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1 ransport in the erythrocyte is attributed to potassium chloride cotransporter 1 (KCC1).
2 vity of a bumetanide-sensitive NKCC1 (sodium potassium chloride cotransporter 1)-like chloride cotran
3 t CA1 pyramidal neurons because of increased potassium chloride cotransporter 2 (KCC2) expression and
4 ride shift caused by significant decrease in potassium chloride cotransporter 2 (Kcc2) mRNA expressio
5 sitive hypotension, with depletion of sodium potassium chloride cotransporter 2 and aquaporin 2.
6 y gamma-aminobutyric acid and glycine due to potassium chloride cotransporter-2 (KCC2) down-regulatio
7                            Disruption of the potassium/chloride cotransporter 3 (KCC3), encoded by th
8 s of Tbx1 or Slc12a2, which encodes a sodium-potassium-chloride cotransporter and is also necessary f
9 s associated with reduced dorsal spinal cord potassium chloride cotransporter expression and impaired
10 ction is downregulated, including the sodium-potassium-chloride cotransporter gene nkcc1 (slc12a2) an
11                Here, we show that the Sodium-Potassium-Chloride Cotransporter Isoform-1 (NKCC1) provi
12                       This study asks if the potassium-chloride cotransporter (K:Cl) and the calcium-
13 aying behavior and discovered mutations in a potassium chloride cotransporter, kcc-2.
14                                          The potassium chloride cotransporter KCC2 plays a major role
15 nds on chloride gradients established by the potassium chloride cotransporter KCC2.
16 w that specific markers of excitability, the potassium-chloride cotransporter KCC2 and GABAA receptor
17 n associated with dysfunction or loss of the potassium-chloride cotransporter KCC2 in a subset of pyr
18 decrease in the functional expression of the potassium-chloride cotransporter KCC2 in spinal cord dor
19      Once in the cortex, upregulation of the potassium-chloride cotransporter KCC2 is both necessary
20 ion ([Cl(-)](i)), which is maintained by the potassium-chloride cotransporter KCC2.
21 s is largely mediated by the neuron-specific potassium-chloride cotransporter KCC2.
22                     The furosemide-sensitive potassium-chloride cotransporter (KCC2) plays an importa
23 (hepatocyte nuclear factor 4alpha), SLC12A5 (potassium-chloride cotransporter member 5), CDH22 (cadhe
24  receptors can physically associate with the potassium-chloride cotransporter protein, KCC2, which se
25 novel association between the GABABR and the potassium-chloride cotransporter protein, KCC2.

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