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1 haracterizing a premotor area, rather than a prefrontal area.
2 rtex, right middle occipital gyrus and right prefrontal area.
3 ment of activation in the mesial frontal and prefrontal areas.
4  cortex, with very few connections to medial prefrontal areas.
5 in contrast to lateral premotor and inferior prefrontal areas.
6 included high beta (24-30 Hz) frequencies in prefrontal areas.
7 ntal areas, including the bilateral inferior prefrontal areas.
8 rontoparietal network, in addition to medial prefrontal areas.
9         All components revealed increases in prefrontal areas.
10 n the anterior cingulate cortex and adjacent prefrontal areas.
11 nterior cingulate cortex as well as adjacent prefrontal areas.
12 n, which are mainly due to processing in the prefrontal areas.
13 e showing decreased connectivity to multiple prefrontal areas.
14 ng neural activity in, and sensory drive to, prefrontal areas.
15 medial parietal, lateral temporal and medial prefrontal areas.
16 he anterior cingulate in addition to lateral prefrontal areas.
17 nal interactions between medial temporal and prefrontal areas.
18 l representations in downstream parietal and prefrontal areas.
19 r injections in orbital, medial, and lateral prefrontal areas.
20 uding several not classically recognized as "prefrontal" areas.
21 es of supragranular pyramidal cells in human prefrontal (area 10) and secondary occipital (area 18) c
22 r (area 4), prestriate visual (area 18), and prefrontal (area 10) cortices of developing chimpanzees
23 eas 3b/3a/1/2), motor (area 4), frontopolar (prefrontal area 10), and visual (areas 17/18) neocortex
24 Injections that included rostral and orbital prefrontal areas (10, 46 rostral, 12) labeled the rostra
25                  The projections from medial prefrontal areas 10m, 25, and 32 end predominantly in th
26 the PAG that arise primarily from the medial prefrontal areas 25, 32, and 10m, anterior cingulate, an
27  anterior cingulate area 24b/c and in medial prefrontal areas 32 and 32V.
28                                   The medial prefrontal areas 32, 24, 14, and 25 (mPFC) form part of
29 intenance, was associated with activation of prefrontal area 46 of the dorsal lateral prefrontal cort
30 erebral cortex in primary visual area 17 and prefrontal area 46.
31                                 Dorsolateral prefrontal areas 46 and 10 are involved in distinct aspe
32  connections are with the ventral portion of prefrontal areas 46, 11, 12, and 13 as well as with the
33 activation of the midcingulate cortex (MCC), prefrontal area 6/44, and the somatosensory cortex, area
34 aintenance was associated with activation of prefrontal area 8 and the intraparietal cortex.
35  dextran amine (BDA) into layer 3 of macaque prefrontal area 9 and examined the labeled intrinsic axo
36  Cell density was reexamined in dorsolateral prefrontal area 9 as an internal control.
37  the capacity to release normal DA levels in prefrontal areas after a pharmacological challenge is pr
38 l premotor, bilateral parietal and bilateral prefrontal areas along with increased activation of bila
39  that has advanced our understanding of this prefrontal area and how its functions are shaped through
40 unction of specific brain systems, including prefrontal areas and cingulate cortex (both involved in
41 hronically ill patients showed lower flow in prefrontal areas and higher flow in thalamic and cerebel
42  impact on executive responses in widespread prefrontal areas and in the pulvinar increased when the
43 led that this delta activity originated from prefrontal areas and modulated posterior alpha power.
44 ening of connectivity between reward-related prefrontal areas and sensorimotor areas in the basal gan
45  between the working memory functions in the prefrontal areas and the long-term memory encoding in th
46 ed decreased connectivity between insula and prefrontal areas, and between amygdala and globus pallid
47                 These changes below specific prefrontal areas appear to be linked through a cascade o
48 e emotional state, and that the dorsolateral prefrontal areas are not involved in emotion in these wa
49 ion of the 5-HTT-IR profiles in dorsolateral prefrontal area between neurodegenerative diseases and c
50  5-HT2cR mRNA differentially in striatal and prefrontal areas between HI and LI rats, and selectively
51 Value furthermore modulated coupling between prefrontal areas, brainstem, and spinal cord, which migh
52                  The local fields in the two prefrontal areas, but not the cortex immediately posteri
53 l cortex of macaque monkeys (with a focus on prefrontal areas) by using light and electron microscopi
54                 Thus, acting together, these prefrontal areas can ensure that our behavior is most ef
55                                          The prefrontal areas Cg and M2 in turn connect to different
56       A greater NODs in the left Broca's and prefrontal areas combined and fewer NODs in the left cin
57 rated a greater NODs in the left Broca's and prefrontal areas combined, left cingulate gyrus and left
58 stence of these corticopontine pathways from prefrontal areas concerned with multiple domains of high
59 ing blood oxygen level-dependent response in prefrontal areas connected by these tracts.
60         Both orbitofrontal and ventrolateral prefrontal areas contribute to updating these valuations
61       However, it is still unknown how these prefrontal areas convey the necessary signal to the prim
62 ed different degrees of selectivity, and the prefrontal areas demonstrated different strengths of sus
63 role in perceptual processing, whereas three prefrontal areas demonstrated sustained activity over me
64                           Medial and ventral prefrontal areas differentially responded to favorable a
65 ity was significantly reduced bilaterally in prefrontal areas encompassing fronto-striatal connection
66  and OFC changed the relationships among all prefrontal areas examined, and could indirectly affect L
67 ggest that specific orbitofrontal and medial prefrontal areas exert a direct influence on the hypotha
68 onnectivity between the ventral striatum and prefrontal areas exerting top-down control on the mesoli
69 nd a hierarchical ordering, with sensory and prefrontal areas exhibiting shorter and longer timescale
70                                Ventrolateral prefrontal areas have been studied less extensively, and
71  spatial organization of neurons within this prefrontal area in humans takes place after the postwean
72 ity was increased between MT/MST and lateral prefrontal areas in congenitally blind relative to sight
73 rm experimentally the causal role of lateral prefrontal areas in the modification of attentional bias
74  expressions, and showed lower activation in prefrontal areas, including orbitofrontal cortex and ins
75 orimotor, premotor, supplementary-motor, and prefrontal areas, including the bilateral inferior prefr
76 beled neurons in the dentate varied with the prefrontal area injected.
77                                          All prefrontal areas investigated received projections from
78 mediate visual perceptual processing and the prefrontal areas involved in the active maintenance of i
79 sponsive to novel information, and the right prefrontal area is associated with the maintenance of co
80 etwork of occipital, parietal, premotor, and prefrontal areas maximally activated by tactile stimulat
81  altered organization of connectivity of the prefrontal areas may reflect the role of the prefrontal
82 argeting of sensory tiers of TRN by specific prefrontal areas may underlie attentional regulation for
83                    We propose that these two prefrontal areas mediate shifting away from disadvantage
84 th epilepsy, such as the mesial temporal and prefrontal areas, no controlled trials have investigated
85 nal anisotropy of white matter tracts in the prefrontal area of 10 schizophrenic patients was determi
86 reases in induced gamma-band activity in the prefrontal areas of healthy subjects but that control-re
87  < .05) was mediated by cortical thinning in prefrontal areas of the right hemisphere.
88                                              Prefrontal area perfusion showed less association with s
89             Injections in lateral eulaminate prefrontal areas primarily labeled neurons in the poster
90                        We propose that these prefrontal areas produce the above patterns of LC activi
91 cialization, from occipital through multiple prefrontal areas, regarding each area's relative contrib
92 ple the superior colliculus (SC), as well as prefrontal areas responsible for top-down control.
93 ted to a relative delay in the maturation of prefrontal areas, resulting in the increase of impulsive
94                                         Some prefrontal areas select and interpret conscious events f
95  reduction of synchronization over the right prefrontal area showed a linear univariate correlation w
96 terior PFC and the domain-specific posterior prefrontal areas (superior frontal sulcus and left infer
97 -risk demonstrated a greater NOD in the left prefrontal area than good and average readers born at lo
98 -risk demonstrated a greater NOD in the left prefrontal area than those born at low-risk and term.
99  'pre-PMd', are, in many respects, more like prefrontal areas than motor areas.
100                                           In prefrontal areas, the orbitofrontal cortex (OFC) has bee
101 eral visual, temporal, parietal, and lateral prefrontal areas, the vast majority overlapping with the
102 includes diffuse projections from restricted prefrontal areas to the thalamic reticular nucleus (RE),
103  neurons labeled after virus injections into prefrontal areas were located in regions spatially separ
104                          The medial and left prefrontal areas were the most altered (lower F18-DOPA r
105 to the ventrolateral orbital cortex (VLO), a prefrontal area where chemosensory, visual, and spatial
106 e connections are particularly prevalent for prefrontal areas, where they may play a prominent role i
107 ty that the brain responses to a meal in the prefrontal areas (which may be involved in the inhibitio
108 attern of reduced functional connectivity of prefrontal areas with limbic-paralimbic structures and e

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