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1 R (pre-BCR) signaling, spontaneously develop pre-B cell leukemia.
2 oduced by the t(1;19) translocation in human pre-B cell leukemia.
3 ted oncogene product, v-Abl, and also causes pre-B-cell leukemia.
4 n implicated in the development of childhood pre-B-cell leukemias.
5 ave not been detected in patients with human pre-B-cell leukemias.
7 nce for the epigenetic changes that occur in pre-B-cell leukemia and other B-cell-related diseases.
8 erated from the bone marrow of patients with pre-B-cell leukemias and these findings should facilitat
14 g protein (PBXIP1/HPIP) is a co-repressor of pre-B-cell leukemia homeobox 1 (PBX1) and is also known
20 malignancies, but are consistently absent in pre-B cell leukemias induced by the chimeric oncoprotein
21 as the product of a proto-oncogene in acute pre-B-cell leukemia, is a global regulator of embryonic
23 1;19) chromosomal translocation of pediatric pre-B cell leukemia produces chimeric oncoprotein E2a-Pb
26 show here the generation of autologous anti-pre-B-cell leukemia-specific cytolytic T-cell lines from
27 f Pbx1, E2a-Pbx1, is an oncoprotein in human pre-B cell leukemia that strongly suppresses differentia
28 se inhibitors showed that the homeoproteins, pre B-cell leukemia transcription factor 1 (PBX1) and PB
29 es the transcriptional activation of IL10 by pre-B cell leukemia transcription factor-1b and another
30 ic deletion of CDH-implicated genes encoding pre-B cell leukemia transcription factors (Pbx) led to l
32 d ecotropic viral insertion site (MEIS), and pre-B-cell leukemia transcription factor 1 (PBX) may reg
33 s of CDKIs, we observed a cellular gene, the pre-B-cell leukemia transcription factor 1 (Pbx1) gene,
34 nockout mice for one of the candidate genes, pre-B-cell leukemia transcription factor 1 (Pbx1), and i
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