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3 the human TM by elevated IOP: interleukin-6, preprotachykinin-1, secretogranin-II, cathepsin-L, strom
4 d and dramatic increase in the expression of preprotachykinin A (a precursor of SP) mRNA and SP in pr
6 e P, and of other neurokinins encoded by the preprotachykinin A (PPT-A) gene, from unmyelinated nerve
8 e we report that mice with disruption of the preprotachykinin A gene, which encodes SP and neurokinin
9 activity and the neuroprotection observed in preprotachykinin A null mice are caused by the extinctio
14 recently raised mice with a deletion of the preprotachykinin-A gene, which encodes the peptides subs
15 and noncatecholaminergic neurons containing preprotachykinin and prepro-enkephalin (PPE) mRNAs were
16 e P (SP)-like peptide that is encoded by the preprotachykinin C (PPT-C) gene recently identified in m
20 s among reporter gene activities, mRNA (beta-preprotachykinin I), and protein levels for substance P.
22 in the expression of genes for cytokines and preprotachykinin-I (PPT-I) in both BCCs and stromal cell
25 ptides (eg, substance P [SP]) encoded by the preprotachykinin-I gene mediate distinct hematopoietic e
29 ydroxytryptamine; 5-HT) neurotransmission on preprotachykinin (PPT) and preproenkephalin (PPE) mRNA l
31 on were used to assess the expression of the preprotachykinin (PPT) gene, substance P, and NK1 in dev
32 ation affects intraneuronal levels of SP and preprotachykinin (PPT) messenger RNA (mRNA) in the senso
34 phetamine to increase preprodynorphin (PPD), preprotachykinin (PPT), preproenkephalin (PPE), and secr
35 site were subjected to Northern analysis of preprotachykinin (PPT), preproenkephalin (PPE), and zif/
36 r with increased (by 82%; P < 0.05) mRNA for preprotachykinin (the substance P precursor) in the trig
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