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1 and early in both normal pregnancies and in pregnancy-induced hypertension.
3 l diabetes was more strongly associated with pregnancy-induced hypertension among women who received
4 measured serum perfluorinated compounds and pregnancy-induced hypertension and a negative associatio
6 ons of PFAS to be positively associated with pregnancy-induced hypertension and preeclampsia in a pop
7 ool entry, gravidity, maternal age at birth, pregnancy-induced hypertension, antepartum hemorrhage, a
9 for cancer progression; it is also linked to pregnancy-induced hypertension called preeclampsia and t
10 gram of home visitation by nurses can reduce pregnancy-induced hypertension, childhood injuries, and
11 , induced abortion, puerperal infection, and pregnancy-induced hypertension), knowledge of the contri
12 mes of preterm birth (< 37 weeks gestation), pregnancy-induced hypertension, low birth weight (< 2,50
13 nd PFOS were both positively associated with pregnancy-induced hypertension (n = 106), with adjusted
14 enerate case groups of stillbirth (n = 106), pregnancy-induced hypertension (n = 224), preterm birth
16 l, 1.3 to 1.5), and preterm delivery without pregnancy-induced hypertension (odds ratio, 1.1; 95 perc
17 95 percent confidence interval, 1.3 to 1.7), pregnancy-induced hypertension (odds ratio, 1.4; 95 perc
18 linked to the survey, PFOA was unrelated to pregnancy-induced hypertension or preterm birth but show
20 have shown an increased sympathetic drive in pregnancy-induced hypertension (PIH) and preeclampsia (P
22 tion between birth weight and later risk for pregnancy-induced hypertension (PIH), a disorder associa
23 ween estimated PFOA exposure and stillbirth, pregnancy-induced hypertension, preterm birth, or indice
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