ライフサイエンスコーパス: pressor response

1 infused mouse is a valid model for the slow pressor response.

2 e were hypotensive and exhibited a decreased pressor response.

3 Among these is a marked pressor response.

4 ly reduced expression of the naloxone-evoked pressor response.

5 blood pressure without affecting the delayed pressor response.

6 SP, 5 nmol) caused a short- and long-lasting pressor response.

7 to the pathways activated during the muscle pressor response.

8 ation of arterial pressure during the muscle pressor response.

9 nt depressor response followed by pronounced pressor response.

10 ithout restraint in the setting of increased pressor response.

11 le afferents to the sympathetically mediated pressor response.

12 ated in the CB(1)R-evoked sympathoexcitation/pressor response.

13 tes displaying blunted slope and exaggerated pressor response.

14 ials), and which were often accompanied by a pressor response.

15 riction in children with exaggerated hypoxic pressor responses.

16 g b.wt (i.v.) were effective in altering the pressor responses.

17 ute rises in SNA was accompanied by enhanced pressor responses.

18 low catestatin predicts augmented adrenergic pressor responses.

19 kg (i.c.v.) abolished central ANG II-induced pressor responses.

20 This contraction augmented the pressor response (37+/-4 mmHg) and HR (61+/-11 bpm) with

21 One type consisted of pressor responses accompanied by bradycardia.

22 lus doses of phenylephrine evoked attenuated pressor responses after CIH (P<0.01).These data suggest

23 SNA and an initial hypotension followed by a pressor response and a longer lasting hypotensive respon

24 Stimulation of trigeminal nerve induces pressor response and improves cerebral blood flow (CBF)

25 T(B2) (and presumably ET(A))-mediated portal pressor response and stellate cell constriction.

26 The [Pyr(1) ]apelin-13-mediated pressor response and the increased low frequency spectra

27 , phenylephrine induced significantly higher pressor responses and greater vasoconstrictions in the o

28 Prenatal exposure to caffeine increased pressor responses and vasoconstrictions to phenylephrine

29 ubtype 1 (VR1), inducing a neurally mediated pressor response, and (2) activation of ATP-sensitive P2

30 otentiated the magnitude and duration of the pressor response as well as the phosphatase inhibition e

31 showed a delayed rise by days 9 to 13 (slow pressor response) at the lower rates of AngII infusion.

32 bute to the blunted sympathetically mediated pressor responses, because bolus doses of phenylephrine

33 Associated with the pressor responses, c-fos expression in the cardiovascula

34 icrol) to conscious rats produced a biphasic pressor response characterized by an initial transient i

35 hrine with Ang II, which restored the Ang II pressor response, did not alter the protective effects o

36 f 10 and 30 micromol/kg iv 20 attenuated the pressor responses due to the administration of exogenous

37 and an increase in TPR, followed by a brief pressor response, effects which were unaffected by SR141

38 key signaling intermediate in the transient pressor response elicited by acute injection of Ang II d

39 We show that the pressor response elicited by intra-RVLM ethanol (10 mug)

40 efferent processing of the BJR, and (2) the pressor responses elicited by alpha-methyl-5-HT were not

41 sms implicated in the sympathoexcitation and pressor responses elicited by central CB(1)R activation

42 The pressor responses elicited from the aforementioned site

43 The AngII slow pressor response enhances renal cortical O(2)(.-) and p2

44 traction and muscle stretch, but the initial pressor response evoked by static contraction was attenu

45 s for increasing MAP, and by correlating the pressor response evoked by these peptides to reported K(

46 this loss reduces the sympathoexcitatory and pressor responses evoked by RVLM stimulation.

47 A stimulation at acupoints P5-P6 reduced the pressor responses for at least 60-min.

48 site responses (viz., defensive behavior and pressor responses from the lateral column vs. quiescence

49 al artery was ligated evoked a larger reflex pressor response (i.e. exercise pressor reflex) than did

50 tle response to cold water stress elicited a pressor response in all rats, the hemodynamic response p

51 the central CB(1)R-evoked sympathoexcitation/pressor response in conscious rats.

52 ression was associated with prolonged portal pressor response in isolated livers.

53 ective agonist [phenylephrine (PE)] caused a pressor response in KO mice, but the final arterial pres

54 travenous infusion of EGF induced an initial pressor response in rats followed by a prolonged decreas

55 vivo as demonstrated by the big ET-1-induced pressor response in rats.

56 maximally inhibited the endothelin-1-induced pressor response in rats.

57 c.v.) angiotensin (ANG) II causes a reliable pressor response in the fetus at 90% gestation.

58 and RVM, pulmonary vascular remodeling, and pressor responses in chronically hypoxic mice, suggestin

59 different effects on central ANG II-induced pressor responses in fetuses at late gestation, and that

60 The two pressor responses involve different neurochemical mechan

61 nses that are variable in rats such that the pressor response is attributable to either a large incre

62 findings demonstrate that the MK-801-induced pressor response is dependent upon the integrity of the

63 findings demonstrate that the L-NAME-induced pressor response is dependent upon the integrity of the

64 The mechanism of this pressor response is unclear.

65 transmitter systems through which the apelin pressor response may occur within the RVLM.

66 Because the pressor response may, in part, be caused by central nerv

67 inhibiting 50% of the Ang II-induced maximal pressor response of 25.5 mg/kg) relative to losartan.

68 nd function, which likely contributes to the pressor response of these hormones.

69 rtensive rats (SHR) respond with exaggerated pressor responses of central origin in response to pharm

70 In WKYLJ CRF specifically lowered the pressor response on trial 1 compared to control, an effe

71 ffect on seizure-induced sympathoexcitation, pressor responses, or tachycardia but abolished the prol

72 lly infused tyramine produced dose-dependent pressor responses, predicted by family history of hypert

73 the enhancement of a carbachol (CCh)-evoked pressor response produced by prior NPY administration in

74 of ATP-sensitive P2X receptors enhances the pressor response seen when muscle mechanoreceptors are e

75 ignificantly increases blood pressure with a pressor response sufficient to reduce catecholamine admi

76 ischemic handgrip exercise, despite a normal pressor response, suggests that enhanced vasoconstrictio

77 hoexcitation (p </= 0.05), and abolished the pressor responses, tachycardia, and QT interval prolonga

78 Cl produces a greater sympathoexcitatory and pressor response than infusion of hypertonic mannitol/so

79 naloxone resulted in a significantly greater pressor response than lactic acid alone, while administr

80 3, an alpha 1A/C-selective agonist, caused a pressor response that was lost in the KO and reduced but

81 atment with prazosin reversed the HS-induced pressor response to a hypotensive response (from 121 +/-

82 The pressor response to acute infusion of Ang II was signifi

83 man angiotensinogen and markedly blunted the pressor response to administration of purified recombina

84 The pressor response to Ang I before and after valsartan was

85 The pressor response to Ang I was blunted significantly by v

86 the dose of ACE inhibitors was doubled, the pressor response to Ang I was no longer different from t

87 lts demonstrate that OA-NO(2) diminishes the pressor response to Ang II and inhibits AT(1)R-dependent

88 uption of the EP1 receptor blunted the acute pressor response to Ang II and reduced chronic Ang II-dr

89 Deletion of p47phox attenuated the pressor response to Ang II; however, coinfusion of pheny

90 s and that AT2 receptors do not modulate the pressor response to AngII.

91 level of ACE inhibition was assessed by the pressor response to angiotensin (Ang) I in patients who

92 portantly, 7 days after virus infection, the pressor response to angiotensin (Ang) II (200 pmol intra

93 n I (AI) was measured and normalized for the pressor response to angiotensin II (AII) to assess inhib

94 xpression by C1 neurons is essential for the pressor response to angiotensin II and that this pathway

95 tly elevated in the transgenic mice, but the pressor response to angiotensin II was augmented.

96 The pressor response to ascending doses of Ang I was evaluat

97 for recording blood pressure, modulated the pressor response to aversive stress.

98 this pathway plays an important role in the pressor response to aversive stress.

99 , an ASIC agonist, but did not attenuate the pressor response to capsaicin, a TRPV1 agonist.

100 nses to lactic acid, but also attenuated the pressor response to capsaicin.

101 cies with respect to causing the exaggerated pressor response to contraction seen in rats with ligate

102 In the six ligated rats, however, the pressor response to contraction was attenuated by L16198

103 ited by N-nitro-L-arginine methyl ester, the pressor response to dexfenfluramine is greatly enhanced.

104 le autonomic traits: baroreflex function and pressor response to environmental stress.

105 Similarly, the pressor response to EP1-selective agonists sulprostone a

106 There was a significant increase in the peak pressor response to ET (10 microg/kg intravenously) in p

107 The pressor response to ET in vitro was significantly enhanc

108 The pressor response to ET-1 was determined in vitro using i

109 The pressor response to exogenous angiotensin I (AI) was mea

110 tential for a counteracting, anti-dipsogenic pressor response to hindbrain AngII allows for lingering

111 mg/kg, i.c.v.) showed a potentiation of the pressor response to i.c.v. ANG II, accompanied by bradyc

112 plasma prostacyclin levels and a significant pressor response to indomethacin in PHT animals.

113 ot in platelets and exhibited an exaggerated pressor response to infused iPF(2alpha)-III compared wit

114 Agtr1a -/-Agtr1b -/- mice have no systemic pressor response to infusions of angiotensin II, but the

115 The pressor response to intravenous injection of Big endothe

116 The rapid, systemic pressor response to intravenous LTC4 was also diminished

117 Changes in platelet 5-HT uptake and the pressor response to intravenous tyramine were assessed f

118 By contrast, both the pressor response to iPF(2alpha)-III and its effects on p

119 ked the diazepam-induced potentiation of the pressor response to isoguvacine.

120 f raise training of the dominant limb on the pressor response to isometric exercise of the triceps su

121 unted both the increase in plasma NE and the pressor response to L-DOPS in all patients

122 amiloride and APETx2 greatly attenuated the pressor response to lactic acid, an ASIC agonist, but di

123 sure response to right handgrip, whereas the pressor response to left handgrip did not change.

124 subjects, unlike MSA subjects, although the pressor response to mental arithmetic was reduced.

125 R-floxed mice enabled demonstration that the pressor response to microinjection of angiotensin II int

126 receptors at the dorsal horn level blunt the pressor response to muscle activity.

127 ion by alpha,beta-methylene ATP enhanced the pressor response to muscle stretch by 42% in control ani

128 At 6 hrs, there was an attenuated pressor response to norepinephrine (p < .01) despite blo

129 In vivo, the pressor response to norepinephrine was markedly reduced

130 The pressor response to norepinephrine was reduced following

131 le, this strain responds with an exaggerated pressor response to pharmacological stimulation of centr

132 e afferents and the baroreflex evoked by the pressor response to phenylephrine (3-25 microg kg(-1), i

133 The slow pressor response to prolonged infusions of angiotensin I

134 lated hypertension; they also show a reduced pressor response to salt loading.

135 in animals, ruling out an effect of enhanced pressor response to stress following prenatal protein re

136 n-treated and DCM rats displayed a decreased pressor response to the intra-arterial administration of

137 The pressor response to tyramine differentially distinguishe

138 e higher dose would differentially blunt the pressor response to tyramine, a marker for NE uptake.

139 Thus, we hypothesized that the pressor response to VR1 stimulation would be smaller and

140 In MSA patients, the pressor response to yohimbine and the decrease in SBP wi

141 The present results suggest that pressor responses to AngII are mediated by the activatio

142 Pressor responses to AngII were attenuated by candesarta

143 hibit significant hypertension and increased pressor responses to angiotensin II and endothelin-1; th

144 asal blood pressure was similar, however the pressor responses to both acute and chronic angiotensin

145 he sympathoexcitatory (splanchnic nerve) and pressor responses to electrical stimulation of the RVLM

146 decreased vascular resistance, and elevated pressor responses to environmental (cold) stress.

147 Systemic pressor responses to ET-1 and angiotensin II were simila

148 pulmonary vascular resistance and pulmonary pressor responses to ET-1, angiotensin II, and hypoxia;

149 hronically block or, for L-164,282, to spare pressor responses to exogenous Ang II.

150 ermittent contraction but did not reduce the pressor responses to femoral arterial injection of compo

151 Importantly, AV3V lesions attenuated pressor responses to increasing doses of 5-HT (3 +/- 1,

152 In anesthetized rats, the pressor responses to increasing doses of norepinephrine

153 Pressor responses to increasing doses of phenylephrine a

154 echanical stimulus, but had no effect on the pressor responses to intra-arterial injection of alpha,b

155 In summary, pressor responses to intravenous 5-HT are diminished by

156 Pressor responses to intravenous norepinephrine, vasopre

157 t, the high dose of amiloride attenuated the pressor responses to lactic acid, but also attenuated th

158 The tendency to show exaggerated pressor responses to mental stress is a significant inde

159 l laminae of the dorsal horn potentiates the pressor responses to microinjection of L-glutamate.

160 Pressor responses to norepinephrine and PNU-37883A (a va

161 mals transfected with the eNOS gene, whereas pressor responses to norepinephrine and U46619 were not

162 mpathetic nerve activity (~70%), and blunted pressor responses to phenylephrine and angiotensin II.

163 renal sodium excretion or volume expansion; pressor responses to phenylephrine were enhanced and bar

164 The tendency to show exaggerated pressor responses to psychological demands may be a sign

165 Pressor responses to static handgrip were also attenuate

166 od too rapidly for accurate measurement, and pressor responses to the injection of drug were greatly

167 The initial pressor response was also attenuated by pretreatment wit

168 The pressor response was not antagonized by prior injection

169 lity to suppress BMI-induced tachycardic and pressor responses was additive.

170 The late, but not the early pressor response, was prevented by pretreatment with chl

171 Both pressor responses were abolished by i.c.v. pretreatment

172 These pressor responses were accompanied by a significant tach

173 Higher mental stress pressor responses were associated with more constriction

174 The chemoreflex sympathoexcitatory pressor responses were attenuated by an acute systemic a

175 Pressor responses were elicited by microinjections of l-

176 Central ANG II-induced fetal pressor responses were not altered by PD123319 (0.8 mg/k

177 Rs in C1 neurons induced a greater sustained pressor response when compared to the control viral-inje

178 N) in conscious unrestrained mice elicited a pressor response, which was abolished by ICV preinjectio

179 In rats, intravenous 16a blocks big ET pressor responses with 30-fold greater potency than 1.

180 Another type consisted of pressor responses without any change in heart rate; such