ライフサイエンスコーパス: potentiation

1 on of dopamine converts this depression into potentiation.

2 e in diheteromeric receptors is required for potentiation.

3 high-conductance state, leading to synaptic potentiation.

4 g routes to the temporal profile of synaptic potentiation.

5 lpha4 subunit interface, did not reduce CMPI potentiation.

6 ation potentiation was stronger than pursuit potentiation.

7 ysiologic deficits such as reduced long-term potentiation.

8 (Delta109-116)), supports normal DAG-induced potentiation.

9 of persistent spines, and impaired long-term potentiation.

10 accessory optic system severely impairs OKR potentiation.

11 tion and excitation in hippocampal long-term potentiation.

12 receptor antagonists attenuated serotonin's potentiation.

13 gomers on synapses and hippocampal long-term potentiation.

14 ha4 subunit extracellular interface in nAChR potentiation.

15 may also lead to alternate outcomes, such as potentiation.

16 izures and interictal spikes induce synaptic potentiation.

17 AD had impaired DLPFC plasticity (mean [SD] potentiation, 1.18 [0.25]) compared with controls (mean

18 18 [0.25]) compared with controls (mean [SD] potentiation, 1.40 [0.35]; F1,44 = 5.90; P = .02; betwee

19 Here we show that firing rate potentiation, a form of intrinsic plasticity mediated by

20 ynapse maturation and abolishes post-tetanic potentiation, a form of synaptic plasticity.

21 nvironment but also caused loss of long-term potentiation after theta-burst stimulation.

22 ocampal subfields exhibit impaired long-term potentiation, an electrophysiological correlate of learn

23 4A5 in hippocampal neurons impairs long-term potentiation and attenuates the formation of hippocampus

24 Spike timing-dependent long-term potentiation and depression (st-LTP and st-LTD) were con

25 t the mossy fiber-granule cell synapse, with potentiation and depression symmetrically distributed wi

26 primarily for generating long-term synaptic potentiation and depression, AMPARs are the main fast tr

27 nsible for synaptic vesicle cycle, long-term potentiation and depression, and neurotrophin and retrog

28 emonstrated over a million (2(20)) epochs of potentiation and depression, suggesting that our devices

29 ity genes that are both induced by long-term potentiation and downregulated in the aged brain.

30 with IQ and are thought to promote long-term potentiation and enhance memory consolidation.

31 Ca(2+)-dependent potentiation and inactivation of TRPA1 were selectively

32 le calpain-2 activation limits the extent of potentiation and is neurodegenerative.

33 will be a key target for modulating synaptic potentiation and learning.

34 uctural plasticity during chemical long-term potentiation and long-term depression.

35 ents the stabilization of synaptic long-term potentiation and markedly impairs long-term fear and obj

36 balance throughout learning to avoid runaway potentiation and memory interference.

37 d postsynaptic structural defects, long-term potentiation and miniature postsynaptic current defects.

38 ignal, broadens the time window for synaptic potentiation and modulates the outcome of hippocampal ST

39 ufficient to recapitulate transient synaptic potentiation and reinstate cocaine seeking.

40 he sequence of events that mediates synaptic potentiation and reinstated cocaine seeking induced by c

41 form, is important for maintaining long-term potentiation and storing memory.

42 ed expression of genes involved in long-term potentiation and synaptic transmission, cancer and neuro

43 the inhibition seen with alpha2 subunits to potentiation and the potentiation seen with alpha4 subun

44 ity in the indirect pathway toward long-term potentiation (and possibly also through more complex mec

45 currents prevents the induction of long-term potentiation, and also interferes with long-lasting pote

46 spinous synapses, was incapable of long-term potentiation, and less effectively patterned STN activit

47 aptic excitability, occlusion of firing rate potentiation, and reductions in BK currents in vestibula

48 ctivated T cells (NFAT) signaling, long-term potentiation, and responsiveness to adrenergic stimulati

49 ng self-administration in rats and that this potentiation appears to involve corticosterone-induced b

50 lices with TIMP2 antibody prevents long-term potentiation, arguing for previously unknown roles for T

51 tent of modulation and kinetic properties of potentiation as determined in previous single-channel st

52 ent stresses resulting from loss of synaptic potentiation associated with disrupted structure of syna

53 y, only patients' NMDAR-Ab prevent long-term potentiation at glutamatergic synapses, while leaving NM

54 ansmission and decreased levels of long-term potentiation at hippocampal Schaffer collateral-CA1 syna

55 presynaptically expressed form of long-term potentiation at mossy cell outputs, shedding light on th

56 drenoceptors promoted STD long-term synaptic potentiation at mouse hippocampal excitatory synapses by

57 sion and blunted depotentiation of long-term potentiation at the Schaffer collateral/cornu ammonis 1

58 OXTR signaling in the induction of long-term potentiation at the synapses between the entorhinal cort

59 the nucleus accumbens, as well as increased potentiation at these synapses as measured by AMPA/N-met

60 We established that CICR potentiation begins in vivo.

61 layed a defect in the induction of long-term potentiation, but not long-term depression, at the synap

62 s at alpha4Gln-124 and alpha4Thr-126 reduced potentiation by CMPI and NS9283, indicating that their b

63 66 were critical for (alpha4)3(beta2)2 nAChR potentiation by CMPI, but not by NS9283, whereas amino a

64 e to Rett-like encephalopathy and that their potentiation by D-serine treatment may underlie the asso

65 ccompanied by an increased susceptibility to potentiation by fewer pre-post spike pairs, indicating a

66 responses at the single-cell level and their potentiation by IL-33.

67 fibrosis transmembrane conductance regulator potentiation by ivacaftor is a novel therapeutic approac

68 ctance, brief mean open lifetime, and strong potentiation by NS-9283, whereas receptors with the comp

69 ls have more recently proposed that synaptic potentiation can occur by the recruitment of additional

70 e both the depression component (Dc-ODP) and potentiation component (Pc-ODP) of plasticity independen

71 rs CREB, SRF, and MEF2 in the depression and potentiation components of ODP in vivo, therefore better

72 2-Aminoethoxydiphenyl borate (2-APB) elicits potentiation current (Ip) on Ca(2+) release-activated Ca

73 mitantly rescues their hippocampal long-term potentiation deficit.

74 indicated that the presence of inhibition or potentiation depended on the source of the extracellular

75 The potentiation depends on the transport of AMPH into the c

76 ntrolling synaptic strength during long-term potentiation/depression and homeostatic scaling.

77 city with heterogeneity, including long-term potentiation/depression and spike-timing-dependent plast

78 l synapses: unilateral connection, long-term potentiation/depression, a spike-timing-dependent plasti

79 wave activity during sleep reflects synaptic potentiation during wake, and that its homeostatic decre

80 Another possibility is net synaptic potentiation during wake: stronger coupling among neuron

81 escence analysis of single-synapse long-term potentiation (FASS-LTP).

82 ceptors counteracted unpaired stratum oriens potentiation following pairing in stratum radiatum.

83 l-nitrilotriacetic acid (C18-NTA)) increased potentiation for low Co(2+) concentrations, indicating t

84 Thus, alpha7 potentiation has emerged as a therapeutic strategy for s

85 7 occludes NMDAR antagonist-induced synaptic potentiation in an intact circuit, confirming the role o

86 hat these connections may underpin long-term potentiation in M1, our findings may lead to novel thera

87 channels were involved in mediating synaptic potentiation in oriens, whereas NMDA and adenosine recep

88 lasticity to facilitate associative synaptic potentiation in prefrontal excitatory circuits.

89 Mice exposed to cNIC exhibited long-term potentiation in response to high-frequency stimulation i

90 nsmission in the dentate gyrus and long-term potentiation in the CA1 region of the hippocampus.

91 hysiological level, TBI suppressed long-term potentiation in the hippocampus, which was fully restore

92 euronal plasticity by facilitating long-term potentiation in the hippocampus.

93 receptor-dependent facilitation of long-term potentiation in the IL-PFC.

94 y of LTD without affecting that of long-term potentiation in the insular cortex.

95 ct recognition memory and impaired long-term potentiation in the prefrontal cortex.

96 ration, similar to cocaine-induced long-term potentiation in the VTA.

97 ype current blockade also prevented synaptic potentiation induced by postsynaptic action potential tr

98 ation, and also interferes with long-lasting potentiation induced either by postsynaptic trains of ac

99 to a complete block of subsequent long-term potentiation induction and a facilitation of long-term d

100 NRG3 facilitated the conversion of long-term potentiation into long-term depression at cortical layer

101 the hypothesis that fast Hebbian short-term potentiation is a key WM mechanism.

102 synaptic plasticity (associative short-term potentiation) is a possible mechanism for WM encoding an

103 of synaptic strength necessary for long-term potentiation, learning, and memory.

104 , APLP1, APLP2), develop defective long-term potentiation (LTP) and aged mice display spatial learnin

105 Long-term potentiation (LTP) and depression (LTD) at glutamatergic

106 Both long-term potentiation (LTP) and depression (LTD) of excitatory sy

107 n is required for the induction of long-term potentiation (LTP) and is generally neuroprotective, whi

108 At glutamatergic synapses, both long-term potentiation (LTP) and long-term depression (LTD) can be

109 ort such bidirectional changes are long-term potentiation (LTP) and long-term depression (LTD) forms

110 sufficient for the maintenance of long-term potentiation (LTP) and long-term memory (LTM).

111 rations of Abeta are necessary for long-term potentiation (LTP) and memory.

112 ic protein CaMKII is necessary for long-term potentiation (LTP) and memory.

113 healthy brain, enhance hippocampal long-term potentiation (LTP) and memory.

114 ICANCE STATEMENT: Various types of long-term potentiation (LTP) are correlated with distinct phases o

115 y, and synaptic changes induced by long-term potentiation (LTP) are thought to underlie memory format

116 ed short-term plasticity (STP) and long-term potentiation (LTP) at perforant path-DG synapses in naiv

117 tic deletion of Calstabin2 reduced long-term potentiation (LTP) at the hippocampal CA3-CA1 connection

118 Long-term potentiation (LTP) at the Schaffer collateral-commissura

119 d and the resulting suppression of long term potentiation (LTP) by Abeta oligomers was prevented.

120 Remarkably, long-term potentiation (LTP) can override the masking effect of th

121 ducibility of associative synaptic long-term potentiation (LTP) due to saturation after sleep depriva

122 1 is required for the induction of long-term potentiation (LTP) elicited by theta-burst stimulation i

123 The synapse specificity of long-term potentiation (LTP) ensures that no interference arises f

124 hat TBI inhibits the expression of long-term potentiation (LTP) evoked by high-frequency stimulation

125 Since the discovery of long-term potentiation (LTP) in 1973, thousands of papers have bee

126 Rs) enhances both the induction of long-term potentiation (LTP) in hippocampal CA1 pyramidal cells an

127 BSTRACT: NMDA receptor independent long-term potentiation (LTP) in hippocampal stratum oriens-alveus

128 of corticostriatal fibres produces long-term potentiation (LTP) in striatal projection neurons when m

129 mpletely suppressed development of long-term potentiation (LTP) in the CA1-subiculum, but not in the

130 mpletely suppressed development of long-term potentiation (LTP) in the CA1-subiculum, but not in the

131 t long-term depression switches to long-term potentiation (LTP) in the former, timing-dependent LTP i

132 physiology experiments showed that long-term potentiation (LTP) in the hippocampus, which is dependen

133 Although long-term potentiation (LTP) in the lateral amygdala (LA) plays an

134 Long-term potentiation (LTP) in the rat hippocampus is the most ex

135 Long-term potentiation (LTP) is a rapid and persistent increase in

136 Long-term potentiation (LTP) is an activity-dependent and persiste

137 Long-term potentiation (LTP) is widely perceived as a memory subst

138 (LFS) to the sciatic nerve induced long-term potentiation (LTP) of C-fiber-evoked potentials, reveali

139 repress the expression of synaptic long-term potentiation (LTP) of C-fiber-evoked potentials.

140 Long-term potentiation (LTP) of excitatory synaptic transmission h

141 addiction indicate that persistent long-term potentiation (LTP) of excitatory synaptic transmission o

142 We found that long-term potentiation (LTP) of mossy fiber input invoked a large

143 ysis of APA following induction of long-term potentiation (LTP) of mouse hippocampal CA3-CA1 synapses

144 Long-term potentiation (LTP) of NMDA receptor (NMDAR)-mediated glu

145 fiber input is known to exhibit a long-term potentiation (LTP) of synaptic efficacy through a combin

146 (LTD) of synaptic transmission but long-term-potentiation (LTP) of synaptic signals in HIL cells.

147 osure, induces glomerulus-specific long-term potentiation (LTP) of synaptic strength selectively at t

148 Surprisingly, we find that long-term potentiation (LTP) of synaptic transmission at the Schaf

149 uronal synapse, phenomena known as long-term potentiation (LTP) or long-term depression (LTD), respec

150 Cortical plasticity mechanisms of long-term potentiation (LTP) or of long-term depression (LTD) were

151 een characterized, their effect on long-term potentiation (LTP) remains unknown.

152 Long-lasting forms of long-term potentiation (LTP) represent one of the major cellular m

153 yl-d-aspartate) receptor-dependent long-term potentiation (LTP) shapes neural circuits and mediates l

154 shroom spines and higher-magnitude long-term potentiation (LTP) than SR synapses.

155 NMDA-receptor-independent form of long-term potentiation (LTP) that requires postsynaptic brain-deri

156 emory and increase the duration of long-term potentiation (LTP), a form of hippocampal synaptic plast

157 icity is a fundamental property of long-term potentiation (LTP), but it is not known if learning is m

158 monstrated exaggerated hippocampal long-term potentiation (LTP), consistent with deficits in hippocam

159 its electrophysiological surrogate long-term potentiation (LTP), effects that may be mediated by intr

160 We show that long-term potentiation (LTP), in the CA1 region of hippocampal sli

161 (NMDA) receptor (NMDAR)-dependent long-term potentiation (LTP), remains unclear.

162 n of letrozole virtually abolished long-term potentiation (LTP), whereas it did not prevent the gener

163 ost likely mediated by fast-acting long-term potentiation (LTP), which relies on the precise timing o

164 oth long-term depression (LTD) and long-term potentiation (LTP).

165 cilitating an anti-Hebbian form of long-term potentiation (LTP).

166 Reelin stimulation of hippocampal long-term potentiation (LTP).

167 nd sufficient for the induction of long-term potentiation (LTP).

168 uired to sustain the late phase of long-term potentiation (LTP).

169 ticity loss evident by a decreased long-term potentiation (LTP).

170 pairments, we examined hippocampal long-term potentiation (LTP).

171 smission and are unable to sustain long-term potentiation (LTP).

172 of Alzheimer's disease (AD), late long-term potentiation (LTP; L-LTP) and its associative plasticity

173 Synaptic plasticity (e.g., long-term potentiation [LTP]) is considered the cellular correlate

174 c and is expressed as an increase (long-term potentiation, LTPGABA) or a decrease (long-term depressi

175 e case for hippocampal presynaptic long-term potentiation (LTPpre), which is expressed as an increase

176 pocampal area CA1 are produced by a synaptic potentiation notably different from Hebbian plasticity.

177 Abeta aggregation in vitro as well as apoE4 potentiation of Abeta cytotoxicity.

178 such as anti-inflammatory interventions and potentiation of adaptive immunity.

179 Rbeta signaling pathways as evidenced by the potentiation of apoptosis induced by dutasteride or fina

180 f this regulon is to limit acetate-dependent potentiation of cell death in staphylococcal populations

181 lines and was due to senescence rather than potentiation of cell death.

182 stolonidiol is responsible for the resulting potentiation of ChAT activity.

183 ing in decreased checkpoint activity and the potentiation of chromosomal instability.

184 We have previously shown that potentiation of CNGA1 channels by transition metals requ

185 molecular mechanism underlies the behavioral potentiation of cocaine self-administration by sigma1R a

186 Plasticity of the DLPFC measured as potentiation of cortical-evoked activity using paired as

187 VDP manifests as a potentiation of current amplitude, hyperpolarizing shift

188 This potentiation of depolarization-induced calcium transient

189 Feedback potentiation of distal dendritic inhibition by CA1 place

190 By contrast, potentiation of endogenous compensatory vasoactive pepti

191 ce preference to cocaine and cocaine-induced potentiation of excitation and reduction of GABAergic in

192 but did not further increase cocaine-induced potentiation of excitation, leading to the restoration o

193 mpal slices resulted in an acute and lasting potentiation of excitatory synaptic responses in CA2 pyr

194 l stratum oriens exhibit a form of long-term potentiation of excitatory transmission that is independ

195 Potentiation of eye acceleration exhibited different pro

196 of ketamine, along with regionally selective potentiation of GABAergic synapses.

197 Moreover, the immediate potentiation of gamma activity after plasticity onset po

198 well as in juveniles, MD caused an immediate potentiation of gamma activity, suggesting a novel mecha

199 ges in single unit activity and a remarkable potentiation of gamma oscillations.

200 Potentiation of glucagon-like peptide-1 (GLP-1) action t

201 uced initiation and maintenance of long-term potentiation of glutamatergic synapses.

202 OFC neurons displayed a persistent long-term potentiation of glutamatergic synaptic transmission foll

203 ntification of 3, which demonstrated ex vivo potentiation of glycine-activated current in mouse dorsa

204 vity-dependent plasticity, exercise-mediated potentiation of hippocampal neurogenesis is also diminis

205 ch it modulates plant physiology through the potentiation of host defense mechanisms still needs furt

206 The PAF potentiation of IL-10 production was dependent on protea

207 terns that contributes to the initiation and potentiation of inflammation in many disease processes.

208 bitory engrams" can form through homeostatic potentiation of inhibition onto postsynaptic cells that

209 uld be produced in vivo in a single trial by potentiation of input that arrived seconds before and af

210 eceptor trafficking for their role in GLP-1R potentiation of insulin secretion in pancreatic beta-cel

211 on, which speeds PIP2 dispersion, attenuated potentiation of KCNQ2/3 currents by SMIT1-mediated myo-i

212 ine, displayed a selective D2R/beta-arrestin potentiation of locomotion.

213 The question whether inhibition or potentiation of mGluR5 could be beneficial depends, amon

214 tages such as temporal, regional, and phasic potentiation of natural signaling, and that of receptor

215 The potentiation of neurite shortening by difopein in G2019S

216 ext of myeloid-specific deletion of Ttp, the potentiation of neutrophil deployment protected mice aga

217 ith this prediction UBP684 displayed greater potentiation of NMDARs with only the GluN1 LBD locked co

218 ound that disruption of PNNs allows synaptic potentiation of normally plasticity-resistant excitatory

219 a key mechanistic step required for morphine potentiation of P2X7R function.

220 y of small-molecule compounds, we found that potentiation of p53 (via inhibition of MDM2) or impairme

221 These data identify potentiation of phasic GABA signalling as a novel therap

222 Production of CRHBP blocks the CRH-induced potentiation of postsynaptic layer 2/3 pyramidal cell ac

223 PKA inactivates ARPP-16 leading to selective potentiation of PP2A signaling.SIGNIFICANCE STATEMENT We

224 Here we found that activity-dependent potentiation of presynaptic voltage-gated calcium channe

225 ing the agonist pair of GABA and propofol or potentiation of responses to a low concentration of GABA

226 surface diffusion markedly impairs synaptic potentiation of Schaffer collaterals and commissural inp

227 phosphorylation is required for H2O2-induced potentiation of sensory behavior and ASH neuron function

228 acellular [K(+)], and KCNQ2 was required for potentiation of SMIT activity by myo-inositol preincubat

229 ynaptic Cortactin is necessary for the rapid potentiation of spontaneous release frequency that takes

230 We also provide evidence that potentiation of ST8SIA2 by 9-cis-retinoic acid and artif

231 er G-proteins, is essential for GPCR-induced potentiation of Syk phosphorylation downstream of CLEC-2

232 sympathetic stimulation of heart cells, and potentiation of synaptic strength in neurons.

233 lockade: memory enhancement and long-lasting potentiation of synaptic transmission in CA1 ex vivo.

234 ndent phosphorylation, abolishes DAG-induced potentiation of synaptic transmission in hippocampal neu

235 lation is an essential step in PKC-dependent potentiation of synaptic transmission, acting downstream

236 oved inhibition of efflux relative to 1, and potentiation of the activity of novobiocin and erythromy

237 E enhances odor responses not through direct potentiation of the afferent signal per se, but rather b

238 The consequent potentiation of the autocrine Wnt/beta-catenin signallin

239 results indicate that in the setting of HF, potentiation of the CB chemoreflex is strongly associate

240 uM with an EC50 value of 159 muM, whereas no potentiation of the glutamate-evoked response was observ

241 Also, the potentiation of the GlyR-mediated tonic current by ethan

242 nducted an analysis of direct activation and potentiation of the human alpha1beta2gamma2L GABAA recep

243 ecision via top-down extrasynaptic aminergic potentiation of the primary osmosensory neurons to incre

244 tations did not significantly affect sensory potentiation of the response.

245 ral responses are mediated by a long-lasting potentiation of the rewarding properties of food and tha

246 pairs of pre- and postsynaptic spikes, with potentiation of the synapse when a presynaptic spike pre

247 Potentiation of the ventilatory and sympathetic drive in

248 ed to excitation of tufted cells at rest and potentiation of their odor responses.

249 clonal diversity and imply that therapeutic potentiation of thymopoiesis might either prevent or rev

250 The "memory-less" potentiation of Treg suppressor function may help avoid

251 enhanced TRPA1 sensitivity and Ca(2+)-evoked potentiation of TRPA1 at low Ca(2+), but inhibited TRPA1

252 y whether those changes result in perceptual potentiation of visual drives on auditory regions of the

253 The potentiation of YAP1 activity by HIF-2alpha was not via

254 rons, basal synaptic transmission, long-term potentiation or expression of LTD.

255 r point mutations could affect the extent of potentiation or inhibition, indicating that a more exten

256 AB2 and KCNIP1, and induces a neuronal hyper-potentiation phenotype in iPSC-derived human cortical ne

257 -frequency stimulation leads to post-tetanic potentiation (PTP) at many types of synapses.

258 Post-tetanic potentiation (PTP) is a widespread form of short-term sy

259 KEY POINTS: Post-tetanic potentiation (PTP) is attributed mainly to an increase i

260 ged Orai1-V102C/A/G mutant channels, and the potentiation ratio was highest on Orai1-V102C with an in

261 bined with PE-induced anti-Hebbian long-term potentiation reported in a previous study could result i

262 (required for Dc-ODP), and CREB in long-term potentiation (required for Pc-ODP).SIGNIFICANCE STATEMEN

263 This cAMP-driven synaptic potentiation requires the activation of both protein kin

264 liorates synapse loss and augments long-term potentiation, resulting in protection of memory.

265 with alpha2 subunits to potentiation and the potentiation seen with alpha4 subunits to inhibition.

266 nd informational considerations suggest that potentiation should occur primarily during wake, when an

267 dendritic spines during structural long-term potentiation (sLTP) in hippocampal CA1 pyramidal neurons

268 three-molecule model of structural long-term potentiation (sLTP) of murine dendritic spines, implicat

269 l plasticity, including structural long-term potentiation (sLTP), which is a correlate of an animal's

270 deprivation and stimulus-selective response potentiation (SRP) resulting from enriched visual experi

271 40) as a contributor to abnormal short-term potentiation (STP), a major form of short-term synaptic

272 of the hippocampal and neocortical synaptic potentiation studies we examined.

273 halamus generates a novel rhythm under GABAA potentiation such as under propofol, its hyperpolarizati

274 tory amino acid transporter 3 (EAAT3) blocks potentiation, suggesting that EAAT3 internalization incr

275 ted with the induction of transient synaptic potentiation (t-SP) at glutamatergic synapses on medium

276 ced drug seeking requires transient synaptic potentiation (t-SP) of cortical glutamatergic synapses o

277 twork resulted in an inhibition of NMDA EPSC potentiation that was rescued by adding extracellular D-

278 ide the depolarization necessary for Hebbian potentiation, these results suggest that activity-depend

279 le mice, to spike-timing-dependent long-term potentiation (tLTP) in DID mice, an effect that was tota

280 ed alpha critically relies on propofol GABAA potentiation to alter the intrinsic spindling mechanisms

281 fficient to confer the anaesthetic-dependent potentiation to the GABA current.

282 This selective potentiation underlines that the alpha4:alpha4 interface

283 osphorylated state and becomes permissive of potentiation upon phosphorylation.

284 exhibit a behavior called voltage-dependent potentiation (VDP), which appears to be a specialization

285 The endomorphin-mediated potentiation was a result of a leftward shift of the act

286 , and not observed in the absence of IP3 IP3 potentiation was also blocked by ryanodine receptor anta

287 Sustained ASIC current potentiation was also observed in neurons pretreated wit

288 ent between the two MA groups, but long-term potentiation was greater in ampakine-treated rats.

289 More importantly, hippocampal CA1 long-term potentiation was markedly impaired in parabiotic wild-ty

290 ticipatory pursuit, suggesting that fixation potentiation was not overridden by certainty of an immin

291 The potentiation was significantly greater in DRG neurons is

292 ong pursuit trials, suggesting that fixation potentiation was stronger than pursuit potentiation.

293 d proinsulin secretory ratios during glucose potentiation were higher in PI-CF, suggesting impaired p

294 agnitude of spike timing-dependent long-term potentiation were significantly higher at muscle afferen

295 (Ca(2+)) plateau potentials produced a large potentiation with an asymmetric seconds-long time course

296 on of these strategies [which we termed "p53 potentiation with checkpoint abrogation" (PPCA)] display

297 fibrosis transmembrane conductance regulator potentiation with ivacaftor.

298 dil, RO 25-6981, and RO 04-5595, inhibit the potentiation without affecting basal-evoked NMDA current

299 e, and a significant disruption of long-term potentiation without alteration of long-term depression,

300 e, OIH and pronociceptive synaptic long-term potentiation without altering antinociception.