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1 dvanced prostatic cancer, endometriosis, and precocious puberty.
2 decline, neuropsychiatric comorbidities and precocious puberty.
3 boys and girls with gonadotropin-independent precocious puberty.
4 nosed in 24% of tested patients, and 12% had precocious puberty.
5 noted were weight loss, growth failure, and precocious puberty.
6 s, others lead to vision loss, proptosis, or precocious puberty.
7 prostate cancer as well as endometriosis and precocious puberty.
8 in the mechanism underlying both normal and precocious puberty.
9 ng in 40 members of 15 families with central precocious puberty.
10 ic-pituitary-gonadal axis results in central precocious puberty.
11 ders were growth hormone deficiency (12.5%), precocious puberty (12.2%), thyroid-stimulating hormone
12 ight loss, macrocephaly, growth failure, and precocious puberty also suggest presence of an intracran
13 adult height in children with LHRH-dependent precocious puberty and is prescribed by some practitione
16 f the lesions correlate with the presence of precocious puberty, and does the type of lesion influenc
17 have shown that some forms of familial male precocious puberty are associated with mutations of the
18 pendent disorders of reproduction, including precocious puberty, endometriosis, and metastatic prosta
19 sis colorectal cancer (HNPCC), familial male precocious puberty (FMPP), Carney complex (CNC), Doyne's
21 e or developmental impairment and those with precocious puberty had significantly larger lesions invo
25 ture reactivation of GnRH neurons results in precocious puberty in mice and humans, but the mechanism
28 e rats undergoing normal puberty or in which precocious puberty was induced by treatment with the glu
29 9-year-old boy, with a diagnosis of CAH and precocious puberty, who was referred to our department f
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