ライフサイエンスコーパス: powdery mildew

1 tif, and confer broad-spectrum resistance to powdery mildew.

2 ncrease in penetration success by the barley powdery mildew.

3 ributors to NHR in Arabidopsis against wheat powdery mildew.

4 ol the primary foliar diseases, Botrytis and powdery mildew.

5 A than wild type in response to infection by powdery mildew.

6 ngus Erysiphe cichoracearum, causal agent of powdery mildew.

7 about the corresponding avirulence genes in powdery mildew.

8 defenses upon inoculation with a nonadapted powdery mildew.

9 patible interactions between Arabidopsis and powdery mildew.

10 marker PR1, and fully enhanced resistance to powdery mildew.

11 omised basal and RPW8-mediated resistance to powdery mildew.

12 ective pre-invasive immune responses against powdery mildews.

13 orted to exhibit biological activity against powdery mildews.

14 k1-4 in a forward genetic screen with barley powdery mildew and consequently characterized it by path

15 es, including enhanced disease resistance to powdery mildew and enhanced ethylene-induced senescence;

16 Powdery mildews and other obligate biotrophic pathogens

17 Powdery mildews are phytopathogens whose growth and repr

18 By contrast, the nonhost barley powdery mildew Blumeria graminis f. sp. hordei (Bgh) typ

19 or full penetration resistance to the barley powdery mildew Blumeria graminis f. sp. hordei.

20 s from infection studies with the nonadapted powdery mildew Blumeria graminis f. sp. hordei.

21 ically important race-specific resistance to powdery mildew (Blumeria graminis).

22 Barley powdery mildew, Blumeria graminis f. sp. hordei (Bgh), i

23 n-host penetration resistance against barley powdery mildew, Blumeria graminis f. sp. hordei, and we

24 We present the genome analysis of barley powdery mildew, Blumeria graminis f.sp. hordei (Blumeria

25 HvRBK1 might function in basal resistance to powdery mildew by influencing microtubule organization.

26 and Pseudozyma flocculosa for the control of powdery mildew by two Canadian research programs is pres

27 re, we describe two barley (Hordeum vulgare) powdery mildew candidate secreted effector proteins, CSE

28 previously elusive 5' sequence of the barley powdery mildew chitin synthase gene, BgChs2, which inclu

29 aces, is not associated with the collapse of powdery mildew colonies.

30 genes MLO2, MLO6 and MLO12 not only restrict powdery mildew colonization, but also affect interaction

31 ese are associated with defense responses to powdery mildew disease but function in different signali

32 ed treatment provided primed defence against powdery mildew disease caused by the biotrophic fungal p

33 utation of Arabidopsis confers resistance to powdery mildew disease caused by the fungus Erysiphe cic

34 may provide a novel strategy for controlling powdery mildew disease in crops.

35 the complex inheritance of resistance to the powdery mildew disease in the model plant Arabidopsis th

36 This study suggests that the Arabidopsis powdery mildew disease will be a suitable model system i

37 inis f. sp hordei (Bgh), the causal agent of powdery mildew disease.

38 inis f.sp. hordei (Bgh), the causal agent of powdery mildew disease.

39 nis f. sp. hordei (Bgh), the causal agent of powdery mildew disease.

40 raminis f. sp. hordei (Bgh), casual agent of powdery mildew disease.

41 Powdery mildew diseases are economically important disea

42 ptible, and accession Ms-0 was resistant, to powdery mildew diseases caused by Erysiphe cruciferarum

43 nst Golovinomyces spp., the causal agents of powdery mildew diseases on multiple plant species.

44 ngal pathogens Golovinomyces spp. that cause powdery mildew diseases on multiple plant species.

45 uitable model system in which to investigate powdery mildew diseases.

46 fundamental changes in our understanding of powdery mildews (Erysiphales).

47 Arabidopsis thaliana is a host to the powdery mildew Erysiphe cichoracearum and nonhost to Blu

48 Attack by the host powdery mildew Erysiphe cichoracearum usually results in

49 -) and to the nonadapted pea (Pisum sativum) powdery mildew Erysiphe pisi However, PLC2-silenced plan

50 negative phenotype, conferring resistance to powdery mildew (Erysiphe cichoracearum) and enhancing et

51 o shows enhanced sensitivity to the pathogen powdery mildew (Erysiphe cruciferarum) and fails to indu

52 two additional inappropriate biotrophs, pea powdery mildew (Erysiphe pisi) and potato late blight (P

53 ew to those inoculated with a virulent, host powdery mildew, Erysiphe cichoracearum.

54 Barley powdery mildew, Erysiphe graminis f.sp. hordei, is an ob

55 n-transgenic tomato variety resistant to the powdery mildew fungal pathogen using the CRISPR/Cas9 tec

56 Powdery mildew fungal pathogens penetrate the plant cell

57 ense signaling in nonhost resistance against powdery mildew fungi and put PLDdelta forward as the mai

58 d potential and quality, the three rusts and powdery mildew fungi have historically caused major crop

59 es on which spores of adapted and nonadapted powdery mildew fungi showed reduced germination.

60 on by otherwise virulent obligate biotrophic powdery mildew fungi such as Golovinomyces orontii.

61 ypotheses for viruses and for rust, smut and powdery mildew fungi that infect 473 plant species natur

62 Penetration resistance to powdery mildew fungi, conferred by localized cell wall a

63 ion phenotype against adapted and nonadapted powdery mildew fungi.

64 elivery of defense components in response to powdery mildew fungi.

65 alization of PEN3-GFP after inoculation with powdery mildew fungi.

66 d postinvasive immunity against a nonadapted powdery mildew fungus (Blumeria graminis f.

67 2-mediated resistance against the biotrophic powdery mildew fungus (Blumeria graminis f.sp. hordei),

68 exhibit a wide spectrum of resistance to the powdery mildew fungus (PM), Erysiphe necator (Schw.) Bur

69 ced lesion formation due to infection by the powdery mildew fungus Blumeria f. sp. tritici.

70 asis of NHR in Arabidopsis against the wheat powdery mildew fungus Blumeria graminis f. sp. tritici (

71 dopsis (Arabidopsis thaliana) and the barley powdery mildew fungus Blumeria graminis f.sp. hordei (Bg

72 in the interaction of barley with the barley powdery mildew fungus Blumeria graminis f.sp. hordei.

73 proach to enhance resistance of wheat to the powdery mildew fungus Blumeria graminis f.sp. tritici, a

74 defense responses to a host-adapted virulent powdery mildew fungus but were impaired in preinvasive d

75 We identify AVR(a10) and AVR(k1) of barley powdery mildew fungus, Blumeria graminis f sp hordei (Bg

76 nous Arabidopsis thaliana against the barley powdery mildew fungus, Blumeria graminis f. sp. hordei.

77 terised two MAP kinase genes from the barley powdery mildew fungus, Blumeria graminis.

78 on resistance against another nonadapted pea powdery mildew fungus, Erysiphe pisi.

79 VPS9a also mediates defense to an adapted powdery mildew fungus, thus regulating a durable type of

80 Investigating Arabidopsis infection with the powdery mildew fungus, we find that the Arabidopsis atyp

81 against all known isolates of the widespread powdery mildew fungus.

82 teins that are homologous with gEgh16 of the powdery mildew fungus.

83 ncing effector-induced HR in response to the powdery mildew fungus.

84 ncing effector-induced HR in response to the powdery mildew fungus.

85 mal plant cell inaccessible for the invading powdery mildew fungus.

86 plete penetration resistance to the virulent powdery mildew Golovinomyces cichoracearum due to enhanc

87 lants 6 h post inoculation with the virulent powdery mildew Golovinomyces cichoracearum.

88 e confer enhanced resistance to infection by powdery mildew (Golovinomyces cichoracearum).

89 We evaluated the effects of grape powdery mildew (GPM) as supplemental food, and habitat s

90 The host-adapted powdery mildew had a reduced reproductive fitness on rop

91 is synchronized with a higher expression of powdery mildew haustorial effectors, a sharp decline in

92 is mutants deficient in resistance to barley powdery mildew identified PENETRATION3 (PEN3).

93 regulates SA-dependent basal defense against powdery mildew in Arabidopsis.

94 -DNA insertion allele in DRP1E did not cause powdery mildew-induced lesions, suggesting that this phe

95 BTH protected wheat systemically against powdery mildew infection by affecting multiple steps in

96 the hypersensitive response (HR) to restrict powdery mildew infection via the salicylic acid-dependen

97 istant transgenic lines during both types of powdery mildew infection, and neither the salicylic acid

98 E1 (EDR1) gene confer enhanced resistance to powdery mildew infection, enhanced senescence, and enhan

99 with papilla formation in the epidermis upon powdery mildew infection.

100 expressed in development and associated with powdery mildew infection.

101 ant and made Arabidopsis hypersusceptible to powdery mildew infection.

102 sponses, which do not effectively block host powdery mildew infections.

103 ce pathways played a role in blocking barley powdery mildew infections.

104 tempted penetration by Bgh during the barley-powdery mildew interaction but is independent of gene-fo

105 We conclude that edr1-mediated resistance to powdery mildew is mediated, in part, by enhanced ABA sig

106 Powdery mildews may already be responding to climate cha

107 Powdery mildews, obligate biotrophic fungal parasites on

108 Powdery mildew of barley, caused by Erysiphe graminis f.

109 gical aberrations and altered sensitivity to powdery mildews of rop6(DN) plants result from perturbat

110 ent has been well demonstrated for rusts and powdery mildews of small grain crops.

111 edr1-mediated enhanced disease resistance to powdery mildew or spontaneous lesions, indicating that t

112 displayed enhanced disease resistance to the powdery mildew pathogen Erysiphe cichoracearum and ident

113 that do not support the normal growth of the powdery mildew pathogen Erysiphe cichoracearum.

114 disease resistance (edr2) to the biotrophic powdery mildew pathogen Erysiphe cichoracearum.

115 Interactions between barley and the powdery mildew pathogen, Blumeria graminis f. sp. hordei

116 nts that do not support normal growth of the powdery mildew pathogen, Erysiphe cichoracearum.

117 hat show resistance to a wild isolate of the powdery mildew pathogen, Erysiphe cichoracearum.

118 Columbia (gl1), which is susceptible to the powdery mildew pathogen, were performed.

119 wild-type plants inoculated with a virulent powdery mildew pathogen.

120 resistance, resulting in reduced growth of a powdery mildew pathogen.

121 nhost to Blumeria graminis f. sp hordei, the powdery mildew pathogenic on barley (Hordeum vulgare).

122 ell as a comparison with the analysis of two powdery mildews pathogenic on dicotyledonous plants.

123 ually control resistance to a broad range of powdery mildew pathogens.

124 haliana confers broad-spectrum resistance to powdery mildew pathogens.

125 the growth and reproduction of the virulent powdery mildew (PM) Golovinomyces orontii on Arabidopsis

126 factors exhibited altered expression at the powdery mildew (PM) infection site, with subsets of thes

127 Tissue colonization by grape powdery mildew (PM) pathogen Erysiphe necator (Schw.) Bu

128 Wheat powdery mildew (PM), caused by Blumeria graminis f.

129 We identified and mapped three independent powdery mildew quantitative disease resistance loci, whi

130 RKY6, the MAP kinases AtMPK6 and AtMPK3, the powdery mildew R proteins RPW8.1 and RPW8.2, EDS1 and PR

131 essed by inoculations with non-host and host powdery mildews relative to non-inoculated control plant

132 are) cultivar C.I. 16151 (harboring the Mla6 powdery mildew resistance allele) and its fast neutron-d

133 PAD4, EDS5, NPR1 and SGT1b for activation of powdery mildew resistance and HR.

134 rts negative regulation on HR cell death and powdery mildew resistance by limiting the transcriptiona

135 In barley (Hordeum vulgare), the Mla13 powdery mildew resistance gene confers Rar1-dependent, A

136 ne for N-mediated resistance to TMV and some powdery mildew resistance genes in barley provide the fi

137 s similar to that conferred by "late-acting" powdery mildew resistance genes of wheat and barley.

138 we show that RPW8.2 from A. lyrata conferred powdery mildew resistance in A. thaliana, suggesting tha

139 ced basal resistance and effector-triggered, powdery mildew resistance locus A12-mediated resistance

140 alleles of the Mla locus can dictate similar powdery mildew resistance phenotypes yet still require d

141 quivalent region from the orthologous barley powdery mildew resistance protein, MLA10, but is similar

142 In accessions Wa-1, Kas-1, Stw-0 and Su-0, powdery mildew resistance was encoded by a semi-dominant

143 tig from barley cv Morex that spans the Mla (powdery mildew) resistance locus.

144 Ug99 lineage, and are homologs of the barley powdery mildew-resistance protein MLA10.

145 Mapping studies revealed that powdery mildew resistances in Kas-1, Wa-1, Te-0, Su-0 an

146 nalyses indicated that four loci, designated powdery mildew resistant 1-4 (pmr1-4), are defined by th

147 Surprisingly, here we found that powdery mildew resistant 4 (pmr4), a mutant lacking path

148 the infection phenotypes of two independent powdery mildew-resistant triple mutant lines with a rang

149 ed transgenic Arabidopsis lines that express POWDERY MILDEW RESISTANT4 (PMR4), which encodes a stress

150 phthora root rot (Rps1, Rps2, and Rps3), and powdery mildew (rmd).

151 Resistance to powdery mildew (Rmd-c), Phytophthora stem and root rot (

152 5 gene rendered Arabidopsis resistant to the powdery mildew species Erysiphe cichoracearum and Erysip

153 Powdery mildew species Oidium neolycopersici (On) can ca

154 ptible to E. orontii, a very closely related powdery mildew, suggesting that a very specific resistan

155 ere more susceptible to the fungal pathogen, powdery mildew, than wild type as measured by conidiopho

156 ng of plant surfaces among distantly related powdery mildews that is based on KCS6-derived wax compon

157 For plant pathogenic fungi, such as powdery mildews, that survive only on a limited number o

158 ytopathogen but rather a biocontrol agent of powdery mildews; this relationship makes it unique for t

159 bidopsis inoculated with the non-host barley powdery mildew to those inoculated with a virulent, host

160 ell death and enhanced disease resistance to powdery mildew via the SA-dependent signaling pathway.

161 e-like cell death and enhanced resistance to powdery mildew via the salicylic acid-dependent signalin