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1 e in potassium absorption induced by dietary potassium depletion.
2 between superficial and deep nephrons during potassium depletion.
3 rocessing with phenylarsine oxide or chronic potassium depletion.
4 l membrane is increased in hypertonicity and potassium depletion.
5 lock endocytosis, namely, chlorpromazine and potassium depletion.
6 hand, consistent with the in vitro data both potassium depletion and hypertonic sucrose, which have b
10 This KCC1 mRNA is substantially increased by potassium depletion but only minimally by sodium depleti
12 ractional excretion of lithium (FELi) during potassium depletion, free-flow micropuncture was perform
13 blocked by hypertonic sucrose as well as by potassium depletion (inhibitors of clathrin-dependent en
15 RNA abundance and KCC1 protein expression in potassium depletion of the rat colonic basolateral membr
16 drial function occurred before intracellular potassium depletion or reduced cell viability occurred.
17 hibition of Na,K-ATPase either by ouabain or potassium depletion prevented the formation of tight jun
19 subunit mRNA and protein expression, whereas potassium depletion up-regulates H,K-ATPase beta-subunit
21 ysis in this system is resistant to cellular potassium depletion, which further distinguishes this pr
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