戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 SUR2A, SUR2B, or ROMK (renal outer medullary potassium channel).
2 ceptor tyrosine kinase FGFR1 and cardiac IKS potassium channel.
3 onal expression of the renal outer medullary potassium channel.
4 hibition, mediated by a GABAB receptor and a potassium channel.
5  by mutations in the voltage-dependent Kv3.3 potassium channel.
6 erely impair the correct functioning of this potassium channel.
7 ng the extent of clustering of KcsA, a model potassium channel.
8 g of a G-protein-coupled inwardly rectifying potassium channel.
9 ng to identify genetic variants of the KCNQ1 potassium channel.
10 acterial MscL channel and certain eukaryotic potassium channels.
11 ctivation and the subsequent closure of KCNQ potassium channels.
12 polarization caused by stretch activation of potassium channels.
13 ting the normal stoichiometry of presynaptic potassium channels.
14 yramidal neuron AIS voltage-gated sodium and potassium channels.
15 ion of G-protein-coupled inwardly rectifying potassium channels.
16 ule cells through modification of Kv4 A-type potassium channels.
17 ed potassium (BK) and subthreshold-activated potassium channels.
18  potassium channels and not by ATP-sensitive potassium channels.
19 nd the Kir4.1 subunit of inwardly rectifying potassium channels.
20 d in silico at the central cavities of hERG1 potassium channels.
21 ough the activation of voltage-gated KCNQ2-5 potassium channels.
22 afferent input impedance by closing calyceal potassium channels.
23 o activate at a rate similar to conventional potassium channels.
24 H-sensitive potassium channel encoded by the potassium channel, 2-pore domain, subfamily K, member 3
25              The TWIK-related acid-sensitive potassium channel 3 (TASK-3; KCNK9) tandem pore potassiu
26                    The selectivity filter in potassium channels, a main component of the ion permeati
27 ent excitability by the closure of KCNQ-type potassium channels, a potential mechanism for the painfu
28                              Deletion of the potassium channel abolishes this response.
29          Slo2 channels are large-conductance potassium channels abundantly expressed in the nervous s
30 assium (BK) channels and Kv3.3 voltage-gated potassium channels accompanies the inability of Purkinje
31 ism involving both T-type calcium and A-type potassium channel activation, but are independent of gap
32 tions but required T-type calcium and A-type potassium channel activation.
33 porter SGLT1, or by closure of ATP-sensitive potassium channels after glucose metabolism.
34  beta cells, including the T2DM-linked KCNQ1 potassium channel alpha subunit.
35 tor-mediated inhibition of a two-pore domain potassium channel and A1 receptor-mediated opening of a
36 a novel regulatory mechanism for a mammalian potassium channel and for T-cell activation, and highlig
37 iated long-QT syndrome by targeting the hERG potassium channel and inhibiting the related current (IK
38 GIRK2), which encodes an inwardly rectifying potassium channel and maps to the Down syndrome critical
39 ignatures of infection, such as induction of potassium channels and amino acid transporters, derepres
40  PV+ cells by regulating the localization of potassium channels and AMPA receptors, respectively.
41 ociation, we found that BK calcium-activated potassium channels and Kv2 channels both make major cont
42    This work shows that BK calcium-activated potassium channels and Kv2 voltage-activated potassium c
43 traethylammonium-sensitive calcium-dependent potassium channels and not by ATP-sensitive potassium ch
44 dopts the Kunitz fold known to mostly act on potassium channels and serine proteases.
45 iptional regulation of Kv1 voltage-dependent potassium channels and the resulting postnatal switch to
46 hesis that activation of inwardly rectifying potassium channels and the sodium-potassium ATPase pump,
47                          Expression of other potassium channels and two different light-activated cha
48 y through rapid and slowed delayed rectifier potassium channels) and that block of the rapid delayed
49 fic BacNav orthologues, an inward-rectifying potassium channel, and connexin-43 in primary human fibr
50  This also depends on slow delayed-rectifier potassium channels, and preferred theta ranges shift whe
51                 EAG-like (ELK) voltage-gated potassium channels are abundantly expressed in the brain
52 l role in essential physiological processes, potassium channels are common targets for animal toxins.
53                                        KCNQ2 potassium channels are critical for normal brain functio
54 ated human ether-a-go-go-related gene (hERG) potassium channels are critical for the repolarization o
55                                  The BK-type potassium channels are exclusively linked to the N-type
56                Kv3.1 and Kv3.2 voltage-gated potassium channels are expressed on parvalbumin-positive
57          Intracellular Na(+) -activated Slo2 potassium channels are in a closed state under normal ph
58  POINTS: Intracellular Na(+) -activated Slo2 potassium channels are in a closed state under normal ph
59                                              Potassium channels are opened by ligands and/or membrane
60                                              Potassium channels are responsible for the selective per
61                        Two-pore domain (K2P) potassium channels are the major molecular correlates of
62 iated optical control of inwardly rectifying potassium channels, as well as adenylyl cyclase.
63         Oligomers of homomeric voltage-gated potassium channels associate early in biogenesis as the
64     Mice with germline deletion of the KCNE2 potassium channel beta subunit exhibited NAFLD as early
65 TIONALE: Large-conductance calcium-activated potassium channels (BK) are composed of pore-forming BKa
66 e, voltage-gated, calcium (Ca(2+))-activated potassium channel (BKCa) plays an important role in regu
67                              ATP-inactivated potassium channel block has no effect on PVEM inducibili
68                                          The potassium channel blocker 4-aminopyridine reliably induc
69 s and the isolated guinea pig brain with the potassium channel blocker 4-aminopyridine.
70  (Na(+))-channel blocker, and d,l-sotalol, a potassium channel blocker, were studied in littermate mi
71 e treat embryonic chick cardiac cells with a potassium channel blocker, which leads to the initiation
72 h this idea, administration of voltage-gated potassium channel blockers restores conduction and resul
73                                              Potassium channel blockers, such as 4-aminopyridine, ind
74 potassium channels and Kv2 voltage-activated potassium channels both regulate action potentials in do
75  for the large conductance calcium-activated potassium channel brings new thinking about regulation o
76  depolarizes neurons by a partial closure of potassium channels but decreases the vesicle release pro
77 her sensory modalities express many types of potassium channels, but how they combine to control firi
78 pharmacological targeting of a mitochondrial potassium channel can lead to ROS-mediated selective apo
79 rns and understanding the role of particular potassium channels can help to guide new pharmacological
80 s in hERG (encoding the Kv11.1 voltage-gated potassium channel) cause long-QT syndrome type 2 (LQT2)
81  GIRK (G protein-coupled inwardly rectifying potassium channels) channels (IK,Ado).
82 6.3%): 3 (2.7%) had TPO-Ab and voltage-gated potassium channel complex (VGKCc) Ab, 2 (1.8%) had GAD65
83 1 (LGI1) is a component of the voltage-gated potassium channel complex.
84 tylcholine receptor (20%), voltage-gated Kv1 potassium channel-complex (13%), and alpha-amino-3-hydro
85 frequent were aquaporin-4, voltage-gated Kv1 potassium channel-complex related proteins (leucine-rich
86 ly and organizations of sodium, calcium, and potassium channel complexes within specific subcellular
87  independently of TIM in Drosophila to alter potassium channel conductance in arousal neurons after l
88 OMPC ARs to mechanoinsensitive voltage-gated potassium channels confers mechanosensitivity to the chi
89 ifying potassium channel-related spinal cord potassium channel) contributes to nodose ganglia (NG) ma
90 scovered that yeast cells lacking endogenous potassium channels could be rescued by WT ROMK but not b
91 t firing by decreasing the calcium-activated potassium channel current (SK), as well as elevates dopa
92 e block of stretch-sensitive ATP-inactivated potassium channels curtailed VF occurrence in a porcine
93  (human Ether-a'-go-go-Related Gene) cardiac potassium channel delays cardiac repolarization and can
94               Increased sodium and decreased potassium channel densities in the initial axon segment
95 ion, 5F8 is clearly among the best synthetic potassium channels developed over the past decades.
96 nd interventions targeted towards correcting potassium channel dysfunction in ataxia need to be tailo
97 r-mediated pathway that impinges on specific potassium channel effectors.
98 sitive K(+) channel (TASK)-1 [a pH-sensitive potassium channel encoded by the potassium channel, 2-po
99 ne and serotonin receptors as well as Kv11.1 potassium channels encoded by KCNH2.
100                          Inwardly rectifying potassium channels enforce tight control of resting memb
101                                              Potassium channels exhibit a modular design with distinc
102                                   Sodium and potassium channels exhibit a one-dimensional periodicity
103         The slow spontaneous inactivation of potassium channels exhibits classic signatures of transm
104  BK, Slo1, MaxiK, KCNMA1) is the predominant potassium channel expressed at the plasma membrane of rh
105                     Kv1.3 is a voltage-gated potassium channel expressed on myelin-reactive T cells f
106                    KV10.1 is a voltage-gated potassium channel expressed selectively in the mammalian
107                       C-type inactivation of potassium channels fine-tunes the electrical signaling i
108               Comprehensive analysis of this potassium channel for the four variants expressed in fro
109                                Voltage-gated potassium channels formed by KCNQ2 and KCNQ3 are essenti
110                                 The roles of potassium channels from the Shaker family in stomatal mo
111 ting tumor viability and invasion, including potassium channel function and EPH receptor signaling.
112 (KCNK3) and TASK-3 (KCNK9) tandem pore (K2P) potassium channel function and stimulate breathing.
113 echanism contributes to the understanding of potassium channel function in general and might lead to
114 assium channel 3 (TASK-3; KCNK9) tandem pore potassium channel function is activated by halogenated a
115 restores small-conductance calcium-dependent potassium channel function, normalizing the firing activ
116 indered by specific genetic perturbations to potassium channel gating.
117                        Among those, we found potassium channel genes KCNA4 and KCNIP4, involved in el
118                             Mutations in the potassium channel genes were the most common cause (n=39
119 -93 mV, indicative of an inwardly rectifying potassium channel (GIRK) mechanism.
120 cium channels and activate G-protein coupled potassium channels (GIRK and TREK channels), both mechan
121 ivity was assessed via virally overexpressed potassium channels (GIRK2) in medium spiny neurons (MSNs
122 of the Kir3 subfamily of G-protein-activated potassium channels (GIRKs), plays several roles in the n
123 ion of G-protein-coupled inwardly rectifying potassium channels (GIRKs).
124 ificant results, including calcium-activated potassium channels (GO:0016286; P=2.30 x 10(-5)), cognit
125     We found that the EAG2 (Ether-a-go-go 2) potassium channel has an evolutionarily conserved functi
126              Kv1.2 and related voltage-gated potassium channels have a highly conserved N-linked glyc
127                                         Slo2 potassium channels have a very low open probability unde
128                       C-type inactivation in potassium channels helps fine-tune long-term channel act
129 toxicity related to anti-human ether-a-go-go potassium channel (hERG) activity of the first-generatio
130              The human ether-a-go-go-related potassium channel (hERG, Kv11.1) is a voltage-dependent
131 -positive specimens had higher voltage-gated potassium channel-IgG immunoprecipitation values (0.33nm
132 f intermediate-conductance calcium-activated potassium channels (IKCa) and TRPV4 channels near the mi
133  of the rapidly activating delayed rectifier potassium channel (IKr), which is important for cardiac
134  of the rapidly activating delayed rectifier potassium channel (IKr).
135                                  The cardiac potassium channel, IKr, and the adrenergic-sensitive car
136 inhibits the function of the Drosophila KCNQ potassium channel in a heterologous expression system.
137 ion, our results reveal a role for the KCNQ1 potassium channel in the regulation of human growth, and
138 he voltage-sensing domain (VSD) of the Kv1.2 potassium channel in the resting state and by exploring
139 agonists play any roles in the regulation of potassium channels in Muller cells and subsequently in t
140 reby demonstrating the involvement of A-type potassium channels in prolonging pauses evoked by GABAer
141 ht on the special function of five different potassium channels in the distal nephron, encoded by the
142 regions and increased expression of specific potassium channels in the NAc may promote abstinence fro
143                                              Potassium channels in the two-pore domain family (K2P) h
144 e affected the activity of voltage-dependent potassium channels in these neurons.
145 tead to proteins that are complexed with the potassium channel, in particular leucine-rich, glioma-in
146 leads to epigenetic repression of Kv1.1-type potassium channels, increased excitability, and impaired
147                         A dataset of 55 hERG potassium channel inhibitors collected from Kramer et al
148  hippocampal cultures and asked how distinct potassium channels interact in determining the basal spi
149 vitro and in vivo, binds epilepsy-associated potassium channel-interacting proteins including KCNAB2
150 that the activation of the calcium-activated potassium channel is sufficient to induce NOX-independen
151    The ether a go-go family of voltage-gated potassium channels is structurally distinct.
152 e isoform of the human ether-a-go-go-related potassium channel, is associated with impaired cognition
153 ltage-dependent sodium and fast-inactivating potassium channels just below spike threshold, amplifyin
154 roach, we discovered that a pair of two-pore potassium channel (K2P) subunits, largely dispensable ea
155                         In contrast to other potassium channels, K2P channels use a selectivity filte
156 sitive human adipocytes, a set that included potassium channel K3 (KCNK3) and mitochondrial tumor sup
157 h Ringer solution (control), a ATP-sensitive potassium channel (KATP ) inhibitor, an intermediate cal
158          ABSTRACT: Sarcolemmal ATP-sensitive potassium channel (KATP channel) activation in isolated
159                    Sarcolemmal ATP-sensitive potassium channels (KATP channels) in cardiac myocytes a
160 hage polarization by targeting ATP sensitive potassium channels (KATP).
161 inhibitor, an intermediate calcium-dependent potassium channel (KCa ) inhibitor, a non-specific KCa c
162          Large-conductance calcium-activated potassium channel (KCa1.1; BK, Slo1, MaxiK, KCNMA1) is t
163                        The calcium-activated potassium channel KCa3.1 controls different cellular pro
164                                              Potassium channels (KChs) are the most diverse ion chann
165                       Mutations in the Kv3.3 potassium channel (KCNC3) cause cerebellar neurodegenera
166                            The voltage-gated potassium channel Kcnh4a, which is expressed in all Hcrt
167 kinase 4 (WNK4) inhibits the activity of the potassium channel KCNJ1 (ROMK) in the distal nephron, th
168                                  Variants in potassium channel KCNJ2 cause Andersen-Tawil Syndrome (A
169         Either of 2 somatic mutations in the potassium channel KCNJ5 (G151R and L168R, hereafter refe
170          Here, we identify a two-pore-domain potassium channel, KCNK3, as a built-in rheostat negativ
171 in lipid bilayers using neutron diffraction: potassium channel KcsA and the transmembrane domain of M
172                              Here, using the potassium channel KcsA as a model, we contribute new evi
173 occurs via activation of inwardly rectifying potassium channels (KIR ), and synthesis of nitric oxide
174 ainate receptor GluR6/7 and inward rectifier potassium channel Kir2.1, closely associated with SAP102
175 ough co-expression of an inwardly rectifying potassium channel (Kir2.1).
176 n of G-protein-activated inwardly rectifying potassium channel (Kir3.X) (GIRK) conductance by submaxi
177      Here, we tested the hypothesis that the potassium channel Kir4.1 is the potassium sensor of DCT
178 receptor 1 (SUR1) or the inwardly rectifying potassium channel Kir6.2, respectively, results in conge
179 ng of MC4R to closure of inwardly rectifying potassium channel, Kir7.1.
180  KCNJ13 gene encodes the inwardly rectifying potassium channel, Kir7.1.
181 , and identify upregulated voltage-dependent potassium channel (KV) number in cerebral arterial myocy
182 ue, in part, to suppression of voltage-gated potassium channels (Kv ) in pulmonary arterial smooth mu
183 ue, in part, to suppression of voltage-gated potassium channels (Kv ) in pulmonary arterial smooth mu
184 MRP has been confirmed to bind voltage-gated potassium channels (Kv 3.1 and Kv 4.2) mRNAs and regulat
185                 Changes in voltage-dependent potassium channels (Kv channels) associate to proliferat
186                                Voltage-gated potassium channels (Kv), which play a role in controllin
187 ng in transcripts encoding the voltage-gated potassium channel Kv1.1 converts an isoleucine to valine
188  editing of at least one other ADAR2 target, potassium channel Kv1.1, is decreased after SCI.
189 ype 1 is caused by missense mutations of the potassium channel Kv1.1, which is abundantly expressed i
190 ent de novo mutations in KCNA2, encoding the potassium channel KV1.2, in six isolated patients with e
191                                          The potassium channel Kv1.3 is highly expressed in the mitoc
192 umour-specific T cells by overexpressing the potassium channel Kv1.3 lowers [K(+)]i and improves effe
193                        The voltage-dependent potassium channel Kv1.3 plays essential physiological fu
194 t and selective blocker of the voltage-gated potassium channel Kv1.3, which is a highly promising tar
195 s treatable by blockade of the voltage-gated potassium channel Kv1.3.
196 nnels at the node of Ranvier and Shaker-type potassium channel (Kv1.2) at the juxtaparanode.
197  composition and distribution of shaker-type-potassium channels (Kv1 channels) within the nodal compl
198 ransmembrane segment, S2, of a voltage-gated potassium channel, Kv1.3, as a model to probe this issue
199 ed by the reduction in the expression of the potassium channel Kv2.1 at the surface of motor neurons.
200 on and localization of the delayed-rectifier potassium channel, Kv2.1.
201 may be related to the expression of the fast potassium channel Kv3.1b, which in rat interneurons is a
202 echanism by which mutations in voltage-gated potassium channels lead to EA is still unknown and there
203 iated inactivation of presynaptic Kv1-family potassium channels, leading to action potential broadeni
204  the human ether a go-go-related gene (hERG) potassium channel, many of which cause misfolding and de
205  subclasses of voltage- and/or calcium-gated potassium channels may provide an important approach to
206 d that a G-protein-coupled inward-rectifying potassium channel mediated regulation of dendritic plate
207 d by cell atrophy, which by restoring normal potassium channel membrane density, re-establishes pacem
208 c nucleotide binding domain of the bacterial potassium channel MloK1.
209        Following the recent development of a potassium channel modulator, AUT1-an imidazolidinedione
210 h the increased activity of barium-sensitive potassium channels, most consistent with inwardly rectif
211 rome of myoclonus epilepsy and ataxia due to potassium channel mutation (MEAK), including cellular el
212 ulfonylurea treatment for most patients with potassium channel mutations.
213  and calcium (Kcnma1, Kcnn1 and Kcnn2)-gated potassium channels observed in the NAc of nonaddicted ra
214                                Voltage-gated potassium channels of the KCNQ (Kv7) subfamily are essen
215 d Slack (Slo2.2, KCNT1) are high-conductance potassium channels of the Slo family.
216                        Two-pore domain (K2P) potassium channels perform essential roles in neuronal f
217  cyclic-nucleotide-gated (HCN) and transient potassium channels play critical roles in regulating the
218 uman human ether-a-go-go-related gene (hERG) potassium channel plays a critical role in the repolariz
219 olution, the key conformational changes of a potassium channel pore domain as it progresses along its
220    Here, we show that inhibition of Kv1.3, a potassium channel preferentially expressed by Tem cells,
221 rotein (EYFP) under the control of the Kv3.1 potassium channel promoter.
222 ibutable to interdependence of voltage-gated potassium channel properties.
223 ene targets included 5 RAB family members, 3 potassium channel proteins, and 2 peroxisome family memb
224 all-intermediate family of calcium-activated potassium channel proteins.
225 to involve exclusively small conductance (SK potassium channels), recent findings have shown that BK
226 es, we randomly labeled tetrameric KirBac1.1 potassium channels, reconstituted them into lipid nanodi
227 vated calcium currents, and independently of potassium channel regulation, membrane potential changes
228  TRESK (2-pore-domain weak inward-rectifying potassium channel-related spinal cord potassium channel)
229  channels are the dominant delayed rectifier potassium channels responsible for action potential repo
230 xpressed the intermediate-conductance KCa3.1 potassium channel, revealing a strong functional couplin
231 confirmed; thereafter, reduced levels of the potassium channel ROMK and kinases SGK1 and WNK1 were ob
232 um channel (ENaC), the renal outer medullary potassium channel (ROMK), and other transport pathways.
233                                     Although potassium channels shape neuronal activity, their roles
234 ther the BK channel SLO-1 or the Shaker type potassium channel SHK-1.
235                                      Several potassium channels showed decreased mRNA production, whe
236 aspartate receptors and weakened by cAMP-PKA-potassium channel signaling in dendritic spines.
237          Small conductance calcium-activated potassium channels (SK channels) are present in spines a
238 and the small conductance, calcium-activated potassium channel, SK2.
239 and the small conductance, calcium-activated potassium channel, SK2.
240                         The sodium-activated potassium channels Slick (Slo2.1, KCNT2) and Slack (Slo2
241                                       Unlike potassium channels, sodium channel alpha-subunits are be
242                            The voltage-gated potassium channel subfamily A member 3 (Kv1.3) dominantl
243      However, the pathophysiological role of potassium channel subfamily K member 3 (KCNK3) in PAH is
244                  Drug block of voltage-gated potassium channel subtype 11.1 human ether-a-go-go relat
245 -coupled G protein-coupled inward rectifying potassium channel subunit 2 (GIRK2) channel.
246  slow inhibition and reveal that one type of potassium channel subunit is important for mediating the
247 jury-induced downregulation of voltage-gated potassium channel subunit Kcna2 in the dorsal root gangl
248 nctional expression of the voltage-dependent potassium channel subunit Kv1.1 substantially contribute
249                        The voltage-dependent potassium channel subunit Kv3.3 is expressed at high lev
250  caused by genetic disruption of a mammalian potassium channel subunit.
251 D afferents express KCNQ3, KCNQ4, and ERG1-3 potassium channel subunits.
252            The ability of NMDARs to regulate potassium channel surface expression and thus, beta-cell
253 upled receptor (GPR4) and a proton-modulated potassium channel (TASK-2) whose transcripts are undetec
254            Here we demonstrated that KCTD12, potassium channel tetramerization domain containing 12,
255  novel proteins (including cullin1, ephexin, potassium channel tetramerization domain containing prot
256 e, is caused by mutations in a voltage-gated potassium channel that contributes to the generation of
257 evealed the three-dimensional structure of a potassium channel that has a central role in regulating
258 f toxins for one such orphan target, KcsA, a potassium channel that has been fundamental to delineati
259                     KCNQ1 is a voltage-gated potassium channel that is modulated by the beta-subunit
260 er a go-go, EAG1 or KV10.1), a voltage-gated potassium channel that is predominantly expressed in the
261 flux and the intermediate-conductance KCa3.1 potassium channel that promotes an outward tail current
262         Slack (Slo2.2) is a sodium-activated potassium channel that regulates neuronal firing activit
263             Kv11.1 (hERG) is a voltage-gated potassium channel that shows very slow ionic current act
264 dent leak currents through a two-pore-domain potassium channel that we term Sandman.
265 es with the recovery of the density of these potassium channels that accompanies cell atrophy.
266 lepsy gene Kcna1 encodes voltage-gated Kv1.1 potassium channels that act to dampen neuronal excitabil
267 ed Kv7 (KCNQ) channels are voltage-dependent potassium channels that are activated at resting membran
268 mammalian small- to intermediate-conductance potassium channels that are activated by calcium-calmodu
269 on of the Kv7.2 subunit of the Kv7 family of potassium channels that control neuronal excitability.
270 f the axon with a high density of sodium and potassium channels that defines the site of action poten
271  among the enriched genes are those encoding potassium channels that down-regulate neuronal activity,
272                          We investigated the potassium channels that shape the reliability of signal
273  not capable of activating small-conductance potassium channels, the intrinsic excitability of VTA do
274 essed in Xenopus laevis oocytes with various potassium channels, the newly discovered segment preserv
275                     Unlike potassium ions in potassium channels, the sodium ions in these channels ap
276  toxin pull-down assay with immobilised KcsA potassium channel to isolate a novel KcsA-binding toxin
277 hways, and uncover a mechanism for different potassium channels to functionally cooperate and regulat
278  report a comprehensive analysis of putative potassium channel toxins (KTxs) from the cDNA library of
279                Here we report that the KCNE2 potassium channel transmembrane regulatory subunit is ex
280 ve increased activation of the 2-pore domain potassium channel TRESK (2-pore-domain weak inward-recti
281 anism of regulation of the proton pump and a potassium channel, two essential elements in K(+) uptake
282 nce imaging has linked chronic voltage-gated potassium channel (VGKC) complex antibody-mediated limbi
283 e extracellular domains of the voltage-gated potassium channel (VGKC) complex proteins, leucine-rich
284 ntigenic components within the voltage-gated potassium channel (VGKC) complex.
285         Antibodies against the voltage-gated potassium channel (VGKC) were first recognised as having
286 , the glycine receptor (GlyR), voltage-gated potassium channel (VGKC)-complex and the associated prot
287 n 11 patients, uncharacterised voltage-gated potassium channel (VGKC)-complex antigens in four patien
288                                         EAG2 potassium channel was enriched at the trailing edge of m
289 was caused by oxidative dysfunction of Kv4.3 potassium channels, was recently identified in transgeni
290 le structures of TRPV1 and voltage-activated potassium channels, we engineered chimeras wherein trans
291 ccompanied by increased mRNA levels of these potassium channels when compared with mRNA expression in
292 ian hnRNP U, result in dysfunction of a Slo2 potassium channel, which is critical to neuronal functio
293 ery even now, buried in the structure of the potassium channel, which was completely unknown at the t
294 s by inactivating dendritic Kv1.1-containing potassium channels, which increased dendrite excitabilit
295 Trp434-Asp447 indole hydrogen bond in Shaker potassium channels with a non-hydrogen bonding homologue
296 blocking small conductance calcium-activated potassium channels with apamin.
297 diated expression of exogenous transmembrane potassium channels with high contrast and resolution.
298 blocking large conductance calcium-activated potassium channels with iberiotoxin, and is abolished by
299  mediated mainly by fast-activating Kv3-type potassium channels, with clustered hotspots at boutons a
300                 More than two dozen types of potassium channels, with different biophysical and regul

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top