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1 epicardial asynchrony and QRS vector and LV preexcitation.
2 nimal and endocardial AT vector indicated LV preexcitation.
3 penetrant accessory pathways and ventricular preexcitation.
4 rosus was disrupted resulting in ventricular preexcitation.
5 ical abnormalities, particularly ventricular preexcitation.
6 ovide the anatomic substrate for ventricular preexcitation.
7 lar, resynchronization, and left ventricular preexcitation.
8 ry care population with electrocardiographic preexcitation.
9 elucidating the pathogenesis of ventricular preexcitation.
10 hort (75-millisecond) PR interval to achieve preexcitation.
13 ndicate sinoatrial node dysfunction, whereas preexcitation and atrioventricular block reveal abnormal
14 e for a syndrome associated with ventricular preexcitation and early onset of atrial fibrillation and
16 were used to show the mechanistic effects of preexcitation and to determine the optimal stimulation s
17 onset (at 8 to 17 years of age), ventricular preexcitation, and asymptomatic elevations of two serum
21 Wolff-Parkinson-White pattern and persistent preexcitation at maximum exercise undergoing invasive ri
22 sible for a familial syndrome of ventricular preexcitation, atrial fibrillation, conduction defects,
23 glycogen storage eliminated the ventricular preexcitation but did not affect the excessive cardiac g
25 The human phenotype consists of ventricular preexcitation, conduction abnormalities, and cardiac hyp
26 d a higher hazard of death for patients with preexcitation >/=65 years (HR, 1.85; 95% CI, 1.07-3.18).
27 e remainder of the population, patients with preexcitation had higher adjusted hazards of atrial fibr
28 In this large ECG study, individuals with preexcitation had higher hazards of atrial fibrillation
29 lar activation (eg, resulting from pacing or preexcitation in patients with Wolff-Parkinson-White syn
30 y glycogen-engorged myocytes as the cause of preexcitation in Pompe, Danon, and other glycogen storag
31 clinical course of patients with ventricular preexcitation in the ECG originates from tertiary center
32 ertrophy followed by dilatation, ventricular preexcitation involving multiple accessory pathways, and
34 was the longest (349+/-6 ms) in the area of preexcitation leading to high average base-to-apex ARI d
37 ional BrS patients, we performed decremental preexcitation of the RVOT before endocardial RV mapping.
38 AP conduction, as demonstrated by recurrent preexcitation or change in retrograde ventriculoatrial a
41 rated the TG(R302Q) mice to have ventricular preexcitation (PR interval 10+/-2 versus 33+/-5 ms in TG
44 nd electrocardiograms suggesting ventricular preexcitation revealed four LAMP2 and seven PRKAG2 mutat
46 ndle-branch block, ventricular paced rhythm, preexcitation syndrome, or previous revascularization wi
48 hough the pathology and electrophysiology of preexcitation syndromes are well characterized, the deve
51 igher hazard of death among individuals with preexcitation when looking across all age groups (HR, 1.
53 Studies reporting asymptomatic patients with preexcitation who did not undergo ablation were included
54 ted members of both families had ventricular preexcitation with conduction abnormalities and cardiac
55 ectrophysiologic abnormalities, particularly preexcitation (Wolff-Parkinson-White syndrome) and atrio
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