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1 and are important to define in treatment of premenstrual syndrome.
2 and was well tolerated by women with severe premenstrual syndrome.
3 or placebo on symptoms in 20 women with the premenstrual syndrome.
4 contributed only modestly to the etiology of premenstrual syndrome.
5 effects of B vitamins in the development of premenstrual syndrome.
6 tory response is unknown in the treatment of premenstrual syndrome.
7 onin reuptake inhibitor in women with severe premenstrual syndrome and determined the effects of post
8 ogenous 3alpha,5alpha-THP withdrawal such as premenstrual syndrome and postpartum or postmenopausal d
9 reuptake inhibitors are effective for severe premenstrual syndrome and premenstrual dysphoric disorde
10 menopausal women, alleviate the symptoms of premenstrual syndrome, and reduce persistent urinary tra
11 fter adjusting for smoking, body mass index, premenstrual syndrome, hot flashes, poor sleep, health s
16 (4) and delta subunits, using a rat model of premenstrual syndrome (PMS) in which 1-3 mM alcohol pref
17 her minerals might impact the development of premenstrual syndrome (PMS) through multiple mechanisms,
18 onses to placebo medication of patients with premenstrual syndrome (PMS) who were randomly assigned i
19 rlying hypertension might also contribute to premenstrual syndrome (PMS), but whether women with PMS
24 About 5-8% of women thus suffer from severe premenstrual syndrome (PMS); most of these women also me
25 ion, including history of depression, severe premenstrual syndrome, poor sleep, age, race, and employ
26 roids have been implicated in the genesis of premenstrual syndrome, postpartum depression, and other
27 depressant to determine whether efficacy for premenstrual syndrome/premenstrual dysphoric disorder is
32 received the same regimen or in 5 women with premenstrual syndrome who were given placebo hormone dur
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