ライフサイエンスコーパス: potassium

1 95% CI: 0.74, 0.92 per SD increment in serum potassium).

2 activity-dependent increase of extracellular potassium.

3 e effects may be driven by changes in plasma potassium.

4 (LIBs) due to the abundance and low cost of potassium.

5 otassium and urinary fractional excretion of potassium.

6 ivation through the release of intracellular potassium.

7 nt regulator, possibly via effects on plasma potassium.

8 sodium intake and to the ratio of sodium to potassium.

9 Potassium (560mg/100g) was the predominant element follo

10 nt that alters cell membrane permeability to potassium affected the maintenance and establishment of

11 at the intracellular pH sensor perturbs the potassium affinity at the extracellular selectivity filt

12 in the cross-coupling of acyl chlorides with potassium alkyltrifluoroborates.

13 with 24-hour urinary excretion of sodium and potassium among US adults.

14 veloped; R(2)cal were 0.99 for magnesium and potassium and 0.97 for ash.

15 ound a significant interaction between serum potassium and aldosterone (P = 0.046).

16 d predators encoding insect specific sodium, potassium and calcium channel blockers for their ability

17 rskolin) that act upon glucose transporters, potassium and calcium channels, and G-protein-coupled re

18 ich are insensitive to the contribution from potassium and do not provide model-independent informati

19 source of vitamin C, carotenoid, phenolics, potassium and fiber.

20 centrations were associated with lower serum potassium and higher urinary excretion of potassium, but

21 one may modify the association between serum potassium and incident diabetes.

22 und no significant association between serum potassium and incident diabetes.

23 nt method to quantify concentrations of ash, potassium and magnesium and present the potential to cla

24 chemometrics for at-line monitoring of ash, potassium and magnesium content of GF flours: tapioca, p

25 Concentrations of ash, potassium and magnesium were determined with reference m

26 n the surface of the soma: the voltage-gated potassium and sodium channels Kv1.4 and Nav1.6 and the g

27 in the polymeric membrane with creatininium, potassium and sodium confirms the strong selective inter

28 ufacturers and some agar types and also with potassium and thymidine for S. pneumoniae For all other

29 osterone and MR activity, assessed via serum potassium and urinary fractional excretion of potassium.

30 a protein-embedded tunnel runs between these potassium and water sites and a helix controlling the cy

31 rgy and sodium intakes, less for protein and potassium, and minimally for nutrient densities.

32 This process reduced the copper, potassium, and phosphorus content, but had little effect

33 elines recommend routine monitoring of serum potassium, and renal function in patients treated with a

34 Potassium ( approximately 5g/100g), magnesium (2g/100g)

35 ty-four-hour urinary excretion of sodium and potassium are unaffected by recall errors and represent

36 Mean potassium at discharge was 4.3+/-0.48 mEq/L.

37 strictor that inhibits the membranous sodium-potassium ATPase pump across cell types and can cause ra

38 t the opposite was true in low phosphorus or potassium availability.

39 ide an overview of factors affecting overall potassium balance and factors modulating potassium dialy

40 nstead, a DCT-ASDN coupling process controls potassium balance in health and becomes aberrantly activ

41 WNK signaling during physiological shifts in potassium balance.

42 energies of cryptomelane, sodium birnessite, potassium birnessite and calcium birnessite are all sign

43 in, it is reported for the first time that a potassium bis(fluoroslufonyl)imide (KFSI)-dimethoxyethan

44 ssion of large-conductance calcium-activated potassium (BK) channels and Kv3.3 voltage-gated potassiu

45 Large-conductance Ca(2+)-activated potassium (BKCa) channels are key determinants of vascul

46 cin (mTOR) activation, loss of glutamate and potassium buffering capacity, loss of astrocyte coupling

47 ith participants who received placebo, urine potassium but not serum potassium increased significantl

48 Potassium, but not chloride, permease activity required

49 um potassium and higher urinary excretion of potassium, but only when renin was suppressed.

50 secretion, activation of NCC helps to retain potassium by increasing electroneutral NaCl reabsorption

51 e spatial distribution of specific analytes (potassium, calcium, manganese, iron, and zinc), and disc

52 The use of Earth-abundant potassium cation as a catalyst for C-H bond functionaliz

53 intracellular activities, and extracellular potassium changes demonstrates that SLEs in the piriform

54 toxicity related to anti-human ether-a-go-go potassium channel (hERG) activity of the first-generatio

55 roach, we discovered that a pair of two-pore potassium channel (K2P) subunits, largely dispensable ea

56 ABSTRACT: Sarcolemmal ATP-sensitive potassium channel (KATP channel) activation in isolated

57 Large-conductance calcium-activated potassium channel (KCa1.1; BK, Slo1, MaxiK, KCNMA1) is t

58 nnels at the node of Ranvier and Shaker-type potassium channel (Kv1.2) at the juxtaparanode.

59 ntigenic components within the voltage-gated potassium channel (VGKC) complex.

60 beta cells, including the T2DM-linked KCNQ1 potassium channel alpha subunit.

61 tor-mediated inhibition of a two-pore domain potassium channel and A1 receptor-mediated opening of a

62 The potassium channel blocker 4-aminopyridine reliably induc

63 s and the isolated guinea pig brain with the potassium channel blocker 4-aminopyridine.

64 6.3%): 3 (2.7%) had TPO-Ab and voltage-gated potassium channel complex (VGKCc) Ab, 2 (1.8%) had GAD65

65 independently of TIM in Drosophila to alter potassium channel conductance in arousal neurons after l

66 (human Ether-a'-go-go-Related Gene) cardiac potassium channel delays cardiac repolarization and can

67 nd interventions targeted towards correcting potassium channel dysfunction in ataxia need to be tailo

68 r-mediated pathway that impinges on specific potassium channel effectors.

69 BK, Slo1, MaxiK, KCNMA1) is the predominant potassium channel expressed at the plasma membrane of rh

70 ting tumor viability and invasion, including potassium channel function and EPH receptor signaling.

71 Among those, we found potassium channel genes KCNA4 and KCNIP4, involved in el

72 ion, our results reveal a role for the KCNQ1 potassium channel in the regulation of human growth, and

73 Either of 2 somatic mutations in the potassium channel KCNJ5 (G151R and L168R, hereafter refe

74 ainate receptor GluR6/7 and inward rectifier potassium channel Kir2.1, closely associated with SAP102

75 Here, we tested the hypothesis that the potassium channel Kir4.1 is the potassium sensor of DCT

76 ng in transcripts encoding the voltage-gated potassium channel Kv1.1 converts an isoleucine to valine

77 The potassium channel Kv1.3 is highly expressed in the mitoc

78 The voltage-dependent potassium channel Kv1.3 plays essential physiological fu

79 s treatable by blockade of the voltage-gated potassium channel Kv1.3.

80 ed by the reduction in the expression of the potassium channel Kv2.1 at the surface of motor neurons.

81 Following the recent development of a potassium channel modulator, AUT1-an imidazolidinedione

82 vated calcium currents, and independently of potassium channel regulation, membrane potential changes

83 confirmed; thereafter, reduced levels of the potassium channel ROMK and kinases SGK1 and WNK1 were ob

84 aspartate receptors and weakened by cAMP-PKA-potassium channel signaling in dendritic spines.

85 The voltage-gated potassium channel subfamily A member 3 (Kv1.3) dominantl

86 nctional expression of the voltage-dependent potassium channel subunit Kv1.1 substantially contribute

87 D afferents express KCNQ3, KCNQ4, and ERG1-3 potassium channel subunits.

88 Kv11.1 (hERG) is a voltage-gated potassium channel that shows very slow ionic current act

89 Here we report that the KCNE2 potassium channel transmembrane regulatory subunit is ex

90 KCNJ13 gene encodes the inwardly rectifying potassium channel, Kir7.1.

91 xpressed the intermediate-conductance KCa3.1 potassium channel, revealing a strong functional couplin

92 anism of regulation of the proton pump and a potassium channel, two essential elements in K(+) uptake

93 ian hnRNP U, result in dysfunction of a Slo2 potassium channel, which is critical to neuronal functio

94 -positive specimens had higher voltage-gated potassium channel-IgG immunoprecipitation values (0.33nm

95 g of a G-protein-coupled inwardly rectifying potassium channel.

96 ng to identify genetic variants of the KCNQ1 potassium channel.

97 onal expression of the renal outer medullary potassium channel.

98 TIONALE: Large-conductance calcium-activated potassium channels (BK) are composed of pore-forming BKa

99 Sarcolemmal ATP-sensitive potassium channels (KATP channels) in cardiac myocytes a

100 occurs via activation of inwardly rectifying potassium channels (KIR ), and synthesis of nitric oxide

101 Small conductance calcium-activated potassium channels (SK channels) are present in spines a

102 Slo2 channels are large-conductance potassium channels abundantly expressed in the nervous s

103 assium (BK) channels and Kv3.3 voltage-gated potassium channels accompanies the inability of Purkinje

104 porter SGLT1, or by closure of ATP-sensitive potassium channels after glucose metabolism.

105 ignatures of infection, such as induction of potassium channels and amino acid transporters, derepres

106 PV+ cells by regulating the localization of potassium channels and AMPA receptors, respectively.

107 dopts the Kunitz fold known to mostly act on potassium channels and serine proteases.

108 KCNQ2 potassium channels are critical for normal brain functio

109 Kv3.1 and Kv3.2 voltage-gated potassium channels are expressed on parvalbumin-positive

110 POINTS: Intracellular Na(+) -activated Slo2 potassium channels are in a closed state under normal ph

111 scovered that yeast cells lacking endogenous potassium channels could be rescued by WT ROMK but not b

112 Sodium and potassium channels exhibit a one-dimensional periodicity

113 The slow spontaneous inactivation of potassium channels exhibits classic signatures of transm

114 C-type inactivation in potassium channels helps fine-tune long-term channel act

115 reby demonstrating the involvement of A-type potassium channels in prolonging pauses evoked by GABAer

116 regions and increased expression of specific potassium channels in the NAc may promote abstinence fro

117 hippocampal cultures and asked how distinct potassium channels interact in determining the basal spi

118 subclasses of voltage- and/or calcium-gated potassium channels may provide an important approach to

119 Voltage-gated potassium channels of the KCNQ (Kv7) subfamily are essen

120 Two-pore domain (K2P) potassium channels perform essential roles in neuronal f

121 The selectivity filter in potassium channels, a main component of the ion permeati

122 leads to epigenetic repression of Kv1.1-type potassium channels, increased excitability, and impaired

123 Unlike potassium channels, sodium channel alpha-subunits are be

124 le structures of TRPV1 and voltage-activated potassium channels, we engineered chimeras wherein trans

125 ough the activation of voltage-gated KCNQ2-5 potassium channels.

126 afferent input impedance by closing calyceal potassium channels.

127 o activate at a rate similar to conventional potassium channels.

128 ctivation and the subsequent closure of KCNQ potassium channels.

129 son of transport of monovalent electrolytes [potassium chloride (KCl), sodium chloride (NaCl)], 2:1 e

130 rved ejection fraction to oral KNO3 (n=9) or potassium chloride (n=3).

131 s associated with reduced dorsal spinal cord potassium chloride cotransporter expression and impaired

132 ium reduction and its partial replacement by potassium chloride in pizza dough and crusts prepared by

133 us was modulated by focal microinjections of potassium chloride into the nucleus reuniens, during whi

134 hermodynamic chemical activity of sodium and potassium chloride, as well as the effect of the salts o

135 tion at unchanged cytosolic Ca(2+) levels in potassium chloride-constricted intact arteries isolated

136 hat are composed of eutectic silver chloride-potassium chloride.

137 large white pigs by intravenous injection of potassium chloride.

138 s of Tbx1 or Slc12a2, which encodes a sodium-potassium-chloride cotransporter and is also necessary f

139 receptors can physically associate with the potassium-chloride cotransporter protein, KCC2, which se

140 latile elements and compounds, such as zinc, potassium, chlorine, and water, provide key evidence for

141 The stability of potassium complex was enhanced up to two hours and the s

142 Blood potassium concentration ([K(+)]) influences the electroc

143 The elevated potassium concentration also depolarized the postsynapti

144 e mechanisms by which DCT cells sense plasma potassium concentration and transmit the information to

145 Increased potassium concentration in the cleft maintained the hair

146 Plasma potassium concentration strongly and negatively correlat

147 assium transport, and unresponsive to plasma potassium concentration.

148 d we review data linking serum and dialysate potassium concentrations with arrhythmias, cardiovascula

149 Postsynaptically, the opioids activate a potassium conductance through the mu-opioid receptor (MO

150 hyperexcitable due to decreased chloride and potassium conductances.

151 ltage-clamp experiments, 2-AG reduced A-type potassium current (IA) through a cannabinoid receptor-in

152 lum Ca2+ concentrations, inwardly rectifying potassium current (IK1) density, and gap junction conduc

153 ate the slowly activating, voltage-dependent potassium current (IKs) in the heart that controls the r

154 tion, which increases slow-delayed rectifier potassium current (IKs).

155 gnificant reduction of the transient outward potassium current (Ito) in EPI but not in endocardial (E

156 revealed downregulation of transient outward potassium current (Ito; P<0.05).

157 associated with decreased transient outward potassium current and Kv4.2 and KChIP2 protein expressio

158 ings are consistent with the hypothesis that potassium current downregulation leads to abnormal repol

159 esults from frequency-dependent reduction of potassium current during spike repolarization.

160 paper unveils the critical role of the brake potassium current IKD in damage-triggered cold allodynia

161 Dysfunction of the fast-inactivating Kv3.4 potassium current in dorsal root ganglion (DRG) neurons

162 led that the isoflurane-activated background potassium current observed in cortical pyramidal neurons

163 ation of the MOR and reduced activation of a potassium current over the same time course.

164 t reduction, but the contribution to overall potassium current reduction was almost always much small

165 , there was a component of calcium-dependent potassium current that showed frequency-dependent reduct

166 Reduction of potassium current was also seen with multimeric G85R SOD

167 dent reduction of overall spike-repolarizing potassium current was identified as Kv3 current by its s

168 ion of G protein-coupled inwardly rectifying potassium current was measured using whole-cell voltage-

169 n resulted in lack of time-dependent outward potassium currents in guard cells, higher rates of water

170 NTS: Kv2 channels underlie delayed-rectifier potassium currents in various neurons, although their ph

171 y consistent reductions in voltage-activated potassium currents near the action potential threshold a

172 elayed repolarization from downregulation of potassium currents, and multiple reentry circuits during

173 Elevated potassium depolarizes the postsynaptic afferent by alter

174 all potassium balance and factors modulating potassium dialysate fluxes in dialysis, and we review da

175 action to methylisothiazolinone, cobalt, and potassium dichromate.

176 le for the inflammasome sensor NLRP3 and for potassium efflux in T. gondii-induced IL-1beta productio

177 axis induces a cell death program initiating potassium efflux upstream of NLRP3.

178 Potassium elevations associated to GABAA receptor-mediat

179 rolyte forms a uniform SEI on the surface of potassium enabling reversible potassium plating/strippin

180 , and an inverse association between urinary potassium excretion and blood pressure, in a nationally

181 There were significant interactions by potassium excretion for circumferential strain.

182 onship of calibrated estimates of sodium and potassium excretion with cardiovascular outcomes.

183 d with progressively higher sodium and lower potassium excretion; in comparison with the lowest quart

184 Amperometric analysis was performed with potassium ferricyanide as an electron mediator under arg

185 s, limiting currents from the reduction of a potassium ferrocyanide (K4[Fe(CN)6].3H2O) were measured

186 (glucose oxidase, horseradish peroxidase and potassium ferrocyanide as mediator of the electron trans

187 cattle manure at a single dose equivalent to potassium fertilizer (ORG) or double dose (2xORG).

188 vation of G-protein-coupled inward rectifier potassium (GIRK) channels and hyperpolarization, but in

189 tion of previously reported ((Ar)L)FeCl with potassium graphite furnished a low-spin (S = 1/2) iron c

190 cal ((Ar)L)FeCl((*)N(C6H4-p-(t)Bu)) (2) with potassium graphite furnished the corresponding high-spin

191 of these devices was then evaluated by using potassium hexacyanoferrate(II) as redox probe.

192 uctures and supports the notion that altered potassium homeostasis and unmyelinated fibers may repres

193 The proper maintenance of potassium homeostasis is crucial for cell viability.

194 ical for blood pressure (BP), acid-base, and potassium homeostasis.

195 y a solid-solid insertion protocol that uses potassium hydride as a redox-controlled reducing agent t

196 nediaminetetraacetic acid (EDTA), 2mmolL(-1) potassium hydrogen phthalate (KHP) in 1% v/v methanol (p

197 Potassium hydroxide caused a total reduction of aflatoxi

198 unfortunately, sensors capable of providing potassium images in vivo are still a future proposition.

199 eived placebo, urine potassium but not serum potassium increased significantly among participants ran

200 Fast-rising and sustained extracellular potassium increases associated to interneuronal network

201 l(-) cotransporter (NCC) is activated by low potassium intake and by hypokalemia.

202 Higher levels of sodium and lower levels of potassium intake are associated with higher blood pressu

203 ed the estimation of mean dietary sodium and potassium intakes.

204 A graphite-potassium intercalation compound (KC8) was dispersed in

205 mparable values for all samples, except when potassium iodate (KIO3) was used in the recipe.

206 Administration of potassium iodide as a source of free iodide led to a dim

207 Scavenging of apoplastic H2O2 by potassium iodide repressed lignin formation, in line wit

208 d Methods Inkjet cartridges were filled with potassium iodide solutions (600 mg/mL) and prints were r

209 Using a microfluidic approach, we find that potassium ion channel-mediated electrical signaling gene

210 underlies the structural basis for gating in potassium ion channels.

211 e electrodes, with and without an additional potassium ion-selective membrane (ISM) coating, followin

212 Potassium-ion batteries (PIBs) are interesting as one of

213 ells and their afferent neurons to show that potassium ions accumulating in the synaptic cleft modula

214 ity with experimental results for sodium and potassium ions in propylene carbonate by obtaining over

215 during the cardiac action potential, passing potassium ions outward to repolarize ventricular myocyte

216 formance for the insertion and extraction of potassium ions.

217 electrochemical energy storage devices using potassium-ions as charge carriers are attractive due to

218 difficult because the large ionic radius of potassium-ions causes structural distortion and instabil

219 However, the accommodation of potassium-ions with satisfactory capacity and cyclabilit

220 Here, we show that inwardly rectifying potassium (Irk) channels regulate release of the Drosoph

221 l shape and turgor under conditions in which potassium is limiting.

222 yocytes, there are several Ca(2+) -sensitive potassium (K(+) ) currents such as the slowly activating

223 Adaptation of the organism to potassium (K(+)) deficiency requires precise coordinatio

224 nts, but little is known about their role in potassium (K(+)) nutrition.

225 ment experiment (nitrogen, N; phosphorus, P; potassium, K).

226 Two-pore domain potassium (K2P) channel ion conductance is regulated by

227 thermo- and mechanosensitive two-pore domain potassium (K2P) channels of the TREK subfamily generate

228 esponse by ATP-sensitive inwardly rectifying potassium (KATP) channels.

229 enclamide, an inhibitor of the ATP-dependent potassium (KATP)-channels, thus suggesting a possible me

230 Small-conductance calcium-activated potassium (KCa2) channels have also been implicated in e

231 l-molecule inhibitor of the inward rectifier potassium (Kir) channel and diuretic target, Kir1.1.

232 ining structural feature of inward-rectifier potassium (Kir) channels is the unique Kir cytoplasmic d

233 bunits which resemble metazoan voltage-gated potassium (Kv1-Kv4) channels in assembly and gating prop

234 We subsequently found that internal potassium levels and TORC1 activity are linked.

235 Likewise, persistence of abnormal potassium levels was linked to a higher risk of death in

236 modialysis prescription is to maintain serum potassium levels within a narrow normal range during bot

237 er reverse-dialysis to elevate extracellular potassium levels.

238 , 73%, and 41% in nitrate-, phosphorus-, and potassium-limiting conditions, respectively.

239 distal tubules of mice subjected to dietary potassium loading and restriction, KS-WNK1 knockout mice

240 However, potassium metal anodes suffer from poor reversibility du

241 Rechargeable potassium metal batteries have recently emerged as alter

242 The prognostic value of long-term potassium monitoring and dynamics in heart failure has n

243 well as with interventions to increase serum potassium more than was achieved with our intervention,

244 To determine (1) the dose-response effect of potassium nitrate (KNO3) on exercise capacity; (2) the p

245 Potassium normalization was independently associated wit

246 ncluded 2164 patients with a total of 16 116 potassium observations.

247 tilizer components (nitrogen, phosphorus and potassium) on the development of potted rice plants and

248 ncentration (0.5wt%) gelation was induced by potassium or calcium chloride.

249 for sodium, and 0.38 (95% CI, 0.17-0.87) for potassium (P<0.01 for trends).

250 repeats, were probed with dimethylsulphate, potassium permanganate and S1 nuclease.

251 Here we used potassium permanganate footprinting, DNase I footprintin

252 Oxidation with potassium permanganate gave good yields of related diols

253 Both chloride and potassium permeability linearly correlated with the mass

254 d MnCl2.4H2O) concentrations and buffers/pH (potassium phosphate buffer pH 6-8, Tris buffer pH 8-10)

255 the surface of potassium enabling reversible potassium plating/stripping electrochemistry with high e

256 Competition from sodium and potassium present in synthetic and real urine did not si

257 on of iodosylbenzene with the poorly soluble potassium (pseudo)halide salts), typically higher conver

258 teria, the kdp operon encodes a four-subunit potassium pump that maintains intracellular homeostasis,

259 cident diabetes than did those in the lowest potassium quartile [OR (95% CI): 0.61 (0.39, 0.97) and 0

260 dL, n = 1163), participants in the highest 2 potassium quartiles had significantly lower odds of inci

261 s were a function of self-reported sodium-to-potassium ratio from a food frequency questionnaire, age

262 rd ratio for a 20% increase in the sodium-to-potassium ratio was 1.13 (95% confidence interval (CI):

263 explore whether factors related to dialytic potassium removal can be modified to improve clinical ou

264 I, RbI, CsI; also investigation of different potassium salts (acetate, iodide, nitrate, chloride, dih

265 lyzed process revealed that use of sodium or potassium salts was crucial for achieving high stereosel

266 te the salty and bitter tastes of sodium and potassium salts.

267 C-dependent changes in the driving force for potassium secretion are not sufficient to explain hyperk

268 tions, reduced chloride secretion, increased potassium secretion, and increased sodium absorption wer

269 e modified K(+) NS allows fluorescence-based potassium sensing in the range of 20 mM to 1 M.

270 sis that the potassium channel Kir4.1 is the potassium sensor of DCT cells.

271 outward currents coupled with extracellular potassium shifts, abolished by pharmacological blockade

272 NLRP3 inhibitor MCC950 or with extracellular potassium significantly reduced IL-1beta cleavage and re

273 ting the molecular anions to create multiple potassium sites within initially dense molecular layers,

274 l of a small conductance Ca(2)(+) -activated potassium (SK) channel was developed and incorporated in

275 Small conductance calcium-activated potassium (SK) channels are required for the slow inhibi

276 iated by small-conductance calcium-activated potassium (SK) channels in the MNTB neurons from rats of

277 ution of small-conductance calcium-activated potassium (SK) channels.

278 and the small conductance Ca(2+) -activated potassium (SK) current (ISK ).

279 Perovskite potassium sodium niobates, K1-xNaxNbO3, are promising le

280 roduced by extracellular application of high-potassium solution (20 mm, K20), in nociceptors incubate

281 respectively, in the presence of 20 equiv of potassium t-butoxide relative to catalyst.

282 h hydrosilanes under the catalytic action of potassium tert-butoxide alone.

283 in (REP), which contains less phosphorus and potassium than soy and casein proteins, as a supplementa

284 trino flux, specifically those associated to potassium, the mantle and the core.

285 chanism of the Ullmann-type reaction between potassium thioacetate (KSAc) and iodobenzene (PhI) catal

286 transmission and is mediated by diffusion of potassium to deep cortical layers.

287 superfamilies, the mechanism by which uphill potassium transport through KdpA is coupled with ATP hyd

288 ere depolarized, nearly devoid of conductive potassium transport, and unresponsive to plasma potassiu

289 omyces cerevisiae are the Trk1 high affinity potassium transporter and the functionally redundant Hal

290 ation of the membrane ATP-hydrolysing sodium/potassium transporter Na(+)/K(+)-ATPase (NKA) into a mon

291 bunit (KdpA) belonging to the superfamily of potassium transporters and another pump-like subunit (Kd

292 Among the major determinants of potassium uptake in the model organism Saccharomyces cer

293 e, the multivariable-adjusted association of potassium values and mortality revealed a nonlinear asso

294 t to determine the association between serum potassium values collected at follow-up with all-cause m

295 In a minimally adjusted model, serum potassium was a significant predictor of incident diabet

296 Furthermore, dynamic changes in potassium were independently associated with substantial

297 Median intakes for fiber and potassium were lower than Adequate Intakes.

298 Especially for potassium, whose content is strictly controlled for chro

299 associations of serum, dietary, and urinary potassium with incident diabetes.

300 multivariable-adjusted association of serum potassium with mortality was assessed by using comprehen