ライフサイエンスコーパス: postural

1 ases are characterized clinically by ataxia, postural abnormalities and cognitive decline.

2 features (postural imbalance, rigidity, and postural abnormalities) and increasingly reported proble

3 nesia, tremor, rigidity, postural imbalance, postural abnormalities) and non-motor features of Parkin

4 eous group of disorders all characterised by postural abnormalities, motor deficits and cerebellar de

5 These results show that, during postural activity, a key role of PTNs is the feedback co

6 al vertebrate condition that originated as a postural adaptation for pectoral control of head orienta

7 This postural adaptation was based on the transformation of t

8 s through calf vibrations were used to study postural adaptation.

9 ge gaps due to an inability in making subtle postural adaptations that are requisite for this task.

10 that allows the measurement of anticipatory postural adjustment of human legs to be synchronized wit

11 decrease in the variability of anticipatory postural adjustments (APAs) occurs when performing cued

12 art of the neural substrate for anticipatory postural adjustments and speculate that dysfunction of t

13 n the present study we explored anticipatory postural adjustments made by infants to one of the most

14 gion and has neurons related to anticipatory postural adjustments preceding step initiation as well a

15 0 ms before takeoff, flies begin a series of postural adjustments that determine the direction of the

16 ironment can evoke self-motion illusions and postural adjustments.

17 untary movement was driven by an involuntary postural aftercontraction of the deltoid muscle of the s

18 Postural and clinical assessments along with structural

19 that muscles operate very differently under postural and dynamic conditions.

20 action tremor and a variable combination of postural and kinetic components with resting tremors les

21 xhibit unsteady gait, as well as involuntary postural and kinetic movements, indicating a disturbance

22 movement disorder characterized mainly by a postural and kinetic tremor of the upper extremities.

23 resence of head tremor and arm tremor (rest, postural and kinetic).

24 information into motor commands that support postural and locomotor control.

25 nding the brain's ability to ensure accurate postural and motor control, as well as perceptual stabil

26 iffness is well adapted for the continuum of postural and movement activity and has a substantial pos

27 gnificant change in phase difference between postural and rest tremor.

28 population activity in area 5d may represent postural and spatial information in the reference frame

29 adipose (three depots), and muscle (truncal postural and thigh locomotive) FFA uptake using [(11)C]p

30 o the suggestion that beta activity promotes postural and tonic contraction, possibly at a cost to th

31 lti-modal display, including song as well as postural and wing movements.

32 These studies show that visual, tactile, postural, and anatomical information all contribute to t

33 t to estimate in individuals due to diurnal, postural, and measurement variation.

34 is preserved in humans as well; E2D induces postural avoidance, increases physiological arousal, and

35 In both groups the postural behavior inside the magnetic resonance was corr

36 ss of this compensation process by measuring postural behaviour in adult survivors of childhood cance

37 R-R variation, abnormal Valsalva ratio, and postural blood pressure changes had significantly higher

38 iation with paced breathing, Valsalva ratio, postural blood pressure changes, and autonomic symptoms)

39 ated differences exist in the time course of postural BP responses, with abnormal responses taking lo

40 timulation gave rise to phase entrainment of postural, but not kinetic, tremor, whereas cerebellar st

41 herent with the cortical EEG recorded during postural, but not kinetic, tremor.

42 ls, the hemodynamic response to reclining, a postural change that increases retinal perfusion pressur

43 Similarly, greater SBP variability with postural change was associated with an increased risk of

44 in participant variability in SBP related to postural change, expressed as coefficient of variation.

45 tolic blood pressure (DBP) within 3 min from postural change.

46 y generated for the index finger by the same postural change.

47 3-month-old infants showed systematic global postural changes during Approach and Contact, but not du

48 t (i) complete SCI disrupts the influence of postural changes on the representation of the deafferent

49 imultaneously did not induce any significant postural changes.

50 ing 60 cms(-1), despite body compression and postural changes.

51 In postural conditions the EMG spectrum is relatively flat

52 gated the effects of changes in the internal postural configuration of the hand on the perceived dist

53 anical contexts created by altering animals' postural configuration.

54 oals of observed actions, while manipulating postural congruency between their own body posture and p

55 Cerebrally, this perceptual postural congruency effect modulates activity in a porti

56 ecordings, and is independent of operator or postural context in its interpretation.

57 lacement positively correlated with those of postural control and gluteus medius muscle activities, r

58 less research attention than its effects on postural control and locomotion.

59 neurons in the vestibular nuclei involved in postural control and self-motion perception.

60 ssing in the vestibular pathways involved in postural control and the computation of self-motion perc

61 ic efferent terminals has been implicated in postural control deficits in Parkinson's disease.

62 In quadrupeds, the most critical aspect of postural control during locomotion is lateral stability.

63 and trunk neural mechanisms contributing to postural control during locomotion.

64 mastoid vibration (MV) could affect dynamic postural control during walking.

65 veal deficits and visuospatial attention and postural control impairment in PD remain challenging res

66 ew work on molluscan feeding, maintenance of postural control in cats and humans, simulations of loco

67 l dual-task interference effects with stance postural control in healthy young adults.

68 her a tailored tai chi program could improve postural control in patients with idiopathic Parkinson's

69 and their thalamic efferents play a role in postural control in patients with Parkinson's disease, p

70 Standing postural control is complex, meaning that it is dependen

71 Knowledge of brain correlates of postural control is limited by the technical difficultie

72 e (COPr) can quantify the behavior of cardio-postural control loop.

73 keletal and feedback components of the human postural control loop.

74 ood cancer may suffer late effects of poorer postural control manifested as reduced contribution of v

75 eased EMG activity of the gluteus medius and postural control muscles during leg swing, and increased

76 conclude that activity in the nMLF provides postural control of tail orientation and thus steers the

77 were investigated: IJV stenosis, reversal of postural control of the cerebral venous outflow pathways

78 In the control group, IJV stenosis (P=0.12), postural control reversal of the cerebral venous outflow

79 In the study group, IJV stenosis, postural control reversal of the cerebral venous outflow

80 on, while the later response arises from the postural control system attempting to align the body wit

81 nction depends in part on the ability of the postural control system to integrate visual, propriocept

82 ctivity, followed by incoordination, loss of postural control, and eventually, sedation.

83 s of interactions with tandem Romberg stance postural control, and that interactions within the spati

84 y of motor activities, including locomotion, postural control, and voluntary movements.

85 addition to its acknowledged importance for postural control, gaze stabilization, and spatial naviga

86 the majority of rays retain a high degree of postural control, indicating significant functional resi

87 Human postural control, which relies on information from vesti

88 omatosensory and vestibular contributions to postural control.

89 been shown to decrease dizziness and improve postural control.

90 assessments of single and dual-task standing postural control.

91 confirming that the decreased variability of postural coordination that is evident following acutely

92 icrocircuits are equally active, but, during postural correction, fish differentially engage these mi

93 The postural corrections critical to standing balance and na

94 These results suggest that (i) postural costs, especially at low speeds, may result in

95 lar dysfunction have visual, perceptual, and postural deficits.

96 Postural deformities are frequent and disabling complica

97 The evidence to date suggests that postural deformities have a multifactorial pathophysiolo

98 d understanding of the mechanisms underlying postural deformities in PD might ultimately lead us to m

99 Camptocormia is an axial postural deformity characterised by abnormal thoracolumb

100 Freezing of gait, postural deformity, and motor fluctuations were common l

101 terchangeably to describe a laterally flexed postural deviation in Parkinson's disease (PD), the imag

102 , particularly for symptoms such as gait and postural difficulties refractory to dopaminergic treatme

103 mes to loud stimuli correlated with gait and postural disturbance.

104 ts were not significantly different from the postural effects in the younger subjects.

105 brain exploits these properties of synergies-postural equivalence, low dimensionality, and topographi

106 ck rule based on an optimal tradeoff between postural error and neural effort explained patterns of m

107 measures of intention ET were different from postural ET but not apparently different from those of c

108 (termed intention ET) and the other without (postural ET).

109 kexEMG coherence than those in patients with postural ET.

110 a lower spikexEMG phase lead than those with postural ET.

111 This postural evolution included anatomical changes of the fo

112 were randomized to four training groups: (1) postural exercises designed to stimulate brainstem pathw

113 ment (n = 3,000), we tested the causality of postural expansion (vs. contraction) on attraction using

114 of romantic attraction, we demonstrate that postural expansiveness makes humans more romantically ap

115 Postural expansiveness-expanding the body in physical sp

116 Normal postural function depends in part on the ability of the

117 Arm tremor is also frequent (postural>kinetic>rest), occurring in up to one-third of

118 Gestures were scored separately for postural (hand/arm positioning) and kinematic (amplitude

119 causes and new pharmacological management of postural hypotension and other cardiovascular diseases.

120 , with the most serious manifestations being postural hypotension and paradoxical supine hypertension

121 demonstrated sufficient activity to produce postural hypotension in its host.

122 The primary outcome was the absence of postural hypotension on postoperative day 1, defined as

123 The primary outcome, absence of postural hypotension on postoperative day 1, occurred in

124 dergoing abdominal surgery, the incidence of postural hypotension or adrenal insufficiency is similar

125 s orthostatic hypotension (OH; also known as postural hypotension).

126 the lymphocytes of a patient with idiopathic postural hypotension.

127 lear palsy and two others with either severe postural imbalance or an isolated short-term memory defi

128 r (hypo- and bradykinesia, tremor, rigidity, postural imbalance, postural abnormalities) and non-moto

129 e cases developed additional motor features (postural imbalance, rigidity, and postural abnormalities

130 Because freezing of gait and postural impairments have been related to frontal lesion

131 The aim of this study was to determine the postural influence on the difference between simultaneou

132 Hand postural information, encoded through kinematic synergie

133 cusing on three key irreversible milestones: postural instability (Hoehn and Yahr 3), dementia and de

134 At 10 years, 55% had died, 68% had postural instability and 46% dementia.

135 arkinson's disease patients with predominant postural instability and gait difficulties (PIGD) may ex

136 as significantly higher in the subgroup with postural instability and gait difficulties compared with

137 association between genotype and tremor and postural instability and gait difficulty (PIGD) scores.

138 oor initial levodopa treatment response, and postural instability and gait difficulty motor PD subtyp

139 ms: Parkinson disease, parkinsonism, tremor, postural instability and gait difficulty, and Parkinson

140 movement sleep behavioural disorder and the postural instability and gait disorder phenotype.

141 , short-latency afferent inhibition, age and postural instability and gait disorder score (Movement D

142 trolling for age, posture and gait symptoms (Postural Instability and Gait Disorder score-Movement Di

143 Gait freezing and postural instability are disabling features of Parkinson

144 s, with most dying or developing dementia or postural instability by 10 years from diagnosis, but a q

145 : tremor dominant (TD), intermediate (I), or postural instability gait difficulty (PIGD), based on pr

146 ral subtypes, such as tremor-dominant PD and postural instability gait difficulty form of PD, have be

147 The pathophysiology of postural instability in Parkinson's disease remains poor

148 kinsonian patients with gait disturbance and postural instability refractory to other treatment modal

149 l symptoms (bradykinesia, rigor, tremor, and postural instability) are used for disease staging and a

150 come at 10 years (surviving free of dementia/postural instability).

151 ressive supranuclear palsy, characterized by postural instability, early unexplained falls, vertical

152 opment of "nondopaminergic" features such as postural instability, falling, and dementia that are not

153 clinical features (rapid progression, early postural instability, poor levodopa responsiveness and s

154 , pre-prepared movement in gait freezing and postural instability, relieved by pedunculopontine nucle

155 u181 concentrations were associated with the postural instability-gait disturbance-dominant phenotype

156 bradykinesia, rigidity, resting tremor, and postural instability.

157 phenotype and comorbidity predicted earlier postural instability.

158 malities that include akinesia, rigidity and postural instability.

159 resting tremor, rigidity, bradykinesia, and postural instability.

160 onian patients with severe gait freezing and postural instability.

161 oked GRF vector towards the axis of baseline postural instability.

162 rigidity, bradykinesia, resting tremor, and postural instability.

163 resting tremor, rigidity, bradykinesia, and postural instability.

164 ghting of sensory feedback gains to minimize postural instability.

165 activation between tremor dominant (TD) and postural instability/gait difficulty (PIGD) subtype pati

166 specific cognitive resources contributed to postural interference in young adults (n=9) in a dual-ta

167 diurnal/awake period, it may not affect the postural IOP effect during the diurnal and the nocturnal

168 The postural IOP effect for the older subjects was 4.7 +/- 0

169 The average postural IOP effects during the diurnal/wake period and

170 These postural IOP effects were not significantly different fr

171 To characterize the effect of postural IOP elevation and pharmacological IOP lowering

172 n all five component tremor subscores: rest, postural, kinetic, proximal and distal.

173 h falls or cognitive impairment, but without postural lightheadedness or near syncope.

174 Postural limb reflexes (PLRs) represent a substantial co

175 Postural metrics obtained during dynamic visual stimuli

176 We assessed whether a postural modification to the Valsalva manoeuvre could im

177 and, and add to a growing literature showing postural modulation of touch.

178 The descending signal for postural muscle atonia during REM sleep is thought to or

179 X-linked myopathy with postural muscle atrophy is a novel X-linked myopathy cau

180 X-linked myopathy with postural muscle atrophy patients consistently showed ele

181 X-linked myopathy with postural muscle atrophy was associated with reduced exer

182 ducing body myopathy, X-linked myopathy with postural muscle atrophy, rigid spine syndrome (RSS) and

183 hological hallmark of X-linked myopathy with postural muscle atrophy; a characteristic spongious stru

184 movements, autonomic activation and loss of postural muscle tone (atonia).

185 one to cataplexy experience sudden losses of postural muscle tone without a corresponding loss of con

186 d decrease in energy consumption of hindlimb postural muscles when sitting, we hypothesise that a cha

187 expected to decipher nonlinear causal cardio-postural-musculoskeletal interactions.

188 The causal interaction between cardio-postural-musculoskeletal systems is critical in maintain

189 s to control the activity of extraocular and postural neurons, thus completing a fundamental three-ne

190 al role assumed by the target muscle, either postural or agonist.

191 recordings, we excluded the possibility that postural or mirror movements could exclusively account f

192 with reduced blood volume contributes to the postural orthostatic tachycardia syndrome (POTS) and tha

193 Patients with the postural orthostatic tachycardia syndrome (POTS) are pri

194 Women with the postural orthostatic tachycardia syndrome (POTS) report

195 s a key component of the pathogenesis of the postural orthostatic tachycardia syndrome (POTS), simila

196 Fibroblasts from a patient with postural orthostatic tachycardia syndrome (POTS), who pr

197 alled appropriate sinus tachycardia and from postural orthostatic tachycardia syndrome, with which ov

198 Postural performance was significantly influenced by the

199 tes motor coordination deficits and impaired postural phenotypes.

200 We suggest that postural reactions generated by these four mechanisms ar

201 Due to postural reactions, locomotion with normal body configur

202 h glaucoma on the basis of the assessment of postural reactivity to dynamic visual stimuli using a vi

203 The neural encoding of spatial and postural reference frames in posterior parietal cortex h

204 In addition, improved postural reflexes correlated with decreased rCBF in the

205 sponsible for reduction of extensor tone and postural reflexes during spinal shock.

206 al phenotype including ataxia, impairment of postural reflexes, and hyperactivity in early stages fol

207 taneous information contribute to whole-body postural reorganization in humans.

208 Contrary to expectation, the evoked postural response consisted of two sequential components

209 r visual-flow speed similarly influences the postural response to a discrete, unidirectional rotation

210 y proposed and predicts a decreasing gain of postural response with increasing visual motion speed.

211 as well as the muscle actions generating the postural response.

212 (vection) and corresponding visually evoked postural responses (VEPR).

213 We conclude that automatic postural responses for laterally moving visual stimuli a

214 On the basis that automatic visually evoked postural responses in high fidelity virtual environments

215 ata from four experiments on visually evoked postural responses show that: 1) visually evoked postura

216 More generalized postural responses such as conditioned freezing can occu

217 the anterior-posterior plane induces robust postural responses that are not modulated by the presenc

218 to acceleration scaling found previously in postural responses to perturbations, initial burst ampli

219 Different postural responses were observed for real and virtual vi

220 and movement activity and has a substantial postural role eliminating the need for continuous muscle

221 d nigrostriatal dopaminergic denervation, on postural sensory integration function in Parkinson's dis

222 We conclude that postural sensory integration function of subjects with P

223 l component analysis was performed to reduce postural sensory organization functions to robust factor

224 The frequency distribution of postural sequences is heavy-tailed with a core of freque

225 Here we infer a postural shift in this species based on a novel combinat

226 nfant behavioural changes and recorded their postural shifts on a pressure mat in three phases: (i) a

227 Global postural shifts were also more phase differentiated in o

228 he scaling of a tuned neuronal response by a postural signal, may help support neuronal computation.

229 ty (62%), bradykinesia (44%), gait (49%) and postural stability (56%) (paired t-tests: P < 0.001).

230 ed with control subjects, patients had worse postural stability and altered patterns of cerebellar DN

231 in UPDRS 3 scores in rigidity, bradykinesia, postural stability and gait correlate with rCBF response

232 Trunk motor control is crucial for postural stability and propulsion after low thoracic spi

233 ts (STD) were calculated as a measurement of postural stability and reported in Newton meters (Nm).

234 Patients underwent evaluation of postural stability by a force platform during presentati

235 ess capable of adapting their APAs to ensure postural stability during movement.

236 e impact of forest canopy characteristics on postural stability in humans.

237 m assessment, neuropsychological testing and postural stability testing.

238 oskeletal systems is critical in maintaining postural stability under orthostatic challenge.

239 Postural stability was also tested in a completely dark

240 Shifts in postural stability were evident immediately after onset

241 trength, vibration perception thresholds and postural stability, in addition to the Expanded Disabili

242 Patients with MS had worse postural stability, widespread alterations in most white

243 e and direction depend on the flies' initial postural state.

244 of syncope as a result of their exposure to postural stress during the biological night.

245 system modulates cardiovascular responses to postural stress, leading to increased susceptibility to

246 n system affects cardiovascular responses to postural stress, resulting in greater susceptibility to

247 Swimmers had significantly lower postural sway (beta = -5.23 cm(2), P < 0.05) and shorter

248 control (EMG --> SBP) as well as control the postural sway (EMG --> COPr) through the significantly h

249 esholds (coefficient = -4.37, P = 0.021) and postural sway (P < 0.001).

250 h Scale, vibration perception thresholds and postural sway (P < 0.05 in all cases).

251 ower glutamate-glutamine predicted increased postural sway (P = 0.017).

252 een patients and healthy control subjects in postural sway and DTI diffusion-tensor imaging parameter

253 the magnitude of step to step adjustments in postural sway and lateral foot placement positively corr

254 consensus has developed on which measures of postural sway can identify those at greatest risk of fal

255 on demonstrated that older adults with lower postural sway complexity in both single and dual-task co

256 Postural sway complexity was quantified using multiscale

257 We hypothesized that older adults with lower postural sway complexity would experience more falls in

258 ned 52.2% of the variance, mainly reflecting postural sway during sensory organization test Condition

259 A weighted amalgam of postural sway elements may identify individuals at great

260 Diminished physiologic complexity of postural sway has been linked to reduced ability to adap

261 ural responses show that: 1) visually evoked postural sway in the lateral direction is modulated by t

262 Conversely, traditional postural sway metrics or SPPB performance did not associ

263 sks produced significantly greater levels of postural sway than either the auditory-object or visual-

264 Postural sway was measured in 161 elderly individuals (8

265 i-scale complexity contained within standing postural sway-particularly during dual task conditions-

266 Regression analysis of the Conditions 1-3 postural sway-related factor [R(2)adj = 0.123, F(5,109)

267 as visual anchors have different effects on postural sway; 3) visual motion in the anterior-posterio

268 rocnemius and soleus) are unlikely to signal postural sways on account of balance-related modulation

269 is a root cause of the severe oculomotor and postural symptoms of unilateral vestibular loss, and tha

270 earance of the characteristic oculomotor and postural symptoms, there is a marked increase in GABA re

271 Recognition of specific postural syndromes might have differential diagnostic va

272 Importantly, hand postural synergies were predicted by neural activation p

273 ntal stimuli can be quantified as changes in postural syntax: worms show different preferences for di

274 on, while the later component stems from the postural system realigning the body with gravity.

275 Rs) represent a substantial component of the postural system responsible for stabilization of dorsal-

276 Exercise induced postural tachycardia in one third of GWI subjects (Stres

277 Postural tachycardia syndrome (POTS) induces disabling c

278 Postural tachycardia syndrome (POTS) is characterized by

279 patients into orthostatic hypotension (OH), postural tachycardia syndrome (POTS), or normal HUT grou

280 improves heart rate control in patients with postural tachycardia syndrome during upright posture.

281 inspiratory resistance) in 26 patients with postural tachycardia syndrome in a randomized, single-bl

282 D) on orthostatic tolerance in patients with postural tachycardia syndrome.

283 the ability to allocate attention between a postural task and a secondary cognitive task was impaire

284 ombined performance of a challenging primary postural task and three specific, yet categorically diss

285 rol in the context of reaching movements and postural tasks have produced divergent findings, and thi

286 a sudden transient loss of consciousness and postural tone with spontaneous recovery; the most common

287 isual guidance differentially affected their postural trajectories.

288 at onset of tremor (P < 0.0001), more severe postural tremor (P < 0.0001), and more severe kinetic tr

289 ic stimulation in essential tremor modulated postural tremor amplitude according to the timing of sti

290 Stimulation near the postural tremor frequency entrained tremor.

291 n tremor-dominant Parkinson's disease and 20 postural tremor recordings in essential tremor, and vali

292 tremor frequency, whereas that of essential postural tremor reduced when tremor frequency departed f

293 Parkinsonian rest and essential postural tremor severity (i.e. tremor amplitude) differe

294 anecdotal sense that head tremor in ET is a postural tremor that dissipates when a patient lies down

295 n phase, while a concurrent voluntary input (postural tremor) would lead more frequently to an out-of

296 140 patients with arm tremor, all presented postural tremor, 103 patients (73.6%) presented also a k

297 hy, optic papillitis, inflammatory myelitis, postural tremor, and cerebellar ataxia.

298 physiological tremor of the hand-kinetic and postural tremor.

299 g predominates in local regions of space and postural tuning predominates over the larger workspace.

300 ilar in both groups, five of the eight trunk postural variables of the scoliotic group were significa