ライフサイエンスコーパス: premature ventricular contraction

1 ystolic potentials and pace-maps matching VT/premature ventricular contraction.

2 subjects (n=10) and 1 subject diagnosed with premature ventricular contractions.

3 onduction blocks, as well as bradycardia and premature ventricular contractions.

4 S:R ratios, polyphasic R-waves and frequent premature ventricular contractions.

5 RVOT-VAs (10 with frequent [>1000/24 hours] premature ventricular contractions, 14 with ventricular

6 , the number of hourly episodes of nocturnal premature ventricular contractions (66+/-117 versus 18+/

7 ut of 334 consecutive patients undergoing VT/premature ventricular contraction ablation, 7 patients u

8 ts referred for left ventricle outflow tract premature ventricular contraction ablation, an aortic va

9 a/atrial fibrillation accounted for >50% and premature ventricular contractions accounted for <10% of

10 ong-term isoproterenol infusion also induced premature ventricular contractions and atrioventricular

11 tric instability, including the frequency of premature ventricular contractions and sustained ventric

12 Premature ventricular contractions and ventricular tachy

13 trial tachycardia/atrial fibrillation; 16.6% premature ventricular contractions; and 8.6% captured as

14 stimulation elicited arrhythmias, including premature ventricular contractions, atrioventricular hea

15 (213 men, age 41.5+/-16 years) referred for premature ventricular contractions evaluation or suspect

16 14%) compared with 0 of 30 (0%) (p = 0.048), premature ventricular contractions in 17 of 30 (57%) com

17 ves appeared in 3 of 18 (17%), superior axis premature ventricular contractions in 21 of 25 (84%), an

18 only beta2-stimulation promoted Ca-mediated premature ventricular contractions in heart failure.

19 In contrast, VT or premature ventricular contractions in the setting of a s

20 vents, sinus pauses, atrioventricular block, premature ventricular contractions, non-sustained ventri

21 Superior axis premature ventricular contractions occurred only in gene

22 , absence epilepsy (petit mal) episodes, and premature ventricular contractions of the heart.

23 plex ventricular ectopic activity, including premature ventricular contractions of the outflow tract

24 ng PI3Kgamma (PI3Kgamma(-/-)) showed runs of premature ventricular contractions on adrenergic stimula

25 ion fraction 59+/-7.3%) with drug refractory premature ventricular contractions or ventricular tachyc

26 l VT and borderline if polymorphic couplets, premature ventricular contractions, or nonsustained mono

27 f 9 [78%] vs. 1 of 10 [10%], p = 0.006), and premature ventricular contractions originating from the

28 Premature ventricular contractions originating in the le

29 y reduced ventricular arrhythmias (-70+/-22% premature ventricular contractions; P<0.05).

30 Variability in premature ventricular contraction (PVC) coupling interva

31 High idiopathic premature ventricular contractions (PVC) burden has been

32 study sought to examine whether suppressing premature ventricular contractions (PVC) using radiofreq

33 tricular tachycardia (NSVT) in patients with premature ventricular contractions (PVCs) and heart fail

34 resenting for catheter ablation suggest that premature ventricular contractions (PVCs) are a modifiab

35 tricular tachycardia (VT) and high burden of premature ventricular contractions (PVCs) are common in

36 ctive of this study was to determine whether premature ventricular contractions (PVCs) arising from t

37 Left ventricular (LV) dyssynchrony caused by premature ventricular contractions (PVCs) has been propo

38 Monomorphic premature ventricular contractions (PVCs) have been show

39 However, certain patients with frequent premature ventricular contractions (PVCs) or VT and tach

40 f idiopathic ventricular tachycardia (VT) or premature ventricular contractions (PVCs) originating fr

41 Premature ventricular contractions (PVCs), couplets, and

42 tained ventricular tachycardia (NSVT), or 3) premature ventricular contractions (PVCs).

43 +/- 14 years, 21 female) with outflow tract premature ventricular contractions (PVCs)/VT.

44 cardia (VT) and frequent ventricular ectopy (premature ventricular contractions [PVCs] >10/h) was ass

45 Premature ventricular contraction rate increased with ex

46 cular tachycardia, ventricular couplets, and premature ventricular contractions showed greater amount

47 aused numerous cardiac arrhythmias including premature ventricular contractions, tachycardia, and hig

48 tained ventricular tachycardia, couplets, or premature ventricular contractions, the extent of interm

49 leads to the development of closely coupled premature ventricular contractions via a phase 2 reentra

50 dical history of hypertension and occasional premature ventricular contractions was found on routine

51 e patients with left ventricle outflow tract premature ventricular contraction were included.

52 Premature ventricular contractions were common in ARVC p

53 ositive if there were either (1) polymorphic premature ventricular contractions with >/=1 couplet or

54 hic morphology of ventricular tachycardia or premature ventricular contractions with left bundle bran

55 and rare sinus beats competing with numerous premature ventricular contractions with right and/or lef