1 h suggests it operates via the activation of prejunctional A1 adenosine receptors.

2                  All the alpha agonists have prejunctional (alpha 2) as well as postjunctional (alpha

3 kDa fragments, which occurred ahead of other prejunctional and postjunctional components, suggesting

4 ine exocytosis was enhanced by activation of prejunctional angiotensin AT1-receptors and attenuated b

5             The M2 receptor is an inhibitory prejunctional autoreceptor.

6                                              Prejunctional beta2-adrenoceptors can enhance neuronal a

7 pe mice, adenosine (500 microm-1 mm) reduced prejunctional Ca(2+) currents simultaneously with a redu

8 released with sufficient rapidity to mediate prejunctional depression, outside-out patches containing

9  induces formation of autophagic vacuoles in prejunctional dopaminergic axons with associated decreas

10 is followed rapidly by adrenoceptor-mediated prejunctional enhancement of parasympathetic nerve-smoot

11                    We previously showed that prejunctional histamine H3-receptors downregulate norepi

12                                      Because prejunctional histamine H3-receptors inhibit transmitter

13 2, act to inhibit cholinergic responses by a prejunctional mechanism in human isolated bronchi.

14 ne, is the primary physiological mediator of prejunctional neuromuscular depression.

15 than a postjunctional mediator; (b) VIP is a prejunctional neurotransmitter that causes release of ev

16 he anti-nociceptive actions of adenosine via prejunctional P1(A1) purinoceptors in the spinal cord an

17 ously decreased the calcium component of the prejunctional perineural current TPA and PDBu also inhib

18 e potentials, EPPs) and the component of the prejunctional perineural voltage change associated with