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1 ain and touch pathways through both pre- and postsynaptic inhibition.
2 suggesting an exclusive role for glycine in postsynaptic inhibition.
3 ely to be involved in presynaptic as well as postsynaptic inhibition.
4 c GABA receptors can mediate presynaptic and postsynaptic inhibition.
5 ligand-gated chloride channels that mediate postsynaptic inhibition.
6 e role of KCC2 in neuronal Cl- extrusion and postsynaptic inhibition.
7 citatory synaptic inputs and thereby produce postsynaptic inhibition.
8 of previously unknown proteins that regulate postsynaptic inhibition and might contribute to developm
9 level of KCC2 function sets the strength of postsynaptic inhibition and suggests that the downregula
10 spiratory rhythm following interference with postsynaptic inhibition and the subsequent discovery of
11 antagonism, discounting both presynaptic and postsynaptic inhibition as sources of cone opponency.
15 d dendrites, where GIRK channels may mediate postsynaptic inhibition, GIRK1 proteins were also found
16 e- and postsynaptic inhibition, we show that postsynaptic inhibition has a predominant role, suggesti
18 ry synaptic input and strong presynaptic and postsynaptic inhibition in both ON and OFF pathways.
19 ings emphasize the importance of glycinergic postsynaptic inhibition in motor neurons and challenge t
20 imals, there are conflicting results showing postsynaptic inhibition in motor neurons by corelease of
21 differentially mediate slow presynaptic and postsynaptic inhibition in PVIs and can contribute to th
24 centration at low levels, a prerequisite for postsynaptic inhibition mediated by GABA and glycine.
25 an active Cl- extrusion pathway important in postsynaptic inhibition mediated by ligand-gated anion c
26 ynaptic inhibition of auditory afferents and postsynaptic inhibition of an identified auditory intern
29 e of GABA, NPY, and SST, leading to pre- and postsynaptic inhibition of excitatory hippocampal circui
30 sion occurred even when LTP was inhibited by postsynaptic inhibition of exocytosis or PKA (protein ki
31 te release by glucocorticoids was blocked by postsynaptic inhibition of G-protein activity with intra
32 f glutamatergic transmission is abolished by postsynaptic inhibition of G-protein signaling with GDPb
34 e afferent neurons together with distributed postsynaptic inhibition of several downstream interneuro
35 dial medulla neurons in the mediation of the postsynaptic inhibition of spinal motoneurons necessary
36 ferent classes of neuron are responsible for postsynaptic inhibition of these interneurons, and the p
37 terneurons responsible for the nonreciprocal postsynaptic inhibition of trigeminal motoneurons that o
39 f KCC2 function would reduce the strength of postsynaptic inhibition, physiological evidence is still
41 The relative significance of presynaptic and postsynaptic inhibition to opioid analgesia is essential
42 upled receptor agonists involves coordinated postsynaptic inhibition via G protein-coupled inwardly r
43 elative contributions of GABA and glycine to postsynaptic inhibition, we performed in vivo intracellu
44 tive activation and inactivation of pre- and postsynaptic inhibition, we show that postsynaptic inhib
45 and (3) SST(+)-neuron-mediated pathways and postsynaptic inhibition within preBotC modulate breathin
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