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1 onally considered solely as ion carriers for postsynaptic depolarization.
2 term depression relied on NMDA receptors and postsynaptic depolarization.
3 ulation or pairing afferent stimulation with postsynaptic depolarization.
4 ced by presynaptic activity but prevented by postsynaptic depolarization.
5 e receptors (AMPARs) mediate the majority of postsynaptic depolarization.
6 pairing of photoreleased glutamate and brief postsynaptic depolarization.
7 ion or low-frequency stimulation paired with postsynaptic depolarization.
8 utamate-dependent coincident presynaptic and postsynaptic depolarization.
9 ing rate, or changes in the average level of postsynaptic depolarization.
10 w-frequency synaptic stimulation paired with postsynaptic depolarization.
11 TD) when presynaptic activity coincides with postsynaptic depolarization.
12 presynaptic glutamate release coincides with postsynaptic depolarization.
13                            We also find that postsynaptic depolarization alone can evoke GABA release
14           Fluorometric imaging revealed that postsynaptic depolarization also reduces presynaptic cal
15                              Further, strong postsynaptic depolarizations also induced a short-lived
16 ed) increased in amplitude and duration with postsynaptic depolarization, although in two (of 4) bask
17 sm at these synapses, which does not require postsynaptic depolarization and acts via presynaptic AMP
18 entials (dendritic spikes) contribute to the postsynaptic depolarization and calcium entry necessary
19    Induction of striatal LTD is dependent on postsynaptic depolarization and calcium influx through L
20 rm of presynaptic depression is triggered by postsynaptic depolarization and is likely to play an imp
21                                   Associated postsynaptic depolarization and RyR activation, therefor
22 methylester (FPL64176), combined with modest postsynaptic depolarization and synaptic activation, is
23 ignals than previously recognized, including postsynaptic depolarization and the activation of NMDARs
24 inhibition of carbachol-induced (3.0 microM) postsynaptic depolarizations and increases in input resi
25 unction, the nicotinic ACh receptor mediates postsynaptic depolarization, and acetylcholinesterase (A
26 collateral synapses requires NMDA receptors, postsynaptic depolarization, and postsynaptic calcium, a
27 sed from DMH neurons in response to repeated postsynaptic depolarizations, and acts in an autocrine f
28 potentials were highly correlated with total postsynaptic depolarization at rest.
29  tetanic stimulation that produced prolonged postsynaptic depolarization (but not spikes), LTP was de
30 neurons, coincident presynaptic activity and postsynaptic depolarization do not generate long-term po
31 anti-Hebbian' LTP because it is prevented by postsynaptic depolarization during afferent activity, an
32 cy afferent stimulation paired with moderate postsynaptic depolarization elicited an N-methyl-d-aspar
33 gulated release, we confirm a model in which postsynaptic depolarization elicits calcium-dependent re
34 h ionotropic receptor blockade to dissociate postsynaptic depolarization from receptor activation.
35  of these features to presynaptic firing and postsynaptic depolarization from regions either close to
36 of mossy fibers, either alone or paired with postsynaptic depolarization, gives rise to long-term dep
37 ated potassium (BK) channels, which suppress postsynaptic depolarization in A17s and limit Ca(v) chan
38 ynaptic GABA(A) receptor activation triggers postsynaptic depolarizations in mouse TRN neurons.
39 oM) did not have a significant effect on the postsynaptic depolarization, increase in input resistanc
40  Associating single-cell RyR activation with postsynaptic depolarization increased intracellular free
41 icotine; (2) pulsed nicotine did not enhance postsynaptic depolarizations induced by iontophoreticall
42 rt-term depression is typically triggered by postsynaptic, depolarization-induced calcium rises, wher
43  high-frequency presynaptic stimulation with postsynaptic depolarization induces Hebbian long-term po
44 brief presynaptic activation coincident with postsynaptic depolarization is required to induce the He
45 over") from stimulated synapses, paired with postsynaptic depolarization, is sufficient to induce LTP
46 ic stimulation with strong (spike eliciting) postsynaptic depolarization, LTP was dependent on VGCCs
47 ay requires both presynaptic stimulation and postsynaptic depolarization, making it a central compone
48 ults are consistent with the notion that the postsynaptic depolarization of cerebellar interpositus n
49 n-GABAA chloride channels contributes to the postsynaptic depolarization of these cells after olfacto
50 n of LTP is postsynaptic; it is dependent on postsynaptic depolarization, on the influx of Ca2+ into
51  which were transiently suppressed by either postsynaptic depolarization or a brief train of action p
52 rade synaptic inhibition that are induced by postsynaptic depolarization or activation of metabotropi
53 ransmission--its induction is independent of postsynaptic depolarization, postsynaptic Ca2+ influx, o
54 ity and facilitated dendritic propagation of postsynaptic depolarization, potentially improving coinc
55 ssary and sufficient to produce the critical postsynaptic depolarization required for associative LTP
56 l a new form of synaptic plasticity in which postsynaptic depolarization results in enhancement of in
57  show that pairing synaptic stimulation with postsynaptic depolarization results in synapse unsilenci
58                    However, when paired with postsynaptic depolarization sufficient to activate L-typ
59 owever, CA1 HC PV cells exhibited a stronger postsynaptic depolarization than PFC PV cells.
60        Long-term potentiation (LTP) requires postsynaptic depolarization that can result from EPSPs p
61                   AMPA receptors mediate the postsynaptic depolarization that initiates neuronal firi
62  layer 5 PFC pyramidal neurons to identify a postsynaptic depolarization that was evoked by action po
63 cline was followed by a reciprocal rise in a postsynaptic depolarization that was largely restricted
64                     Glutamate generates fast postsynaptic depolarization throughout the CNS.
65           Paired stimuli (PS), consisting of postsynaptic depolarization to 0 mV and presynaptic stim
66 owever, 10 Hz-induced LTD does not depend on postsynaptic depolarization, unlike HFS-induced LTD.
67                                       Direct postsynaptic depolarization was decreased at more positi
68 otentiation and long-term depression require postsynaptic depolarization, which many current models a
69  a subsequent volley, leading to a depressed postsynaptic depolarization with repetitive stimulation.
70  by a subsequent volley, leading to enhanced postsynaptic depolarization with repetitive stimulation.

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