ライフサイエンスコーパス: preterm birth

1 as associated with a 16% increase in odds of preterm birth.

2 problems in children and adults that survive preterm birth.

3 rment of the function of this barrier during preterm birth.

4 come the educational burdens that may follow preterm birth.

5 ors to prevent preterm cervical ripening and preterm birth.

6 mature rupture of membranes, and spontaneous preterm birth.

7 l improve late respiratory morbidities after preterm birth.

8 pre-eclampsia, fetal growth restriction, and preterm birth.

9 Moderate and late preterm birth.

10 of labor or cesarean delivery and subsequent preterm birth.

11 ymase to systemic GBS infection and rates of preterm birth.

12 y insult is a primary trigger of spontaneous preterm birth.

13 gonist (-)-naloxone, in infection-associated preterm birth.

14 cal conditions are suspected to be causes of preterm birth.

15 ng enterocolitis and late-onset sepsis after preterm birth.

16 ntitis is considered to be a risk factor for preterm birth.

17 most highly related to BMI and sex, but not preterm birth.

18 eproductive outcomes such as infertility and preterm birth.

19 inhibitor atosiban in women with threatened preterm birth.

20 tary nitrite intake may increase the risk of preterm birth.

21 ot significantly associated with spontaneous preterm birth.

22 prevent adverse pregnancy outcomes, such as preterm birth.

23 or tertiary amines, has been associated with preterm birth.

24 with fetal growth restriction and iatrogenic preterm birth.

25 nts at the EBF1, EEFSEC, and AGTR2 loci with preterm birth.

26 al ripening in the second trimester predicts preterm birth.

27 e for gestational age, low birth weight, and preterm birth.

28 had low predictive accuracy for spontaneous preterm birth.

29 was due to iatrogenic instead of spontaneous preterm birth.

30 ginal colonization, ascending infection, and preterm birth.

31 alternatives to progesterone for preventing preterm birth.

32 es compared to membranes from term and other preterm births.

33 nicians counseling families facing extremely preterm births.

34 embrane (pPROM) is associated with 30-40% of preterm births.

35 ; hazard ratio, 0.71; 95% CI, 0.45 to 1.14), preterm birth (116 cases among 1774 exposed pregnancies

36 ) and 3.58 (95% CI: 1.57 to 8.14) after very preterm birth (28 to 31 weeks).

37 n was associated with reductions in rates of preterm birth (-3.77% [95% CI -6.37 to -1.16]; ten studi

38 There was no risk increase after moderately preterm birth (32 to 36 weeks) (relative risk: 1.36; 95%

39 % other), of whom 474 (5.0%) had spontaneous preterm births, 335 (3.6%) had medically indicated prete

40 In women with threatened preterm birth, 48 h of tocolysis with nifedipine or atos

41 iciency was associated with a higher risk of preterm birth (adjusted risk ratio = 1.21, 95% CI: 0.99,

42 rone prophylaxis given to reduce the risk of preterm birth affects neonatal and childhood outcomes.

43 conjunction with dietary nitrite intake and preterm birth among 496 mothers of preterm infants and 5

44 ent supplements also had a greater effect on preterm births among underweight pregnant women (BMI <18

45 (95% CI 1.06-2.76, I(2)=56.1%, p=0.058) for preterm birth and 1.41 (95% CI 0.90-2.21, I(2)=0.0%, p=0

46 fied 30 of 410 women (7.3%) with spontaneous preterm birth and 31 of 384 (8.1%) at 22 to 30 weeks.

47 A statewide nested case-control study of preterm birth and air pollution by source and compositio

48 iologic studies suggest associations between preterm birth and ambient air pollution.

49 xed effects to estimate associations between preterm birth and average pollutant concentrations durin

50 -based pregnancy dating to assess impacts on preterm birth and fetal growth in all studies.

51 health outcomes associated with BV, such as preterm birth and human immunodeficiency virus type 1 ac

52 ent with RSG reduces the rate of LPS-induced preterm birth and improves neonatal outcomes by reducing

53 ediated proinflammatory response, preventing preterm birth and improving neonatal outcomes.

54 Other factors associated with preterm birth and low birth weight included treatment wi

55 ngenital heart disease, neural tube defects, preterm birth and low birth weight, birth asphyxia, and

56 ncer survivors may have an increased risk of preterm birth and low birth weight, suggesting that addi

57 gnificant risks of 2 key perinatal outcomes, preterm birth and low birth weight.

58 Preterm birth and low birthweight were the most common a

59 ture of membranes, which are associated with preterm birth and neonatal disease.

60 tration has been shown to reduce the risk of preterm birth and neonatal morbidity in women at high ri

61 ammation (IUI), an important risk factor for preterm birth and neurodevelopmental outcomes, has not b

62 , vaccine effectiveness, vaccine uptake, and preterm birth and of the association of influenza illnes

63 examined their associations with the risk of preterm birth and offspring birth size.

64 A similar effect with protection from preterm birth and perinatal death, and partial correctio

65 y cascade of preterm parturition, to prevent preterm birth and perinatal death.

66 paration of the placenta) is associated with preterm birth and perinatal mortality, but associations

67 r support the efficacy of DNAC in preventing preterm birth and prematurity-related outcomes.

68 udy was to determine the association between preterm birth and risk of incident heart failure (HF) in

69 The association between preterm birth and risk of incident HF was analyzed by us

70 e examined by conditioning on intermediates (preterm birth and small for gestational age) with sensit

71 th restriction and with an increased risk of preterm birth and small size for gestational age at birt

72 tern is associated with a lower incidence of preterm birth and with larger birth size in an Asian pop

73 cystis jirovecii colonization is frequent in preterm births and could be a risk factor to develop res

74 HIV-infected women with preterm births and spontaneous preterm births had signif

75 and child anthropometry and haemoglobin and preterm birth) and socioenvironmental determinants (ie,

76 outcome measures were: perinatal mortality, preterm birth, and being small-for-gestational age (SGA)

77 es, including eclampsia, stroke, stillbirth, preterm birth, and low birth weight; screening and risk

78 rse fetal outcomes: stillbirth, miscarriage, preterm birth, and low birthweight.

79 ted with higher risk for preterm birth, very preterm birth, and neonatal death.

80 ociated with higher risk of stillbirth, very preterm birth, and neonatal death; and ZDV-3TC-LPV-R was

81 impairment, plus GBS-associated stillbirth, preterm birth, and neonatal encephalopathy.

82 Neonatal death, preterm birth, and pregnancy loss occurred in 2%, 8%, an

83 n increase the odds for developing BPD after preterm birth, and that maternal smoking is strongly ass

84 m births, 335 (3.6%) had medically indicated preterm births, and 8601 (91.4%) had term births.

85 ingleton gestations, no previous spontaneous preterm births, and cervical lengths of 25 mm or less at

86 e management of labour and delivery, care of preterm births, and treatment of serious infectious dise

87 ombined with other clinical risk factors for preterm birth [any one of a history in a previous pregna

88 ure of fetal membranes (FMs), and subsequent preterm birth are associated with local infection and in

89 s contributing to chronic lung disease after preterm birth are incompletely understood.

90 endoscopy during pregnancy was not linked to preterm birth (ARR, 1.03; 95% CI, 0.84-1.27).

91 doscopy during pregnancy was associated with preterm birth (ARR, 1.16) but not with small for gestati

92 ncy was associated with an increased risk of preterm birth (ARR, 1.54; 95% confidence interval [CI],

93 ambient air pollutants were associated with preterm birth; associations were observed in all exposur

94 use, resulted in a lower rate of spontaneous preterm birth at less than 34 weeks of gestation.

95 pessary would reduce the rate of spontaneous preterm birth at less than 34 weeks of gestation.

96 The primary end point was spontaneous preterm birth at less than 34 weeks of gestation.

97 Spontaneous preterm birth at less than 37 weeks was the primary outc

98 e applied risk ratios to estimate numbers of preterm births attributable to GBS.

99 as significantly associated with spontaneous preterm birth based on adjusted models of temporal expos

100 ancy and infant outcomes in women at risk of preterm birth (because of previous spontaneous birth at

101 There was a strong association between preterm birth before 32 weeks of gestation and HF in chi

102 Spontaneous preterm birth before 32 weeks was a secondary outcome.

103 with preterm birth before 37 weeks and with preterm birth before 34 weeks were characterized by an i

104 highest quintile had 0.79 times the risk of preterm birth before 37 weeks (95% confidence interval:

105 Primary outcomes were preterm birth before 37 weeks and before 32 weeks, small

106 tinuous associations of arginine intake with preterm birth before 37 weeks and with preterm birth bef

107 d spontaneous (aRR, 1.34; 95% CI, 1.20-1.53) preterm birth, being small for gestational age at birth

108 on, obstetric delivery, gestational age (for preterm birth), birth weight, birth weight in relation t

109 ully adjusted models, impaired fetal growth, preterm birth, breech presentation and cesarean section

110 nal smoking not only will lower the risk for preterm birth but also will improve late respiratory mor

111 as associated with a small increased risk of preterm birth but no increased risk of small for gestati

112 ngleton pregnancies and no prior spontaneous preterm birth but with short cervical length on transvag

113 ations between exposure to air pollution and preterm birth, but evidence of a relationship with PROM

114 s a key role in brain injury associated with preterm birth, but little is known about the microglial

115 ations between phthalate metabolites and all preterm birth by 8-isoprostane, with the greatest estima

116 ship between maternal phthalate exposure and preterm birth by oxidative stress with repeated measurem

117 ween phthalate exposure during pregnancy and preterm birth by oxidative stress.

118 estational time after 37 weeks in studies of preterm birth) can lead to overestimation of any true be

119 As preterm birth causes plasma estrogen level to drop 100-f

120 Among women with spontaneous preterm birth, cervical length of 25 mm or less occurred

121 Among mothers: preterm birth, cesarean delivery, and hypertensive disea

122 ntervals, fetal growth restriction (FGR) and preterm birth, child nutrition and infection, and enviro

123 The leading under-5 causes were preterm birth complications (1.055 million [95% uncertai

124 , 95% uncertainty range [UR] 28 400-45 200), preterm birth complications (30 900 deaths, 24 200-40 80

125 Preterm birth complications and pneumonia were both impo

126 Preterm birth complications are the leading cause of dea

127 rovinces had lower respiratory infections or preterm birth complications as the leading causes of YLL

128 ause was pneumonia in sub-Saharan Africa and preterm birth complications in southern Asia.

129 The contribution of preterm birth complications to mortality decreased after

130 n of deaths due to congenital abnormalities, preterm birth complications, and injuries nationally, an

131 matory condition that increases the risk for preterm birth, death, and disability because of persiste

132 their possible association with the risk of preterm birth (defined as birth occurring before 37 comp

133 In women with threatened preterm birth, delay of delivery by 48 h allows antenata

134 mmon complications, such as preeclampsia and preterm birth, display developmental phenotypes that rel

135 Hence, preterm birth disrupts interneuron neurogenesis in the M

136 Here, we show that preterm birth disrupts interneuron neurogenesis in the m

137 ts (n=477) in a nested case-control study of preterm birth drawn from a prospective birth cohort of p

138 ith high nitrite intake were associated with preterm birth during the first (AHR = 1.84, 95% CI: 1.14

139 PM2.5 elemental carbon) were associated with preterm birth [e.g., odds ratios for interquartile range

140 Particulate matter exposure and preterm birth: estimates of U.S. attributable burden and

141 Preterm birth explained 89% of the association of matern

142 Antenatal magnesium sulphate given prior to preterm birth for fetal neuroprotection prevents CP and

143 were estimated by repeated ultrasounds, and preterm birth (gestational age <37 wk) and small size fo

144 Women with threatened preterm birth (gestational age 25-34 weeks) were randoml

145 h a preterm first birth and at least 1 later preterm birth had a HR of CVD of 1.65 (95% CI, 1.20-2.28

146 ed women with preterm births and spontaneous preterm births had significantly increased levels of sCD

147 tcomes of interest were perinatal mortality, preterm birth, hospital attendance for asthma exacerbati

148 arean section (HR, 1.17; 95% CI, 1.01-1.34), preterm birth (HR, 1.24; 95% CI, 1.07-1.43), birth weigh

149 ikely explanation for these findings is that preterm birth impedes function of the cerebellum even in

150 reduce maternal anemia in late gestation and preterm birth in comparison with women consuming a norma

151 remature cervical ripening and prevention of preterm birth in humans.

152 ne particulate matter, nitrogen dioxide, and preterm birth in New York City.

153 f universal screening to predict spontaneous preterm birth in nulliparous women using serial measurem

154 iation between GBS maternal colonization and preterm birth in order to inform estimates of the burden

155 C at MR imaging is associated with impending preterm birth in patients with a short sonographic cervi

156 r was negatively associated with spontaneous preterm birth in the adjusted model (OR = 0.90; 95% CI:

157 strategies for fetal protection and delaying preterm birth in the clinical setting.

158 Air pollution and preterm birth in the U.S. state of Georgia (2002-2006):

159 We calculated detectable risk ratios for preterm birth in vaccinated versus unvaccinated women an

160 Preterm birth incorporates an increased risk for cerebel

161 tes, we found that maternal smoking prior to preterm birth increased the odds of having an infant wit

162 Preterm birth (infants born at <37 wk of gestational age

163 r cause of newborn mortality associated with preterm birth, infection, hypoxia, and malformations inc

164 er-individual susceptibility to injury after preterm birth.Inflammation mediated by microglia plays a

165 risk of gestational diabetes, pre-eclampsia, preterm birth, instrumental and caesarean births, infect

166 , 2.0; 95% CI, 1.2 to 3.2, respectively) and preterm birth (IRR, 5.8; 95% CI, 5.3 to 6.5 and IRR, 1.6

167 Preterm birth is a common adverse birth outcome known to

168 Spontaneous preterm birth is a leading cause of infant mortality.

169 Spontaneous preterm birth is a major cause of perinatal morbidity an

170 Preterm birth is a major risk factor for adverse neurolo

171 is review, there is evidence to suggest that preterm birth is associated with maternal GBS colonizati

172 various birth outcomes, but the evidence for preterm birth is mixed.

173 Conversely, the risk of preterm birth is reported to correlate with size of cerv

174 dvanced maternal age and low birth weight or preterm birth is statistically and substantively negligi

175 Preterm birth is the leading cause of neonatal and infan

176 Preterm birth is the leading cause of neonatal mortality

177 wer birth weight (birth weight <2,500 g) and preterm birth (length of gestation <37 weeks).

178 Adverse birth outcomes included preterm birth, low birth weight, and fetal or neonatal d

179 Preterm birth, low birth weight, and greater infant weig

180 Prevalence of preterm birth, low birth weight, small-for-gestational-a

181 ed antiretroviral therapy is associated with preterm birth, low birthweight, small for gestational ag

182 d 11 perinatal outcomes: preterm birth, very preterm birth, low birthweight, very low birthweight, te

183 ements from stationary monitors, and risk of preterm birth (< 37 weeks of gestation) in the U.S. stat

184 interval [CI]: 7.96 to 36.3) after extremely preterm birth (<28 weeks) and 3.58 (95% CI: 1.57 to 8.14

185 d any severe adverse outcome, including very preterm birth (<32 weeks), very SGA (<3rd percentile of

186 Preterm birth (<37 gestational weeks), small for gestati

187 om investigator groups on the association of preterm birth (<37 weeks' gestation) and maternal GBS co

188 Trials in which women considered at risk of preterm birth (<37 weeks' gestation) were randomised to

189 the risk of low birth weight (<2,500 g) and preterm birth (<37 weeks' gestation), we compared betwee

190 The prevalence of preterm birth (<37 weeks) and small for gestational age

191 nfluenza vaccine (MIV) during pregnancy with preterm birth (<37 weeks) and small-for-gestational-age

192 adverse birth outcome, including stillbirth, preterm birth (<37 weeks), small size for gestational ag

193 al age, birth length and head circumference, preterm birth (<37 wk), maternal weight gain, and anemia

194 tational age, low birthweight [<2,500 g], or preterm birth [<37 wk]) in paucigravidae (first or secon

195 low in young age, our findings indicate that preterm birth may be a previously unknown risk factor fo

196 neurodevelopmental disorders associated with preterm birth may result from neurological insults that

197 Up to 3.5 million preterm births may be attributable to GBS.

198 While the odds of preterm birth more than doubled in studies reporting con

199 This nested case-control study of preterm birth (n = 130 cases, 352 controls) included wom

200 tically transmitted, have been implicated in preterm birth, neonatal infections, and chronic lung dis

201 ation, the increase in GBS dissemination and preterm births observed in MCPT4-deficient mice was abol

202 Preterm birth occurred less frequently among pregnancies

203 outcomes compared with unexposed offspring (preterm birth odds ratio [OR], 1.47 [95% CI, 1.40-1.55];

204 loyment was associated with a 3% decrease in preterm birth odds.

205 ciated with significantly increased risks of preterm birth (odds ratio [OR], 1.56; 95% CI, 1.25-1.94;

206 emely challenging to detect (risk ratios for preterm birth of 0.9 to 1.0) and will require sample siz

207 Survivors of preterm birth often present with medical morbidities; ho

208 gnesium treatment, given to women at risk of preterm birth on important maternal and fetal outcomes,

209 rone was not associated with reduced risk of preterm birth or composite neonatal adverse outcomes, an

210 A reduction in SGA births, but not preterm birth or perinatal mortality, was observed in th

211 ancy to be associated with increased risk of preterm birth or small for gestational age, but not of c

212 isk of 25(OH)D concentrations <50 nmol/L for preterm birth or small size for gestational age were 17.

213 Significant reductions were observed in preterm birth (OR = 0.33; 95% CI: 0.12, 0.89) and anemia

214 antidepressant exposure was associated with preterm birth (OR, 1.34 [95% CI, 1.18-1.52]) but not wit

215 of pregnancy (OR, 2.82; 95% CI, 1.58-5.04), preterm birth (OR, 1.56; 95% CI, 1.02-2.38), and use of

216 R score) was associated with a lower risk of preterm births (OR: 0.67; 95% CI: 0.50, 0.91), higher po

217 r sex, parental education, low birth weight, preterm birth, parental social class, maternal smoking a

218 sociation between maternal levels of B12 and preterm birth (per each 1-standard-deviation increase in

219 Preterm birth places infants in an adverse environment t

220 had a significantly increased prevalence of preterm birth (prevalence ratio [PR], 1.52; 95% CI, 1.34

221 t an alternative new therapeutic approach to preterm birth prevention.

222 d (GCF) and serum samples between women with preterm birth (PTB) and full-term birth (FTB) and correl

223 iculate matter (PM2.5) during pregnancy with preterm birth (PTB) and low birth weight (LBW) but disag

224 Racial/ethnic disparities in preterm birth (PTB) are well documented in the epidemiol

225 Preterm birth (PTB) contributes significantly to infant

226 Preterm birth (PTB) has been associated with exposure to

227 Preterm birth (PTB) is a leading cause of neonatal death

228 Preterm birth (PTB) is commonly accompanied by in utero

229 Preterm birth (PTB) is the leading cause of neonatal mor

230 Preterm birth (PTB) is the leading cause of neonatal mor

231 Preterm birth (PTB) is the leading cause of neonatal mor

232 Preterm birth (PTB) is the leading cause of neonatal mor

233 osition methods to understand disparities in preterm birth (PTB) prevalence between births of non-His

234 Preterm birth (PTB) rates (11.4% in 2013) in the United

235 Ambient air pollutants may increase preterm birth (PTB) risk, but critical exposure windows

236 egnancy has high potential for prediction of preterm birth (PTB), a problem affecting 15 million newb

237 and maternal anemia, low birth weight (LBW), preterm birth (PTB), and stillbirth in rural Ethiopia.

238 Molecular mechanisms regulating preterm birth (PTB)-associated cervical remodeling remai

239 tivity (PA) during pregnancy and the risk of preterm birth (PTB).

240 om heightened mTORC1 signaling is a cause of preterm birth (PTB).

241 oxidative stress are known risk factors for preterm birth (PTB); however, the mechanisms and pathway

242 h outcomes [small for gestational age (SGA), preterm birth (PTB)].In an observational study in 987 ne

243 nces associate with up to 40% of spontaneous preterm births (PTB).

244 NAC administration significantly reduced the preterm birth rate and altered placental immune profile

245 nthropometrics, gestational weight gain, and preterm birth rate, but not in maternal age, parity, soc

246 This demonstrates mediation of the phthalate-preterm birth relationship by oxidative stress, and the

247 tion is associated with an increased risk of preterm birth (relative risk 1.50, 95% CI 1.24-1.82), lo

248 a strong protective effect of vaccination on preterm birth (relative risk = 0.79, 95% confidence inte

249 y linked to premature uterine senescence and preterm birth, results in aberrant lipid signatures with

250 IRS protection, >90% IRS protection reduced preterm birth risk (risk ratio, 0.35; 95% confidence int

251 hat IRS may significantly reduce malaria and preterm birth risk among pregnant women with HIV receivi

252 n and meteorological parameters, to increase preterm birth risk has received significant attention wo

253 Preterm birth risk tended to increase with first-trimest

254 as observed, but associations with increased preterm birth risk were found for both increased atmosph

255 Our study lends support for an increase in preterm birth risk with atmospheric pressure.

256 ere was also some evidence of an increase in preterm birth risk with first-trimester average temperat

257 Preterm birth risks associated with air pollution and me

258 to the duration of gestation and the risk of preterm birth, robust associations with genetic variants

259 eks gestation provided greater reductions in preterm birth (RR 0.89, 95% CI 0.85-0.93; p=0.03).

260 one of a history in a previous pregnancy of preterm birth, second trimester loss, preterm premature

261 sociation between smoke-free legislation and preterm birth, showing some degree of bias.

262 olic acid (FA) alone, on risk of spontaneous preterm birth (SPB) and the impact of supplementation ti

263 proportion mediated observed for spontaneous preterm births specifically.

264 owth restriction and the risk of spontaneous preterm birth (sPTB).

265 Main models were stratified by preterm birth status.The prevalence of exclusive breastf

266 -term variation in population-level rates of preterm birth, stillbirth, and perinatal death in Ontari

267 ciated with short-term variation in rates of preterm birth, stillbirth, or perinatal death.

268 se activity, we examined pregnancy outcomes (preterm birth, stillbirth, small for gestational age, or

269 at can occur earlier in pregnancy leading to preterm births, stillbirths, or late-onset neonatal infe

270 ittle by the reason the woman was at risk of preterm birth, the gestational age at which magnesium su

271 s, pelvic inflammatory disease, and possibly preterm birth, tubal factor infertility, and ectopic pre

272 s the relationship between air pollution and preterm birth using 2008-2010 New York City (NYC) birth

273 TC-LPV-R was associated with higher risk for preterm birth, very preterm birth, and neonatal death.

274 etroviral therapy and 11 perinatal outcomes: preterm birth, very preterm birth, low birthweight, very

275 fied between maternal HIV infection and very preterm birth, very small for gestational age, very low

276 distributions were compared for spontaneous preterm birth vs all other births.

277 ociation between dietary arginine intake and preterm birth warrants further investigation.

278 The frequency of preterm birth was 5.0%.

279 In small clinical studies, preterm birth was associated with altered cardiac struct

280 Preterm birth was associated with GBS bacteriuria in coh

281 The positive association with preterm birth was due to iatrogenic instead of spontaneo

282 The rate of preterm birth was not associated with circulating influe

283 A nested case-control study of preterm birth was performed in 2011 from women enrolled

284 FGR and preterm birth was the leading risk factor cluster in all

285 iratory disease during early childhood after preterm birth, we performed a prospective, longitudinal

286 of the association of influenza illness with preterm birth were identified from the published literat

287 Small-for-gestational-age (SGA) and preterm births were examined as secondary outcomes.

288 onally, increased GBS systemic infection and preterm births were observed in MCPT4-deficient mice ver

289 ment in obstetrics that could greatly impact preterm birth, which currently has no successful treatme

290 a-amniotic infections are strongly linked to preterm birth, which is the leading cause of perinatal m

291 was associated with a 5% increase in odds of preterm birth, while second-trimester unemployment was a

292 Among women without prior spontaneous preterm birth who had asymptomatic singleton pregnancies

293 inal follow-up of all survivors of extremely preterm birth who were born in Victoria, Australia, in t

294 such as the reason the woman was at risk of preterm birth, why treatment was given, the gestational

295 ticipating in a nested case-control study of preterm birth with 116 cases and 323 controls.

296 1, EEFSEC, and AGTR2 showed association with preterm birth with genomewide significance.

297 We estimated the risk ratio (RR) for preterm birth with maternal GBS colonization to be 1.21

298 ed to significant adverse sequelae including preterm birth, with cone depth and radicality of treatme

299 Benefit is seen regardless of the reason for preterm birth, with similar effects across a range of pr

300 ta suggest associations with infertility and preterm birth, yet the attributable risk for female geni