ライフサイエンスコーパス: postsynaptic action

1 blockade likely increased excitability by a postsynaptic action.

2 ppears to be a more important determinant of postsynaptic action.

3 een the morphology of an interneuron and its postsynaptic action.

4 yperpolarized the cells, suggesting a direct postsynaptic action.

5 in a Ca2+-free solution, indicating a direct postsynaptic action.

6 tetrodotoxin (1 microm), indicating a direct postsynaptic action.

7 sting that this inhibition was mediated by a postsynaptic action.

8 l documented, little is known about possible postsynaptic actions.

9 influences of release yet have antagonistic postsynaptic actions.

10 ic synthesis, vesicular storage, release and postsynaptic actions.

11 released from multiple sites has independent postsynaptic actions.

12 ease glutamate transmission by both pre- and postsynaptic actions.

13 , including those of dynorphin A release and postsynaptic actions, and of CGRP secretion.

14 The convergence or divergence of these postsynaptic actions awaits further investigation.

15 findings provide evidence for segregation of postsynaptic actions between two targets of RT cells and

16 3 receptor activation may be partly a direct postsynaptic action but part may also be due to facilita

17 Lack of postsynaptic action currents in the mutants suggests tha

18 hin inhibits the hypocretin system by direct postsynaptic actions (hyperpolarization, decreased spike

19 Noc has inhibitory postsynaptic actions in CA1, CA3, and the dentate and se

20 have therapeutic effects through unexpected postsynaptic actions in dlPFC, strengthening synaptic co

21 e release of serotonin (5HT) or increase the postsynaptic action of 5HT.

22 While much is now known about the postsynaptic action of neurosteroids, far less is known

23 orporation of synaptic NMDARs and document a postsynaptic action of this major SNARE protein relevant

24 alyze the relative contributions of pre- and postsynaptic actions of a particular gene product in neu

25 ession during hypoxia/cerebral ischemia, but postsynaptic actions of A1Rs are less clear.

26 les to differentiate between presynaptic and postsynaptic actions of BDNF.

27 ese mice, GABAB activity and presynaptic and postsynaptic actions of ethanol were investigated.

28 ynaptic potential blockade demonstrated that postsynaptic actions of Hcrt/Orx alone could evoke prolo

29 Postsynaptic actions of NPY, mediated by both Y1- and Y2

30 GAL has been shown to antagonize the postsynaptic actions of other cosecreted neurotransmitte

31 erally for discriminating presynaptic versus postsynaptic actions of other neurotransmitters and neur

32 h pharmacological studies have described the postsynaptic actions of vestibular efferent stimulation

33 however, the ability of GAL to modulate the postsynaptic actions of VP has not been assessed.

34 We also studied 'postsynaptic' actions of these drugs through their modul

35 gic dendrites and, thus, implicates abundant postsynaptic action on GABAergic neurons via M(2).

36 ind that ACh produces significant excitatory postsynaptic actions on young MGB neurons, probably medi

37 mGluR postsynaptic actions over spinal neurons display the pha

38 In knockout mice, the speed of pre-to-postsynaptic action potential (AP) transmission was redu

39 nt neurotransmitter is released to trigger a postsynaptic action potential (AP).

40 electrically compact granule cells, a single postsynaptic action potential can retard escape of gluta

41 of the amygdala (LA) is only modified when a postsynaptic action potential closely follows a synaptic

42 ited high probability of firing a well timed postsynaptic action potential during high-frequency stim

43 synaptic potentiation that does not require postsynaptic action potential firing in the axon.

44 wn whether LTP can be induced by patterns of postsynaptic action potential firing that occur in these

45 of presynaptic activity properly timed with postsynaptic action potential output can not only increa

46 o prevented synaptic potentiation induced by postsynaptic action potential trains.

47 r some conditions, information regarding the postsynaptic action potential, carried by backpropagatin

48 ity exploiting the time relationship between postsynaptic action potentials (APs) and EPSPs.

49 Repeated induction of pre- and postsynaptic action potentials (APs) at a fixed time dif

50 Ca2+ signals triggered by postsynaptic action potentials (APs) drive the induction

51 y postsynaptic potentials (EPSPs) led single postsynaptic action potentials (APs) within a narrow tem

52 larized (LTP) or hyperpolarized (LTD) and no postsynaptic action potentials are evoked.

53 lso show that coincident individual pre- and postsynaptic action potentials are only capable of induc

54 n re-established the effectiveness of single postsynaptic action potentials at inducing LTP in adult

55 Presynaptic stimulation paired with single postsynaptic action potentials became progressively less

56 ong-term potentiation (LTP), indicating that postsynaptic action potentials can modulate synaptic pla

57 ptic potentials (EPSPs) were paired with two postsynaptic action potentials in a theta-burst pattern,

58 postsynaptic MNTB neurons and induced extra postsynaptic action potentials in response to presynapti

59 lying oscillatory somatic [Ca2+]i by evoking postsynaptic action potentials in SCN neurons during a p

60 Near coincidental pre- and postsynaptic action potentials induce associative long-t

61 Initially, the timing of presynaptic and postsynaptic action potentials must be translated into s

62 ned by the precise timing of presynaptic and postsynaptic action potentials on a millisecond timescal

63 thermore, coincident synaptic activation and postsynaptic action potentials rapidly restrict diffusio

64 z and that the timing of individual pre- and postsynaptic action potentials relative to one another i

65 When glutamate was paired with postsynaptic action potentials, however, the NMDA-recept

66 synaptic glutamate receptors, and 26% was on postsynaptic action potentials, in approximate accord wi

67 tion rate; however, pairs of presynaptic and postsynaptic action potentials, repeated at frequencies

68 llowed 30 ms later by depolarization-induced postsynaptic action potentials.

69 o the rate and the timing of presynaptic and postsynaptic action potentials.

70 critically on the timing of presynaptic and postsynaptic action potentials.

71 ptic stimulation, and the timing of pre- and postsynaptic action potentials.

72 fficacy can depend on the timing of pre- and postsynaptic action potentials.

73 modified according to the timing of pre- and postsynaptic action potentials.

74 , depends on the relative timing of pre- and postsynaptic action potentials.

75 by STDP compete for control of the timing of postsynaptic action potentials.

76 ly increased likelihood of firing well-timed postsynaptic action potentials.

77 ugh glutamate and GABA have clearly distinct postsynaptic actions, we are just beginning to appreciat

78 Both presynaptic and postsynaptic actions were specific because they were not

79 atergic transmission through a CRF1-mediated postsynaptic action, whereas Ucn I facilitated synaptic