ライフサイエンスコーパス: prenatal period

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1 ildren exposed to antidepressants during the prenatal period.

2 vide information on exposure timing over the prenatal period.

3 ngements in the brain takes place during the prenatal period.

4 ks would be highest for exposures during the prenatal period.

5 s of this malformation at any age, including prenatal period.

6 gluconeogenic enzymes are not induced in the prenatal period.

7 operating prior to conception and during the prenatal period.

8 ton species) after E16 through the remaining prenatal period.

9 ored regularly in such trials throughout the prenatal period.

10 egions of mantle dentine formed at different prenatal periods.

11 ests that environmental exposures during the prenatal period and early childhood years increase the r

12 ources of the disparities should include the prenatal period and early life.

13 re effective interventions are needed in the prenatal period and first 3 years after birth for the mo

14 nterfere with perceptual learning during the prenatal period and provide additional evidence for an o

15 ect in allergy may be established during the prenatal period and that the protective effect may origi

16 ssed in neural tissue, especially during the prenatal period, and enriched for biological pathways in

17 would occur more in the postpartum than the prenatal period, and would be more prevalent with bipola

18 s and lack of detailed information about the prenatal period are major problems with current approach

19 al brain development to date, confirming the prenatal period as a time of considerable epigenomic pla

20 ng critical examination of preconception and prenatal periods as vulnerable to environmental insults

21 Much of this work has focused on the prenatal period, because infections during pregnancy hav

22 The authors explored this issue during the prenatal period by pharmacologically elevating physiolog

23 re than 40% of women hospitalized during the prenatal period for a bipolar or a psychotic condition w

24 ys were more vulnerable to stress during the prenatal period, girls were more affected by postnatal s

25 that the availability of choline during the prenatal period influences neural and cognitive developm

26 rexpress CAT in RPTCs) were studied from the prenatal period into adulthood.

27 We found that the prenatal period is a critical time for shaping the immun

28 exposure to stressful life events during the prenatal period is associated with an increased risk of

29 enia and with accumulating evidence that the prenatal period is involved in the origins of this disea

30 The early prenatal period is therefore an important new window for

31 However, treatment to the mother, in the prenatal period, may provide the possibility of preventi

32 s of the effects of imprinted genes from the prenatal period onwards.

33 The fastest changes occur during the prenatal period, slow down markedly after birth and cont

34 rformance and exposure to mercury during the prenatal period, the neonatal period (birth to 28 days),

35 When treatment was begun in the prenatal period, the rate of HIV transmission was 6.1 pe

36 -COMP-induced chondrocyte pathology from the prenatal period to adolescence.

37 n-based cohort of children followed from the prenatal period to age 13.

38 lying social and emotional behavior from the prenatal period to the end of life.

39 environmental exposures beginning during the prenatal period) to identify modifiable factors that aff

40 Rather than focusing exclusively on the prenatal period, we describe a life cycle approach to im

41 ignaling required for lung maturation in the prenatal period were epigenetically deregulated and coul

42 ensitive to thyroid hormone during the early prenatal period, when the fetus is entirely dependent on

43 rat, several cell types are born during the prenatal period, while others are born postnatally.

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