ライフサイエンスコーパス: preinvasive lesion

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1 g cancer types (30 of 40; 75%) but not in 10 preinvasive lesions.

2 mutagenesis in both prostate cancer and its preinvasive lesions.

3 lung cancers and in 78% of the preneoplastic/preinvasive lesions.

4 of 30 mo, range 4-152) of 164 subjects with preinvasive lesions (80 high grade and 84 low grade at i

5 In preinvasive lesions, amplification of the PIK3CA and ove

6 even lesions of the head and neck, including preinvasive lesions and benign lesions associated with c

7 lung cancers, 70% of normal or preneoplastic/preinvasive lesions associated with lung cancer, and 49%

8 ic infiltration even around the lowest grade preinvasive lesions, but immunosuppressive cells, includ

9 0% of all EL-Kras/Tgfbr1(+/-) mice developed preinvasive lesions compared with 100% of EL-Kras (wild-

10 creasing molecular characterisation of these preinvasive lesions, data will be available for how fact

11 mors because the molecular phenotypes of the preinvasive lesions differed at various sites.

12 Presence of preinvasive lesions, especially high-grade lesions, may

13 47 normal, mildly abnormal, or preneoplastic/preinvasive lesions found in smokers without lung cancer

14 Effector T cells, however, were scarce in preinvasive lesions, found in only a subset of advanced

15 We found a significant increase in CN in preinvasive lesions graded severe dysplasia or higher.

16 1%) of the adenocarcinomas and preneoplastic/preinvasive lesions had smaller chromosome areas of 3p a

17 mas and the frequency of PIK3CA mutations in preinvasive lesions has not been explored.

18 n individual tumors; occurs in preneoplastic/preinvasive lesions in smokers with and without lung can

19 ied six susceptibility loci that affect mean preinvasive lesion multiplicity.

20 We asked whether we could classify preinvasive lesions of airway epithelium according to th

21 (c) address the interpretation of benign and preinvasive lesions of the mouse lung.

22 LCs occur both at preinvasive lesion sites and elsewhere in the bronchial

23 94% of human breast carcinomas and in 95% of preinvasive lesions, such as ductal and lobular carcinom

24 wth appear to occur after the development of preinvasive lesions, suggesting that these agents inhibi

25 lleles of Nkx3.1 leads to the development of preinvasive lesions that resemble PIN.

26 allow the progression from a precancerous or preinvasive lesion to a malignant tumor.

27 The progression from preinvasive lesion to invasive carcinoma is a critical s

28 rs can become lethal when they progress from preinvasive lesions to invasive carcinomas.

29 ell populations from prostate cancer and its preinvasive lesions using laser capture microdissection,

30 xpression levels in invasive lesions than in preinvasive lesions using samples obtained by laser capt

31 iary care referral center, 164 subjects with preinvasive lesions were monitored up to 12.5 years by r

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