ライフサイエンスコーパス: postsynaptic current

1 ala pyramidal neurons, generating excitatory postsynaptic currents.

2 sue exhibit decreased spontaneous inhibitory postsynaptic currents.

3 e is essential for fast, glutamate-activated postsynaptic currents.

4 quency of glutamatergic miniature excitatory postsynaptic currents.

5 increased amplitude of miniature inhibitory postsynaptic currents.

6 and frequency changes in miniature GABAergic postsynaptic currents.

7 , but not frequency, of miniature excitatory postsynaptic currents.

8 s a rapid increase of spontaneous excitatory postsynaptic currents.

9 robust monosynaptic GABAergic and nicotinic postsynaptic currents.

10 ase in the amplitude of miniature inhibitory postsynaptic currents.

11 A(A) receptor (GABA(A)R)-mediated inhibitory postsynaptic currents.

12 plitude of alpha3*-nAChR-mediated excitatory postsynaptic currents.

13 requency of GABAergic spontaneous inhibitory postsynaptic currents.

14 ion restricts the potentiation of excitatory postsynaptic currents.

15 AMPA receptor-mediated miniature excitatory postsynaptic currents.

16 mber and reduces the amplitude of inhibitory postsynaptic currents.

17 ease in the amplitude of GABAergic miniature postsynaptic currents.

18 mplitude of spontaneous miniature excitatory postsynaptic currents.

19 , accounting for the disappearance of evoked postsynaptic currents.

20 gnal-to-noise ratio power ratio (SNR) of its postsynaptic currents.

21 termined by analysis of miniature excitatory postsynaptic currents.

22 action potentials and spontaneous excitatory postsynaptic currents.

23 tio (SNR) from its presynaptic arrays to its postsynaptic currents.

24 precise interaction of excitatory-inhibitory postsynaptic currents.

25 ncreased frequency of spontaneous inhibitory postsynaptic currents.

26 ease in synaptic AMPA receptors (AMPARs) and postsynaptic currents.

27 ecording 5-HT1A receptor-mediated inhibitory postsynaptic currents (5-HT1A -IPSCs) generated by the a

28 wer rate of spontaneous miniature inhibitory postsynaptic current activity.

29 xcitatory transmitter release and excitatory postsynaptic currents also were heightened at synapses b

30 uced decay time of AMPAR-mediated excitatory postsynaptic currents (AMPAR-EPSCs), enhanced depression

31 artly by an increase in miniature excitatory postsynaptic current amplitude and partly by a mechanism

32 speptin neurons, estradiol reduced miniature postsynaptic current amplitude independent of time of da

33 affinity, the decrease in arcuate miniature postsynaptic current amplitude was attributed to decreas

34 zepam treatment reduced miniature inhibitory postsynaptic current amplitude, which returned to contro

35 eficits in membrane properties and GABAergic postsynaptic current amplitude.

36 t frequencies, although miniature excitatory postsynaptic current amplitudes remained similar.

37 type serotonin receptor to reduce excitatory postsynaptic current amplitudes, an effect previously sh

38 hyrin clusters and mean miniature inhibitory postsynaptic current amplitudes, whereas a dominant nega

39 nt kinetics and reduced miniature inhibitory postsynaptic current amplitudes.

40 metabolic advantage over quantal excitatory postsynaptic currents--an advantage that may have driven

41 notypes, decreased excitatory and inhibitory postsynaptic current and reduced c-Fos immunoreactivity

42 They contribute to the excitatory postsynaptic current and to the detection of painful aci

43 eptors in MSNs, consisting of an increase in postsynaptic currents and a decrease of presynaptic inhi

44 KO of NL1 impaired NMDAR-mediated excitatory postsynaptic currents and abolished NMDAR-dependent LTP.

45 ed AMPA receptor (AMPAR)-mediated excitatory postsynaptic currents and AMPAR surface expression in pr

46 causes a decrease in NMDA-receptor-mediated postsynaptic currents and an increase in presynaptic glu

47 potentiation, altered amplitude of miniature postsynaptic currents and elevated dopamine in basal for

48 n of these channels reduces PF-PC excitatory postsynaptic currents and excitatory postsynaptic potent

49 Measurements of spontaneous glycinergic postsynaptic currents and GlyR immunolabeling revealed t

50 lectrophysiological recordings of excitatory postsynaptic currents and hippocampal long-term potentia

51 nock out (KO) mice display larger excitatory postsynaptic currents and increased spontaneous activity

52 lectrophysiological recordings of excitatory postsynaptic currents and long-term potentiation in brai

53 KO mice, AMPAR-mediated miniature excitatory postsynaptic currents and synaptic levels of AMPAR subun

54 en in the frequency of spontaneous miniature postsynaptic currents and the size of the readily releas

55 in-2 elicited both inhibitory and excitatory postsynaptic currents and triggered network bursting in

56 ese ASIC-1as contribute to the generation of postsynaptic currents and, more relevant, to calcium inf

57 taneous GABA(A) receptor-mediated inhibitory postsynaptic currents, and are blocked by gabazine or te

58 x into the cytosol, a decrease of inhibitory postsynaptic currents, and ultimately a shift of GABA-el

59 Spike-independent miniature postsynaptic currents are generally stochastic and are t

60 Spontaneous postsynaptic currents are lower in amplitude and have fa

61 ynamins, dynamin 1 and 3, smaller excitatory postsynaptic currents are observed due to an impairment

62 gnificant decrease in spontaneous excitatory postsynaptic currents at both two and twenty four hours,

63 in the frequency and amplitude of GABAergic postsynaptic currents beginning approximately 14 d post-

64 te that hESC-derived neurons receive unitary postsynaptic currents both in vitro and in vivo and adop

65 tion and charge transfer rate of spontaneous postsynaptic current bursts at the neuromuscular junctio

66 The persistent current of cholinergic postsynaptic current bursts is mostly mediated by levami

67 that motoneurons control muscle by producing postsynaptic current bursts.

68 trol animals, IL-6 did not affect excitatory postsynaptic currents but selectively and reversibly red

69 localized ASICs contribute to the excitatory postsynaptic current by responding to the transient acid

70 the depression of AMPAR-mediated excitatory postsynaptic currents by SNRIs required p38 kinase activ

71 The reversal potential of the compound postsynaptic currents (combined simultaneous EPSCs and I

72 litude and frequency of miniature inhibitory postsynaptic currents compared with those in wild-type s

73 Our estimates indicate that APs and postsynaptic currents contribute similar proportions of

74 ower kinetics in vitro and slower excitatory postsynaptic current decays in neurons.

75 The IL-6-induced inhibitory postsynaptic currents decrease was inhibited by drugs in

76 inhibited by tetrodotoxin (TTX), inhibitory postsynaptic currents decreased and currents were no lon

77 The frequency of miniature excitatory postsynaptic currents decreased, accompanied by dendriti

78 efects, long-term potentiation and miniature postsynaptic current defects.

79 This steady-state postsynaptic current does not increase overall synaptic

80 chanisms, we identified a previously unknown postsynaptic current during neurotransmission that was m

81 ty, but induces a steady-state, asynchronous postsynaptic current during stimulus trains.

82 techniques, evoked or spontaneous excitatory postsynaptic currents (eEPSCs or sEPSCs) were recorded f

83 Excitatory postsynaptic currents (eEPSCs) were evoked by the stimul

84 amplitude of evoked NMDA-mediated excitatory postsynaptic currents (eEPSCs), without affecting AMPA-m

85 Inhibitory postsynaptic currents (eIPSCs) were evoked after the sti

86 mplitude of GABAA-mediated evoked inhibitory postsynaptic currents (eIPSCs).

87 increased amplitudes of miniature excitatory postsynaptic currents, enhancement of LTP, and eliminati

88 itional properties, Q and average excitatory postsynaptic current (EPSC) amplitude, were unaffected b

89 n of these failures leads to mean excitatory postsynaptic current (EPSC) amplitudes that are independ

90 release, as measured by miniature excitatory postsynaptic current (EPSC) analysis and FM 1-43 stainin

91 normal mice, a glutamate-mediated excitatory postsynaptic current (EPSC) between mitral cells emerged

92 steady-state NMDA currents, NMDAR excitatory postsynaptic current (EPSC) decay kinetics, progressive

93 vealed by a decrease in miniature excitatory postsynaptic current (EPSC) frequency and in the input-o

94 geminal afferent fibres evoked an excitatory postsynaptic current (EPSC) in trigeminal neurones with

95 miniature and stimulation-evoked excitatory postsynaptic current (EPSC).

96 o reverse ion influxes generating excitatory postsynaptic currents (EPSCs) and action potentials.

97 l fiber synapses, mGluR1-mediated excitatory postsynaptic currents (EPSCs) and associated calcium tra

98 MS patients on glutamate-mediated excitatory postsynaptic currents (EPSCs) and excitotoxic damage in

99 NMDAR-mediated excitatory postsynaptic currents (EPSCs) and puff NMDA-elicited cur

100 ic gene Bax in stem cells reduced excitatory postsynaptic currents (EPSCs) and spine density in matur

101 THC (1 microM) inhibited excitatory postsynaptic currents (EPSCs) and whole-cell I(Ca) evoke

102 utamate uncaging sites from which excitatory postsynaptic currents (EPSCs) could be evoked ("hotspot

103 increased duration of spontaneous excitatory postsynaptic currents (EPSCs) during the symptomatic pha

104 ed neurons were patch-clamped and excitatory postsynaptic currents (EPSCs) evoked by electrically sti

105 ced the amplitude of monosynaptic excitatory postsynaptic currents (EPSCs) evoked from the dorsal roo

106 aviors and associated deficits in excitatory postsynaptic currents (EPSCs) generated in apical dendri

107 duction of AMPA receptor-mediated excitatory postsynaptic currents (EPSCs) in cortical neurons.

108 -dependent inhibition of autaptic excitatory postsynaptic currents (EPSCs) in cultured hippocampal ne

109 rease in frequency of spontaneous excitatory postsynaptic currents (EPSCs) in layer V pyramidal neuro

110 increased the amplitude of basal excitatory postsynaptic currents (EPSCs) in MAGL(-/-) mice but not

111 Excitatory postsynaptic currents (EPSCs) in monosynaptic nTS neuron

112 ST stimulation elicits excitatory postsynaptic currents (EPSCs) in NTS neurons mediated by

113 Here we examined excitatory postsynaptic currents (EPSCs) of dorsal horn neurons evo

114 n the amplitude of NMDAR-mediated excitatory postsynaptic currents (EPSCs) of dorsal horn neurons evo

115 Here, we found that evoked NMDAR-excitatory postsynaptic currents (EPSCs) of retrogradely labeled sp

116 partate receptor (NMDAR)-mediated excitatory postsynaptic currents (EPSCs) onto VTA dopamine neurons.

117 The excitatory postsynaptic currents (EPSCs) recorded in laminae III-V

118 Evaluation of excitatory postsynaptic currents (EPSCs) revealed that rCASP6 rapid

119 been shown to arise from complex excitatory postsynaptic currents (EPSCs) that are evoked in SNR neu

120 -cell voltage clamp recordings of excitatory postsynaptic currents (EPSCs) were performed on dorsal h

121 Excitatory postsynaptic currents (EPSCs) were recorded from superfi

122 he time course of Purkinje neuron excitatory postsynaptic currents (EPSCs).

123 reased the frequency of miniature excitatory postsynaptic currents (EPSCs).

124 (2)AR, and AMPA receptor-mediated excitatory postsynaptic currents (EPSCs).

125 iniature excitatory transmission (excitatory postsynaptic currents, EPSCs).

126 nd increased frequency and mean amplitude of postsynaptic current events.

127 We find that postsynaptic currents exhibit a decreasing ability to ge

128 of an initial fast glutamatergic excitatory postsynaptic current followed by a slow-rising cholinerg

129 inputs, we recorded light-evoked excitatory postsynaptic currents following viral-mediated expressio

130 , together with reduced miniature excitatory postsynaptic current frequencies and behavioral defects.

131 ntly, by a reduction of miniature excitatory postsynaptic current frequencies, although miniature exc

132 sociated with increased miniature excitatory postsynaptic current frequency and amplitude, suggesting

133 imulation of astrocytes increased excitatory postsynaptic current frequency through a metabotropic gl

134 Inhibitory postsynaptic current frequency, measured on pyramidal ne

135 Here we show that postsynaptic currents frequently occur in bursts at the

136 ic glutamate receptors is decreased, but the postsynaptic current from activation of NMDA-specific gl

137 ed by these synapses, we recorded excitatory postsynaptic currents from mammalian retinal ganglion ce

138 bition of GABAA receptor-mediated inhibitory postsynaptic currents (GABAAR IPSCs) is associated with

139 y, whereas in naive mice DAMGO inhibits GABA postsynaptic currents, GABAergic currents are potentiate

140 However, unitary postsynaptic currents generated by individual, GAD67-exp

141 reduced frequencies of miniature excitatory postsynaptic currents, i.e., to defects in synaptic tran

142 e D in the generation of the slow excitatory postsynaptic current in cerebellar Purkinje cells.

143 revealed a concomitant nonquantal excitatory postsynaptic current in the calyx terminal that was caus

144 tion of electrically evoked local excitatory postsynaptic current in the saline and ShA groups.

145 However, the average postsynaptic current in the VNLL fails to elicit APs in

146 release from cones was measured by recording postsynaptic currents in Ambystoma tigrinum horizontal o

147 We find that postsynaptic currents in bipolar MSO neuron models gener

148 that the frequency of spontaneous excitatory postsynaptic currents in dentate gyrus granule cells is

149 n pyramidal neurons and supported inhibitory postsynaptic currents in distal dendrites better than GI

150 t not the amplitude, of miniature excitatory postsynaptic currents in dorsal horn neurons that could

151 n terminals, which was detected by recording postsynaptic currents in downstream neurons.

152 lisecond flashes of blue light produced fast postsynaptic currents in HA neurons, with a high connect

153 m influx, and increased miniature excitatory postsynaptic currents in hippocampal neurons.

154 d to the hippocampus of adult mice triggered postsynaptic currents in host pyramidal neurons in acute

155 d decrements in apically targeted excitatory postsynaptic currents in layer V pyramidal cells of PFC.

156 axons resulting in characteristic "doublet" postsynaptic currents in LSO neurons.

157 lc7a10-null mice and spontaneous glycinergic postsynaptic currents in motor neurons show substantiall

158 glutamatergic interneuron-evoked inhibitory postsynaptic currents in motor neurons.

159 spine density/diameter, increased excitatory postsynaptic currents in mPFC layer V pyramidal neurons,

160 ally for receptors containing GluK5, as were postsynaptic currents in neurons expressing recombinant

161 rate of spontaneous and miniature excitatory postsynaptic currents in pyramidal neurons and hippocamp

162 cacy of spontaneous and miniature inhibitory postsynaptic currents in pyramidal neurons.

163 ncreased frequency of spontaneous excitatory postsynaptic currents in spinal cord nociceptive neurons

164 ly block NMDAR-mediated miniature excitatory postsynaptic currents in the absence of Mg(2+).

165 cts of mu-opioid receptor modulation of GABA postsynaptic currents in the mouse VTA 1 d after chronic

166 ty and the frequency of miniature excitatory postsynaptic currents in the mPFC were prevented by PKMz

167 Examination of the light-evoked postsynaptic currents in these ON-type orientation-selec

168 tor (GABAAR)-mediated spontaneous inhibitory postsynaptic currents in WT mice, indicating that astroc

169 in driving the activity of, or in producing postsynaptic currents in, Purkinje cells in cerebellar s

170 ynaptic transmission (spontaneous excitatory postsynaptic currents) in spinal lamina IIo nociceptive

171 ency of excitatory miniature and spontaneous postsynaptic currents increased.

172 -1 (TRPV1)- and TNF-alpha-induced excitatory postsynaptic current increases and TNF-alpha-evoked N-me

173 t in nanodomains stabilizes the amplitude of postsynaptic currents, indicating that, in addition to t

174 Specifically, miniature excitatory postsynaptic currents, input resistance, hippocampal lon

175 potential (LFP), thought to be dominated by postsynaptic currents integrated over a larger volume of

176 This inhibitory postsynaptic current (IPSC) erosion resulted from a fail

177 a GABA(A) receptor-mediated slow inhibitory postsynaptic current (IPSC) in a PN and an autaptic IPSC

178 oduced a 5-HT1A receptor-mediated inhibitory postsynaptic current (IPSC) that resulted in only a tran

179 OF increased depression of inhibitory postsynaptic currents (IPSCs) along IPSC trains evoked b

180 sure in vivo caused a decrease in inhibitory postsynaptic currents (IPSCs) and an increase in the AMP

181 ll mutations prolong the decay of inhibitory postsynaptic currents (IPSCs) and induce spontaneous Gly

182 pronounced initial depression of inhibitory postsynaptic currents (IPSCs) followed by modest long-te

183 rminals evoked robust D2-receptor inhibitory postsynaptic currents (IPSCs) in GIRK2-expressing MSNs t

184 of the frequency potentiation of inhibitory postsynaptic currents (IPSCs) in hypoglossal motoneurons

185 induces rhythmic, theta-frequency inhibitory postsynaptic currents (IPSCs) in pyramidal cells, even w

186 nd that GABA(A)-receptor-mediated inhibitory postsynaptic currents (IPSCs) in the inhibitory low-thre

187 entiated GABAA and GABAB-mediated inhibitory postsynaptic currents (IPSCs) in VTA dopamine neurons, a

188 ked I-LTD and acute depression of inhibitory postsynaptic currents (IPSCs) induced by D(2) dopamine r

189 ong-lasting enhancement of evoked inhibitory postsynaptic currents (IPSCs) mediated by D1-type recept

190 inergic spontaneous and miniature inhibitory postsynaptic currents (IPSCs) of lamina II neurons.

191 hibition by morphine of GABAergic inhibitory postsynaptic currents (IPSCs) recorded from neurons in t

192 Here the duration of inhibitory postsynaptic currents (IPSCs) was dependent on the numbe

193 ntaneous inhibitory transmission (inhibitory postsynaptic currents, IPSCs) in the DR.

194 ces, NMDAR-dependent LTD of AMPAR excitatory postsynaptic currents is abolished in neurons expressing

195 ndeed, the amplitude of miniature excitatory postsynaptic currents is enhanced in both Vac14(-/-) and

196 atios ( approximately 40:1), fast inhibitory postsynaptic current kinetics (tau(decay) = 2.5 ms) and

197 ered the time course of miniature inhibitory postsynaptic current kinetics and reduced miniature inhi

198 By regulating KAR excitatory postsynaptic current kinetics, Neto1 can control synapti

199 the light-evoked and spontaneous excitatory postsynaptic currents (light-evoked EPSCs and sEPSCs) fr

200 uency of GABAA receptor-mediated spontaneous postsynaptic currents (LTP-GABAA) and this mechanism is

201 e cerebellum also leads to a slow excitatory postsynaptic current mediated by nonselective TRPC3 cati

202 clustering at synapses, miniature excitatory postsynaptic currents mediated by TARPless AMPARs were r

203 y describes spontaneous miniature inhibitory postsynaptic currents mediated by vesicular dopamine rel

204 ceptor (AMPAR)-mediated miniature excitatory postsynaptic current (mEPSC) amplitudes due to postsynap

205 ry synapses assessed by miniature excitatory postsynaptic current (mEPSC) and electron microscopy.

206 educes the frequency of miniature excitatory postsynaptic current (mEPSC), and reduces AMPA-induced m

207 uency of AMPAR-mediated miniature excitatory postsynaptic current (mEPSC), while expressing wild-type

208 up of the amplitude of miniature excitatory postsynaptic currents (mEPSCs) and of synaptic levels of

209 scaling, all of a cell's AMPAergic miniature postsynaptic currents (mEPSCs) are increased or decrease

210 the NMDAR component of miniature excitatory postsynaptic currents (mEPSCs) in CA1 interneurons but n

211 mplitude of spontaneous miniature excitatory postsynaptic currents (mEPSCs) in cultured mouse hippoca

212 tch clamp recordings of miniature excitatory postsynaptic currents (mEPSCs) revealed that these behav

213 Miniature excitatory postsynaptic currents (mEPSCs) were recorded from CA1 py

214 and charge transfer of miniature excitatory postsynaptic currents (mEPSCs) were significantly reduce

215 d the amplitude of AMPA miniature excitatory postsynaptic currents (mEPSCs), and shRNA-mediated GRIP1

216 Excitatory miniature postsynaptic currents (mEPSCs), but not miniature inhibi

217 n microscopy, and giant miniature excitatory postsynaptic currents (mEPSCs), reflecting more transmit

218 reduction in evoked and miniature excitatory postsynaptic currents (mEPSCs).

219 ase in the frequency of miniature excitatory postsynaptic currents (mEPSCs).

220 mplitude of spontaneous miniature excitatory postsynaptic currents (mEPSCs); however, synaptic facili

221 to reduced frequency of miniature excitatory postsynaptic currents (mini-EPSCs) and smaller release-r

222 In contrast, spontaneous postsynaptic currents ("minis") resulting from stochasti

223 1 significantly reduced miniature inhibitory postsynaptic current (mIPSC) amplitudes and GABA levels

224 exhibit increased basal miniature inhibitory postsynaptic current (mIPSC) frequency but showed a decr

225 ltered sIPSC amplitude, miniature inhibitory postsynaptic current (mIPSC) frequency, and mIPSC amplit

226 uency of miniature inhibitory and excitatory postsynaptic currents (mIPSCs and mEPSCs, respectively)

227 creased EtOH sensitivity of GABAAR miniature postsynaptic currents (mIPSCs) correlated with EtOH depe

228 lopment the duration of miniature inhibitory postsynaptic currents (mIPSCs) mediated by GABAA recepto

229 lopment the duration of miniature inhibitory postsynaptic currents (mIPSCs) mediated by GABAA recepto

230 n potential independent miniature inhibitory postsynaptic currents (mIPSCs) was significantly increas

231 and charge transfer of miniature inhibitory postsynaptic currents (mIPSCs) were significantly increa

232 rents (mEPSCs), but not miniature inhibitory postsynaptic currents (mIPSCs), increase in amplitude af

233 s) and the frequency of miniature inhibitory postsynaptic currents (mIPSCs).

234 ids act tonically to regulate AMPA miniature postsynaptic current (mPSC) frequency in embryonic motor

235 uration of GABAA receptor (GABAAR) miniature postsynaptic currents (mPSCs) was substantially blunted

236 generated primarily by the summation of the postsynaptic currents of cortical principal cells.

237 and the amplitude of monosynaptic excitatory postsynaptic currents of dorsal horn neurons in SNL rats

238 capacitance changes from individual rods and postsynaptic currents of second-order neurons.

239 the frequency and the amplitude of GABAergic postsynaptic currents of SF-1 GFP-positive neurons.

240 mall DRG neurons and monosynaptic excitatory postsynaptic currents of spinal dorsal horn neurons evok

241 us or endogenous IL-6 on electrically evoked postsynaptic currents on a cortical rat slice preparatio

242 ut not the frequency of miniature inhibitory postsynaptic currents or expression of the glutamic acid

243 sociate with changes in miniature excitatory postsynaptic currents or paired-pulse facilitation, sugg

244 ortical neurons exhibited reduced inhibitory postsynaptic current peak amplitudes, prolonged current

245 All postsynaptic currents (PSCs) in developed hcrt/orx cells

246 Thus rapid postsynaptic currents (PSCs) mediated by adenosine (equi

247 Spontaneous postsynaptic currents (PSCs) provide key information abo

248 GABAergic and glutamatergic postsynaptic currents (PSCs), both of which can excite t

249 n frequency, but not amplitude, of miniature postsynaptic currents (PSCs).

250 e exhibit a decrease in NMDA/AMPA excitatory postsynaptic current ratio in area CA1 of the hippocampu

251 d more actively with an increased excitatory postsynaptic current response upon the application of ni

252 mbrane excitability and divergent inhibitory postsynaptic current responses to CRF application.

253 ency of spontaneous and miniature excitatory postsynaptic currents (s/mEPSCs).

254 cs of sound-evoked excitatory and inhibitory postsynaptic currents (seEPSCs and seIPSCs, respectively

255 cing the frequency of spontaneous excitatory postsynaptic current (sEPSCs).

256 affected, we examined spontaneous excitatory postsynaptic currents (sEPSCs) and dopamine (DA) modulat

257 We recorded spontaneous excitatory postsynaptic currents (sEPSCs) from lamina II neurons in

258 requency of spontaneous glutamate excitatory postsynaptic currents (sEPSCs) in >90% of NTS-TH-EGFP ne

259 ased the frequency of spontaneous excitatory postsynaptic currents (sEPSCs) only in Ndufs4(KO) CA1 ne

260 currents (sIPSCs) and spontaneous excitatory postsynaptic currents (sEPSCs) three- and two-fold highe

261 ased the frequency of spontaneous excitatory postsynaptic currents (sEPSCs) without affecting their a

262 ch clamp recordings of spontaneous miniature postsynaptic currents show that patient antibodies decre

263 -1 increased both the spontaneous inhibitory postsynaptic current (sIPSC) amplitudes and frequency by

264 ad no clear effect on spontaneous inhibitory postsynaptic current (sIPSC) frequency at IN-PC synapses

265 d large reductions in spontaneous inhibitory postsynaptic current (sIPSC) frequency in both granule c

266 Spontaneous and miniature inhibitory postsynaptic currents (sIPSCS and mIPSCs) were recorded

267 sented frequencies of spontaneous inhibitory postsynaptic currents (sIPSCs) and spontaneous excitator

268 uced the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) but did not alter sIPSC a

269 es and frequencies of spontaneous inhibitory postsynaptic currents (sIPSCs) in CA1 pyramidal neurons.

270 on the properties of spontaneous inhibitory postsynaptic currents (sIPSCs) in cultured rat hippocamp

271 xpression and reduced spontaneous inhibitory postsynaptic currents (sIPSCs) in D1-type, but not D2-ty

272 (A) receptor-mediated spontaneous inhibitory postsynaptic currents (sIPSCs) in dlBnST target neurons,

273 ased the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) in downstream hippocampal

274 et charge transfer of spontaneous inhibitory postsynaptic currents (sIPSCs) recorded from CA1 pyramid

275 Spontaneous inhibitory postsynaptic currents (sIPSCs) were not differentially a

276 ency and amplitude of spontaneous inhibitory postsynaptic currents (sIPSCs) were significantly decrea

277 ant increases in both spontaneous excitatory postsynaptic current (spEPSC) amplitude and RRP size (es

278 tude and frequency of spontaneous excitatory postsynaptic currents (spEPSCs) increased during PTP in

279 etics of GABAA receptor-mediated spontaneous postsynaptic currents (sPSCs) or tonic currents in GnRH

280 lled the amplitude and variance of simulated postsynaptic currents, suggesting several ways in which

281 ase in the frequency of miniature excitatory postsynaptic currents that could be rapidly and fully su

282 d, nociceptin, and GABA(B) receptors induced postsynaptic currents that desensitized within minutes,

283 ed by depression accompanied by asynchronous postsynaptic currents that increase steadily during the

284 g steps to the hair cell were used to elicit postsynaptic currents that occurred at constant phase fo

285 , trains of depolarizations evoke excitatory postsynaptic currents that show facilitation followed by

286 Dalpha7-nAChRs boosted miniature excitatory postsynaptic currents, the ensuing increase in Shal chan

287 First, two distinct excitatory postsynaptic currents transmit signals on different time

288 and a reduction of spontaneous glutamatergic postsynaptic currents, underscoring the relevance of aff

289 We also recorded postsynaptic currents using in vitro whole cell recordin

290 ame visual stimulus is presented repeatedly, postsynaptic currents vary in amplitude.

291 tle consequences for nerve-evoked excitatory postsynaptic currents: vesicle heterogeneity, refractori

292 quency of GABAA receptor-mediated inhibitory postsynaptic currents was depressed in medium spiny neur

293 mice, the frequency of GABAergic spontaneous postsynaptic currents was increased in the PM; this incr

294 because alpha(2)-AR-mediated slow inhibitory postsynaptic currents were depressed in wt but not betaa

295 uced, whereas the amplitude and frequency of postsynaptic currents were enhanced compared with contro

296 le-cell patch clamp recordings of inhibitory postsynaptic currents were performed from ventral tegmen

297 Both evoked and spontaneous excitatory postsynaptic currents were predominantly glutamatergic.

298 versibly reduced the amplitude of inhibitory postsynaptic currents with a postsynaptic effect.

299 mplex dendritic arbors and display GABAergic postsynaptic currents with immature kinetics.

300 reases the frequency of miniature inhibitory postsynaptic currents with no effect on amplitude, which