates AMPA receptors on the postsynaptic terminal forming functional synapses.

292 A total of 1 318 700 single pre- or postsynaptic terminals were analysed.

293 orchestrate the function of presynaptic and postsynaptic terminals.

294 axonal microtubules and missorts to pre- and postsynaptic terminals.

295 Drosophila brain (A2 and B1 cells) that are postsynaptic to antennal vibration receptors.

296 link to critical regulators of dendritic and postsynaptic trafficking.

297 as the interneuron is hyperpolarized, or by postsynaptic trains of action potentials in the absence

298 long-lasting potentiation induced either by postsynaptic trains of action potentials or by pairing p

299 tioning increases excitatory presynaptic and postsynaptic transmission in rat PL-mPFC pyramidal neuro

300 aggregation in synapses (pre > pre + post > postsynaptic) was observed.