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1 tion of the spread, as did the ATP-activated potassium channel blocker.
2 l fluid containing 4-aminopyridine (4-AP), a potassium channel blocker.
3 could be abolished by barium, a non-specific potassium channel blocker.
4 mol/L) plus glibenclamide (10 micromol/L), a potassium channel blocker.
5 monly used symptomatic treatment option is a potassium channel blocker [3,4-diaminopyridine (3,4-DAP)
6 l of central hyperexcitability that uses the potassium channel blocker 4-aminopyridine (4-AP) to indu
7 ves a 48-hour preconditioning step using the potassium channel blocker 4-aminopyridine (4-AP), at a l
9 pioid inhibition was reduced by 65% with the potassium channel blocker 4-aminopyridine (4-AP; 100 mic
11 t hippocampal slices by superfusion with the potassium channel blocker 4-aminopyridine in Mg(2+)-free
14 n mouse thalamocortical slices in vitro, the potassium channel blocker 4-AP and GABAA receptor antago
15 in EPSCs induced by either serotonin or the potassium channel blockers 4-aminopyridine (4-AP) or alp
16 ctural similarity to cone snail and scorpion potassium channel blockers, a mutant molecule, Tk-hefu,
17 WIN 55,212-2 was also investigated using the potassium channel blockers barium and 4-aminopyridine.
19 ections to probe interactions of an exemplar potassium channel blocker, barium, with the inward recti
20 ations to IAA-94 and DIDS were unaffected by potassium channel blockers, but were prevented by elevat
21 hich resulted in differential sensitivity to potassium channel blockers (cesium and barium) and sudde
23 r, SKF525A, but not by the calcium-activated potassium channel blocker, charybdotoxin, indicating a m
31 mall-conductance (SK-type) calcium-activated potassium channel blockers in normally innervated cells
34 lucidates some recent advances in the use of potassium channel-blockers of Kv1.3 and IKCa1 to amelior
35 cium-dependent (KCa) or ATP-sensitive (KATP) potassium channel blockers on pial arteriolar CO2 reacti
36 vide a mechanism for the variable effects of potassium channel blockers on transmitter release magnit
38 sion of cesium acetate (CsAc) and the sodium/potassium channel blocker, QX314, each blocked the effec
39 h this idea, administration of voltage-gated potassium channel blockers restores conduction and resul
41 g electrical stimulation and exposure to the potassium channel blocker tetraethyl ammonium chloride (
42 pathway inhibitor PD98059, the nonselective potassium channel blocker tetraethylammonium (TEA), and
43 2 receptor antagonist cimetidine or with the potassium channel blocker tetraethylammonium chloride.
44 c channels are inhibited by the nonselective potassium channel blockers tetraethylammonium and barium
47 (Na(+))-channel blocker, and d,l-sotalol, a potassium channel blocker, were studied in littermate mi
48 e treat embryonic chick cardiac cells with a potassium channel blocker, which leads to the initiation
49 We investigated whether d-sotalol, a pure potassium-channel blocker with no clinically significant
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