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1 neage restriction and cell commitment during preimplantation development.
2 contents are required for fertilization and preimplantation development.
3 failed fertilization or embryo arrest during preimplantation development.
4 eres comprising a single embryo during human preimplantation development.
5 uct function in supporting fertilization and preimplantation development.
6 s, DNA methylation is globally erased during preimplantation development.
7 ole in maintaining energy homeostasis during preimplantation development.
8 ly disrupts oocyte chromatin methylation and preimplantation development.
9 nd the inner cell mass (ICM) lineages during preimplantation development.
10 Ctr9 and Rtf1, are required during mammalian preimplantation development.
11 tic reprogramming is required for successful preimplantation development.
12 l genome in zygotes is gradually lost during preimplantation development.
13 developing intestinal epithelium and during preimplantation development.
14 ed proteins and multi-component complexes in preimplantation development.
15 e function and chromosome segregation during preimplantation development.
16 ividual HDACs have distinct functions during preimplantation development.
17 the acetylation state of histone H4K5 during preimplantation development.
18 ylases (HDAC) HDAC1, HDAC2, and HDAC3 during preimplantation development.
19 l roles during oogenesis, fertilization, and preimplantation development.
20 Zscan4 thus seems to be essential for preimplantation development.
21 ether PI3K is intrinsically activated during preimplantation development.
22 the question whether they play any roles in preimplantation development.
23 TP production to survive and proceed through preimplantation development.
24 h notable differences between species during preimplantation development.
25 ing oocyte growth and is required for normal preimplantation development.
26 re present in oocytes and persist throughout preimplantation development.
27 tion, which has been shown to persist during preimplantation development.
28 microarrays to explore gene activity during preimplantation development.
29 blastomas arising in Ptc1+/- mice can direct preimplantation development.
30 are required for maintaining imprints during preimplantation development.
31 As representing genes expressed during human preimplantation development.
32 s to follow the fate of their progeny during preimplantation development.
33 has been investigated at different stages of preimplantation development.
34 ns of oocyte-derived Dnmt1o and Dnmt1 during preimplantation development add further support to the n
36 t genome-scale DNA methylation maps of human preimplantation development and embryonic stem cell deri
37 model indicates that Mcl-1 is essential for preimplantation development and implantation, and sugges
38 s directly involved in genome defense during preimplantation development and in PGCs at the time of g
39 GA), begins during the 2-cell stage in mouse preimplantation development and marks a vital transition
40 or the study of gene expression during human preimplantation development and obviate the need for res
41 at embryos without Stella are compromised in preimplantation development and rarely reach the blastoc
42 ally suppressed during oocyte maturation and preimplantation development and that endo-siRNAs, rather
43 horylated Akt was detected throughout murine preimplantation development, and its presence at the pla
44 evelopment to blastocyst, the final stage of preimplantation development, and promoted mitochondrial
45 tage-specific expression in ICM cells during preimplantation development, and show that Mbd3 is requi
46 es exhibiting differential expression during preimplantation development are of unknown identity and/
47 is a prolonged process that extends through preimplantation development, as characterized by slow co
48 are not only expressed in oocytes and during preimplantation development, but also display distinct s
49 e methylation acquired in the oocyte through preimplantation development, but become fully methylated
50 fertilized and their embryos undergo normal preimplantation development, but the embryos fail to imp
51 highlights significant differences in human preimplantation development compared with mouse and prov
53 al low protein diet exclusively during mouse preimplantation development (Emb-LPD) is sufficient to c
56 iling the expression of microRNAs throughout preimplantation development identified several candidate
59 hat seven isotypes of PKC are present during preimplantation development in mouse, some are of matern
61 We focus on how imprinted XCI occurs during preimplantation development, including a brief discussio
62 nal and zygotic Dgcr8 alleles did not impair preimplantation development, including the determination
63 of DNA methylation patterns during mammalian preimplantation development involves the concurrent main
64 me-wide DNA methylation reprogramming during preimplantation development is a dynamic balance between
66 are present in the blastocyst, each stage of preimplantation development is characterized by a differ
73 n of oocyte maturation and the initiation of preimplantation development occur during transcriptional
74 Our findings show that the dynamics during preimplantation development of gene inactivation and acq
76 tivation of the FGF signaling pathway during preimplantation development of the mouse embryo is known
79 he single-cell transcriptional data of mouse preimplantation development, our algorithm outperforms c
80 protein, is correlated with a faster rate of preimplantation development (Ped fast phenotype) in mice
81 ucture of chromatin and its reprogramming in preimplantation development remain poorly understood.
82 vation, the full range of their functions in preimplantation development remains largely unknown.
83 nge in promoter utilization may occur during preimplantation development such that TATA-less promoter
84 duction of HDAC2 has no noticeable effect on preimplantation development, suggesting that individual
86 omplex and hybrid N-glycans in oogenesis and preimplantation development, the Mgat1 gene in oocytes w
90 nt, maternal HDAC3 protein was stable during preimplantation development, thereby preventing an exami
91 ingle-cell gene-expression analysis in human preimplantation development to instruct human stem cell
92 and breast cancer cells could support normal preimplantation development to the blastocyst stage but
93 os segregate three different lineages during preimplantation development: trophoblast, epiblast and h
94 ted that H19 imprinting could be lost during preimplantation development under certain culture condit
95 powerful tool for the study of fertilization/preimplantation development, vertical viral gene transmi
97 tional regulation during human oogenesis and preimplantation development, we defined stage-specific t
98 d trafficks to the eight-cell nucleus during preimplantation development, where it maintains DNA meth
99 rnal LPD treatment during post-fertilization preimplantation development which may reflect the relati
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