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1 95% CI: 0.74, 0.92 per SD increment in serum potassium).
2 activity-dependent increase of extracellular potassium.
3 e effects may be driven by changes in plasma potassium.
4 (LIBs) due to the abundance and low cost of potassium.
5 otassium and urinary fractional excretion of potassium.
6 ivation through the release of intracellular potassium.
7 nt regulator, possibly via effects on plasma potassium.
8 sodium intake and to the ratio of sodium to potassium.
10 nt that alters cell membrane permeability to potassium affected the maintenance and establishment of
11 at the intracellular pH sensor perturbs the potassium affinity at the extracellular selectivity filt
16 d predators encoding insect specific sodium, potassium and calcium channel blockers for their ability
17 rskolin) that act upon glucose transporters, potassium and calcium channels, and G-protein-coupled re
18 ich are insensitive to the contribution from potassium and do not provide model-independent informati
20 centrations were associated with lower serum potassium and higher urinary excretion of potassium, but
23 nt method to quantify concentrations of ash, potassium and magnesium and present the potential to cla
24 chemometrics for at-line monitoring of ash, potassium and magnesium content of GF flours: tapioca, p
26 n the surface of the soma: the voltage-gated potassium and sodium channels Kv1.4 and Nav1.6 and the g
27 in the polymeric membrane with creatininium, potassium and sodium confirms the strong selective inter
28 ufacturers and some agar types and also with potassium and thymidine for S. pneumoniae For all other
29 osterone and MR activity, assessed via serum potassium and urinary fractional excretion of potassium.
30 a protein-embedded tunnel runs between these potassium and water sites and a helix controlling the cy
33 elines recommend routine monitoring of serum potassium, and renal function in patients treated with a
35 ty-four-hour urinary excretion of sodium and potassium are unaffected by recall errors and represent
37 strictor that inhibits the membranous sodium-potassium ATPase pump across cell types and can cause ra
39 ide an overview of factors affecting overall potassium balance and factors modulating potassium dialy
40 nstead, a DCT-ASDN coupling process controls potassium balance in health and becomes aberrantly activ
42 energies of cryptomelane, sodium birnessite, potassium birnessite and calcium birnessite are all sign
43 in, it is reported for the first time that a potassium bis(fluoroslufonyl)imide (KFSI)-dimethoxyethan
44 ssion of large-conductance calcium-activated potassium (BK) channels and Kv3.3 voltage-gated potassiu
46 cin (mTOR) activation, loss of glutamate and potassium buffering capacity, loss of astrocyte coupling
47 ith participants who received placebo, urine potassium but not serum potassium increased significantl
50 secretion, activation of NCC helps to retain potassium by increasing electroneutral NaCl reabsorption
51 e spatial distribution of specific analytes (potassium, calcium, manganese, iron, and zinc), and disc
53 intracellular activities, and extracellular potassium changes demonstrates that SLEs in the piriform
54 toxicity related to anti-human ether-a-go-go potassium channel (hERG) activity of the first-generatio
55 roach, we discovered that a pair of two-pore potassium channel (K2P) subunits, largely dispensable ea
61 tor-mediated inhibition of a two-pore domain potassium channel and A1 receptor-mediated opening of a
64 6.3%): 3 (2.7%) had TPO-Ab and voltage-gated potassium channel complex (VGKCc) Ab, 2 (1.8%) had GAD65
65 independently of TIM in Drosophila to alter potassium channel conductance in arousal neurons after l
66 (human Ether-a'-go-go-Related Gene) cardiac potassium channel delays cardiac repolarization and can
67 nd interventions targeted towards correcting potassium channel dysfunction in ataxia need to be tailo
69 BK, Slo1, MaxiK, KCNMA1) is the predominant potassium channel expressed at the plasma membrane of rh
70 ting tumor viability and invasion, including potassium channel function and EPH receptor signaling.
72 ion, our results reveal a role for the KCNQ1 potassium channel in the regulation of human growth, and
74 ainate receptor GluR6/7 and inward rectifier potassium channel Kir2.1, closely associated with SAP102
76 ng in transcripts encoding the voltage-gated potassium channel Kv1.1 converts an isoleucine to valine
80 ed by the reduction in the expression of the potassium channel Kv2.1 at the surface of motor neurons.
82 vated calcium currents, and independently of potassium channel regulation, membrane potential changes
83 confirmed; thereafter, reduced levels of the potassium channel ROMK and kinases SGK1 and WNK1 were ob
86 nctional expression of the voltage-dependent potassium channel subunit Kv1.1 substantially contribute
91 xpressed the intermediate-conductance KCa3.1 potassium channel, revealing a strong functional couplin
92 anism of regulation of the proton pump and a potassium channel, two essential elements in K(+) uptake
93 ian hnRNP U, result in dysfunction of a Slo2 potassium channel, which is critical to neuronal functio
94 -positive specimens had higher voltage-gated potassium channel-IgG immunoprecipitation values (0.33nm
98 TIONALE: Large-conductance calcium-activated potassium channels (BK) are composed of pore-forming BKa
100 occurs via activation of inwardly rectifying potassium channels (KIR ), and synthesis of nitric oxide
103 assium (BK) channels and Kv3.3 voltage-gated potassium channels accompanies the inability of Purkinje
105 ignatures of infection, such as induction of potassium channels and amino acid transporters, derepres
106 PV+ cells by regulating the localization of potassium channels and AMPA receptors, respectively.
110 POINTS: Intracellular Na(+) -activated Slo2 potassium channels are in a closed state under normal ph
111 scovered that yeast cells lacking endogenous potassium channels could be rescued by WT ROMK but not b
115 reby demonstrating the involvement of A-type potassium channels in prolonging pauses evoked by GABAer
116 regions and increased expression of specific potassium channels in the NAc may promote abstinence fro
117 hippocampal cultures and asked how distinct potassium channels interact in determining the basal spi
118 subclasses of voltage- and/or calcium-gated potassium channels may provide an important approach to
122 leads to epigenetic repression of Kv1.1-type potassium channels, increased excitability, and impaired
124 le structures of TRPV1 and voltage-activated potassium channels, we engineered chimeras wherein trans
129 son of transport of monovalent electrolytes [potassium chloride (KCl), sodium chloride (NaCl)], 2:1 e
131 s associated with reduced dorsal spinal cord potassium chloride cotransporter expression and impaired
132 ium reduction and its partial replacement by potassium chloride in pizza dough and crusts prepared by
133 us was modulated by focal microinjections of potassium chloride into the nucleus reuniens, during whi
134 hermodynamic chemical activity of sodium and potassium chloride, as well as the effect of the salts o
135 tion at unchanged cytosolic Ca(2+) levels in potassium chloride-constricted intact arteries isolated
138 s of Tbx1 or Slc12a2, which encodes a sodium-potassium-chloride cotransporter and is also necessary f
139 receptors can physically associate with the potassium-chloride cotransporter protein, KCC2, which se
140 latile elements and compounds, such as zinc, potassium, chlorine, and water, provide key evidence for
144 e mechanisms by which DCT cells sense plasma potassium concentration and transmit the information to
148 d we review data linking serum and dialysate potassium concentrations with arrhythmias, cardiovascula
149 Postsynaptically, the opioids activate a potassium conductance through the mu-opioid receptor (MO
151 ltage-clamp experiments, 2-AG reduced A-type potassium current (IA) through a cannabinoid receptor-in
152 lum Ca2+ concentrations, inwardly rectifying potassium current (IK1) density, and gap junction conduc
153 ate the slowly activating, voltage-dependent potassium current (IKs) in the heart that controls the r
155 gnificant reduction of the transient outward potassium current (Ito) in EPI but not in endocardial (E
157 associated with decreased transient outward potassium current and Kv4.2 and KChIP2 protein expressio
158 ings are consistent with the hypothesis that potassium current downregulation leads to abnormal repol
160 paper unveils the critical role of the brake potassium current IKD in damage-triggered cold allodynia
161 Dysfunction of the fast-inactivating Kv3.4 potassium current in dorsal root ganglion (DRG) neurons
162 led that the isoflurane-activated background potassium current observed in cortical pyramidal neurons
164 t reduction, but the contribution to overall potassium current reduction was almost always much small
165 , there was a component of calcium-dependent potassium current that showed frequency-dependent reduct
167 dent reduction of overall spike-repolarizing potassium current was identified as Kv3 current by its s
168 ion of G protein-coupled inwardly rectifying potassium current was measured using whole-cell voltage-
169 n resulted in lack of time-dependent outward potassium currents in guard cells, higher rates of water
170 NTS: Kv2 channels underlie delayed-rectifier potassium currents in various neurons, although their ph
171 y consistent reductions in voltage-activated potassium currents near the action potential threshold a
172 elayed repolarization from downregulation of potassium currents, and multiple reentry circuits during
174 all potassium balance and factors modulating potassium dialysate fluxes in dialysis, and we review da
176 le for the inflammasome sensor NLRP3 and for potassium efflux in T. gondii-induced IL-1beta productio
179 rolyte forms a uniform SEI on the surface of potassium enabling reversible potassium plating/strippin
180 , and an inverse association between urinary potassium excretion and blood pressure, in a nationally
183 d with progressively higher sodium and lower potassium excretion; in comparison with the lowest quart
184 Amperometric analysis was performed with potassium ferricyanide as an electron mediator under arg
185 s, limiting currents from the reduction of a potassium ferrocyanide (K4[Fe(CN)6].3H2O) were measured
186 (glucose oxidase, horseradish peroxidase and potassium ferrocyanide as mediator of the electron trans
188 vation of G-protein-coupled inward rectifier potassium (GIRK) channels and hyperpolarization, but in
189 tion of previously reported ((Ar)L)FeCl with potassium graphite furnished a low-spin (S = 1/2) iron c
190 cal ((Ar)L)FeCl((*)N(C6H4-p-(t)Bu)) (2) with potassium graphite furnished the corresponding high-spin
192 uctures and supports the notion that altered potassium homeostasis and unmyelinated fibers may repres
195 y a solid-solid insertion protocol that uses potassium hydride as a redox-controlled reducing agent t
196 nediaminetetraacetic acid (EDTA), 2mmolL(-1) potassium hydrogen phthalate (KHP) in 1% v/v methanol (p
198 unfortunately, sensors capable of providing potassium images in vivo are still a future proposition.
199 eived placebo, urine potassium but not serum potassium increased significantly among participants ran
202 Higher levels of sodium and lower levels of potassium intake are associated with higher blood pressu
208 d Methods Inkjet cartridges were filled with potassium iodide solutions (600 mg/mL) and prints were r
209 Using a microfluidic approach, we find that potassium ion channel-mediated electrical signaling gene
211 e electrodes, with and without an additional potassium ion-selective membrane (ISM) coating, followin
213 ells and their afferent neurons to show that potassium ions accumulating in the synaptic cleft modula
214 ity with experimental results for sodium and potassium ions in propylene carbonate by obtaining over
215 during the cardiac action potential, passing potassium ions outward to repolarize ventricular myocyte
217 electrochemical energy storage devices using potassium-ions as charge carriers are attractive due to
218 difficult because the large ionic radius of potassium-ions causes structural distortion and instabil
222 yocytes, there are several Ca(2+) -sensitive potassium (K(+) ) currents such as the slowly activating
227 thermo- and mechanosensitive two-pore domain potassium (K2P) channels of the TREK subfamily generate
229 enclamide, an inhibitor of the ATP-dependent potassium (KATP)-channels, thus suggesting a possible me
231 l-molecule inhibitor of the inward rectifier potassium (Kir) channel and diuretic target, Kir1.1.
232 ining structural feature of inward-rectifier potassium (Kir) channels is the unique Kir cytoplasmic d
233 bunits which resemble metazoan voltage-gated potassium (Kv1-Kv4) channels in assembly and gating prop
236 modialysis prescription is to maintain serum potassium levels within a narrow normal range during bot
239 distal tubules of mice subjected to dietary potassium loading and restriction, KS-WNK1 knockout mice
243 well as with interventions to increase serum potassium more than was achieved with our intervention,
244 To determine (1) the dose-response effect of potassium nitrate (KNO3) on exercise capacity; (2) the p
247 tilizer components (nitrogen, phosphorus and potassium) on the development of potted rice plants and
254 d MnCl2.4H2O) concentrations and buffers/pH (potassium phosphate buffer pH 6-8, Tris buffer pH 8-10)
255 the surface of potassium enabling reversible potassium plating/stripping electrochemistry with high e
257 on of iodosylbenzene with the poorly soluble potassium (pseudo)halide salts), typically higher conver
258 teria, the kdp operon encodes a four-subunit potassium pump that maintains intracellular homeostasis,
259 cident diabetes than did those in the lowest potassium quartile [OR (95% CI): 0.61 (0.39, 0.97) and 0
260 dL, n = 1163), participants in the highest 2 potassium quartiles had significantly lower odds of inci
261 s were a function of self-reported sodium-to-potassium ratio from a food frequency questionnaire, age
262 rd ratio for a 20% increase in the sodium-to-potassium ratio was 1.13 (95% confidence interval (CI):
263 explore whether factors related to dialytic potassium removal can be modified to improve clinical ou
264 I, RbI, CsI; also investigation of different potassium salts (acetate, iodide, nitrate, chloride, dih
265 lyzed process revealed that use of sodium or potassium salts was crucial for achieving high stereosel
267 C-dependent changes in the driving force for potassium secretion are not sufficient to explain hyperk
268 tions, reduced chloride secretion, increased potassium secretion, and increased sodium absorption wer
271 outward currents coupled with extracellular potassium shifts, abolished by pharmacological blockade
272 NLRP3 inhibitor MCC950 or with extracellular potassium significantly reduced IL-1beta cleavage and re
273 ting the molecular anions to create multiple potassium sites within initially dense molecular layers,
274 l of a small conductance Ca(2)(+) -activated potassium (SK) channel was developed and incorporated in
276 iated by small-conductance calcium-activated potassium (SK) channels in the MNTB neurons from rats of
280 roduced by extracellular application of high-potassium solution (20 mm, K20), in nociceptors incubate
283 in (REP), which contains less phosphorus and potassium than soy and casein proteins, as a supplementa
285 chanism of the Ullmann-type reaction between potassium thioacetate (KSAc) and iodobenzene (PhI) catal
287 superfamilies, the mechanism by which uphill potassium transport through KdpA is coupled with ATP hyd
288 ere depolarized, nearly devoid of conductive potassium transport, and unresponsive to plasma potassiu
289 omyces cerevisiae are the Trk1 high affinity potassium transporter and the functionally redundant Hal
290 ation of the membrane ATP-hydrolysing sodium/potassium transporter Na(+)/K(+)-ATPase (NKA) into a mon
291 bunit (KdpA) belonging to the superfamily of potassium transporters and another pump-like subunit (Kd
293 e, the multivariable-adjusted association of potassium values and mortality revealed a nonlinear asso
294 t to determine the association between serum potassium values collected at follow-up with all-cause m
300 multivariable-adjusted association of serum potassium with mortality was assessed by using comprehen
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