ライフサイエンスコーパス: potassium channel

1 SUR2A, SUR2B, or ROMK (renal outer medullary potassium channel).

2 ceptor tyrosine kinase FGFR1 and cardiac IKS potassium channel.

3 onal expression of the renal outer medullary potassium channel.

4 hibition, mediated by a GABAB receptor and a potassium channel.

5 by mutations in the voltage-dependent Kv3.3 potassium channel.

6 erely impair the correct functioning of this potassium channel.

7 ng the extent of clustering of KcsA, a model potassium channel.

8 g of a G-protein-coupled inwardly rectifying potassium channel.

9 ng to identify genetic variants of the KCNQ1 potassium channel.

10 acterial MscL channel and certain eukaryotic potassium channels.

11 ctivation and the subsequent closure of KCNQ potassium channels.

12 polarization caused by stretch activation of potassium channels.

13 ting the normal stoichiometry of presynaptic potassium channels.

14 yramidal neuron AIS voltage-gated sodium and potassium channels.

15 ion of G-protein-coupled inwardly rectifying potassium channels.

16 ule cells through modification of Kv4 A-type potassium channels.

17 ed potassium (BK) and subthreshold-activated potassium channels.

18 potassium channels and not by ATP-sensitive potassium channels.

19 nd the Kir4.1 subunit of inwardly rectifying potassium channels.

20 d in silico at the central cavities of hERG1 potassium channels.

21 ough the activation of voltage-gated KCNQ2-5 potassium channels.

22 afferent input impedance by closing calyceal potassium channels.

23 o activate at a rate similar to conventional potassium channels.

24 H-sensitive potassium channel encoded by the potassium channel, 2-pore domain, subfamily K, member 3

25 The TWIK-related acid-sensitive potassium channel 3 (TASK-3; KCNK9) tandem pore potassiu

26 The selectivity filter in potassium channels, a main component of the ion permeati

27 ent excitability by the closure of KCNQ-type potassium channels, a potential mechanism for the painfu

28 Deletion of the potassium channel abolishes this response.

29 Slo2 channels are large-conductance potassium channels abundantly expressed in the nervous s

30 assium (BK) channels and Kv3.3 voltage-gated potassium channels accompanies the inability of Purkinje

31 ism involving both T-type calcium and A-type potassium channel activation, but are independent of gap

32 tions but required T-type calcium and A-type potassium channel activation.

33 porter SGLT1, or by closure of ATP-sensitive potassium channels after glucose metabolism.

34 beta cells, including the T2DM-linked KCNQ1 potassium channel alpha subunit.

35 tor-mediated inhibition of a two-pore domain potassium channel and A1 receptor-mediated opening of a

36 a novel regulatory mechanism for a mammalian potassium channel and for T-cell activation, and highlig

37 iated long-QT syndrome by targeting the hERG potassium channel and inhibiting the related current (IK

38 GIRK2), which encodes an inwardly rectifying potassium channel and maps to the Down syndrome critical

39 ignatures of infection, such as induction of potassium channels and amino acid transporters, derepres

40 PV+ cells by regulating the localization of potassium channels and AMPA receptors, respectively.

41 ociation, we found that BK calcium-activated potassium channels and Kv2 channels both make major cont

42 This work shows that BK calcium-activated potassium channels and Kv2 voltage-activated potassium c

43 traethylammonium-sensitive calcium-dependent potassium channels and not by ATP-sensitive potassium ch

44 dopts the Kunitz fold known to mostly act on potassium channels and serine proteases.

45 iptional regulation of Kv1 voltage-dependent potassium channels and the resulting postnatal switch to

46 hesis that activation of inwardly rectifying potassium channels and the sodium-potassium ATPase pump,

47 Expression of other potassium channels and two different light-activated cha

48 y through rapid and slowed delayed rectifier potassium channels) and that block of the rapid delayed

49 fic BacNav orthologues, an inward-rectifying potassium channel, and connexin-43 in primary human fibr

50 This also depends on slow delayed-rectifier potassium channels, and preferred theta ranges shift whe

51 EAG-like (ELK) voltage-gated potassium channels are abundantly expressed in the brain

52 l role in essential physiological processes, potassium channels are common targets for animal toxins.

53 KCNQ2 potassium channels are critical for normal brain functio

54 ated human ether-a-go-go-related gene (hERG) potassium channels are critical for the repolarization o

55 The BK-type potassium channels are exclusively linked to the N-type

56 Kv3.1 and Kv3.2 voltage-gated potassium channels are expressed on parvalbumin-positive

57 Intracellular Na(+) -activated Slo2 potassium channels are in a closed state under normal ph

58 POINTS: Intracellular Na(+) -activated Slo2 potassium channels are in a closed state under normal ph

59 Potassium channels are opened by ligands and/or membrane

60 Potassium channels are responsible for the selective per

61 Two-pore domain (K2P) potassium channels are the major molecular correlates of

62 iated optical control of inwardly rectifying potassium channels, as well as adenylyl cyclase.

63 Oligomers of homomeric voltage-gated potassium channels associate early in biogenesis as the

64 Mice with germline deletion of the KCNE2 potassium channel beta subunit exhibited NAFLD as early

65 TIONALE: Large-conductance calcium-activated potassium channels (BK) are composed of pore-forming BKa

66 e, voltage-gated, calcium (Ca(2+))-activated potassium channel (BKCa) plays an important role in regu

67 ATP-inactivated potassium channel block has no effect on PVEM inducibili

68 The potassium channel blocker 4-aminopyridine reliably induc

69 s and the isolated guinea pig brain with the potassium channel blocker 4-aminopyridine.

70 (Na(+))-channel blocker, and d,l-sotalol, a potassium channel blocker, were studied in littermate mi

71 e treat embryonic chick cardiac cells with a potassium channel blocker, which leads to the initiation

72 h this idea, administration of voltage-gated potassium channel blockers restores conduction and resul

73 Potassium channel blockers, such as 4-aminopyridine, ind

74 potassium channels and Kv2 voltage-activated potassium channels both regulate action potentials in do

75 for the large conductance calcium-activated potassium channel brings new thinking about regulation o

76 depolarizes neurons by a partial closure of potassium channels but decreases the vesicle release pro

77 her sensory modalities express many types of potassium channels, but how they combine to control firi

78 pharmacological targeting of a mitochondrial potassium channel can lead to ROS-mediated selective apo

79 rns and understanding the role of particular potassium channels can help to guide new pharmacological

80 s in hERG (encoding the Kv11.1 voltage-gated potassium channel) cause long-QT syndrome type 2 (LQT2)

81 GIRK (G protein-coupled inwardly rectifying potassium channels) channels (IK,Ado).

82 6.3%): 3 (2.7%) had TPO-Ab and voltage-gated potassium channel complex (VGKCc) Ab, 2 (1.8%) had GAD65

83 1 (LGI1) is a component of the voltage-gated potassium channel complex.

84 tylcholine receptor (20%), voltage-gated Kv1 potassium channel-complex (13%), and alpha-amino-3-hydro

85 frequent were aquaporin-4, voltage-gated Kv1 potassium channel-complex related proteins (leucine-rich

86 ly and organizations of sodium, calcium, and potassium channel complexes within specific subcellular

87 independently of TIM in Drosophila to alter potassium channel conductance in arousal neurons after l

88 OMPC ARs to mechanoinsensitive voltage-gated potassium channels confers mechanosensitivity to the chi

89 ifying potassium channel-related spinal cord potassium channel) contributes to nodose ganglia (NG) ma

90 scovered that yeast cells lacking endogenous potassium channels could be rescued by WT ROMK but not b

91 t firing by decreasing the calcium-activated potassium channel current (SK), as well as elevates dopa

92 e block of stretch-sensitive ATP-inactivated potassium channels curtailed VF occurrence in a porcine

93 (human Ether-a'-go-go-Related Gene) cardiac potassium channel delays cardiac repolarization and can

94 Increased sodium and decreased potassium channel densities in the initial axon segment

95 ion, 5F8 is clearly among the best synthetic potassium channels developed over the past decades.

96 nd interventions targeted towards correcting potassium channel dysfunction in ataxia need to be tailo

97 r-mediated pathway that impinges on specific potassium channel effectors.

98 sitive K(+) channel (TASK)-1 [a pH-sensitive potassium channel encoded by the potassium channel, 2-po

99 ne and serotonin receptors as well as Kv11.1 potassium channels encoded by KCNH2.

100 Inwardly rectifying potassium channels enforce tight control of resting memb

101 Potassium channels exhibit a modular design with distinc

102 Sodium and potassium channels exhibit a one-dimensional periodicity

103 The slow spontaneous inactivation of potassium channels exhibits classic signatures of transm

104 BK, Slo1, MaxiK, KCNMA1) is the predominant potassium channel expressed at the plasma membrane of rh

105 Kv1.3 is a voltage-gated potassium channel expressed on myelin-reactive T cells f

106 KV10.1 is a voltage-gated potassium channel expressed selectively in the mammalian

107 C-type inactivation of potassium channels fine-tunes the electrical signaling i

108 Comprehensive analysis of this potassium channel for the four variants expressed in fro

109 Voltage-gated potassium channels formed by KCNQ2 and KCNQ3 are essenti

110 The roles of potassium channels from the Shaker family in stomatal mo

111 ting tumor viability and invasion, including potassium channel function and EPH receptor signaling.

112 (KCNK3) and TASK-3 (KCNK9) tandem pore (K2P) potassium channel function and stimulate breathing.

113 echanism contributes to the understanding of potassium channel function in general and might lead to

114 assium channel 3 (TASK-3; KCNK9) tandem pore potassium channel function is activated by halogenated a

115 restores small-conductance calcium-dependent potassium channel function, normalizing the firing activ

116 indered by specific genetic perturbations to potassium channel gating.

117 Among those, we found potassium channel genes KCNA4 and KCNIP4, involved in el

118 Mutations in the potassium channel genes were the most common cause (n=39

119 -93 mV, indicative of an inwardly rectifying potassium channel (GIRK) mechanism.

120 cium channels and activate G-protein coupled potassium channels (GIRK and TREK channels), both mechan

121 ivity was assessed via virally overexpressed potassium channels (GIRK2) in medium spiny neurons (MSNs

122 of the Kir3 subfamily of G-protein-activated potassium channels (GIRKs), plays several roles in the n

123 ion of G-protein-coupled inwardly rectifying potassium channels (GIRKs).

124 ificant results, including calcium-activated potassium channels (GO:0016286; P=2.30 x 10(-5)), cognit

125 We found that the EAG2 (Ether-a-go-go 2) potassium channel has an evolutionarily conserved functi

126 Kv1.2 and related voltage-gated potassium channels have a highly conserved N-linked glyc

127 Slo2 potassium channels have a very low open probability unde

128 C-type inactivation in potassium channels helps fine-tune long-term channel act

129 toxicity related to anti-human ether-a-go-go potassium channel (hERG) activity of the first-generatio

130 The human ether-a-go-go-related potassium channel (hERG, Kv11.1) is a voltage-dependent

131 -positive specimens had higher voltage-gated potassium channel-IgG immunoprecipitation values (0.33nm

132 f intermediate-conductance calcium-activated potassium channels (IKCa) and TRPV4 channels near the mi

133 of the rapidly activating delayed rectifier potassium channel (IKr), which is important for cardiac

134 of the rapidly activating delayed rectifier potassium channel (IKr).

135 The cardiac potassium channel, IKr, and the adrenergic-sensitive car

136 inhibits the function of the Drosophila KCNQ potassium channel in a heterologous expression system.

137 ion, our results reveal a role for the KCNQ1 potassium channel in the regulation of human growth, and

138 he voltage-sensing domain (VSD) of the Kv1.2 potassium channel in the resting state and by exploring

139 agonists play any roles in the regulation of potassium channels in Muller cells and subsequently in t

140 reby demonstrating the involvement of A-type potassium channels in prolonging pauses evoked by GABAer

141 ht on the special function of five different potassium channels in the distal nephron, encoded by the

142 regions and increased expression of specific potassium channels in the NAc may promote abstinence fro

143 Potassium channels in the two-pore domain family (K2P) h

144 e affected the activity of voltage-dependent potassium channels in these neurons.

145 tead to proteins that are complexed with the potassium channel, in particular leucine-rich, glioma-in

146 leads to epigenetic repression of Kv1.1-type potassium channels, increased excitability, and impaired

147 A dataset of 55 hERG potassium channel inhibitors collected from Kramer et al

148 hippocampal cultures and asked how distinct potassium channels interact in determining the basal spi

149 vitro and in vivo, binds epilepsy-associated potassium channel-interacting proteins including KCNAB2

150 that the activation of the calcium-activated potassium channel is sufficient to induce NOX-independen

151 The ether a go-go family of voltage-gated potassium channels is structurally distinct.

152 e isoform of the human ether-a-go-go-related potassium channel, is associated with impaired cognition

153 ltage-dependent sodium and fast-inactivating potassium channels just below spike threshold, amplifyin

154 roach, we discovered that a pair of two-pore potassium channel (K2P) subunits, largely dispensable ea

155 In contrast to other potassium channels, K2P channels use a selectivity filte

156 sitive human adipocytes, a set that included potassium channel K3 (KCNK3) and mitochondrial tumor sup

157 h Ringer solution (control), a ATP-sensitive potassium channel (KATP ) inhibitor, an intermediate cal

158 ABSTRACT: Sarcolemmal ATP-sensitive potassium channel (KATP channel) activation in isolated

159 Sarcolemmal ATP-sensitive potassium channels (KATP channels) in cardiac myocytes a

160 hage polarization by targeting ATP sensitive potassium channels (KATP).

161 inhibitor, an intermediate calcium-dependent potassium channel (KCa ) inhibitor, a non-specific KCa c

162 Large-conductance calcium-activated potassium channel (KCa1.1; BK, Slo1, MaxiK, KCNMA1) is t

163 The calcium-activated potassium channel KCa3.1 controls different cellular pro

164 Potassium channels (KChs) are the most diverse ion chann

165 Mutations in the Kv3.3 potassium channel (KCNC3) cause cerebellar neurodegenera

166 The voltage-gated potassium channel Kcnh4a, which is expressed in all Hcrt

167 kinase 4 (WNK4) inhibits the activity of the potassium channel KCNJ1 (ROMK) in the distal nephron, th

168 Variants in potassium channel KCNJ2 cause Andersen-Tawil Syndrome (A

169 Either of 2 somatic mutations in the potassium channel KCNJ5 (G151R and L168R, hereafter refe

170 Here, we identify a two-pore-domain potassium channel, KCNK3, as a built-in rheostat negativ

171 in lipid bilayers using neutron diffraction: potassium channel KcsA and the transmembrane domain of M

172 Here, using the potassium channel KcsA as a model, we contribute new evi

173 occurs via activation of inwardly rectifying potassium channels (KIR ), and synthesis of nitric oxide

174 ainate receptor GluR6/7 and inward rectifier potassium channel Kir2.1, closely associated with SAP102

175 ough co-expression of an inwardly rectifying potassium channel (Kir2.1).

176 n of G-protein-activated inwardly rectifying potassium channel (Kir3.X) (GIRK) conductance by submaxi

177 Here, we tested the hypothesis that the potassium channel Kir4.1 is the potassium sensor of DCT

178 receptor 1 (SUR1) or the inwardly rectifying potassium channel Kir6.2, respectively, results in conge

179 ng of MC4R to closure of inwardly rectifying potassium channel, Kir7.1.

180 KCNJ13 gene encodes the inwardly rectifying potassium channel, Kir7.1.

181 , and identify upregulated voltage-dependent potassium channel (KV) number in cerebral arterial myocy

182 ue, in part, to suppression of voltage-gated potassium channels (Kv ) in pulmonary arterial smooth mu

183 ue, in part, to suppression of voltage-gated potassium channels (Kv ) in pulmonary arterial smooth mu

184 MRP has been confirmed to bind voltage-gated potassium channels (Kv 3.1 and Kv 4.2) mRNAs and regulat

185 Changes in voltage-dependent potassium channels (Kv channels) associate to proliferat

186 Voltage-gated potassium channels (Kv), which play a role in controllin

187 ng in transcripts encoding the voltage-gated potassium channel Kv1.1 converts an isoleucine to valine

188 editing of at least one other ADAR2 target, potassium channel Kv1.1, is decreased after SCI.

189 ype 1 is caused by missense mutations of the potassium channel Kv1.1, which is abundantly expressed i

190 ent de novo mutations in KCNA2, encoding the potassium channel KV1.2, in six isolated patients with e

191 The potassium channel Kv1.3 is highly expressed in the mitoc

192 umour-specific T cells by overexpressing the potassium channel Kv1.3 lowers [K(+)]i and improves effe

193 The voltage-dependent potassium channel Kv1.3 plays essential physiological fu

194 t and selective blocker of the voltage-gated potassium channel Kv1.3, which is a highly promising tar

195 s treatable by blockade of the voltage-gated potassium channel Kv1.3.

196 nnels at the node of Ranvier and Shaker-type potassium channel (Kv1.2) at the juxtaparanode.

197 composition and distribution of shaker-type-potassium channels (Kv1 channels) within the nodal compl

198 ransmembrane segment, S2, of a voltage-gated potassium channel, Kv1.3, as a model to probe this issue

199 ed by the reduction in the expression of the potassium channel Kv2.1 at the surface of motor neurons.

200 on and localization of the delayed-rectifier potassium channel, Kv2.1.

201 may be related to the expression of the fast potassium channel Kv3.1b, which in rat interneurons is a

202 echanism by which mutations in voltage-gated potassium channels lead to EA is still unknown and there

203 iated inactivation of presynaptic Kv1-family potassium channels, leading to action potential broadeni

204 the human ether a go-go-related gene (hERG) potassium channel, many of which cause misfolding and de

205 subclasses of voltage- and/or calcium-gated potassium channels may provide an important approach to

206 d that a G-protein-coupled inward-rectifying potassium channel mediated regulation of dendritic plate

207 d by cell atrophy, which by restoring normal potassium channel membrane density, re-establishes pacem

208 c nucleotide binding domain of the bacterial potassium channel MloK1.

209 Following the recent development of a potassium channel modulator, AUT1-an imidazolidinedione

210 h the increased activity of barium-sensitive potassium channels, most consistent with inwardly rectif

211 rome of myoclonus epilepsy and ataxia due to potassium channel mutation (MEAK), including cellular el

212 ulfonylurea treatment for most patients with potassium channel mutations.

213 and calcium (Kcnma1, Kcnn1 and Kcnn2)-gated potassium channels observed in the NAc of nonaddicted ra

214 Voltage-gated potassium channels of the KCNQ (Kv7) subfamily are essen

215 d Slack (Slo2.2, KCNT1) are high-conductance potassium channels of the Slo family.

216 Two-pore domain (K2P) potassium channels perform essential roles in neuronal f

217 cyclic-nucleotide-gated (HCN) and transient potassium channels play critical roles in regulating the

218 uman human ether-a-go-go-related gene (hERG) potassium channel plays a critical role in the repolariz

219 olution, the key conformational changes of a potassium channel pore domain as it progresses along its

220 Here, we show that inhibition of Kv1.3, a potassium channel preferentially expressed by Tem cells,

221 rotein (EYFP) under the control of the Kv3.1 potassium channel promoter.

222 ibutable to interdependence of voltage-gated potassium channel properties.

223 ene targets included 5 RAB family members, 3 potassium channel proteins, and 2 peroxisome family memb

224 all-intermediate family of calcium-activated potassium channel proteins.

225 to involve exclusively small conductance (SK potassium channels), recent findings have shown that BK

226 es, we randomly labeled tetrameric KirBac1.1 potassium channels, reconstituted them into lipid nanodi

227 vated calcium currents, and independently of potassium channel regulation, membrane potential changes

228 TRESK (2-pore-domain weak inward-rectifying potassium channel-related spinal cord potassium channel)

229 channels are the dominant delayed rectifier potassium channels responsible for action potential repo

230 xpressed the intermediate-conductance KCa3.1 potassium channel, revealing a strong functional couplin

231 confirmed; thereafter, reduced levels of the potassium channel ROMK and kinases SGK1 and WNK1 were ob

232 um channel (ENaC), the renal outer medullary potassium channel (ROMK), and other transport pathways.

233 Although potassium channels shape neuronal activity, their roles

234 ther the BK channel SLO-1 or the Shaker type potassium channel SHK-1.

235 Several potassium channels showed decreased mRNA production, whe

236 aspartate receptors and weakened by cAMP-PKA-potassium channel signaling in dendritic spines.

237 Small conductance calcium-activated potassium channels (SK channels) are present in spines a

238 and the small conductance, calcium-activated potassium channel, SK2.

239 and the small conductance, calcium-activated potassium channel, SK2.

240 The sodium-activated potassium channels Slick (Slo2.1, KCNT2) and Slack (Slo2

241 Unlike potassium channels, sodium channel alpha-subunits are be

242 The voltage-gated potassium channel subfamily A member 3 (Kv1.3) dominantl

243 However, the pathophysiological role of potassium channel subfamily K member 3 (KCNK3) in PAH is

244 Drug block of voltage-gated potassium channel subtype 11.1 human ether-a-go-go relat

245 -coupled G protein-coupled inward rectifying potassium channel subunit 2 (GIRK2) channel.

246 slow inhibition and reveal that one type of potassium channel subunit is important for mediating the

247 jury-induced downregulation of voltage-gated potassium channel subunit Kcna2 in the dorsal root gangl

248 nctional expression of the voltage-dependent potassium channel subunit Kv1.1 substantially contribute

249 The voltage-dependent potassium channel subunit Kv3.3 is expressed at high lev

250 caused by genetic disruption of a mammalian potassium channel subunit.

251 D afferents express KCNQ3, KCNQ4, and ERG1-3 potassium channel subunits.

252 The ability of NMDARs to regulate potassium channel surface expression and thus, beta-cell

253 upled receptor (GPR4) and a proton-modulated potassium channel (TASK-2) whose transcripts are undetec

254 Here we demonstrated that KCTD12, potassium channel tetramerization domain containing 12,

255 novel proteins (including cullin1, ephexin, potassium channel tetramerization domain containing prot

256 e, is caused by mutations in a voltage-gated potassium channel that contributes to the generation of

257 evealed the three-dimensional structure of a potassium channel that has a central role in regulating

258 f toxins for one such orphan target, KcsA, a potassium channel that has been fundamental to delineati

259 KCNQ1 is a voltage-gated potassium channel that is modulated by the beta-subunit

260 er a go-go, EAG1 or KV10.1), a voltage-gated potassium channel that is predominantly expressed in the

261 flux and the intermediate-conductance KCa3.1 potassium channel that promotes an outward tail current

262 Slack (Slo2.2) is a sodium-activated potassium channel that regulates neuronal firing activit

263 Kv11.1 (hERG) is a voltage-gated potassium channel that shows very slow ionic current act

264 dent leak currents through a two-pore-domain potassium channel that we term Sandman.

265 es with the recovery of the density of these potassium channels that accompanies cell atrophy.

266 lepsy gene Kcna1 encodes voltage-gated Kv1.1 potassium channels that act to dampen neuronal excitabil

267 ed Kv7 (KCNQ) channels are voltage-dependent potassium channels that are activated at resting membran

268 mammalian small- to intermediate-conductance potassium channels that are activated by calcium-calmodu

269 on of the Kv7.2 subunit of the Kv7 family of potassium channels that control neuronal excitability.

270 f the axon with a high density of sodium and potassium channels that defines the site of action poten

271 among the enriched genes are those encoding potassium channels that down-regulate neuronal activity,

272 We investigated the potassium channels that shape the reliability of signal

273 not capable of activating small-conductance potassium channels, the intrinsic excitability of VTA do

274 essed in Xenopus laevis oocytes with various potassium channels, the newly discovered segment preserv

275 Unlike potassium ions in potassium channels, the sodium ions in these channels ap

276 toxin pull-down assay with immobilised KcsA potassium channel to isolate a novel KcsA-binding toxin

277 hways, and uncover a mechanism for different potassium channels to functionally cooperate and regulat

278 report a comprehensive analysis of putative potassium channel toxins (KTxs) from the cDNA library of

279 Here we report that the KCNE2 potassium channel transmembrane regulatory subunit is ex

280 ve increased activation of the 2-pore domain potassium channel TRESK (2-pore-domain weak inward-recti

281 anism of regulation of the proton pump and a potassium channel, two essential elements in K(+) uptake

282 nce imaging has linked chronic voltage-gated potassium channel (VGKC) complex antibody-mediated limbi

283 e extracellular domains of the voltage-gated potassium channel (VGKC) complex proteins, leucine-rich

284 ntigenic components within the voltage-gated potassium channel (VGKC) complex.

285 Antibodies against the voltage-gated potassium channel (VGKC) were first recognised as having

286 , the glycine receptor (GlyR), voltage-gated potassium channel (VGKC)-complex and the associated prot

287 n 11 patients, uncharacterised voltage-gated potassium channel (VGKC)-complex antigens in four patien

288 EAG2 potassium channel was enriched at the trailing edge of m

289 was caused by oxidative dysfunction of Kv4.3 potassium channels, was recently identified in transgeni

290 le structures of TRPV1 and voltage-activated potassium channels, we engineered chimeras wherein trans

291 ccompanied by increased mRNA levels of these potassium channels when compared with mRNA expression in

292 ian hnRNP U, result in dysfunction of a Slo2 potassium channel, which is critical to neuronal functio

293 ery even now, buried in the structure of the potassium channel, which was completely unknown at the t

294 s by inactivating dendritic Kv1.1-containing potassium channels, which increased dendrite excitabilit

295 Trp434-Asp447 indole hydrogen bond in Shaker potassium channels with a non-hydrogen bonding homologue

296 blocking small conductance calcium-activated potassium channels with apamin.

297 diated expression of exogenous transmembrane potassium channels with high contrast and resolution.

298 blocking large conductance calcium-activated potassium channels with iberiotoxin, and is abolished by

299 mediated mainly by fast-activating Kv3-type potassium channels, with clustered hotspots at boutons a

300 More than two dozen types of potassium channels, with different biophysical and regul