ライフサイエンスコーパス: potassium chloride

1 al coupling (evidenced by direct response to potassium chloride).

2 large white pigs by intravenous injection of potassium chloride.

3 bition of protein kinase C or high levels of potassium chloride.

4 mide, and by a depolarizing concentration of potassium chloride.

5 but normal with non-nutrient stimuli such as potassium chloride.

6 naptosomes were stimulated by veratridine or potassium chloride.

7 d control individuals were depolarized using potassium chloride.

8 SR was enhanced by chemical stimulation with potassium chloride.

9 port, and disassemble upon depolarization by potassium chloride.

10 g mixtures of kappa and iota carrageenan and potassium chloride.

11 hat are composed of eutectic silver chloride-potassium chloride.

12 the effective supersaturation of the aqueous potassium chloride.

13 ous agarose gel prepared with supersaturated potassium chloride.

14 ministration of thiopental, pancuronium, and potassium chloride.

15 of cortical spreading depressions induced by potassium chloride.

16 nadate sensitive, and slightly stimulated by potassium chloride.

17 e partial substitution of sodium chloride by potassium chloride (0%, 25%, and 50%) and addition of ar

18 ases in perfusion pressures (delta mm Hg) to potassium chloride (30-300 mmol/L) of in vitro perfused

19 e analysis, including particles comprised of potassium chloride and organic nitrogen during the begin

20 Potassium chloride and sodium nitrate also potentiate fi

21 not enhance pressure responses to 125 mmol/L potassium chloride, and failed to increase perfusion pre

22 response to treatment with sodium chloride, potassium chloride, and sorbitol.

23 n (BUN) and creatinine, chemistries (sodium, potassium, chloride, and bicarbonate), complete blood ce

24 Sodium, potassium, chloride, and creatinine excretion were analy

25 l-time concentration measurements of sodium, potassium, chloride, and pH in a small volume of biologi

26 ulation of nonischemic ipsilateral cortex by potassium chloride application (KCl group; n = 7) and sa

27 s shown that simple sugars, amino acids, and potassium chloride are abundant in Belgian endive extrac

28 salts sodium bromide, potassium bromide, and potassium chloride are employed.

29 hermodynamic chemical activity of sodium and potassium chloride, as well as the effect of the salts o

30 undling serum metabolic panel tests (sodium, potassium, chloride, bicarbonate, glucose, blood urea ni

31 The fluids are dominantly sodium chloride-potassium chloride brines, but they also contain divalen

32 n physiologically relevant concentrations of potassium chloride, calcium chloride, and manganese chlo

33 pithelial sodium channel (gammaENaC), sodium-potassium -chloride co-transporter 2 (NKCC2), sodium chl

34 ome type I, showing an absence of the sodium-potassium-chloride co-transporter 2, NKCC2.

35 The potassium-chloride co-transporter KCC2, encoded by SLC12

36 were determined under varying conditions of potassium chloride concentration using a surface plasmon

37 tion at unchanged cytosolic Ca(2+) levels in potassium chloride-constricted intact arteries isolated

38 ransport in the erythrocyte is attributed to potassium chloride cotransporter 1 (KCC1).

39 vity of a bumetanide-sensitive NKCC1 (sodium potassium chloride cotransporter 1)-like chloride cotran

40 t CA1 pyramidal neurons because of increased potassium chloride cotransporter 2 (KCC2) expression and

41 ride shift caused by significant decrease in potassium chloride cotransporter 2 (Kcc2) mRNA expressio

42 sitive hypotension, with depletion of sodium potassium chloride cotransporter 2 and aquaporin 2.

43 s associated with reduced dorsal spinal cord potassium chloride cotransporter expression and impaired

44 The potassium chloride cotransporter KCC2 plays a major role

45 nds on chloride gradients established by the potassium chloride cotransporter KCC2.

46 aying behavior and discovered mutations in a potassium chloride cotransporter, kcc-2.

47 y gamma-aminobutyric acid and glycine due to potassium chloride cotransporter-2 (KCC2) down-regulatio

48 This study asks if the potassium-chloride cotransporter (K:Cl) and the calcium-

49 The furosemide-sensitive potassium-chloride cotransporter (KCC2) plays an importa

50 s of Tbx1 or Slc12a2, which encodes a sodium-potassium-chloride cotransporter and is also necessary f

51 ction is downregulated, including the sodium-potassium-chloride cotransporter gene nkcc1 (slc12a2) an

52 Here, we show that the Sodium-Potassium-Chloride Cotransporter Isoform-1 (NKCC1) provi

53 w that specific markers of excitability, the potassium-chloride cotransporter KCC2 and GABAA receptor

54 n associated with dysfunction or loss of the potassium-chloride cotransporter KCC2 in a subset of pyr

55 decrease in the functional expression of the potassium-chloride cotransporter KCC2 in spinal cord dor

56 Once in the cortex, upregulation of the potassium-chloride cotransporter KCC2 is both necessary

57 s is largely mediated by the neuron-specific potassium-chloride cotransporter KCC2.

58 ion ([Cl(-)](i)), which is maintained by the potassium-chloride cotransporter KCC2.

59 (hepatocyte nuclear factor 4alpha), SLC12A5 (potassium-chloride cotransporter member 5), CDH22 (cadhe

60 receptors can physically associate with the potassium-chloride cotransporter protein, KCC2, which se

61 novel association between the GABABR and the potassium-chloride cotransporter protein, KCC2.

62 Disruption of the potassium/chloride cotransporter 3 (KCC3), encoded by th

63 1 consumed 0.4 +/- 0.06 mEq (mean +/- SD) of potassium chloride daily, and group 2 ate 4.8 +/- 1.0 mE

64 Potassium chloride depolarization caused sustained relea

65 icantly reduced in hippocampal neurons after potassium chloride depolarization.

66 diet supplemented with potassium citrate or potassium chloride (each 4 mmol/d) for 18 weeks.

67 Investigations involve solutions of potassium chloride electrolyte containing potassium ferr

68 Addition of potassium chloride enhanced extraction of HgR from LAEA-

69 ed for characterization was lithium chloride/potassium chloride eutectic (LKE), which has potential a

70 s link earlier observations that both sodium/potassium/chloride exchange and Ca(2+) are required for

71 system and its regulatory control of sodium, potassium, chloride, hydrogen ion, and water homeostasis

72 ium reduction and its partial replacement by potassium chloride in pizza dough and crusts prepared by

73 um ferricyanide, and supporting electrolyte, potassium chloride, in the presence of a magnetic field.

74 us was modulated by focal microinjections of potassium chloride into the nucleus reuniens, during whi

75 nitride membrane dividing water solution of potassium chloride into two compartments connected by th

76 Potassium chloride ion cotransporters (KCCs) are part of

77 ing TMX with a depolarizing concentration of potassium chloride (K+ -30 mM).

78 was sensitive to the level of extracellular potassium chloride (KCl) and depolarizing concentrations

79 itrogen, and creatinine, escalating doses of potassium chloride (KCl) and spironolactone were adminis

80 vasodilator (sodium nitroprusside, SNP), or potassium chloride (KCl) at rest; (2) mild or moderate i

81 ricans.We sought to determine the effects of potassium chloride (KCl) supplements, at a commonly pres

82 We demonstrate here that replacing potassium chloride (KCl) with potassium acetate (KAc) or

83 evoked in arteries via norepinephrine (NE), potassium chloride (KCl), and caffeine, and in veins thr

84 stitution-mediated DS(-) precipitation using potassium chloride (KCl), and excellent peptide recovery

85 son of transport of monovalent electrolytes [potassium chloride (KCl), sodium chloride (NaCl)], 2:1 e

86 Potassium chloride (KCl)-depolarization has been used to

87 mass for either 24 or 65 h showed increased potassium chloride (KCl)-induced and oxytocin-induced co

88 t (+)-N-allylnormetazocine ((+)-SKF10047) on potassium chloride (KCl)-induced calcium influx in RGC-5

89 ma-1 receptor agonist (+)-SKF10047 inhibited potassium chloride (KCl)-induced calcium influx.

90 [Ca2+]i when they were stimulated with 30 mM potassium chloride (KCl).

91 endothelin [ET-1], phorbol ester [PdBu], and potassium chloride [KCl]).

92 ala and responds strongly to stimuli such as potassium chloride, lithium chloride, and protein kinase

93 Measurements were taken in potassium chloride (moderate ion-lipid binding) and tetr

94 rved ejection fraction to oral KNO3 (n=9) or potassium chloride (n=3).

95 rved ejection fraction to oral KNO3 (n=9) or potassium chloride (n=3).

96 meshwork (TM) cells possess a robust sodium-potassium-chloride (Na-K-Cl) cotransport system that fun

97 udy was to examine the effects of sodium and potassium chloride on lipid oxidation in O/W emulsions.

98 cal administration of the depolarizing agent potassium chloride or systemic administration of the ant

99 e to phenylephrine and serotonin, but not to potassium chloride or U46619.

100 incubation of cells in 100 nm insulin, 30 mm potassium chloride, or 0.25 mm diazoxide, indicating tha

101 Addition of sodium chloride, potassium chloride, or sodium sulfate to leptospiral med

102 The AG was defined as (sodium plus potassium) - (chloride plus total carbon dioxide).

103 udies were performed under a range of salts (potassium chloride, potassium phosphate, potassium aceta

104 Brief exposure to 10 mM potassium chloride produced epileptiform bursting and po

105 Elevation of extracellular potassium chloride resulted in spontaneous asynchronous

106 Potassium chloride showed similar impact on lipid oxidat

107 The tolerance limits for sodium chloride, potassium chloride, sodium acetate, sodium fluoride, sod

108 artially denaturing TGGE is carried out with potassium chloride, sodium chloride, or magnesium chlori

109 rontium acetate/sodium fluoride (SrAc2F) and potassium chloride/sodium monofluorophosphate (KCl/MFP)

110 dissolution kinetics of ZnO NPs in buffered potassium chloride solution using anodic stripping volta

111 M phosphate buffer (pH 7.0) containing 0.1 M potassium chloride solution, and a flow rate of 0.8 ml m

112 Potassium chloride-stimulated NO formation was also enha

113 Potassium chloride supplementation of the Sulfolobus ass

114 find that trehalose has a specific impact on potassium chloride that is unlike that of other sugars o

115 ements for growth: 9 amino acids, sodium and potassium chloride, thiamine, iron, zinc, magnesium, hyp

116 of epileptiform activity induced by elevated potassium chloride to induce multiple forms of LTP in ar

117 episodes were induced by application of 3 M potassium chloride to the cortex of adult anesthetized r

118 ter in the kidney loop of Henle and the KCC2 potassium-chloride transporter in neuronal membranes, wo

119 de and ethacrynic acid, which block the KCC1 potassium-chloride transporter in the kidney loop of Hen

120 lobal ischemia caused an upregulation of the potassium-chloride transporter KCC2 in neurons which per

121 ression of active sodium transporters-sodium/potassium/chloride transporter type 2 (NKCC2), sodium/ch

122 dly induced by sodium chloride, drought, and potassium chloride treatments.

123 Perfusion of low potassium chloride Tyrode solution plus quinidine led to

124 ute, and the latency, Km, and sensitivity to potassium chloride were also similar.