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1 of ICU admission was found as performant as posttraumatic amnesia (area under the curve, 0.81; diffe
2 at ICU admission (phase 1) and compared with posttraumatic amnesia duration and the initial Glasgow C
3 s of ICU admission could be as performant as posttraumatic amnesia for predicting traumatic brain inj
7 activator (uPA) from injured brain leads to posttraumatic bleeding by causing premature clot lysis.
9 ence supporting a potential relation between posttraumatic brain hypoxia and microcirculatory derange
11 xiety, panic, phobia, obsession, compulsion, posttraumatic, care management, case management, collabo
12 e of Coping Status-A), posttraumatic stress (Posttraumatic Checklist-Specific Stressor), anxiety (Hos
15 erapy significantly predicted improvement in Posttraumatic Diagnostic Scale scores (beta = -3.86 [95%
16 OP arm had significantly larger decreases in Posttraumatic Diagnostic Scale scores (from 35.0 to 29.1
17 m also had significantly larger decreases in Posttraumatic Diagnostic Scale scores (from 35.0 to 30.1
23 with mild traumatic brain injury (mTBI) with posttraumatic migraines (PTMs) and those without PTMs on
26 le therapy for reducing cartilage injury and posttraumatic osteoarthritis by attenuating Piezo-mediat
30 (234/5153) were designated as feline ocular posttraumatic sarcoma, a tumor previously demonstrated t
31 re also significant differences in scores of posttraumatic stress (AMD, -5.86; 95% CI, -8.53 to -3.19
32 y Adjustment Schedule [WHODAS]), symptoms of posttraumatic stress (measured by the Posttraumatic Stre
33 vised), coping (Measure of Coping Status-A), posttraumatic stress (Posttraumatic Checklist-Specific S
34 gnificant levels of depression, anxiety, and posttraumatic stress among patients and levels of burden
35 tween deployment-acquired TBI and subsequent posttraumatic stress and related disorders among U.S. Ar
36 ttle is understood about how the symptoms of posttraumatic stress develop over time into the syndrome
37 roup analysis revealed a lower prevalence of posttraumatic stress disorder (odds ratio, 0.23; 95% CI,
38 02), bipolar I (OR, 1.5; 95% CI, 1.06-2.05), posttraumatic stress disorder (OR, 1.6; 95% CI, 1.27-2.1
39 ssault-related injury (P<.001), diagnosis of posttraumatic stress disorder (P=.008), and diagnosis of
40 earlier trauma exposure is known to predict posttraumatic stress disorder (PTSD) after subsequent tr
41 f exposure to stressful experiences, such as posttraumatic stress disorder (PTSD) and depression, are
42 an drive neuropsychiatric disorders, such as posttraumatic stress disorder (PTSD) and drug addiction.
43 ales disproportionately have higher rates of posttraumatic stress disorder (PTSD) and experience grea
44 tress reactivity is a predominant feature of posttraumatic stress disorder (PTSD) and may reflect dis
45 anding the neural causes and consequences of posttraumatic stress disorder (PTSD) and mild traumatic
46 ticipants (N=558) were assessed for probable posttraumatic stress disorder (PTSD) and probable depres
47 ticipants (N=558) were assessed for probable posttraumatic stress disorder (PTSD) and probable depres
49 s), obsessive-compulsive disorder (OCD), and posttraumatic stress disorder (PTSD) are common mental d
51 tudies have found that those who suffer from posttraumatic stress disorder (PTSD) are more likely to
52 d was a significant risk for a high level of posttraumatic stress disorder (PTSD) arousal symptoms.
55 e resilient to psychiatric disorders such as posttraumatic stress disorder (PTSD) compared with those
57 atic brain injury (TBI) is a risk factor for posttraumatic stress disorder (PTSD) has been difficult
58 The limited neurobiological understanding of posttraumatic stress disorder (PTSD) has been partially
60 ncreasing predictability of animal models of posttraumatic stress disorder (PTSD) has required active
61 ifferential effects of maternal and paternal posttraumatic stress disorder (PTSD) have been observed
62 or (alpha1AR) antagonist prazosin for combat posttraumatic stress disorder (PTSD) in 67 active duty s
63 lable from medical dispensaries for treating posttraumatic stress disorder (PTSD) in many states of t
64 the neurobiological mechanisms that predict posttraumatic stress disorder (PTSD) in recent trauma su
65 re to stress is a risk factor for developing posttraumatic stress disorder (PTSD) in response to trau
66 al blood during the onset and development of posttraumatic stress disorder (PTSD) indicate widespread
85 econsolidation marker.SIGNIFICANCE STATEMENT Posttraumatic stress disorder (PTSD) is characterized by
93 ly described the paradoxical co-existence in posttraumatic stress disorder (PTSD) of sensory intrusiv
94 interactions between smoking and symptoms of posttraumatic stress disorder (PTSD) on pain intensity,
95 Medication and psychotherapy treatments for posttraumatic stress disorder (PTSD) provide insufficien
97 nced trauma and associated distress-that is, posttraumatic stress disorder (PTSD) severity-more than
100 theater veterans with a high probability of posttraumatic stress disorder (PTSD) were nearly 2 times
101 s process a potential therapeutic target for posttraumatic stress disorder (PTSD), a mental illness c
102 of NPY with trauma-evoked syndromes such as posttraumatic stress disorder (PTSD), although the exact
104 ent among veterans, especially veterans with posttraumatic stress disorder (PTSD), and poses a major
105 sychiatric disorders, such as depression and posttraumatic stress disorder (PTSD), are inadequate.
106 posure therapy is an effective treatment for posttraumatic stress disorder (PTSD), but a comprehensiv
107 smaller hippocampal and amygdala volumes in posttraumatic stress disorder (PTSD), but findings have
108 posure therapy is an effective treatment for posttraumatic stress disorder (PTSD), but many patients
109 to underlie the pathophysiology of pediatric posttraumatic stress disorder (PTSD), but the few studie
110 al patterns of spontaneous brain activity in posttraumatic stress disorder (PTSD), but the findings a
111 r (GR) sensitivity is present in people with posttraumatic stress disorder (PTSD), but the molecular
112 n array of pathological conditions including posttraumatic stress disorder (PTSD), characterized by d
113 d C-reactive protein, have been described in posttraumatic stress disorder (PTSD), generalized anxiet
114 erapy (CPT), an evidence-based treatment for posttraumatic stress disorder (PTSD), has not been teste
115 e genetic basis of three disorder categories-posttraumatic stress disorder (PTSD), major depressive d
116 est were 30-day prevalence postdeployment of posttraumatic stress disorder (PTSD), major depressive e
117 eneralization of fear, a cardinal feature of posttraumatic stress disorder (PTSD), manifests as inapp
118 re-based therapy, an effective treatment for posttraumatic stress disorder (PTSD), relies on extincti
120 factor for cardiovascular disease (CVD), and posttraumatic stress disorder (PTSD), the sentinel stres
121 for developing anxiety disorders, including posttraumatic stress disorder (PTSD), the underlying mec
123 tion to threat is perturbed in patients with posttraumatic stress disorder (PTSD), with some studies
145 ala reactivity is a vulnerability factor for posttraumatic stress disorder (PTSD); however, our under
146 disorder (RR, 1.29 [95% CI, 1.01-1.65]), and posttraumatic stress disorder (RR, 1.47 [95% CI, 1.09-1.
147 hose who screened positive for depression or posttraumatic stress disorder after return from deployme
149 pite significant overlap of symptoms between posttraumatic stress disorder and anxiety disorders, the
150 thasone compared with placebo on symptoms of posttraumatic stress disorder and depression and health-
151 nts, beneficial effects on the occurrence of posttraumatic stress disorder and depression may be pres
153 nea and insomnia), mental health status (eg, posttraumatic stress disorder and depression), nonocular
154 seases; and mental health disorders, such as posttraumatic stress disorder and depression, that are a
160 a burden to daily life and characteristic of posttraumatic stress disorder and other anxiety disorder
161 responses and have been have been linked to posttraumatic stress disorder and other mental health af
162 le target in developing novel treatments for posttraumatic stress disorder and related disorders.
163 s potential therapeutic effects for treating posttraumatic stress disorder are related to altering em
164 ew Version (PSS-I) and the stressor-specific Posttraumatic Stress Disorder Checklist (PCL-S); seconda
165 oms of posttraumatic stress (measured by the Posttraumatic Stress Disorder Checklist [PCL]), personal
166 outcomes were posttraumatic stress symptoms (Posttraumatic Stress Disorder Checklist for DSM-5), func
167 y the Neurobehavioral Symptom Inventory, the Posttraumatic Stress Disorder Checklist Military Version
169 ses including mild traumatic brain injury or posttraumatic stress disorder display alterations on ASL
173 co-occurring psychiatric conditions and with posttraumatic stress disorder having the largest impact.
174 polymorphism, rs4523957, is associated with posttraumatic stress disorder in humans, consistent with
176 health, increasing depression, anxiety, and posttraumatic stress disorder in these same villages.
180 nisms of severe sepsis in the development of posttraumatic stress disorder need further examination.
181 There has been relatively little study of posttraumatic stress disorder or obsessive-compulsive di
182 lthough pathological fear, such as occurs in posttraumatic stress disorder or specific phobias, is al
183 ntify anxiety, stress, caregiver burden, and posttraumatic stress disorder outcomes in informal careg
184 of threat.SIGNIFICANCE STATEMENT Anxiety and posttraumatic stress disorder patients generalize fear t
185 sing a locally validated version of the UCLA Posttraumatic Stress Disorder Reaction Index (range, 0-4
186 (OR, 1.15; 95% CI, 1.02-1.30; P = .02), and posttraumatic stress disorder score (OR, 1.04; 95% CI, 1
187 fied between overall DE symptom severity and posttraumatic stress disorder scores and tear breakup ti
188 ation manual was associated with significant posttraumatic stress disorder symptom reduction at 2 mon
189 e pooled prevalences of clinically important posttraumatic stress disorder symptoms (95% CI) were 25%
190 ationship to whole-brain cortical thickness, posttraumatic stress disorder symptoms (PTSD) and depres
191 turned out to be a significant predictor of posttraumatic stress disorder symptoms at 3-month follow
192 atients with general anxiety, depression, or posttraumatic stress disorder symptoms during 2-year fol
193 lihood of remission from general anxiety and posttraumatic stress disorder symptoms during follow-up.
194 e individuals, increased lifetime stress and posttraumatic stress disorder symptoms explained the maj
197 rathreshold general anxiety, depression, and posttraumatic stress disorder symptoms ranged from 38% t
198 ore was 17 (9-24), and pooled prevalences of posttraumatic stress disorder symptoms were 17% (10-26%)
202 s associated with a significant reduction in posttraumatic stress disorder symptoms, 2) a self-help r
203 significant general anxiety, depression, or posttraumatic stress disorder symptoms, and these sympto
210 acotherapies and device-based treatments for posttraumatic stress disorder that have been developed v
214 lthough symptoms of anxiety, depression, and posttraumatic stress disorder were found to be common 1
218 , 20.2%; generalized anxiety disorder, 7.9%; posttraumatic stress disorder, 9.8%; alcohol dependence,
219 specific psychiatric disorders (depression, posttraumatic stress disorder, addiction, social anxiety
221 chronic symptoms of postconcussive disorder, posttraumatic stress disorder, and depression and for th
222 eported symptoms of postconcussive disorder, posttraumatic stress disorder, and depression were deter
223 ast 6-month generalized anxiety disorder and posttraumatic stress disorder, and past 12-month alcohol
224 tions, such as generalized anxiety disorder, posttraumatic stress disorder, and social anxiety disord
227 hopathology (indiscriminate social behavior, posttraumatic stress disorder, attention-deficit/hyperac
228 ive disorders (mean scores >35), followed by posttraumatic stress disorder, borderline personality di
229 ocial outcomes (depression, anxiety, stress, posttraumatic stress disorder, burden, activity restrict
231 antify the impact of psychiatric conditions (posttraumatic stress disorder, depression, anxiety, and
232 ty disorders, obsessive-compulsive disorder, posttraumatic stress disorder, depressive disorders, bip
234 younger, have more obesity, depression, and posttraumatic stress disorder, less obstructive coronary
235 y a role in the formation and persistence of posttraumatic stress disorder, of which sleep impairment
236 nalyses used a priori seeds relevant to TMS, posttraumatic stress disorder, or MDD (subgenual anterio
237 suicidal plan, bipolar disorder, psychosis, posttraumatic stress disorder, substance dependence, cur
238 memories underlie anxiety disorders, such as posttraumatic stress disorder, the key neural and molecu
239 h symptom levels of depression, anxiety, and posttraumatic stress disorder, which are strongly associ
264 nd 78 patients (13.8%) who received placebo (posttraumatic stress disorder: odds ratio, 0.82; 95% CI,
265 yment (depression: OR, 1.4; 95% CI, 1.1-1.9; posttraumatic stress disorder: OR, 2.4; 95% CI, 2.1-2.8)
266 increases in the rates of major depression; posttraumatic stress disorder; other anxiety disorders;
267 erapy for anxiety, obsessive-compulsive, and posttraumatic stress disorders and to evaluate whether a
269 Other conditions represented were psychotic, posttraumatic stress or anxiety, somatoform, neurocognit
271 equal to 14 (interquartile range, 5-20) and Posttraumatic Stress Scale equal to 22 (interquartile ra
272 tal Anxiety and Depression Scale; p = 0.010, Posttraumatic Stress Scale), resilience (p = 0.012, Hosp
274 ange of postdisaster trajectories of chronic posttraumatic stress symptom (PTSS) and depression sympt
275 g strategy use is a significant predictor of posttraumatic stress symptom severity 60 days after hosp
277 traumatic and, as a consequence, experience posttraumatic stress symptomatology at clinical levels.
279 the Impact of Event Scale-Revised assessing posttraumatic stress symptoms 60 days after hospital dis
280 50, p<0.001) were better predictors of later posttraumatic stress symptoms than coping strategies 5 d
281 d social support, cognitive functioning, and posttraumatic stress symptoms were also assessed using s
287 gnificant levels of anxiety, depression, and posttraumatic stress, as well as cognitive function and
288 nosis before transplantation (schizophrenia, posttraumatic stress, major depressive, and bipolar diso
289 cantly different regarding the prevalence of posttraumatic stress-related symptoms (52.3 vs 50%, resp
290 tionnaire and the Impact of Event Scale (for posttraumatic stress-related symptoms) 3 months later.
293 ent negative life events, and depressive and posttraumatic-stress-disorder symptom scores with placen
294 ered the Acute Stress Disorder Scale and the Posttraumatic Symptom Scale-10 to assess symptoms of acu
296 eficits belong to the most prevalent chronic posttraumatic symptoms, most likely due to diffuse axona
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