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1 s and four nest types (plate, cup, dome, and pouch).
2 tion or neutrophil infiltration into the air pouch.
3 such as neutrophil infiltration into the air pouch.
4 rates of cell proliferation within the wing pouch.
5 ty is down regulated in the notum and distal pouch.
6 t-sided structures, called the left coelomic pouch.
7 owed the presence of globular bodies in this pouch.
8 was considered false positive due to Dacron pouch.
9 ination of oral ectoderm that forms Rathke's pouch.
10 l roles in patterning and growth of Rathke's pouch.
11 Gli2 occurs prior to the closure of Rathke' pouch.
12 s and form ectopic foci surrounding Rathke's pouch.
13 le novelty among vertebrates, the male brood pouch.
14 externally located gills, possibly housed in pouches.
15 tion of Wnt11r and Fgf8a rescues tbx1 mutant pouches.
16 ll micromeres to the left and right coelomic pouches.
17 ation of the more anterior arch arteries and pouches.
18 emaining at high levels in the more anterior pouches.
19 e from the endoderm of the caudal pharyngeal pouches.
20 ed into mouse synovial-like subcutaneous air pouches.
21 n and are not incorporated into the coelomic pouches.
22 mp7 and PRDC are expressed in the pharyngeal pouches.
23 nan-induced inflammation in footpads and air pouches.
24 increases junctional E-cadherin in maturing pouches.
25 enerate a late-forming portion of pharyngeal pouch 1 (termed late-p1) and skeletal elements adjacent
26 119 patients (3.2%) required excision of the pouch, 32 (0.8%) had a nonfunctioning pouch, and 46 pati
29 gastrojejunal (GJ) anastomosis (37), gastric pouch (4), gastric remnant (2), jejuno-jejunostomy (1),
31 s of the pituitary are derived from Rathke's pouch, a pocket formed by an invagination of the oral ec
32 packs and 40% of RYO (Roll-Your-Own) tobacco pouches, almost exclusively lower priced brands, were di
33 d prematurely differentiated within Rathke's pouch along with correlated ectopic expression of Mash1
34 opy evaluation confirmed the presence of the pouch along with some indication of residual degenerated
38 rmine whether immediate colectomy with ileal pouch-anal anastamosis (IPAA) after diagnosis of severe
39 ), 118 subtotal colectomies (19%), 134 ileal pouch-anal anastomoses (21%), 23 segmental colectomies (
40 le-center experience of transabdominal ileal pouch-anal anastomoses (IPAA) redo surgery for a failed
44 hough restorative proctocolectomy with ileal pouch-anal anastomosis (IPAA) substantially reduces the
46 compare surgical outcome of transanal ileal pouch-anal anastomosis (ta-IPAA) with transabdominal min
49 f surgery among patients who underwent ileal pouch-anal anastomosis was associated with higher 90-day
50 ients, 71 (65.7%) underwent subsequent ileal pouch-anal anastomosis, 2 died of other causes, and 3 we
55 er of transcription factors expressed in the pouch and by inductive signals from the ventral dienceph
57 lish distinct molecular domains in the third pouch and control the subsequent separation of these org
58 eded to maintain progenitors within Rathke's pouch and for the restriction of differentiated cells to
59 ver, total leukocyte infiltration in the air pouch and IL-1beta production were attenuated in Cryo(-Z
60 at RvE1 decreases neutrophil influx into the pouch and increases neutrophil phagocytosis of P. gingiv
63 full-thickness plications within the gastric pouch and stoma using the StomaphyX device with SerosFus
64 in highly regionalised patterns in the third pouch and that sprouty gene deletion results in upregula
65 However, the shape and organization of the pouch and the anterior/intermediate pituitary lobes are
66 dies have shown that development of Rathke's pouch and the generation of distinct populations of horm
67 wo mouse models of inflammation, zymosan air pouch and thioglycolate-induced peritonitis models, the
68 and 'normal' phosphate (Pi) supply using a 'pouch and wick' system, and had been screened previously
69 luding 11 (0.36%) with adenocarcinoma of the pouch and/or the anal-transitional zone (ATZ), 1 (0.03%)
71 f foxY results in a failure to form coelomic pouches and disrupts the expression of virtually all tra
72 ononuclear cell recruitment in vivo into air pouches and injured muscles depends on the heterocomplex
73 r and the Pak2a kinase in the development of pouches and the posterior facial skeleton that depends o
75 randomized left-right asymmetry of coelomic pouches, and disorganized circumesophageal muscle causin
76 ling in patterning the vertebrate pharyngeal pouches, and show that Hh signalling may be involved in
77 e propose that signals intrinsic to Rathke's pouch are necessary for cell specification between e11.5
78 st of the cell divisions in the central wing pouch are oriented along the proximal-distal (P-D) axis
83 were measured at the middle height level of pouch (AreaM) and the highest level of pouch (AreaH).
84 or a notochord, cartilaginous arcualia, gill pouches, articulations within the proboscis, and multipl
88 egulated and cells are able to exit Rathke's pouch but have lost their migrational cues and form ecto
91 ctively in myeloid cells infiltrating an air pouch cavity upon injection of carrageenan failed becaus
92 r contributor to the adult, but how coelomic pouch cells (CPCs) are specified during embryogenesis is
93 gnaling induce the rearrangement of maturing pouch cells into bilayers through junctional localizatio
94 n of beta-catenin in small clusters of canal pouch cells prevents their resorption, causing instead t
96 Hodgkin lymphomas with golden hamster cheek pouch cells, resulting in serially-transplanted (over 5-
99 ve gastrectomy (approximately 100 mL gastric pouch), closed duodenal stump, end-to-side duodenoileost
100 ariable analyses among patients in the ileal pouch cohort (odds ratio, 1.38; 95% CI, 1.05-1.82).
101 ns were observed among patients in the ileal pouch cohort who received anti-TNF agents preoperatively
105 f the DiGeorge syndrome gene TBX1, a lack of pouches correlates with severe craniofacial defects, yet
107 regate pharyngeal arch artery and pharyngeal pouch defects in RA receptor mutants, and show that RA s
108 es of wnt11r and fgf8a phenocopy tbx1 mutant pouch defects, and mesoderm-specific restoration of Wnt1
109 e conducted a birth dating study of Rathke's pouch derivatives to determine whether the location of s
110 the Hoxa3 null mutant lacks third pharyngeal pouch derivatives, the thymus and parathyroids by E18.5,
112 as required for survival of third pharyngeal pouch-derived organs, but expression in either tissue wa
114 -specific roles of Hoxa3 in third pharyngeal pouch development, we analyzed tissue-specific mutants u
116 nclude stomal stenosis, malpositioned bands, pouch dilation, band slippage, perforation, gastric volv
119 for assessment of polyp burden in the rectum/pouch, duodenal polyposis and pre-colectomy groups, resp
124 ial body epithelium emerging from pharyngeal pouch endoderm in early organogenesis, differential Foxa
125 icate NCCs in regulating patterning of third pouch endoderm into thymus- versus parathyroid-specified
126 oderm, core paraxial mesoderm and pharyngeal pouch endoderm of the mandibular arch as well as more ca
127 is among the prominent markers of pharyngeal pouch endoderm, but to date no role has been assigned to
130 phB-Pak2a signaling is required to stabilize pouch epithelial cells at the end of branching morphogen
131 ver, an unexplored possibility is that brood pouch evolution was partly shaped by parent-offspring or
132 with good functional results, and may avoid pouch excision and permanent ileostomy in carefully sele
133 sensitivities were analysed, and changes in pouch faecal and mucosal microbiota assessed by 16S rRNA
134 l pouch surgery was the primary indicator of pouch failure (hazard ratio, 3.691; 95% confidence inter
141 atrices were randomly placed in subcutaneous pouches for 7 and 28 days and evaluated by histology and
143 head, endodermal branches, called pharyngeal pouches, form through the transient stratification, coll
145 rly phase during which FGFs drive pharyngeal pouch formation, and a later phase when they directly re
147 Wnt-dependent epithelial destabilization of pouch-forming cells with their collective migration towa
149 the mesoderm to promote the morphogenesis of pouch-forming endoderm through wnt11r and fgf8a expressi
150 he proportion of patients with a functioning pouch, frequency of defecation and incidence of incontin
152 The clinical significance of these cementum pouches has yet to be determined but bacterial contamina
154 0.069) and an increased rate of giant venous pouch in children in whom no mutation was identified (p
155 tion of the small micromeres to the coelomic pouches in the sea urchin embryo provides an exceptional
157 nd timing of key regional markers within the pouch, including Tbx1, Bmp4 and Fgf8, were altered.
159 in inflammatory bacteria are associated with pouch inflammation in patients with UC who underwent pou
161 However, cells at the periphery of the wing pouch instead tend to orient their divisions perpendicul
162 hat endodermal Eph-ephrin signaling promotes pouch integrity by targeting Pak2a to the plasma membran
164 lar to the Hesx1-deficient embryos, Rathke's pouch is initially expanded in Six3+/- ;Hesx1Cre/+ compo
166 c bypass are radiologically contained GJ and pouch leaks and can be safely managed nonoperatively.
167 M evaluation showed the presence of a narrow pouch-like opening between cementum and enamel in 15 of
168 This study reports on the presence of a pouch-like opening between the enamel and cementum in ma
169 ermined but bacterial contamination of these pouch-like structures in areas of furcation periodontal
171 ed glass and two commercial bag-in-box (BIB) pouches (low density polyethylene - LDPE and ethylene vi
173 proliferating cells are enriched around the pouch lumen, and they appear to delaminate as they exit
175 anal-transitional zone (ATZ), 1 (0.03%) with pouch lymphoma, 3 with squamous cell cancer of the ATZ,
178 t mice were subjected to the carrageenan-air pouch model of inflammation and then treated with D-T(4)
179 om M(r) 5,000 to 1,500,000 in the rodent air pouch model of inflammation to determine their potential
184 induced neutrophilic inflammation in the air-pouch model of synovitis, and they show decreased joint
188 ites have inflammatory properties in the air pouch model, and rHuPH20 can instead inhibit some aspect
191 cells (BMSCs) to form bone in a mouse muscle pouch model, exhibiting specificity that BMPs lack.
199 d signal, Hedgehog, is required for coelomic pouch morphogenesis and institution of laterality, but d
201 chemical inhibitor approaches, we show that pouch morphogenesis requires Bmp signaling from 10-18 h
202 n the pharyngeal arches, aberrant endodermal pouch morphogenesis, and hypoplastic cranial cartilages.
203 t in jawed vertebrates, including pharyngeal pouch morphogenesis, patterning of the oral skeleton and
205 20 years were 1.0%, 2.0%, 3.7%, and 6.9% for pouch neoplasia and 0.6%, 1.4%, 2.1%, and 3.3% for pouch
206 We calculated the cumulative incidence of pouch neoplasia and performed a case-control study to id
209 med to determine the cumulative incidence of pouch neoplasia in patients with IBD and identify risk f
211 ized features, risk factors, and outcomes of pouch neoplasia in patients with inflammatory bowel dise
212 e Cox model, the risk factor associated with pouch neoplasia was a preoperative diagnosis of UC-assoc
220 hat absence of SOX2 in the developing Rathke pouch of conditional embryos led to severe anterior lobe
221 al disease sites (P = .0025) and presence of pouch of Douglas implants (P = .0045) were associated wi
223 factors asymmetrically in the left coelomic pouch only, which launch the pathway that eventually lea
226 filtered cigars, pipe tobacco, hookah, snus pouches, other smokeless tobacco, dissolvable tobacco, b
227 ics show that Bmp signaling is necessary for pouch outpocketing via the Fibroblast growth factor (Fgf
230 e epithelial integrity of the vertical canal pouch perimeter (presumptive anterior and posterior SSCs
231 gest time to first FAP-related event [rectal/pouch polyposis]), intermediate (duodenal polyposis) and
232 orable pouch voiding mechanics, insufficient pouch pressure generation or failure of external sphinct
233 s originates from bilateral third pharyngeal pouch primordia containing endodermal progenitors of bot
234 rder shift increases the allocation of third pouch progenitors to the thymus domain and corresponding
237 ients with failing IPAA, partial or complete pouch reconstruction can be done safely with good functi
238 (UC) who required partial or complete ileal pouch reconstruction due to poor function or infectious
241 ocalization of Dachs in cells throughout the pouch requires the movement of the Ds transition region
242 6th arch arteries and to the 4th pharyngeal pouch, respectively, suggesting that different regulator
245 ical, and pathologic factors associated with pouch retention were evaluated with log-rank test and Co
250 e RA signaling occur secondary to pharyngeal pouch segmentation defects, although this model has neve
252 Between the two plastics, the LDPE lined pouch showed a considerably higher aroma sorption as com
253 e RYGB model that closely replicates gastric pouch size of human RYGB surgery of about 5% of total ga
255 tive study of patients with UC who underwent pouch surgery (N = 131) from 1981 through 2012 and were
256 ts with familial adenomatous polyposis after pouch surgery (n = 9), individuals with intact colons un
258 Pelvic sepsis developing after redo ileal pouch surgery was the primary indicator of pouch failure
259 a failed ileoanal pouch may be offered redo pouch surgery with a high likelihood of success in terms
267 ng development, the canals are sculpted from pouches that protrude from the otic vesicle, the embryon
269 Diverticular disease is characterized by pouches (that is, diverticulae) due to weakness in the b
271 along the proximodistal axis into the distal pouch, the hinge, the surrounding pleura, and the notum.
273 t Eya1 expression to the somites, pharyngeal pouches, the preplacodal ectoderm (the common precursor
275 hanges in pregnant versus non-pregnant brood pouch tissue and characterize the genomic organization o
277 microcirculation as an in situ assay, cheek pouch tissue was exteriorized in anesthetized (phentobar
278 on in cells as they transition from Rathke's pouch to the anterior lobe appears to be essential for t
281 R) inhibited plasma leakage in hamster cheek pouch topically exposed to tissue culture trypomastigote
282 2.6 (95% confidence interval [CI] 1.1-6.5)), pouch-vaginal fistula (P = 0.01, HR 2.8 (95% CI 1.3-6.4)
284 ted from dysfunctional voiding - unfavorable pouch voiding mechanics, insufficient pouch pressure gen
286 re quantified in the lavage fluid and in the pouch wall after staining with Giemsa or after enzymatic
290 In vivo, Th17 cell recruitment into the air pouch was reduced in CD43(-/-) mice in response to CCL20
294 d ectodermal components, as well as Rathke's pouch, were similarly sampled and profiled using both mi
295 ed or highly reduced in the posterior/caudal pouches, where the parathyroid marker Gcm2 is expressed,
296 o early gene expression defects in the third pouch, whereas thymus hypoplasia is caused by reduced pr
297 males brood their offspring in a specialized pouch, which presumably evolved to facilitate male paren
298 induced neutrophil infiltration into the air pouch, which was not observed with rHuPH20 treatment.
300 roughly uniform throughout most of the wing pouch with a steep transition region that propagates fro
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