ライフサイエンスコーパス: pressor

1 n >2.4 mg/dL, pH < 7.30, and the need for >2 pressors.

2 These findings suggest that the pressor action of [Pyr(1) ]apelin-13 in the RVLM of norm

3 regulate the central nervous system-mediated pressor action of Ang II.

4 olved in circulatory homeostasis through the pressor action of angiotensin II at its AT1 receptor.

5 s, inhibition of which increases sympathetic pressor activity and heart rate.

6 data suggest that tachykinins mediate their pressor activity by increasing the excitability of spina

7 tion." The agents we utilize as sedative and pressor agents have considerable effects on immune funct

8 tested by assessment of the acute effects of pressor agents on mean arterial pressure (MAP) and renal

9 SB334867) attenuated hypercapnic gas-induced pressor and anxiety responses, without altering the robu

10 nt of central prostaglandin synthesis on the pressor and bradycardic effect of cytidine 5'-diphosphoc

11 te in this brain region nearly abolishes the pressor and bradycardic effects of renin infused in the

12 pretreatments almost completely blocked the pressor and bradycardic responses to CDP-choline while n

13 0.5 microg kg(-1); i.a.) that attenuated the pressor and cardioaccelerator responses to both contract

14 e hindlimb muscles were freely perfused, the pressor and cardioaccelerator responses to either contra

15 The pressor and cardioaccelerator responses to either contra

16 (5 microg kg(-1), i.a.) not only blocked the pressor and cardioaccelerator responses to lactic acid a

17 decerebrate cats if amiloride attenuated the pressor and cardioaccelerator responses to static contra

18 projections were found ipsilaterally in the pressor and depressor areas of the medulla and the spina

19 examine RVLMS and control site responses to pressor and depressor challenges during sleep that resul

20 t overall activity declines and increases to pressor and depressor challenges, respectively.

21 Pressor and depressor manipulations are usually followed

22 e CVLM and RVLM were functionally defined by pressor and depressor responses to microinjected GABA (5

23 he subfornical organ impairs Ang II-mediated pressor and drinking responses at least by inhibiting th

24 via Ad-Syn-MIF gene transfer attenuated the pressor and drinking responses produced by icv-injected

25 n levels in the PVN and produced significant pressor and drinking responses that were inhibited by PV

26 allopurinol impaired fetal alpha1-adrenergic pressor and femoral vasopressor responses and enhanced t

27 However, with HBOC-201, pressor and fluid requirements were reduced by half, whi

28 roreflex slopes were obtained using the peak pressor and heart rate responses to increasing doses of

29 In the MI rats, the pressor and RSNA responses to contraction were reduced b

30 heart rate and CO responses and enhanced the pressor and SVR responses to sustained stress (1 min exp

31 The pressor and sympathetic responses to ischaemic muscle co

32 By contrast, the [Pyr(1) ]apelin-13 induced pressor and sympathoexcitatory effects were abolished by

33 Although reduced, pressor and sympathoexcitatory responses due to inhibiti

34 ificantly reduced the central Ang II-induced pressor and sympathoexcitatory responses, decreased base

35 The pressor and tachycardic responses to ARCN stimulation at

36 itatory effects on RVLM neurons resulting in pressor and tachycardic responses, (2) these responses w

37 was reversed i.e., the cold stimulus induced pressor and tachycardic responses, augmented glutamate,

38 gestational age was a greater determinant of pressor and vasopressor reactivity.

39 s responded to acute hypoxaemia with similar pressor and vasopressor responses compared to singleton

40 Pressor and vasopressor responses to all agonists were g

41 ot atmospheric air, resulted in respiratory, pressor, and bradycardic responses, as well as anxiety-l

42 Nox1 overexpression augments the oxidative, pressor, and hypertrophic responses to Ang II, supportin

43 onses in Affs from rabbits infused with slow pressor Ang II are mediated independently by O2- in the

44 Slow-pressor AngII elevated blood pressure in AOF females and

45 es are paralleled in rodents following "slow-pressor" angiotensin II (AngII) administration: young ma

46 o investigate the projections of this caudal pressor area (CPA) to the medulla and pons.

47 l area where rostral ventrolateral medullary pressor area (RVLM) is located.

48 caudal C1 catecholamine neurons, and caudal pressor area receive dense axonal projections, generally

49 ction in CNS neurons plays a key role in the pressor, bradycardic, and dipsogenic actions of Ang II i

50 To extend the in vitro studies, the pressor, bradycardic, and drinking responses to intracer

51 he magnitude of both the sympathoadrenal and pressor but not the adrenocortical response to stress.

52 GsMTx4 reduced the pressor, cardioaccelerator and renal sympathetic nerve r

53 4 into the jugular vein had no effect on the pressor, cardioaccelerator, or renal sympathetic nerve r

54 significantly longer than in quiet sleep for pressor challenges.

55 eased after L-NMMA and saline+PE (volume and pressor control for L-NMMA).

56 rial supply of the hindlimb reduced the peak pressor (control: 24 +/- 2, GsMTx4: 14 +/- 3 mmHg, P < 0

57 rial supply of the hindlimb reduced the peak pressor (control: 24 +/- 5, GsMTx4: 12 +/- 5 mmHg, P < 0

58 Adrenal medullectomy did not affect the pressor (DeltaMAP = 12 +/- 2 mm Hg), tachycardic (DeltaH

59 Some patients with pressor-dependent severe sepsis appear to have relative

60 Accordingly the pressor dose 33 and pressor dose 100 values were lowered following lipopolys

61 ure by 33 and 100 mm Hg (pressor dose 33 and pressor dose 100) was determined.

62 Accordingly the pressor dose 33 and pressor dose 100 values were lowered

63 rease systolic pressure by 33 and 100 mm Hg (pressor dose 33 and pressor dose 100) was determined.

64 An equi-pressor dose of angiotensin II had no effect on myogenic

65 One healthy subject died after a pressor dose of angiotensin II was infused continuously

66 The pressor dose of norepinephrine necessary to increase sys

67 ced renal cortical tissue PO2 more than equi-pressor doses of phenylephrine, probably because it redu

68 ificantly greater than those induced by equi-pressor doses of phenylephrine.

69 Furthermore, at pressor doses vasopressin improved cerebral perfusion.

70 Similar to humans, "slow-pressor" doses of angiotensin II (AngII) increase blood

71 It is concluded that the pressor effect due to a vasoactive oxygen carrier may be

72 in II, but not norepinephrine, had a greater pressor effect in denervated animals.

73 er/thr phosphatases dampen the ERK-dependent pressor effect of ethanol in normotensive rats.

74 cts tonically to attenuate the ERK-dependent pressor effect of ethanol or acetaldehyde in normotensiv

75 th the plasma or urine DHPG/NE ratio and the pressor effect of tyramine.

76 The pressor effect results from conversion of L-DOPS to NE o

77 PyKYNE-losartan fully antagonized the Ang II pressor effect, albeit with 4-fold potency reduction (th

78 PNU-37883A had no pressor effect, except in the presence of pentolinium (p

79 cariporide and epinephrine prompted adequate pressor effects during chest compression and facilitated

80 xtent MC3 receptors in the RVLM, and (3) the pressor effects of ACTH were mediated via sympathetic ac

81 We conclude that the central and pressor effects of angiotensin II are critical for T-cel

82 Rho-kinase pathway in the maintenance of the pressor effects of vasopressin in endotoxemic rats.

83 rbidities, angiotensin II dose and duration, pressor effects, other physiologic and side effects, and

84 ings suggest that CSR1 is a potent tumor sup-pressor gene.

85 600 IU/L, creatinine >2.0 mg/dL, intubation, pressors) had an AUROC for transplant/death of 0.899 whi

86 After 6 hrs with either pressor, hemodynamics were stable.

87 in system and thus dictates the level of the pressor hormone angiotensin-II.

88 ion has focused on mechanisms underlying its pressor hypersensitivity, which contrasts with the vascu

89 Equi-pressor infusion of phenylephrine did not significantly

90 one or both cell types in response to "slow-pressor" infusion of AngII.

91 range, 13 [0-25] vs 15 [0-25]; p = 0.8) and pressor/inotrope-free days (median and interquartile ran

92 that NO synthesized in the brain attenuates pressor mechanisms involving prostaglandin, endothelin a

93 ndogenous inhibitor of NOS, by virtue of its pressor or nitric oxide-depleting effects, or a combinat

94 1.6 to 2.7; P<0.001), hypotension requiring pressors (OR, 1.9 per quintile; 95% CI, 1.5 to 2.3; P<0.

95 ; and comatose state, intubation, receipt of pressors, or death in the emergency department.

96 1.3x10(-)(1)(0)) for the sensitivity to cold pressor pain in males, but not in females.

97 hypnotic analgesia training to control cold pressor pain.

98 The NPY is a potent pressor peptide co-released with catecholamines during s

99 play an important role in counteracting the pressor, proliferative, and profibrotic actions of angio

100 uld produce changes in endogenous levels and pressor reactivity to exogenous norepinephrine and vasop

101 The exercise pressor reflex (a neural drive originating in skeletal m

102 have led to the hypothesis that the exercise pressor reflex (EPR) becomes hyperactive after the devel

103 An exaggerated exercise pressor reflex (EPR) contributes to exercise intolerance

104 The skeletal muscle exercise pressor reflex (EPR) induces increases in heart rate (HR

105 The exercise pressor reflex (EPR) is an important neural mechanism th

106 o activation of the skeletal muscle exercise pressor reflex (EPR) is exaggerated.

107 T: Contraction of muscle evokes the exercise pressor reflex (EPR), which is expressed partly by incre

108 veness is mediated by an overactive exercise pressor reflex (EPR).

109 roxide scavenger, attenuated the exaggerated pressor reflex and reduced reactive oxygen species produ

110 cardiovascular responses during the exercise pressor reflex and simultaneously modulated medullary nN

111 Activation of the exercise pressor reflex during electrically induced static muscle

112 Potentially, effective treatment of exercise pressor reflex dysfunction may reduce the cardiovascular

113 echano-gated Piezo channels, on the exercise pressor reflex evoked by intermittent contraction of the

114 n important role in the exaggerated exercise pressor reflex found in rats with ligated femoral arteri

115 We conclude that GsMTx4 reduced the exercise pressor reflex in decerebrate rats and that the reductio

116 o-gated Piezo channels, reduced the exercise pressor reflex in decerebrate rats.

117 that P2 receptors contribute to the exercise pressor reflex in humans.

118 ys an important role in evoking the exercise pressor reflex in rats with a compromised arterial blood

119 d the hypothesis that the augmented exercise pressor reflex in rats with a ligated femoral artery is

120 eceptors attenuated the exaggerated exercise pressor reflex in rats with ligated femoral arteries.

121 we found no effect of tiron infusion on the pressor reflex in rats with patent femoral arteries.

122 of the mechanical component of the exercise pressor reflex in rats.

123 tery disease, causes an exaggerated exercise pressor reflex in response to muscle contraction.

124 ory that selectively stimulates the exercise pressor reflex independent of central command and/or the

125 (ASICs), their contribution to the exercise pressor reflex is not known.

126 erents, sense the decrease in pH and evoke a pressor reflex known to increase mean arterial pressure.

127 However, given that exercise pressor reflex overactivity is known to elicit enhanced

128 10-min on the gallbladder induced consistent pressor reflex responses.

129 acid and E-2 led to a significantly greater pressor reflex than lactic acid alone in the presence of

130 ion of somatosensory input from the exercise pressor reflex to this resetting during exercise.

131 arger reflex pressor response (i.e. exercise pressor reflex) than did static contraction of the contr

132 d (to simulate exercising muscle and evoke a pressor reflex), endomorphin-2 and naloxone resulted in

133 an increase in blood pressure (i.e. exercise pressor reflex).

134 e in the metabolic component of the exercise pressor reflex.

135 voke the metabolic component of the exercise pressor reflex.

136 ension is mediated by an overactive exercise pressor reflex.

137 Aergic neurotransmission during the exercise pressor reflex.

138 king the metabolic component of the exercise pressor reflex.

139 eurotransmission that regulates the exercise pressor reflex.

140 voke the metabolic component of the exercise pressor reflex.

141 e ASIC currents and the lactic acid-mediated pressor reflex.

142 uscle play an important role in the exercise pressor reflex.

143 ension is mediated by an overactive exercise pressor reflex.

144 omponent of this reflex, termed the exercise pressor reflex.

145 regulate muscle nociception and the exercise pressor reflex.

146 te to evoke this reflex, termed the exercise pressor reflex.

147 n decerebrated rats exaggerates the exercise pressor reflex.

148 h regulating muscle nociception and exercise pressor reflexes (EPRs), and P2Y1 has been linked to hea

149 onary resuscitation, mechanical ventilation, pressors, rescue thrombolysis, or surgical embolectomy,

150 al artery was ligated evoked a larger reflex pressor response (i.e. exercise pressor reflex) than did

151 Stimulation of trigeminal nerve induces pressor response and improves cerebral blood flow (CBF)

152 The [Pyr(1) ]apelin-13-mediated pressor response and the increased low frequency spectra

153 otentiated the magnitude and duration of the pressor response as well as the phosphatase inhibition e

154 key signaling intermediate in the transient pressor response elicited by acute injection of Ang II d

155 We show that the pressor response elicited by intra-RVLM ethanol (10 mug)

156 The AngII slow pressor response enhances renal cortical O(2)(.-) and p2

157 s for increasing MAP, and by correlating the pressor response evoked by these peptides to reported K(

158 tle response to cold water stress elicited a pressor response in all rats, the hemodynamic response p

159 the central CB(1)R-evoked sympathoexcitation/pressor response in conscious rats.

160 c.v.) angiotensin (ANG) II causes a reliable pressor response in the fetus at 90% gestation.

161 transmitter systems through which the apelin pressor response may occur within the RVLM.

162 inhibiting 50% of the Ang II-induced maximal pressor response of 25.5 mg/kg) relative to losartan.

163 nd function, which likely contributes to the pressor response of these hormones.

164 the enhancement of a carbachol (CCh)-evoked pressor response produced by prior NPY administration in

165 of ATP-sensitive P2X receptors enhances the pressor response seen when muscle mechanoreceptors are e

166 Cl produces a greater sympathoexcitatory and pressor response than infusion of hypertonic mannitol/so

167 naloxone resulted in a significantly greater pressor response than lactic acid alone, while administr

168 atment with prazosin reversed the HS-induced pressor response to a hypotensive response (from 121 +/-

169 The pressor response to acute infusion of Ang II was signifi

170 lts demonstrate that OA-NO(2) diminishes the pressor response to Ang II and inhibits AT(1)R-dependent

171 uption of the EP1 receptor blunted the acute pressor response to Ang II and reduced chronic Ang II-dr

172 Deletion of p47phox attenuated the pressor response to Ang II; however, coinfusion of pheny

173 portantly, 7 days after virus infection, the pressor response to angiotensin (Ang) II (200 pmol intra

174 xpression by C1 neurons is essential for the pressor response to angiotensin II and that this pathway

175 tly elevated in the transgenic mice, but the pressor response to angiotensin II was augmented.

176 for recording blood pressure, modulated the pressor response to aversive stress.

177 this pathway plays an important role in the pressor response to aversive stress.

178 , an ASIC agonist, but did not attenuate the pressor response to capsaicin, a TRPV1 agonist.

179 nses to lactic acid, but also attenuated the pressor response to capsaicin.

180 cies with respect to causing the exaggerated pressor response to contraction seen in rats with ligate

181 In the six ligated rats, however, the pressor response to contraction was attenuated by L16198

182 le autonomic traits: baroreflex function and pressor response to environmental stress.

183 Similarly, the pressor response to EP1-selective agonists sulprostone a

184 tential for a counteracting, anti-dipsogenic pressor response to hindbrain AngII allows for lingering

185 mg/kg, i.c.v.) showed a potentiation of the pressor response to i.c.v. ANG II, accompanied by bradyc

186 The rapid, systemic pressor response to intravenous LTC4 was also diminished

187 unted both the increase in plasma NE and the pressor response to L-DOPS in all patients

188 amiloride and APETx2 greatly attenuated the pressor response to lactic acid, an ASIC agonist, but di

189 R-floxed mice enabled demonstration that the pressor response to microinjection of angiotensin II int

190 ion by alpha,beta-methylene ATP enhanced the pressor response to muscle stretch by 42% in control ani

191 At 6 hrs, there was an attenuated pressor response to norepinephrine (p < .01) despite blo

192 In vivo, the pressor response to norepinephrine was markedly reduced

193 The pressor response to norepinephrine was reduced following

194 e afferents and the baroreflex evoked by the pressor response to phenylephrine (3-25 microg kg(-1), i

195 lated hypertension; they also show a reduced pressor response to salt loading.

196 in animals, ruling out an effect of enhanced pressor response to stress following prenatal protein re

197 n-treated and DCM rats displayed a decreased pressor response to the intra-arterial administration of

198 Thus, we hypothesized that the pressor response to VR1 stimulation would be smaller and

199 The initial pressor response was also attenuated by pretreatment wit

200 Rs in C1 neurons induced a greater sustained pressor response when compared to the control viral-inje

201 ubtype 1 (VR1), inducing a neurally mediated pressor response, and (2) activation of ATP-sensitive P2

202 hrine with Ang II, which restored the Ang II pressor response, did not alter the protective effects o

203 and an increase in TPR, followed by a brief pressor response, effects which were unaffected by SR141

204 le afferents to the sympathetically mediated pressor response.

205 ated in the CB(1)R-evoked sympathoexcitation/pressor response.

206 tes displaying blunted slope and exaggerated pressor response.

207 ithout restraint in the setting of increased pressor response.

208 One type consisted of pressor responses accompanied by bradycardia.

209 lus doses of phenylephrine evoked attenuated pressor responses after CIH (P<0.01).These data suggest

210 , phenylephrine induced significantly higher pressor responses and greater vasoconstrictions in the o

211 Prenatal exposure to caffeine increased pressor responses and vasoconstrictions to phenylephrine

212 efferent processing of the BJR, and (2) the pressor responses elicited by alpha-methyl-5-HT were not

213 sms implicated in the sympathoexcitation and pressor responses elicited by central CB(1)R activation

214 A stimulation at acupoints P5-P6 reduced the pressor responses for at least 60-min.

215 different effects on central ANG II-induced pressor responses in fetuses at late gestation, and that

216 The two pressor responses involve different neurochemical mechan

217 hibit significant hypertension and increased pressor responses to angiotensin II and endothelin-1; th

218 asal blood pressure was similar, however the pressor responses to both acute and chronic angiotensin

219 decreased vascular resistance, and elevated pressor responses to environmental (cold) stress.

220 ermittent contraction but did not reduce the pressor responses to femoral arterial injection of compo

221 In anesthetized rats, the pressor responses to increasing doses of norepinephrine

222 Pressor responses to increasing doses of phenylephrine a

223 echanical stimulus, but had no effect on the pressor responses to intra-arterial injection of alpha,b

224 Pressor responses to intravenous norepinephrine, vasopre

225 t, the high dose of amiloride attenuated the pressor responses to lactic acid, but also attenuated th

226 Pressor responses to norepinephrine and PNU-37883A (a va

227 mpathetic nerve activity (~70%), and blunted pressor responses to phenylephrine and angiotensin II.

228 renal sodium excretion or volume expansion; pressor responses to phenylephrine were enhanced and bar

229 Pressor responses to static handgrip were also attenuate

230 od too rapidly for accurate measurement, and pressor responses to the injection of drug were greatly

231 Both pressor responses were abolished by i.c.v. pretreatment

232 Central ANG II-induced fetal pressor responses were not altered by PD123319 (0.8 mg/k

233 Another type consisted of pressor responses without any change in heart rate; such

234 bute to the blunted sympathetically mediated pressor responses, because bolus doses of phenylephrine

235 Associated with the pressor responses, c-fos expression in the cardiovascula

236 ffect on seizure-induced sympathoexcitation, pressor responses, or tachycardia but abolished the prol

237 lly infused tyramine produced dose-dependent pressor responses, predicted by family history of hypert

238 hoexcitation (p </= 0.05), and abolished the pressor responses, tachycardia, and QT interval prolonga

239 ute rises in SNA was accompanied by enhanced pressor responses.

240 low catestatin predicts augmented adrenergic pressor responses.

241 kg (i.c.v.) abolished central ANG II-induced pressor responses.

242 saccharide, it caused a striking increase in pressor responsiveness (mean slope after lipopolysacchar

243 ipopolysaccharide caused a large increase in pressor responsiveness above lipopolysaccharide values.

244 y assesses whether alpha-2 agonists increase pressor responsiveness following lipopolysaccharide admi

245 ent testing of endothelial function and cold pressor responsiveness of the brachial artery.

246 enal sympathetic nerve activity and restored pressor responsiveness to both phenylephrine and angiote

247 Pressor responsiveness to norepinephrine decreased sligh

248 ssure, renal sympathetic nerve activity, and pressor responsiveness to phenylephrine and angiotensin

249 renal sympathetic nerve activity and reduced pressor responsiveness to vasopressors persisted.

250 lar to prolonged human sepsis, and decreased pressor sensitivity to norepinephrine.

251 time >12 hours, ICU stay >5 days, 3 or more pressors simultaneously, extensive alcohol abuse, cancer

252 esponses to 3 psychological challenges--cold pressor, star tracing, and video game tasks--were measur

253 on, the baroreflex was activated using brief pressor stimuli and the consequent cardiac (heart rate c

254 ip between SNA and vasoconstriction during a pressor stimulus, which increases BP and may be contra-i

255 gen saturations, but 90% (20 of 22) required pressor support.

256 al excitation augmented chemoreflex-mediated pressor, sympathoexcitatory and minute neural ventilatio

257 The systolic BP effect for the cold pressor task was apparent for women and for whites in ra

258 ndent vasodilation), and in response to cold pressor test (CPT) (reflecting primarily endothelium-dep

259 re, MSNA responses were compared when a cold pressor test (CPT) and lower body negative pressure (LBN

260 yocardial blood flow (MBF) responses to cold pressor test (CPT) and pharmacologic vasodilation was me

261 min or until presyncope, and during the cold pressor test (CPT) and Valsalva manoeuvres.

262 grees head-up tilt (HUT), followed by a cold pressor test (CPT) in HUT.

263 The cold pressor test (CPT) is a powerful sympathoexcitatory stre

264 ted blood pressure (BP) response to the cold pressor test (CPT) is associated with increased risk of

265 A cold pressor test (CPT) was administered for comparison.

266 y Flow Mediated Dilation = BAFMD) and a cold pressor test (CPT).

267 Pain response was assessed using the cold-pressor test (CPT): participants immersed their left han

268 ex-mediated sympathetic system) and the cold pressor test (CPT; a non-specific sympathetic stimulus).

269 ve pressure (-60 mm Hg) and pain by the cold pressor test (ice water exposure).

270 ron emission tomography at rest, during cold pressor test (largely endothelium-dependent), and after

271 oprolol and -2.2 U for placebo, P=0.15; cold pressor test 3.1+/-8.9 U for carvedilol versus 9.0+/-2.7

272 n these variables did not differ when a cold pressor test and lower body negative pressure were super

273 tor responses to isometric handgrip and cold pressor test did not differ between treatment groups.

274 ever, myocardial blood flow response to cold pressor test increased by 47.6% from resting values in i

275 and myocardial blood flow responses to cold pressor test normalized.

276 xcitatory manoeuvres like exercise, the cold pressor test or mental stress.

277 responses to other reflex stimuli (the cold pressor test or Valsalva's manoeuvre) were similar in pa

278 cclusion, but more constriction after a cold pressor test than age-matched controls.

279 y sodium and potassium intervention and cold pressor test vary considerably among individuals.

280 The cold pressor test was used to produce pain, and a modified pr

281 ts either to stress (socially-evaluated cold pressor test) or a control condition (room temperature w

282 Pain (cold pressor test) reduces tissue oxygen saturation in all me

283 thresholds, tonic suprathreshold pain (cold pressor test), and repeated phasic suprathreshold mechan

284 lin-induced wet dog shake model and the cold pressor test).

285 ersing a hand in ice water for 2-4 min (cold pressor test, CPT).

286 P responses to dietary intervention and cold pressor test.

287 0% maximum voluntary contraction) and a cold pressor test.

288 y sodium and potassium intervention and cold pressor test.

289 they were either exposed to a stressor (cold pressor test; CPS) or a warm water control, and immediat

290 to assess MBF regulation in response to cold pressor testing (CPT) and adenosine infusion.

291 yocardial blood flow (MBF) increases to cold pressor testing (CPT) are at increased risk for cardiova

292 tor function was studied in response to cold pressor testing (CPT) in 71 patients with normal angiogr

293 s in myocardial blood flow (MBF) to the cold pressor testing (CPT) method noninvasively with PET corr

294 ium-dependent vasomotion in response to cold pressor testing.

295 d abnormal vasoconstrictor responses to cold pressor tests (CPT) that were similar in primary and sec

296 pressure was not restored until mannitol and pressor therapy were initiated at 120 mins.

297 Ultrasound can help in deciding fluid vs. pressor treatment by evaluating the inferior vena cava a

298 (DGF), which can result from hypotension and pressor use related to the liver transplantation (LT), m

299 At least 5 days after surgery, pressor, vasoconstrictor and cardiac chronotropic respon

300 e effects of stress (socially evaluated cold pressor vs. control procedure) and MR-availability (400