ライフサイエンスコーパス: premature birth

1 ing cause of neurological deficits following premature birth.

2 ory response, and is closely associated with premature birth.

3 d treatment of lung diseases associated with premature birth.

4 e important players in regulating subsequent premature birth.

5 the neonatal lung parenchyma associated with premature birth.

6 glucocorticoid therapies for lung defects in premature birth.

7 most common causes of death associated with premature birth.

8 tors, which in turn may cause miscarriage or premature birth.

9 erized by a thick desquamating epidermis and premature birth.

10 ociation does not seem to be associated with premature birth.

11 y dysplasia (BPD) is a major complication of premature birth.

12 tricular white matter injury in survivors of premature birth.

13 neurodevelopmental morbidity in survivors of premature birth.

14 in the occurrence of other complications of premature birth.

15 d to fetal growth restriction rather than to premature birth.

16 pmental disorders and other complications of premature birth.

17 Is) are associated with approximately 27% of premature births.

18 ; 93.6% [95% CI, 82.5%-98.7%]) and extremely premature birth (3/47; 6.4% [95% CI, 1.3%-17.5%]).

19 Premature birth accounts for the majority of fetal morbi

20 oxin in placental blood were associated with premature birth, acute chorioamnionitis, and elevated pr

21 set of their rheumatic disease, particularly premature births (also seen in RA women after disease on

22 ty lupus during pregnancy leads to increased premature birth and a decrease in live births, with almo

23 fications because of the association between premature birth and accompanying hypoxia, and increased

24 d no underlying medical conditions, although premature birth and asthma were more frequent among hosp

25 tal NTHi disease is strongly associated with premature birth and causes significant morbidity and mor

26 humans that cBF integrity is impaired after premature birth and links neonatal complications with lo

27 pe 1 (HIV-1) infection increases the risk of premature birth and other adverse outcomes of pregnancy.

28 ge in the motor impairment that results from premature birth and suggest that therapies designed to p

29 quadriplegic cerebral palsy associated with premature birth and typical periventricular leukomalacia

30 a (e.g., asthma, cardiovascular diseases, or premature birth) and who were younger than two years of

31 sion in pregnancy, fetal growth restriction, premature birth, and fetal and maternal mortality (1).

32 kinase levels, rates of congenital anomaly, premature birth, and growth parameters in infants were c

33 ubmicroscopic infections on maternal anemia, premature birth, and low birth weight.

34 ystocia, spontaneous and medically indicated premature birth, and stillbirth.

35 sk factor for fetal loss, preeclampsia (PE), premature birth, and the occurrence of any placenta-medi

36 l of UPI and oxygen-fluctuations and removed premature birth as a confounding factor.

37 of respiratory distress syndrome (RDS) after premature birth by altering vasoreactivity and increasin

38 magnesium sulfate, administered soon before premature birth, can reduce the high rate of cerebral pa

39 urity is a sight-threatening complication of premature birth caused by nitro-oxidative insult to the

40 PR-VEP latency is not affected by premature birth, demonstrating that the maturation of th

41 t known whether the stresses associated with premature birth disrupt regionally specific brain matura

42 al and experimental studies demonstrate that premature birth disrupts alveolarization, decreasing the

43 Premature birth disrupts brain development and is associ

44 ns included primiparity in 8 patients (42%), premature birth in 6 (32%), and maternal diabetes mellit

45 second half of pregnancy was associated with premature birth in 8 of 28 cases (28.6%; 95% CI, 13.2%-4

46 Premature birth in conjunction with extremely low birth

47 These results suggest that premature birth in the absence of identifiable retinal o

48 igravidae (OR, 6.09; 95% CI, 1.16-31.95) and premature births in multigravidae (OR, 2.25; 95% CI, 1.1

49 (ROP) affects only premature infants, but as premature births increase in many areas of the world, RO

50 Premature birth is a major risk factor for multiple brai

51 al lipid species early in the progression to premature birth is an important step toward discovering

52 When a premature birth is anticipated, antenatal corticosteroid

53 We report for the first time that premature birth is associated with altered cerebellar me

54 Premature birth is associated with numerous complex abno

55 To determine whether premature birth itself alters visual cortical function,

56 Such a group comprises premature birth, low-birth-weight, congenital anomalies,

57 pregnancy, the higher the incidence of very premature birth (<34 weeks) and severe small for gestati

58 for Congenital Heart Surgery risk category, premature birth, major noncardiac structural anomaly, an

59 To determine the effect of premature birth on neurogenesis, we used a rabbit model

60 effects of neonatal hypoxia associated with premature birth on the central nervous system are well k

61 y and mortality in the infant as a result of premature births or genetic or drug-induced NMDA recepto

62 R, 0.37; 95% CI, 0.19-0.75; P < .001), fewer premature births (OR, 0.44; 95% CI, 0.24-0.79; P < .01),

63 owth in the neonatal period inevitable after premature birth, or is it associated with specific medic

64 Children with urinary tract anomalies, premature birth, or major comorbidities were excluded fr

65 ay, 53 toddlers with other conditions [e.g., premature birth, prenatal drug exposure], 64 toddlers wi

66 n are important predictors of fetal loss and premature birth, respectively.

67 prematurity syndrome (IPS), characterized by premature birth, respiratory distress, and edematous ski

68 tic cohort, children who had had an LBW or a premature birth showed the greatest responses to ozone.

69 erences in the overall rates of miscarriage, premature births, small full-term births, or neonatal de

70 mes, including the frequency of miscarriage, premature births, small full-term infants, perinatal dea

71 nominator for postnatal phenomena related to premature birth, such as neonatal mortality and cerebral

72 crostructure is vulnerable to the effects of premature birth, suggesting a mechanism for the adverse

73 Premature birth terminates the hypoxic in utero environm

74 chronic respiratory disease associated with premature birth that primarily affects infants born at l

75 isability and cerebral palsy in survivors of premature birth, the cellular-molecular mechanisms by wh

76 orly characterized pulmonary complication of premature birth; the current definition is based solely

77 infections predispose low birth weights and premature birth, then such outcomes should be apparent w

78 ciation between family history of asthma and premature birth was found, but it was not associated wit

79 h-club organization remains intact following premature birth, we reveal significant disruptions in bo

80 Of interest, young age and premature birth were independently associated with atten

81 It is observed among survivors of premature birth who have been treated with prolonged sup

82 tes were studied in a baboon model of severe premature birth with respiratory distress.

83 l the end of pregnancy, we hypothesized that premature birth would disrupt interneuron production and