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1 attern and ST-segment elevation in the right precordial leads.
2 II, III, aVR, and aVF and the mid to lateral precordial leads.
3 elevation and T-wave inversion in the right precordial leads.
6 elevation (type 1 Brugada pattern) in right precordial leads at therapeutic concentrations in 2 pati
7 nly present with ST-segment elevation in the precordial leads, chest pain, relatively minor elevation
9 there was a dominant frequency gradient from precordial leads facing the scar region to the contralat
11 ary disease, increasing STdep in the lateral precordial leads is associated with increasing LV mass a
12 ncy spectral area computed from conventional precordial leads, like coronary perfusion pressure and e
13 haracteristics, including QRS morphology and precordial lead morphology, can help distinguish between
15 normalities of repolarization in the lateral precordial leads of the electrocardiogram, as manifested
17 x (the time to the maximum deflection in the precordial leads/QRS duration) was the largest in LV sum
18 eversed QTUc prolongation, especially in the precordial leads (quinidine, 590+/-79 to 479+/-35 [+/-SD
19 pattern of ST-segment elevation in the right precordial leads should not be seen as a marker of a spe
20 a distinct ST-segment elevation in the right precordial leads, the syndrome is associated with a high
21 easured ST depression (STdep) in the lateral precordial leads to the presence of left ventricular hyp
24 nts displayed extensive T-wave inversions in precordial leads V1 through V4, with either persistent o
25 coved-type ST-segment elevation in the right precordial leads (V1 to V3; type 1 Brugada electrocardio
26 terized by ST segment elevation in the right precordial leads, V1-V3 (unrelated to ischemia or struct
27 p = 0.0004), a more depressed ST-segment in precordial lead V5 (p = 0.0002), and a higher coronary a
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