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1 SUR2A, SUR2B, or ROMK (renal outer medullary potassium channel).
2 ceptor tyrosine kinase FGFR1 and cardiac IKS potassium channel.
3 onal expression of the renal outer medullary potassium channel.
4 hibition, mediated by a GABAB receptor and a potassium channel.
5 by mutations in the voltage-dependent Kv3.3 potassium channel.
6 erely impair the correct functioning of this potassium channel.
7 ng the extent of clustering of KcsA, a model potassium channel.
8 g of a G-protein-coupled inwardly rectifying potassium channel.
9 ng to identify genetic variants of the KCNQ1 potassium channel.
10 acterial MscL channel and certain eukaryotic potassium channels.
11 ctivation and the subsequent closure of KCNQ potassium channels.
12 polarization caused by stretch activation of potassium channels.
13 ting the normal stoichiometry of presynaptic potassium channels.
14 yramidal neuron AIS voltage-gated sodium and potassium channels.
15 ion of G-protein-coupled inwardly rectifying potassium channels.
16 ule cells through modification of Kv4 A-type potassium channels.
17 ed potassium (BK) and subthreshold-activated potassium channels.
18 potassium channels and not by ATP-sensitive potassium channels.
19 nd the Kir4.1 subunit of inwardly rectifying potassium channels.
20 d in silico at the central cavities of hERG1 potassium channels.
21 ough the activation of voltage-gated KCNQ2-5 potassium channels.
22 afferent input impedance by closing calyceal potassium channels.
23 o activate at a rate similar to conventional potassium channels.
24 H-sensitive potassium channel encoded by the potassium channel, 2-pore domain, subfamily K, member 3
27 ent excitability by the closure of KCNQ-type potassium channels, a potential mechanism for the painfu
30 assium (BK) channels and Kv3.3 voltage-gated potassium channels accompanies the inability of Purkinje
31 ism involving both T-type calcium and A-type potassium channel activation, but are independent of gap
35 tor-mediated inhibition of a two-pore domain potassium channel and A1 receptor-mediated opening of a
36 a novel regulatory mechanism for a mammalian potassium channel and for T-cell activation, and highlig
37 iated long-QT syndrome by targeting the hERG potassium channel and inhibiting the related current (IK
38 GIRK2), which encodes an inwardly rectifying potassium channel and maps to the Down syndrome critical
39 ignatures of infection, such as induction of potassium channels and amino acid transporters, derepres
41 ociation, we found that BK calcium-activated potassium channels and Kv2 channels both make major cont
42 This work shows that BK calcium-activated potassium channels and Kv2 voltage-activated potassium c
43 traethylammonium-sensitive calcium-dependent potassium channels and not by ATP-sensitive potassium ch
45 iptional regulation of Kv1 voltage-dependent potassium channels and the resulting postnatal switch to
46 hesis that activation of inwardly rectifying potassium channels and the sodium-potassium ATPase pump,
48 y through rapid and slowed delayed rectifier potassium channels) and that block of the rapid delayed
49 fic BacNav orthologues, an inward-rectifying potassium channel, and connexin-43 in primary human fibr
50 This also depends on slow delayed-rectifier potassium channels, and preferred theta ranges shift whe
52 l role in essential physiological processes, potassium channels are common targets for animal toxins.
54 ated human ether-a-go-go-related gene (hERG) potassium channels are critical for the repolarization o
58 POINTS: Intracellular Na(+) -activated Slo2 potassium channels are in a closed state under normal ph
65 TIONALE: Large-conductance calcium-activated potassium channels (BK) are composed of pore-forming BKa
66 e, voltage-gated, calcium (Ca(2+))-activated potassium channel (BKCa) plays an important role in regu
70 (Na(+))-channel blocker, and d,l-sotalol, a potassium channel blocker, were studied in littermate mi
71 e treat embryonic chick cardiac cells with a potassium channel blocker, which leads to the initiation
72 h this idea, administration of voltage-gated potassium channel blockers restores conduction and resul
74 potassium channels and Kv2 voltage-activated potassium channels both regulate action potentials in do
75 for the large conductance calcium-activated potassium channel brings new thinking about regulation o
76 depolarizes neurons by a partial closure of potassium channels but decreases the vesicle release pro
77 her sensory modalities express many types of potassium channels, but how they combine to control firi
78 pharmacological targeting of a mitochondrial potassium channel can lead to ROS-mediated selective apo
79 rns and understanding the role of particular potassium channels can help to guide new pharmacological
80 s in hERG (encoding the Kv11.1 voltage-gated potassium channel) cause long-QT syndrome type 2 (LQT2)
82 6.3%): 3 (2.7%) had TPO-Ab and voltage-gated potassium channel complex (VGKCc) Ab, 2 (1.8%) had GAD65
84 tylcholine receptor (20%), voltage-gated Kv1 potassium channel-complex (13%), and alpha-amino-3-hydro
85 frequent were aquaporin-4, voltage-gated Kv1 potassium channel-complex related proteins (leucine-rich
86 ly and organizations of sodium, calcium, and potassium channel complexes within specific subcellular
87 independently of TIM in Drosophila to alter potassium channel conductance in arousal neurons after l
88 OMPC ARs to mechanoinsensitive voltage-gated potassium channels confers mechanosensitivity to the chi
89 ifying potassium channel-related spinal cord potassium channel) contributes to nodose ganglia (NG) ma
90 scovered that yeast cells lacking endogenous potassium channels could be rescued by WT ROMK but not b
91 t firing by decreasing the calcium-activated potassium channel current (SK), as well as elevates dopa
92 e block of stretch-sensitive ATP-inactivated potassium channels curtailed VF occurrence in a porcine
93 (human Ether-a'-go-go-Related Gene) cardiac potassium channel delays cardiac repolarization and can
96 nd interventions targeted towards correcting potassium channel dysfunction in ataxia need to be tailo
98 sitive K(+) channel (TASK)-1 [a pH-sensitive potassium channel encoded by the potassium channel, 2-po
104 BK, Slo1, MaxiK, KCNMA1) is the predominant potassium channel expressed at the plasma membrane of rh
111 ting tumor viability and invasion, including potassium channel function and EPH receptor signaling.
112 (KCNK3) and TASK-3 (KCNK9) tandem pore (K2P) potassium channel function and stimulate breathing.
113 echanism contributes to the understanding of potassium channel function in general and might lead to
114 assium channel 3 (TASK-3; KCNK9) tandem pore potassium channel function is activated by halogenated a
115 restores small-conductance calcium-dependent potassium channel function, normalizing the firing activ
120 cium channels and activate G-protein coupled potassium channels (GIRK and TREK channels), both mechan
121 ivity was assessed via virally overexpressed potassium channels (GIRK2) in medium spiny neurons (MSNs
122 of the Kir3 subfamily of G-protein-activated potassium channels (GIRKs), plays several roles in the n
124 ificant results, including calcium-activated potassium channels (GO:0016286; P=2.30 x 10(-5)), cognit
125 We found that the EAG2 (Ether-a-go-go 2) potassium channel has an evolutionarily conserved functi
129 toxicity related to anti-human ether-a-go-go potassium channel (hERG) activity of the first-generatio
131 -positive specimens had higher voltage-gated potassium channel-IgG immunoprecipitation values (0.33nm
132 f intermediate-conductance calcium-activated potassium channels (IKCa) and TRPV4 channels near the mi
133 of the rapidly activating delayed rectifier potassium channel (IKr), which is important for cardiac
136 inhibits the function of the Drosophila KCNQ potassium channel in a heterologous expression system.
137 ion, our results reveal a role for the KCNQ1 potassium channel in the regulation of human growth, and
138 he voltage-sensing domain (VSD) of the Kv1.2 potassium channel in the resting state and by exploring
139 agonists play any roles in the regulation of potassium channels in Muller cells and subsequently in t
140 reby demonstrating the involvement of A-type potassium channels in prolonging pauses evoked by GABAer
141 ht on the special function of five different potassium channels in the distal nephron, encoded by the
142 regions and increased expression of specific potassium channels in the NAc may promote abstinence fro
145 tead to proteins that are complexed with the potassium channel, in particular leucine-rich, glioma-in
146 leads to epigenetic repression of Kv1.1-type potassium channels, increased excitability, and impaired
148 hippocampal cultures and asked how distinct potassium channels interact in determining the basal spi
149 vitro and in vivo, binds epilepsy-associated potassium channel-interacting proteins including KCNAB2
150 that the activation of the calcium-activated potassium channel is sufficient to induce NOX-independen
152 e isoform of the human ether-a-go-go-related potassium channel, is associated with impaired cognition
153 ltage-dependent sodium and fast-inactivating potassium channels just below spike threshold, amplifyin
154 roach, we discovered that a pair of two-pore potassium channel (K2P) subunits, largely dispensable ea
156 sitive human adipocytes, a set that included potassium channel K3 (KCNK3) and mitochondrial tumor sup
157 h Ringer solution (control), a ATP-sensitive potassium channel (KATP ) inhibitor, an intermediate cal
161 inhibitor, an intermediate calcium-dependent potassium channel (KCa ) inhibitor, a non-specific KCa c
167 kinase 4 (WNK4) inhibits the activity of the potassium channel KCNJ1 (ROMK) in the distal nephron, th
171 in lipid bilayers using neutron diffraction: potassium channel KcsA and the transmembrane domain of M
173 occurs via activation of inwardly rectifying potassium channels (KIR ), and synthesis of nitric oxide
174 ainate receptor GluR6/7 and inward rectifier potassium channel Kir2.1, closely associated with SAP102
176 n of G-protein-activated inwardly rectifying potassium channel (Kir3.X) (GIRK) conductance by submaxi
178 receptor 1 (SUR1) or the inwardly rectifying potassium channel Kir6.2, respectively, results in conge
181 , and identify upregulated voltage-dependent potassium channel (KV) number in cerebral arterial myocy
182 ue, in part, to suppression of voltage-gated potassium channels (Kv ) in pulmonary arterial smooth mu
183 ue, in part, to suppression of voltage-gated potassium channels (Kv ) in pulmonary arterial smooth mu
184 MRP has been confirmed to bind voltage-gated potassium channels (Kv 3.1 and Kv 4.2) mRNAs and regulat
187 ng in transcripts encoding the voltage-gated potassium channel Kv1.1 converts an isoleucine to valine
189 ype 1 is caused by missense mutations of the potassium channel Kv1.1, which is abundantly expressed i
190 ent de novo mutations in KCNA2, encoding the potassium channel KV1.2, in six isolated patients with e
192 umour-specific T cells by overexpressing the potassium channel Kv1.3 lowers [K(+)]i and improves effe
194 t and selective blocker of the voltage-gated potassium channel Kv1.3, which is a highly promising tar
197 composition and distribution of shaker-type-potassium channels (Kv1 channels) within the nodal compl
198 ransmembrane segment, S2, of a voltage-gated potassium channel, Kv1.3, as a model to probe this issue
199 ed by the reduction in the expression of the potassium channel Kv2.1 at the surface of motor neurons.
201 may be related to the expression of the fast potassium channel Kv3.1b, which in rat interneurons is a
202 echanism by which mutations in voltage-gated potassium channels lead to EA is still unknown and there
203 iated inactivation of presynaptic Kv1-family potassium channels, leading to action potential broadeni
204 the human ether a go-go-related gene (hERG) potassium channel, many of which cause misfolding and de
205 subclasses of voltage- and/or calcium-gated potassium channels may provide an important approach to
206 d that a G-protein-coupled inward-rectifying potassium channel mediated regulation of dendritic plate
207 d by cell atrophy, which by restoring normal potassium channel membrane density, re-establishes pacem
210 h the increased activity of barium-sensitive potassium channels, most consistent with inwardly rectif
211 rome of myoclonus epilepsy and ataxia due to potassium channel mutation (MEAK), including cellular el
213 and calcium (Kcnma1, Kcnn1 and Kcnn2)-gated potassium channels observed in the NAc of nonaddicted ra
217 cyclic-nucleotide-gated (HCN) and transient potassium channels play critical roles in regulating the
218 uman human ether-a-go-go-related gene (hERG) potassium channel plays a critical role in the repolariz
219 olution, the key conformational changes of a potassium channel pore domain as it progresses along its
220 Here, we show that inhibition of Kv1.3, a potassium channel preferentially expressed by Tem cells,
223 ene targets included 5 RAB family members, 3 potassium channel proteins, and 2 peroxisome family memb
225 to involve exclusively small conductance (SK potassium channels), recent findings have shown that BK
226 es, we randomly labeled tetrameric KirBac1.1 potassium channels, reconstituted them into lipid nanodi
227 vated calcium currents, and independently of potassium channel regulation, membrane potential changes
228 TRESK (2-pore-domain weak inward-rectifying potassium channel-related spinal cord potassium channel)
229 channels are the dominant delayed rectifier potassium channels responsible for action potential repo
230 xpressed the intermediate-conductance KCa3.1 potassium channel, revealing a strong functional couplin
231 confirmed; thereafter, reduced levels of the potassium channel ROMK and kinases SGK1 and WNK1 were ob
232 um channel (ENaC), the renal outer medullary potassium channel (ROMK), and other transport pathways.
243 However, the pathophysiological role of potassium channel subfamily K member 3 (KCNK3) in PAH is
246 slow inhibition and reveal that one type of potassium channel subunit is important for mediating the
247 jury-induced downregulation of voltage-gated potassium channel subunit Kcna2 in the dorsal root gangl
248 nctional expression of the voltage-dependent potassium channel subunit Kv1.1 substantially contribute
253 upled receptor (GPR4) and a proton-modulated potassium channel (TASK-2) whose transcripts are undetec
255 novel proteins (including cullin1, ephexin, potassium channel tetramerization domain containing prot
256 e, is caused by mutations in a voltage-gated potassium channel that contributes to the generation of
257 evealed the three-dimensional structure of a potassium channel that has a central role in regulating
258 f toxins for one such orphan target, KcsA, a potassium channel that has been fundamental to delineati
260 er a go-go, EAG1 or KV10.1), a voltage-gated potassium channel that is predominantly expressed in the
261 flux and the intermediate-conductance KCa3.1 potassium channel that promotes an outward tail current
266 lepsy gene Kcna1 encodes voltage-gated Kv1.1 potassium channels that act to dampen neuronal excitabil
267 ed Kv7 (KCNQ) channels are voltage-dependent potassium channels that are activated at resting membran
268 mammalian small- to intermediate-conductance potassium channels that are activated by calcium-calmodu
269 on of the Kv7.2 subunit of the Kv7 family of potassium channels that control neuronal excitability.
270 f the axon with a high density of sodium and potassium channels that defines the site of action poten
271 among the enriched genes are those encoding potassium channels that down-regulate neuronal activity,
273 not capable of activating small-conductance potassium channels, the intrinsic excitability of VTA do
274 essed in Xenopus laevis oocytes with various potassium channels, the newly discovered segment preserv
276 toxin pull-down assay with immobilised KcsA potassium channel to isolate a novel KcsA-binding toxin
277 hways, and uncover a mechanism for different potassium channels to functionally cooperate and regulat
278 report a comprehensive analysis of putative potassium channel toxins (KTxs) from the cDNA library of
280 ve increased activation of the 2-pore domain potassium channel TRESK (2-pore-domain weak inward-recti
281 anism of regulation of the proton pump and a potassium channel, two essential elements in K(+) uptake
282 nce imaging has linked chronic voltage-gated potassium channel (VGKC) complex antibody-mediated limbi
283 e extracellular domains of the voltage-gated potassium channel (VGKC) complex proteins, leucine-rich
286 , the glycine receptor (GlyR), voltage-gated potassium channel (VGKC)-complex and the associated prot
287 n 11 patients, uncharacterised voltage-gated potassium channel (VGKC)-complex antigens in four patien
289 was caused by oxidative dysfunction of Kv4.3 potassium channels, was recently identified in transgeni
290 le structures of TRPV1 and voltage-activated potassium channels, we engineered chimeras wherein trans
291 ccompanied by increased mRNA levels of these potassium channels when compared with mRNA expression in
292 ian hnRNP U, result in dysfunction of a Slo2 potassium channel, which is critical to neuronal functio
293 ery even now, buried in the structure of the potassium channel, which was completely unknown at the t
294 s by inactivating dendritic Kv1.1-containing potassium channels, which increased dendrite excitabilit
295 Trp434-Asp447 indole hydrogen bond in Shaker potassium channels with a non-hydrogen bonding homologue
297 diated expression of exogenous transmembrane potassium channels with high contrast and resolution.
298 blocking large conductance calcium-activated potassium channels with iberiotoxin, and is abolished by
299 mediated mainly by fast-activating Kv3-type potassium channels, with clustered hotspots at boutons a
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