ライフサイエンスコーパス: pressor

1 n >2.4 mg/dL, pH < 7.30, and the need for >2 pressors.

2 These findings suggest that the pressor action of [Pyr(1) ]apelin-13 in the RVLM of norm

3 regulate the central nervous system-mediated pressor action of Ang II.

4 olved in circulatory homeostasis through the pressor action of angiotensin II at its AT1 receptor.

5 s, inhibition of which increases sympathetic pressor activity and heart rate.

6 data suggest that tachykinins mediate their pressor activity by increasing the excitability of spina

7 tion." The agents we utilize as sedative and pressor agents have considerable effects on immune funct

8 anaged with countermeasures and short-acting pressor agents if necessary.

9 SB334867) attenuated hypercapnic gas-induced pressor and anxiety responses, without altering the robu

10 nt of central prostaglandin synthesis on the pressor and bradycardic effect of cytidine 5'-diphosphoc

11 te in this brain region nearly abolishes the pressor and bradycardic effects of renin infused in the

12 pretreatments almost completely blocked the pressor and bradycardic responses to CDP-choline while n

13 0.5 microg kg(-1); i.a.) that attenuated the pressor and cardioaccelerator responses to both contract

14 e hindlimb muscles were freely perfused, the pressor and cardioaccelerator responses to either contra

15 The pressor and cardioaccelerator responses to either contra

16 (5 microg kg(-1), i.a.) not only blocked the pressor and cardioaccelerator responses to lactic acid a

17 decerebrate cats if amiloride attenuated the pressor and cardioaccelerator responses to static contra

18 projections were found ipsilaterally in the pressor and depressor areas of the medulla and the spina

19 e CVLM and RVLM were functionally defined by pressor and depressor responses to microinjected GABA (5

20 he subfornical organ impairs Ang II-mediated pressor and drinking responses at least by inhibiting th

21 via Ad-Syn-MIF gene transfer attenuated the pressor and drinking responses produced by icv-injected

22 n levels in the PVN and produced significant pressor and drinking responses that were inhibited by PV

23 allopurinol impaired fetal alpha1-adrenergic pressor and femoral vasopressor responses and enhanced t

24 However, with HBOC-201, pressor and fluid requirements were reduced by half, whi

25 roreflex slopes were obtained using the peak pressor and heart rate responses to increasing doses of

26 In the MI rats, the pressor and RSNA responses to contraction were reduced b

27 rapidly evolving renal failure, hypotension, pressor and steroid use, and variable nutritional suppor

28 heart rate and CO responses and enhanced the pressor and SVR responses to sustained stress (1 min exp

29 The pressor and sympathetic responses to ischaemic muscle co

30 By contrast, the [Pyr(1) ]apelin-13 induced pressor and sympathoexcitatory effects were abolished by

31 Although reduced, pressor and sympathoexcitatory responses due to inhibiti

32 ificantly reduced the central Ang II-induced pressor and sympathoexcitatory responses, decreased base

33 The pressor and tachycardic responses to ARCN stimulation at

34 itatory effects on RVLM neurons resulting in pressor and tachycardic responses, (2) these responses w

35 was reversed i.e., the cold stimulus induced pressor and tachycardic responses, augmented glutamate,

36 gestational age was a greater determinant of pressor and vasopressor reactivity.

37 s responded to acute hypoxaemia with similar pressor and vasopressor responses compared to singleton

38 Pressor and vasopressor responses to all agonists were g

39 ot atmospheric air, resulted in respiratory, pressor, and bradycardic responses, as well as anxiety-l

40 Nox1 overexpression augments the oxidative, pressor, and hypertrophic responses to Ang II, supportin

41 fter slow breathing, mental arithmetic, cold pressor, and sublingual nitroglycerin.

42 Slow-pressor AngII elevated blood pressure in AOF females and

43 es are paralleled in rodents following "slow-pressor" angiotensin II (AngII) administration: young ma

44 o investigate the projections of this caudal pressor area (CPA) to the medulla and pons.

45 l area where rostral ventrolateral medullary pressor area (RVLM) is located.

46 caudal C1 catecholamine neurons, and caudal pressor area receive dense axonal projections, generally

47 , lateral parabrachial nucleus (LPB), caudal pressor area, and lamina I of the spinal trigeminal nucl

48 ction in CNS neurons plays a key role in the pressor, bradycardic, and dipsogenic actions of Ang II i

49 To extend the in vitro studies, the pressor, bradycardic, and drinking responses to intracer

50 he magnitude of both the sympathoadrenal and pressor but not the adrenocortical response to stress.

51 administration attenuates perceived pain and pressor, but not MSNA burst frequency, responses during

52 GsMTx4 reduced the pressor, cardioaccelerator and renal sympathetic nerve r

53 4 into the jugular vein had no effect on the pressor, cardioaccelerator, or renal sympathetic nerve r

54 panel of tasks: breath-hold challenge, cold-pressor challenge, and heartbeat perception during and o

55 ttempters tolerated the breath-hold and cold-pressor challenges for significantly longer and displaye

56 eased after L-NMMA and saline+PE (volume and pressor control for L-NMMA).

57 rial supply of the hindlimb reduced the peak pressor (control: 24 +/- 2, GsMTx4: 14 +/- 3 mmHg, P < 0

58 rial supply of the hindlimb reduced the peak pressor (control: 24 +/- 5, GsMTx4: 12 +/- 5 mmHg, P < 0

59 Adrenal medullectomy did not affect the pressor (DeltaMAP = 12 +/- 2 mm Hg), tachycardic (DeltaH

60 Accordingly the pressor dose 33 and pressor dose 100 values were lowered following lipopolys

61 ure by 33 and 100 mm Hg (pressor dose 33 and pressor dose 100) was determined.

62 Accordingly the pressor dose 33 and pressor dose 100 values were lowered

63 rease systolic pressure by 33 and 100 mm Hg (pressor dose 33 and pressor dose 100) was determined.

64 Fourteen day administration of a slow-pressor dose of AngII in male mice was associated with t

65 d with minipumps containing saline or a slow-pressor dose of angiotensin II (0.25 mg.kg(-1).d(-1)).

66 An equi-pressor dose of angiotensin II had no effect on myogenic

67 One healthy subject died after a pressor dose of angiotensin II was infused continuously

68 The pressor dose of norepinephrine necessary to increase sys

69 ced renal cortical tissue PO2 more than equi-pressor doses of phenylephrine, probably because it redu

70 ificantly greater than those induced by equi-pressor doses of phenylephrine.

71 Furthermore, at pressor doses vasopressin improved cerebral perfusion.

72 Similar to humans, "slow-pressor" doses of angiotensin II (AngII) increase blood

73 It is concluded that the pressor effect due to a vasoactive oxygen carrier may be

74 in II, but not norepinephrine, had a greater pressor effect in denervated animals.

75 er/thr phosphatases dampen the ERK-dependent pressor effect of ethanol in normotensive rats.

76 cts tonically to attenuate the ERK-dependent pressor effect of ethanol or acetaldehyde in normotensiv

77 PyKYNE-losartan fully antagonized the Ang II pressor effect, albeit with 4-fold potency reduction (th

78 PNU-37883A had no pressor effect, except in the presence of pentolinium (p

79 cariporide and epinephrine prompted adequate pressor effects during chest compression and facilitated

80 xtent MC3 receptors in the RVLM, and (3) the pressor effects of ACTH were mediated via sympathetic ac

81 We conclude that the central and pressor effects of angiotensin II are critical for T-cel

82 Rho-kinase pathway in the maintenance of the pressor effects of vasopressin in endotoxemic rats.

83 rbidities, angiotensin II dose and duration, pressor effects, other physiologic and side effects, and

84 ings suggest that CSR1 is a potent tumor sup-pressor gene.

85 600 IU/L, creatinine >2.0 mg/dL, intubation, pressors) had an AUROC for transplant/death of 0.899 whi

86 esia index, calculated as the change in cold pressor hand withdrawal latency (HWL) before and after d

87 After 6 hrs with either pressor, hemodynamics were stable.

88 in system and thus dictates the level of the pressor hormone angiotensin-II.

89 ion has focused on mechanisms underlying its pressor hypersensitivity, which contrasts with the vascu

90 Equi-pressor infusion of phenylephrine did not significantly

91 one or both cell types in response to "slow-pressor" infusion of AngII.

92 range, 13 [0-25] vs 15 [0-25]; p = 0.8) and pressor/inotrope-free days (median and interquartile ran

93 ndogenous inhibitor of NOS, by virtue of its pressor or nitric oxide-depleting effects, or a combinat

94 1.6 to 2.7; P<0.001), hypotension requiring pressors (OR, 1.9 per quintile; 95% CI, 1.5 to 2.3; P<0.

95 ; and comatose state, intubation, receipt of pressors, or death in the emergency department.

96 1.3x10(-)(1)(0)) for the sensitivity to cold pressor pain in males, but not in females.

97 after both sleep conditions, we tested cold pressor pain tolerance before and 40-min after double-bl

98 hypnotic analgesia training to control cold pressor pain.

99 The NPY is a potent pressor peptide co-released with catecholamines during s

100 uld produce changes in endogenous levels and pressor reactivity to exogenous norepinephrine and vasop

101 The exercise pressor reflex (a neural drive originating in skeletal m

102 Although the exercise pressor reflex (EPR) and the chemoreflex (CR) are recogn

103 gated the interactive effect of the exercise pressor reflex (EPR) and the chemoreflex (CR) on the car

104 An exaggerated exercise pressor reflex (EPR) contributes to exercise intolerance

105 The skeletal muscle exercise pressor reflex (EPR) induces increases in heart rate (HR

106 The exercise pressor reflex (EPR) is an important neural mechanism th

107 The exercise pressor reflex (EPR) is defined by a rise in mean arteri

108 o activation of the skeletal muscle exercise pressor reflex (EPR) is exaggerated.

109 T: Contraction of muscle evokes the exercise pressor reflex (EPR), which is expressed partly by incre

110 ovides a novel working model of the exercise pressor reflex (EPR).

111 veness is mediated by an overactive exercise pressor reflex (EPR).

112 roxide scavenger, attenuated the exaggerated pressor reflex and reduced reactive oxygen species produ

113 cardiovascular responses during the exercise pressor reflex and simultaneously modulated medullary nN

114 Activation of the exercise pressor reflex during electrically induced static muscle

115 Potentially, effective treatment of exercise pressor reflex dysfunction may reduce the cardiovascular

116 echano-gated Piezo channels, on the exercise pressor reflex evoked by intermittent contraction of the

117 n important role in the exaggerated exercise pressor reflex found in rats with ligated femoral arteri

118 We conclude that GsMTx4 reduced the exercise pressor reflex in decerebrate rats and that the reductio

119 o-gated Piezo channels, reduced the exercise pressor reflex in decerebrate rats.

120 that P2 receptors contribute to the exercise pressor reflex in humans.

121 n modifying the exaggeration of the exercise pressor reflex in PAD and a reduction in the activity of

122 n modifying the exaggeration of the exercise pressor reflex in PAD and a reduction of the activity of

123 neficial effects on the exaggerated exercise pressor reflex in PAD rats.

124 ys an important role in evoking the exercise pressor reflex in rats with a compromised arterial blood

125 d the hypothesis that the augmented exercise pressor reflex in rats with a ligated femoral artery is

126 eceptors attenuated the exaggerated exercise pressor reflex in rats with ligated femoral arteries.

127 we found no effect of tiron infusion on the pressor reflex in rats with patent femoral arteries.

128 neficial effects on the exaggerated exercise pressor reflex in rats with peripheral artery disease (P

129 of the mechanical component of the exercise pressor reflex in rats.

130 tery disease, causes an exaggerated exercise pressor reflex in response to muscle contraction.

131 ory that selectively stimulates the exercise pressor reflex independent of central command and/or the

132 (ASICs), their contribution to the exercise pressor reflex is not known.

133 erents, sense the decrease in pH and evoke a pressor reflex known to increase mean arterial pressure.

134 However, given that exercise pressor reflex overactivity is known to elicit enhanced

135 10-min on the gallbladder induced consistent pressor reflex responses.

136 acid and E-2 led to a significantly greater pressor reflex than lactic acid alone in the presence of

137 arger reflex pressor response (i.e. exercise pressor reflex) than did static contraction of the contr

138 d (to simulate exercising muscle and evoke a pressor reflex), endomorphin-2 and naloxone resulted in

139 an increase in blood pressure (i.e. exercise pressor reflex).

140 n decerebrated rats exaggerates the exercise pressor reflex.

141 e in the metabolic component of the exercise pressor reflex.

142 voke the metabolic component of the exercise pressor reflex.

143 ension is mediated by an overactive exercise pressor reflex.

144 Aergic neurotransmission during the exercise pressor reflex.

145 king the metabolic component of the exercise pressor reflex.

146 eurotransmission that regulates the exercise pressor reflex.

147 voke the metabolic component of the exercise pressor reflex.

148 uscle play an important role in the exercise pressor reflex.

149 ension is mediated by an overactive exercise pressor reflex.

150 lobin (P = 0.007) and an attenuated exercise pressor reflex.

151 e ASIC currents and the lactic acid-mediated pressor reflex.

152 omponent of this reflex, termed the exercise pressor reflex.

153 regulate muscle nociception and the exercise pressor reflex.

154 te to evoke this reflex, termed the exercise pressor reflex.

155 h regulating muscle nociception and exercise pressor reflexes (EPRs), and P2Y1 has been linked to hea

156 ce nociceptive signals and modulate exercise pressor reflexes (EPRs).

157 onary resuscitation, mechanical ventilation, pressors, rescue thrombolysis, or surgical embolectomy,

158 The exercise pressor response (DeltaMAP) did not differ between condi

159 al artery was ligated evoked a larger reflex pressor response (i.e. exercise pressor reflex) than did

160 Stimulation of trigeminal nerve induces pressor response and improves cerebral blood flow (CBF)

161 R co-activation with hypoxia potentiates the pressor response and restricts blood flow to contracting

162 The [Pyr(1) ]apelin-13-mediated pressor response and the increased low frequency spectra

163 otentiated the magnitude and duration of the pressor response as well as the phosphatase inhibition e

164 diaphragm fatigue development, and a blunted pressor response compared to men.

165 key signaling intermediate in the transient pressor response elicited by acute injection of Ang II d

166 We show that the pressor response elicited by intra-RVLM ethanol (10 mug)

167 s for increasing MAP, and by correlating the pressor response evoked by these peptides to reported K(

168 the central CB(1)R-evoked sympathoexcitation/pressor response in conscious rats.

169 c.v.) angiotensin (ANG) II causes a reliable pressor response in the fetus at 90% gestation.

170 transmitter systems through which the apelin pressor response may occur within the RVLM.

171 inhibiting 50% of the Ang II-induced maximal pressor response of 25.5 mg/kg) relative to losartan.

172 nd function, which likely contributes to the pressor response of these hormones.

173 the enhancement of a carbachol (CCh)-evoked pressor response produced by prior NPY administration in

174 of ATP-sensitive P2X receptors enhances the pressor response seen when muscle mechanoreceptors are e

175 Cl produces a greater sympathoexcitatory and pressor response than infusion of hypertonic mannitol/so

176 naloxone resulted in a significantly greater pressor response than lactic acid alone, while administr

177 atment with prazosin reversed the HS-induced pressor response to a hypotensive response (from 121 +/-

178 The pressor response to acute infusion of Ang II was signifi

179 lts demonstrate that OA-NO(2) diminishes the pressor response to Ang II and inhibits AT(1)R-dependent

180 uption of the EP1 receptor blunted the acute pressor response to Ang II and reduced chronic Ang II-dr

181 Deletion of p47phox attenuated the pressor response to Ang II; however, coinfusion of pheny

182 portantly, 7 days after virus infection, the pressor response to angiotensin (Ang) II (200 pmol intra

183 xpression by C1 neurons is essential for the pressor response to angiotensin II and that this pathway

184 tly elevated in the transgenic mice, but the pressor response to angiotensin II was augmented.

185 for recording blood pressure, modulated the pressor response to aversive stress.

186 this pathway plays an important role in the pressor response to aversive stress.

187 , an ASIC agonist, but did not attenuate the pressor response to capsaicin, a TRPV1 agonist.

188 nses to lactic acid, but also attenuated the pressor response to capsaicin.

189 cies with respect to causing the exaggerated pressor response to contraction seen in rats with ligate

190 In the six ligated rats, however, the pressor response to contraction was attenuated by L16198

191 le autonomic traits: baroreflex function and pressor response to environmental stress.

192 Similarly, the pressor response to EP1-selective agonists sulprostone a

193 tential for a counteracting, anti-dipsogenic pressor response to hindbrain AngII allows for lingering

194 mg/kg, i.c.v.) showed a potentiation of the pressor response to i.c.v. ANG II, accompanied by bradyc

195 amiloride and APETx2 greatly attenuated the pressor response to lactic acid, an ASIC agonist, but di

196 R-floxed mice enabled demonstration that the pressor response to microinjection of angiotensin II int

197 ion by alpha,beta-methylene ATP enhanced the pressor response to muscle stretch by 42% in control ani

198 At 6 hrs, there was an attenuated pressor response to norepinephrine (p < .01) despite blo

199 In vivo, the pressor response to norepinephrine was markedly reduced

200 The pressor response to norepinephrine was reduced following

201 e afferents and the baroreflex evoked by the pressor response to phenylephrine (3-25 microg kg(-1), i

202 lated hypertension; they also show a reduced pressor response to salt loading.

203 The pressor response to static muscle contraction and alpha,

204 in animals, ruling out an effect of enhanced pressor response to stress following prenatal protein re

205 n-treated and DCM rats displayed a decreased pressor response to the intra-arterial administration of

206 Thus, we hypothesized that the pressor response to VR1 stimulation would be smaller and

207 phragm strength, women and men had a similar pressor response to work-matched inspiratory loading, in

208 Rs in C1 neurons induced a greater sustained pressor response when compared to the control viral-inje

209 ubtype 1 (VR1), inducing a neurally mediated pressor response, and (2) activation of ATP-sensitive P2

210 hrine with Ang II, which restored the Ang II pressor response, did not alter the protective effects o

211 and an increase in TPR, followed by a brief pressor response, effects which were unaffected by SR141

212 us system activity to activate an endogenous pressor response, improve cerebral perfusion, and decrea

213 ithout restraint in the setting of increased pressor response.

214 ludes attenuated blood flow and an augmented pressor response.

215 le afferents to the sympathetically mediated pressor response.

216 ated in the CB(1)R-evoked sympathoexcitation/pressor response.

217 tes displaying blunted slope and exaggerated pressor response.

218 lus doses of phenylephrine evoked attenuated pressor responses after CIH (P<0.01).These data suggest

219 , phenylephrine induced significantly higher pressor responses and greater vasoconstrictions in the o

220 Prenatal exposure to caffeine increased pressor responses and vasoconstrictions to phenylephrine

221 efferent processing of the BJR, and (2) the pressor responses elicited by alpha-methyl-5-HT were not

222 sms implicated in the sympathoexcitation and pressor responses elicited by central CB(1)R activation

223 A stimulation at acupoints P5-P6 reduced the pressor responses for at least 60-min.

224 different effects on central ANG II-induced pressor responses in fetuses at late gestation, and that

225 er, LPA(6) KO mice also displayed attenuated pressor responses to an adrenergic agent and abnormal bl

226 hibit significant hypertension and increased pressor responses to angiotensin II and endothelin-1; th

227 asal blood pressure was similar, however the pressor responses to both acute and chronic angiotensin

228 decreased vascular resistance, and elevated pressor responses to environmental (cold) stress.

229 ermittent contraction but did not reduce the pressor responses to femoral arterial injection of compo

230 In anesthetized rats, the pressor responses to increasing doses of norepinephrine

231 Pressor responses to increasing doses of phenylephrine a

232 echanical stimulus, but had no effect on the pressor responses to intra-arterial injection of alpha,b

233 Pressor responses to intravenous norepinephrine, vasopre

234 t, the high dose of amiloride attenuated the pressor responses to lactic acid, but also attenuated th

235 Pressor responses to norepinephrine and PNU-37883A (a va

236 mpathetic nerve activity (~70%), and blunted pressor responses to phenylephrine and angiotensin II.

237 renal sodium excretion or volume expansion; pressor responses to phenylephrine were enhanced and bar

238 od too rapidly for accurate measurement, and pressor responses to the injection of drug were greatly

239 Central ANG II-induced fetal pressor responses were not altered by PD123319 (0.8 mg/k

240 bute to the blunted sympathetically mediated pressor responses, because bolus doses of phenylephrine

241 Associated with the pressor responses, c-fos expression in the cardiovascula

242 ffect on seizure-induced sympathoexcitation, pressor responses, or tachycardia but abolished the prol

243 lly infused tyramine produced dose-dependent pressor responses, predicted by family history of hypert

244 hoexcitation (p </= 0.05), and abolished the pressor responses, tachycardia, and QT interval prolonga

245 ute rises in SNA was accompanied by enhanced pressor responses.

246 low catestatin predicts augmented adrenergic pressor responses.

247 kg (i.c.v.) abolished central ANG II-induced pressor responses.

248 saccharide, it caused a striking increase in pressor responsiveness (mean slope after lipopolysacchar

249 ipopolysaccharide caused a large increase in pressor responsiveness above lipopolysaccharide values.

250 y assesses whether alpha-2 agonists increase pressor responsiveness following lipopolysaccharide admi

251 enal sympathetic nerve activity and restored pressor responsiveness to both phenylephrine and angiote

252 Pressor responsiveness to norepinephrine decreased sligh

253 ssure, renal sympathetic nerve activity, and pressor responsiveness to phenylephrine and angiotensin

254 renal sympathetic nerve activity and reduced pressor responsiveness to vasopressors persisted.

255 lar to prolonged human sepsis, and decreased pressor sensitivity to norepinephrine.

256 time >12 hours, ICU stay >5 days, 3 or more pressors simultaneously, extensive alcohol abuse, cancer

257 on, the baroreflex was activated using brief pressor stimuli and the consequent cardiac (heart rate c

258 ip between SNA and vasoconstriction during a pressor stimulus, which increases BP and may be contra-i

259 gen saturations, but 90% (20 of 22) required pressor support.

260 al excitation augmented chemoreflex-mediated pressor, sympathoexcitatory and minute neural ventilatio

261 The systolic BP effect for the cold pressor task was apparent for women and for whites in ra

262 re, MSNA responses were compared when a cold pressor test (CPT) and lower body negative pressure (LBN

263 yocardial blood flow (MBF) responses to cold pressor test (CPT) and pharmacologic vasodilation was me

264 min or until presyncope, and during the cold pressor test (CPT) and Valsalva manoeuvres.

265 grees head-up tilt (HUT), followed by a cold pressor test (CPT) in HUT.

266 The cold pressor test (CPT) is a powerful sympathoexcitatory stre

267 ted blood pressure (BP) response to the cold pressor test (CPT) is associated with increased risk of

268 A cold pressor test (CPT) was administered for comparison.

269 y Flow Mediated Dilation = BAFMD) and a cold pressor test (CPT).

270 Pain response was assessed using the cold-pressor test (CPT): participants immersed their left han

271 ex-mediated sympathetic system) and the cold pressor test (CPT; a non-specific sympathetic stimulus).

272 ve pressure (-60 mm Hg) and pain by the cold pressor test (ice water exposure).

273 ron emission tomography at rest, during cold pressor test (largely endothelium-dependent), and after

274 n these variables did not differ when a cold pressor test and lower body negative pressure were super

275 ity burst frequency, responses during a cold pressor test in healthy humans.

276 and myocardial blood flow responses to cold pressor test normalized.

277 xcitatory manoeuvres like exercise, the cold pressor test or mental stress.

278 cclusion, but more constriction after a cold pressor test than age-matched controls.

279 y sodium and potassium intervention and cold pressor test vary considerably among individuals.

280 The cold pressor test was used to produce pain, and a modified pr

281 ts either to stress (socially-evaluated cold pressor test) or a control condition (room temperature w

282 Pain (cold pressor test) reduces tissue oxygen saturation in all me

283 thresholds, tonic suprathreshold pain (cold pressor test), and repeated phasic suprathreshold mechan

284 lin-induced wet dog shake model and the cold pressor test).

285 immersion in 4 degrees C ice-water (ie, cold pressor test).

286 ollows: (1) choice sessions following a cold pressor test, to induce stress, and (2) binge dosing of

287 y sodium and potassium intervention and cold pressor test.

288 P responses to dietary intervention and cold pressor test.

289 they were either exposed to a stressor (cold pressor test; CPS) or a warm water control, and immediat

290 to assess MBF regulation in response to cold pressor testing (CPT) and adenosine infusion.

291 yocardial blood flow (MBF) increases to cold pressor testing (CPT) are at increased risk for cardiova

292 tor function was studied in response to cold pressor testing (CPT) in 71 patients with normal angiogr

293 s in myocardial blood flow (MBF) to the cold pressor testing (CPT) method noninvasively with PET corr

294 ium-dependent vasomotion in response to cold pressor testing.

295 ring each visit, participants completed cold pressor tests (CPT; hand in ~0.4 degrees C ice bath for

296 pressure was not restored until mannitol and pressor therapy were initiated at 120 mins.

297 Ultrasound can help in deciding fluid vs. pressor treatment by evaluating the inferior vena cava a

298 (DGF), which can result from hypotension and pressor use related to the liver transplantation (LT), m

299 At least 5 days after surgery, pressor, vasoconstrictor and cardiac chronotropic respon

300 e effects of stress (socially evaluated cold pressor vs. control procedure) and MR-availability (400