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1 e in potassium absorption induced by dietary potassium depletion.
2 between superficial and deep nephrons during potassium depletion.
3 rocessing with phenylarsine oxide or chronic potassium depletion.
4 l membrane is increased in hypertonicity and potassium depletion.
5 lock endocytosis, namely, chlorpromazine and potassium depletion.
6 hand, consistent with the in vitro data both potassium depletion and hypertonic sucrose, which have b
7                              We suggest that potassium depletion and hyperuricemia in rats exacerbate
8 ctose diets that are given thiazides exhibit potassium depletion and hyperuricemia.
9                                              Potassium depletion but not sodium depletion resulted in
10 This KCC1 mRNA is substantially increased by potassium depletion but only minimally by sodium depleti
11                                              Potassium depletion by thiazide diuretics is associated
12 ractional excretion of lithium (FELi) during potassium depletion, free-flow micropuncture was perform
13  blocked by hypertonic sucrose as well as by potassium depletion (inhibitors of clathrin-dependent en
14                                              Potassium depletion (KD) causes renal chloride wasting,
15 RNA abundance and KCC1 protein expression in potassium depletion of the rat colonic basolateral membr
16 drial function occurred before intracellular potassium depletion or reduced cell viability occurred.
17 hibition of Na,K-ATPase either by ouabain or potassium depletion prevented the formation of tight jun
18              Both dietary sodium and dietary potassium depletion substantially increase active potass
19 subunit mRNA and protein expression, whereas potassium depletion up-regulates H,K-ATPase beta-subunit
20                                              Potassium depletion, which decreased phagocytosis by >90
21 ysis in this system is resistant to cellular potassium depletion, which further distinguishes this pr

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