ライフサイエンスコーパス: potassium ion

1 binding between the substrate camphor and a potassium ion.

2 a significantly different position than the potassium ion.

3 e third phosphonate oxygen, which attracts a potassium ion.

4 rt of sodium ions and counter-transport of a potassium ion.

5 d one proton and the countertransport of one potassium ion.

6 formance for the insertion and extraction of potassium ions.

7 of L-asparagine, D-glucose, D-fructose, and potassium ions.

8 roducibility was achieved for the sensing of potassium ions.

9 oligonucleotide d(T2AG3)4 in the presence of potassium ions.

10 stabilized by physiological concentration of potassium ions.

11 t pH 5.5 the predominant conductance was for potassium ions.

12 use of compensatory changes in extracellular potassium ions.

13 inhibited by valinomycin in the presence of potassium ions.

14 that this current was carried principally by potassium ions.

15 tranded helices in the presence of sodium or potassium ions.

16 ce environment such as a propagating wave of potassium ions.

17 of the parallel GQ core where it aligns with potassium ions.

18 anines called G quartets, in the presence of potassium ions.

19 onium ions than those that contain sodium or potassium ions.

20 ization and, as a consequence, the efflux of potassium ions.

21 cell into the gastric lumen in exchange for potassium ions.

22 ells and their afferent neurons to show that potassium ions accumulating in the synaptic cleft modula

23 with 2.9 nS conductance for the transport of potassium ions across a phospholipid bilayer.

24 ensitive potassium (K(ATP)) channels conduct potassium ions across cell membranes and thereby couple

25 smembrane protein that transports sodium and potassium ions across cell membranes during an activity

26 from ATP hydrolysis to transport sodium and potassium ions across cell membranes in opposite directi

27 as natural molecular nanomachines, transport potassium ions across the plasma membrane of the cell.

28 ATPase asymmetrically distributes sodium and potassium ions across the plasma membrane to generate an

29 for the controlled and selective passage of potassium ions across the plasma membrane via a conserve

30 to potassium accumulation, and suggest that potassium ion action on HCN channels can modulate neurot

31 In this study, brain extracellular potassium ion activity and local cerebral blood flow wer

32 egree in hyperthermic animals, extracellular potassium ion activity showed delayed secondary elevatio

33 No secondary elevation of extracellular potassium ion activity was observed in hypothermic anima

34 he time required to refill the membrane with potassium ions after the ions are swept out of the membr

35 ser fluence on the signal intensities of the potassium ion and cationized TBP was also studied.

36 e acridine ring appears to act as a "pseudo" potassium ion and is positioned above the centre of the

37 rtially compensated for by the presence of a potassium ion and its accompanying coordination sphere.

38 riven proton translocation was stimulated by potassium ions and inhibited by KF, by the pyrophosphate

39 riven proton translocation was stimulated by potassium ions and inhibited by the PP(i) analog aminome

40 olated mass-dense granules was stimulated by potassium ions and inhibited by the pyrophosphate analog

41 lude ATP, ADP, nonfolded protein substrates, potassium ions, and GroES (in the case of GroEL).

42 'cells' between mica sheets are filled with potassium ions, and they provide an environment in which

43 arget molecule such as ATP, HIV, ochratoxin, potassium ions, and thrombin can be developed.

44 Evidence is also presented that potassium ions are indispensable for the cross-talk betw

45 Because potassium ions are potent metabolic vasodilators in the

46 support of partial substitution by sodium or potassium ions are reproduced with the present crystals,

47 druplex structures formed in the presence of potassium ions are significantly more active than those

48 Potassium ions are vital for maintaining functionality o

49 o complexes of similar formula with an added potassium ion, [(ArO)(2)(THF)Dy](2)(mu-eta(2):eta(2)-N(2

50 a phospholipid membrane to the permeation of potassium ions as compared to chloride ions: Potassium i

51 electrochemical energy storage devices using potassium-ions as charge carriers are attractive due to

52 actate) and electrolytes (such as sodium and potassium ions), as well as the skin temperature (to cal

53 r both endowing an exquisite selectivity for potassium ions, as well as for controlling the flow of i

54 s a high-symmetry molecular box containing a potassium ion at its interior.

55 Binding of potassium ion at this site rigidifies the interface and

56 ally relevant dynamic range, (2) response to potassium ions at a physiological ionic strength, and (3

57 replacement of hydrogen ions with sodium or potassium ions at multiple sites along the phosphate bac

58 pumps that expel sodium ions in exchange for potassium ions; (b) that the pump derives energy from th

59 Potassium-ion batteries (PIBs) are interesting as one of

60 ent cations were required for activity, with potassium ions being the most effective.

61 potassium ions as compared to chloride ions: Potassium ions, being larger than sodium ions, interact

62 ecule is stabilized by the coordination of a potassium ion between the two stacked quartets.

63 Potassium ion binding stimulates turnover at physiologic

64 Finally, we observed a potassium ion bound to the cytoplasmic domain for this c

65 between loop 1 and loop 3 in the presence of potassium ions but not sodium ions.

66 le to form triple helices in the presence of potassium ions, but required low pH.

67 Here we demonstrate that potassium ion can act as a modulator of Kex2 activity wi

68 otentially encountering compartments high in potassium ion caused by the action of antiporters such a

69 difficult because the large ionic radius of potassium-ions causes structural distortion and instabil

70 s the bilayer: namely, upon removing ambient potassium ions, changes are seen in the NMR shifts of ca

71 idine (4-AP), a well known voltage-sensitive potassium ion channel (K(v)) blocker.

72 and signaling of an endogenous ATP-sensitive potassium ion channel (KATP) in HepG2C3A, a hepatocellul

73 genes (IFITM1, IFIT1, MXI, and GIP3), and a potassium ion channel (KCNJ1) were up-regulated in all t

74 t had a gene-targeted deletion of the Shaker potassium ion channel (Kv1.3) to elucidate how activity

75 smallest known protein to form a functional potassium ion channel and basically corresponds to the "

76 PLLP functions as a voltage-dependent potassium ion channel and is expressed primarily in kidn

77 ed property of not inhibiting the human hERG potassium ion channel at concentrations at which the FID

78 sfunction and injury; however, sodium and/or potassium ion channel dysfunction at the node of Ranvier

79 e of a complex containing a toxin bound to a potassium ion channel has been solved for the first time

80 a helianthus that inhibits the voltage-gated potassium ion channel Kv1.3, to effectively discover cri

81 w, we examine the link between voltage-gated potassium ion channel pharmacology and the biophysics of

82 molecule ligands and a nanodisc-encapsulated potassium ion channel protein, KcsA-Kv1.3.

83 nthase, GDP-D-mannose 4,6 dehydratase, and a potassium ion channel protein.

84 Using a microfluidic approach, we find that potassium ion channel-mediated electrical signaling gene

85 and signaling of an endogenous ATP-sensitive potassium ion channel.

86 Human sperm cells rely on an unusual type of potassium ion channel.

87 eceptor and human ether-a-go-go-related gene potassium ion channel.

88 e metal in each case almost in line with the potassium ion channel.

89 annels, members of the two-pore domain K(+) (potassium ion) channel family K2P, are expressed almost

90 tial molecule for the proper localization of potassium ion channels at presynaptic nerve terminals, w

91 ny details about the workings of a family of potassium ion channels called GIRK channels.

92 because of decreased availability of A-type potassium ion channels due to transcriptional (loss of c

93 o be a very high density of transient A-type potassium ion channels in dendrites of hippocampal CA1 p

94 cancer cells, and inhibit calcium-dependent potassium ion channels indicate that triphenylmethyl-con

95 on with voltage-gated sodium channels or the potassium ion channels Kv1.1 and Kv1.5 and are thus not

96 Calcium-activated potassium ion channels SK and IK (small and intermediate

97 KCNQ channels belong to a family of potassium ion channels with crucial roles in physiology

98 : KVLQT1, HERG, and Min K encode for cardiac potassium ion channels, and SCN5A encodes for the cardia

99 channels are voltage-gated, noninactivating potassium ion channels, and their down-regulation has be

100 es KV3.1, a subunit of the KV3 voltage-gated potassium ion channels, which are major determinants of

101 underlies the structural basis for gating in potassium ion channels.

102 voltage-dependent calcium and ATP-activated potassium ion channels.

103 5) Ipc and SLu contain specialized fast potassium ion channels.

104 x interplay between voltage-gated sodium and potassium ion channels.

105 s belong to the superfamily of voltage-gated potassium ion channels.

106 re tetrameric like the related voltage-gated potassium ion channels; the order of subunits affects th

107 thium ion complexation relative to sodium or potassium ion complexation.

108 o with normal Tyrode solution (extracellular potassium ion concentration 4 mmol/liter) and were studi

109 An increase of extracellular potassium ion concentration can result in neuronal hyper

110 transient variations of local extracellular potassium ion concentration in the central nervous syste

111 shifts of key residues in the filter as the potassium ion concentration is changed from 50 mM to 1 m

112 ischemia and hyperkalemia, the extracellular potassium ion concentration is elevated.

113 These biochemical data indicate that potassium ion concentration may function as a regulator

114 conductance were both dependent on external potassium ion concentration.

115 ts that were transitory-ie, increased plasma potassium ion concentrations in one patient and a transi

116 milarities and differences in the sodium and potassium ion condensation around DNA, we carried out a

117 ao directly associates with the slow outward potassium ion current (I(Ks)) and recruits both PKA and

118 activation, which increases the slow outward potassium ion current (IKS).

119 In contrast, the permeability to protons and potassium ions decreased sharply by two orders of magnit

120 ependent conformational shifts, and all show potassium ion dependence.

121 Potassium ions diffuse across the cell membrane in a sin

122 he triethylene glycol side chains with added potassium ions drives the formation of helical nanowires

123 r, also provides the critical exit route for potassium ions during neuronal apoptosis via p38 MAPK-de

124 hoenzyme is associated with an influx of two potassium ions; (e) that each half of the working cycle

125 n shown that known Nlrp3 stimuli converge on potassium ion efflux upstream of Nlrp3 activation, the e

126 In this sensor design, a potassium ion-exchanged BK7 glass waveguide was over-coa

127 ecular hydrogen bonding by wrapping around a potassium ion exclusively.

128 The blockers did so by cutting off potassium ion flow to a site in the pore, which then emp

129 In primary macrophages, potassium ion flux and the membrane channel pannexin 1 h

130 (3) Substituting potassium ions for sodium slightly decreased hairpin sta

131 embranes are ideally suited for re-capturing potassium ions from the TTS lumen during, and immediatel

132 G protein-gated inwardly rectifying potassium ion (GIRK/Kir3) channels, which mediate the po

133 exploits preexisting sodium-, chloride-, and potassium ion gradients to catalyze the thermodynamicall

134 In addition, a potassium ion has been located at the dimer interface.

135 2 protein kinase are required for sodium and potassium ion homeostasis and salt tolerance in Arabidop

136 The data suggest that loss of potassium ion homeostasis during re-perfusion after foca

137 dicating that delayed deterioration of brain potassium ion homeostasis was not caused by temperature

138 When presented with low concentrations of potassium ions in a buffer that mimics the composition o

139 erties of electrode materials by focusing on potassium ions in alpha-MnO2.

140 tility for simultaneously sensing sodium and potassium ions in aqueous solutions, human whole blood s

141 ports three sodium ions and then imports two potassium ions in each transport cycle.

142 The potassium ions in leucite are exchangeable for rubidium

143 Unlike potassium ions in potassium channels, the sodium ions in

144 ity with experimental results for sodium and potassium ions in propylene carbonate by obtaining over

145 The results for protons and potassium ions in shorter-chain lipids are consistent wi

146 e G-quartets and an uninterrupted run of six potassium ions in the central channel of the quadruplex.

147 distinct from but in addition to the role of potassium ions in the ion channel at the centre of all q

148 al structure is generated by the presence of potassium ions in the precursor solution within the chan

149 al zinc phthalocyanine dimer, was formed via potassium ion induced dimerization of 4,5,4',5',4'', 5''

150 o determine the kinetics associated with the potassium ion-induced hairpin-to-G4 transition, which is

151 es of the ExoIX:DNA complexes show that this potassium ion interacts directly with a phosphate dieste

152 te battery, which relies on the insertion of potassium ions into a copper hexacyanoferrate cathode an

153 we show that necrosis releases intracellular potassium ions into the extracellular fluid of mouse and

154 inding site in the C-terminal domain where a potassium ion is directly coordinated by five main chain

155 What is more, a potassium ion is found to be able to permeate a membrane

156 Notably, the potassium ion is one of the most abundant cations in bio

157 ed, and a mechanism of activation of Kex2 by potassium ion is proposed.

158 each glutamate ion into the cell, while one potassium ion is transported out of the cell.

159 The third metal ion, most likely a potassium ion, is involved in substrate recognition thro

160 sducer in homogeneous aptasensing system for potassium ion (K(+)) assay in aqueous media.

161 f colonization are dependent on the level of potassium ion (K(+)) but independent of flagella, as ver

162 oduct of arachidonic acid metabolism and the potassium ion (K(+)) have been identified as endothelium

163 TATA spacer relative to ATAT showed that in potassium ion (K(+)) the E2 affinity of the two sequence

164 In the CNS, potassium ion (K+) buffering is dependent on the glia-sp

165 s essential for selectivity in voltage-gated potassium ion (K+) channel pores.

166 ystal structure of a mammalian Shaker family potassium ion (K+) channel.

167 Voltage-dependent potassium ion (K+) channels (Kv channels) conduct K+ ion

168 ygenase cytochrome P450cam (CYP101) requires potassium ion (K+) to drive formation of the characteris

169 Potassium ions (K(+)) are the most abundant cations in p

170 In the presence of sodium ions and no potassium ions, LJM-3064 adopts an antiparallel-stranded

171 ut also the activation of apoptosis, whereas potassium ion loss controls the progression of the cell

172 says that at physiological concentrations of potassium ions NHEIII(1) folds into two coexisting G4 DN

173 channel structure and on the position of the potassium ions occupying the selectivity filter.

174 The effects of potassium ion on the nested allostery of GroEL are due t

175 n with an elevated concentration of external potassium ions on the expression of Kv3.1 channel subuni

176 Each quadruplex has five potassium ions organised in a linear channel, with squar

177 during the cardiac action potential, passing potassium ions outward to repolarize ventricular myocyte

178 ements, NMCCs are slightly more permeable to potassium ions over sodium (PK/PNa = 2.68 +/- 0.21) and

179 sent an optode-based nanosensor selective to potassium ions, owing to the addition of a pH-sensitive

180 tential, 3 muM PD-307243 increased the total potassium ions passed through hERG channels by 8.8 +/- 1

181 nly one trace sodium ion per 160 bp, and one potassium ion per 41 bp.

182 f varying flow velocity upon permeability to potassium ions (PK) of single perfused mesenteric microv

183 rying flow velocity (U) upon permeability to potassium ions (PK) of single perfused mesenteric venule

184 vely low laser fluence, necessary to produce potassium ions, prevents the excessive fragmentation of

185 diffusion of local extracellular calcium and potassium ions, prolonging action-potential duration and

186 se with adenosylcobalamin (coenzyme B12) and potassium ion reacts with molecular oxygen in the absenc

187 Potassium ion recycling is normal in these mutants, but

188 ts in rapid host membrane depolarization and potassium ion release.

189 ese quadruplex structures indicates that the potassium ions required for chair type structures intera

190 ridges one of the phosphonate oxygens with a potassium ion, resulted in insoluble protein.

191 with one or two T4 loops in the presence of potassium ions reveal that sequences with longer loops d

192 loroviruses is that they code for functional potassium ion-selective channel proteins (Kcv) that are

193 In a first example, a potassium ion-selective electrode acts as the reference

194 e electrodes, with and without an additional potassium ion-selective membrane (ISM) coating, followin

195 ed region of the protein is known to chelate potassium ions selectively.

196 Measurements with potassium ion-sensitive electrodes revealed that the net

197 crystal structure also reveals that a single potassium ion stabilizes the K loop; bound potassium is,

198 Potassium ion stimulated surfactin secretion, and the ro

199 No evidence exists for partial sodium or potassium ion substitution for solvent water molecules w

200 the porous silica template; the size of the potassium ion templates the microporosity of alpha-MnO2,

201 mic exchange can be restarted by addition of potassium ions that competitively bind 18-crown-6, thus

202 dedicated to regulated background leakage of potassium ions that serve to control neuronal excitabili

203 nd an outward current carried exclusively by potassium ions that was reduced by 1 mM 4-aminopyridine

204 complexed with the biologically significant potassium ion, the only conformers found to form under t

205 If it is deprived of potassium ions, the Na+/K+ pump is restricted to sodium

206 In the presence of potassium ions, the oligonucleotides that contain at lea

207 get the filter that controls the movement of potassium ions through these channels.

208 Potassium ion titration studies reveal that the site-spe

209 menal potassium, which enabled the influx of potassium ions to depolarize the VNO neurons in vivo.

210 narrowest segment of the pore, permits only potassium ions to diffuse through the pore.

211 Fluctuations allow water, then potassium ions, to reenter the pore; linker-S6 repacking

212 itive Na/K pump, and thus favored passage of potassium ions towards the lumen while preventing K+ ext

213 mission, central nervous system development, potassium ion transport, protein dephosphorylation, and

214 involved in signaling, lipid metabolism, and potassium ion transport.

215 Unexpectedly, potassium ions traversing the channel from the inside co

216 ary structure is assembled in the absence of potassium ion under the experimental conditions.

217 the orthoclase surface to release a surface potassium ion upon uranyl adsorption.

218 ortant influence on the selective passage of potassium ions via the K(+) channel pore.

219 ray crystal structure containing this RNA, a potassium ion was found to be contacted by six oxygen at

220 ed by profuse surface colonies if sufficient potassium ion was present.

221 ability of phospholipid bilayers to protons, potassium ions, water, urea, and glycerol.

222 ochemical microscopy (SECM) for detection of potassium ion were fabricated.

223 Paper-strip ISEs for cadmium, silver, and potassium ions were developed with groundbreaking limits

224 ctures of complexes of FTHFS with cesium and potassium ions were examined and monovalent cation bindi

225 SK channels allow efflux of potassium ions when intracellular calcium increases and

226 y of 1 depends strongly on the proportion of potassium ions, which interfere through host-guest excha

227 llel quadruplex structure in the presence of potassium ions, while earlier NMR results in the presenc

228 e neuroendocrine homolog PC2 is inhibited by potassium ion with all substrates examined.

229 lly allows the replacement of an active site potassium ion with the epsilon-amino nitrogen.

230 However, the accommodation of potassium-ions with satisfactory capacity and cyclabilit

231 vior of Kex2 is also altered upon binding of potassium ion, with opposite effects on acylation and de

232 The diffusion coefficient of a potassium ion within the channel is also calculated usin

233 lete Escherichia coli KdpFABC complex with a potassium ion within the selectivity filter of KdpA and