ライフサイエンスコーパス: preterm infant

1 le is known about the microglial response in preterm infants.

2 guidelines exist for the nutritional care of preterm infants.

3 t of bronchopulmonary dysplasia in extremely preterm infants.

4 useful therapeutically to alleviate FIRS in preterm infants.

5 cohort study of consecutively born extremely preterm infants.

6 table for monitoring the postnatal growth of preterm infants.

7 AMPA receptors in autopsy samples from human preterm infants.

8 ate preterm") infants, the largest cohort of preterm infants.

9 near term, have not been studied among very preterm infants.

10 patent ductus arteriosus (PDA) in extremely preterm infants.

11 itive development and brain abnormalities in preterm infants.

12 to improve rates of successful extubation in preterm infants.

13 ection, and other morbidity and mortality in preterm infants.

14 22.4% late preterm, and 12.5% very/extremely preterm infants.

15 ve yielded standards for postnatal growth in preterm infants.

16 ntion of invasive infection and mortality in preterm infants.

17 d not be used to measure postnatal growth of preterm infants.

18 Extubation failure is common in extremely preterm infants.

19 t and serum omega-3 LCPUFA concentrations in preterm infants.

20 %5mC accompanies the early growth deficit in preterm infants.

21 ISA may be a promising therapy for extremely preterm infants.

22 ients prevent invasive infection or death in preterm infants.

23 morbidity should be reviewed when evaluating preterm infants.

24 h the development of BPD and with late PH in preterm infants.

25 the high prevalence of cognitive problems in preterm infants.

26 ncrease the effectiveness of screening among preterm infants.

27 result in decreased infections in high-risk preterm infants.

28 utcomes have been reported in the studies of preterm infants.

29 isit to a pediatrician and a second test) in preterm infants.

30 infants, and the postnatal growth period of preterm infants.

31 Our analysis included 95 preterm infants.

32 ociated with high morbidity and mortality in preterm infants.

33 al line-associated bloodstream infections in preterm infants.

34 ssociated with bronchopulmonary dysplasia in preterm infants.

35 the neurodevelopmental outcomes in term and preterm infants.

36 during the first year of life in a cohort of preterm infants.

37 h/neurodevelopmental impairment in extremely preterm infants.

38 enting and treating brain injury in term and preterm infants.

39 and clinical application in treating NEC in preterm infants.

40 (BPD), a respiratory condition that affects preterm infants.

41 he incidence of necrotizing enterocolitis in preterm infants.

42 lute FFM accretion during a hospital stay in preterm infants.

43 timodal brain MRI to study a large cohort of preterm infants.

44 on of preexistent pulmonary vulnerability in preterm infants.

45 tential pathogens, is associated with LOS in preterm infants.

46 ke in relation to the estimated needs of the preterm infants.

47 nistration of PN benefits growth outcomes in preterm infants.

48 or damage increasingly observed in extremely preterm infants.

49 he effect of 5 LEs on plasma phytosterols in preterm infants.

50 demonstration of glutamate excitotoxicity in preterm infants.

51 ll children with cerebral palsy, but not for preterm infants.

52 rican and European-American low-birth-weight preterm infants.

53 spiratory and neurodevelopmental outcomes in preterm infants.

54 cluding bronchopulmonary dysplasia (BPD), in preterm infants.

55 develop respiratory distress syndrome among preterm infants.

56 and late preterm (MLPT) births comprise most preterm infants.

57 l vasculature, eye, and brain development of preterm infants.

58 terval and survival and morbidity among very preterm infants.

59 mental safety of hydrocortisone in extremely preterm infants.

60 eading and intractable cause of mortality in preterm infants.

61 plementation in predominantly human milk-fed preterm infants.

62 rum levels of IL-5 and IL-13 but not IL-4 in preterm infants.

63 g enterocolitis and late-onset sepis in very preterm infants.

64 terocolitis, late-onset sepsis, and death in preterm infants.

65 r neurodevelopmental impairment in extremely preterm infants.

66 -term infants (0.03%) was lower than that in preterm infants (0.57%) (P < .001).

67 RNA gene sequencing.Among the 3161 enrolled preterm infants, 106 (3.4%; 95% CI: 2.8%, 4.0%) develope

68 infants (RR 961.7, 95% CI 681.3-1357.5) and preterm infants (141.7, 90.1-222.8).

69 owing themes: 1) nutrient specifications for preterm infants, 2) clinical and practical issues in ent

70 l and practical issues in enteral feeding of preterm infants, 3) gastrointestinal and surgical issues

71 RSV hospitalizations for 2 groups: moderate-preterm infants (32-34 weeks' gestational age) and term

72 Of 707 live-born extremely preterm infants, 486 (68.7%) survived to 6.5 years of ag

73 The study was conducted on 336 preterm infants; 78 were from diabetic mothers and 258 w

74 or no better than CPAP when used to support preterm infants after extubation.

75 that of CPAP as respiratory support for very preterm infants after extubation.

76 alise the source of delta brush events in 10 preterm infants aged 32-36 postmenstrual weeks.

77 Since preterm infants almost universally receive early and oft

78 an be disrupted by perinatal inflammation in preterm infants, although the mechanisms are incompletel

79 Delivery of a small-for-gestation or preterm infant and early-onset preeclampsia (by week 34)

80 ntake and preterm birth among 496 mothers of preterm infants and 5,398 mothers with full-term deliver

81 onset infections commonly occur in extremely preterm infants and are associated with high rates of mo

82 0 ppm on postnatal days 5 to 14 to high-risk preterm infants and continued for 24 days, appears to be

83 e prevalence of P. jirovecii colonization in preterm infants and its possible association with medica

84 ndicator of long-term health and survival in preterm infants and molds circuit formation, but gaps re

85 splasia is a chronic lung disease of extreme preterm infants and results in impaired gas exchange.

86 ve values and recall rates for full-term and preterm infants and sensitivity for milder forms of CAH.

87 develop existing postnatal growth charts for preterm infants and their methodologic quality.

88 ize and quantify early foveal development in preterm infants and to compare this development between

89 % CI, 2.7-20.9], P < .001 for very/extremely preterm infants) and cerebrospinal fluid (CSF) to blood

90 nt of new guidelines for nutritional care of preterm infants, and 2) develop a targeted research agen

91 ular and imaging data from animal models and preterm infants, and find that microglial expression of

92 Preterm infants are at increased risk of cardiometabolic

93 Preterm infants are at risk for a broad spectrum of neur

94 Very preterm infants are at risk of developing encephalopathy

95 Preterm infants are at significant risk of severe infect

96 Preterm infants are at significantly increased risk for

97 Every year, 15 million preterm infants are born, and most spend their first wee

98 yncytial virus (RSV) prophylaxis in moderate-preterm infants are highly debated.

99 terine body composition reference charts for preterm infants are lacking.

100 Preterm infants are most vulnerable to pertussis.

101 h multicentered trials of lactoferrin use in preterm infants are near completion, regulatory burdens

102 Preterm infants are susceptible to inflammation-induced

103 , major causes of mortality and morbidity in preterm infants, are reduced in infants fed their own mo

104 Clinicians aim to extubate preterm infants as early as possible, to minimize the ri

105 spective pilot study, ON parameters in these preterm infants associate weakly with CNS pathology and

106 motor skills were assessed in 33 of the very preterm infants at 18-24 months corrected age.

107 linically-indicated MRS studies conducted on preterm infants at a single institution during a six-yea

108 The marked reduction in %5mC at IGF2 DMR2 in preterm infants at birth compared with term-age supports

109 factors that affect the body composition of preterm infants at discharge.

110 esonance imaging for early identification of preterm infants at risk for childhood cognitive and acad

111 rth (beta=-11.48, p<0.001) and compared with preterm infants at term-corrected age (t=3.13, p=0.01).

112 velopment, and compare term-born neonates to preterm infants at term-equivalent age.

113 on tensor imaging (DTI) was acquired from 90 preterm infants at term-equivalent age.

114 A prospective observational study of preterm infants (birth weight <1500 g and/or gestational

115 One hundred forty-four consecutive admitted preterm infants (birth weight: 500-1249 g) were studied.

116 ur les Petits Ages Gestationnels), including preterm infants born at <32 wk of gestation in France in

117 A total of 141 preterm infants born at <35 wk of gestation and admitted

118 rtality or moderate/severe BPD among similar preterm infants born at 28 weeks or younger following NS

119 Two thousand three hundred ninety extremely preterm infants born at less than 27 weeks' gestational

120 ulation-based cohort study that included all preterm infants born at less than 29 weeks of gestation

121 Preterm infants born at less than 29 weeks' gestation be

122 Among extremely preterm infants born at US academic centers over the las

123 nary dysplasia than a control emulsion among preterm infants born before 29 weeks of gestation and ma

124 558 eligible (37.8%) spontaneously breathing preterm infants born between 23.0 and 26.8 weeks' gestat

125 Preterm infants born between 26 weeks 0 days' and 31 wee

126 For preterm infants born to injured women during the hospita

127 , we measured top-down sensory prediction in preterm infants (born <33 weeks gestation) before infant

128 als published in English, enrolled intubated preterm infants (born <37 weeks' gestation), and reporte

129 ssociated with structural differences in the preterm infant brain.

130 increases the rate of survival of extremely preterm infants, but there are concerns that improved su

131 f bronchopulmonary dysplasia among extremely preterm infants, but they may compromise brain developme

132 opmentally immature epidermal barrier in the preterm infant can permit entry of microorganisms leadin

133 A total of 277 preterm infants completed echocardiogram and BPD assessm

134 Extremely preterm infants contribute disproportionately to neonata

135 ut associated cerebral lesions are common in preterm infants currently not regarded as at highest ris

136 In preterm infants cytomegalovirus (CMV) infection acquired

137 Among preterm infants, delayed cord clamping did not result in

138 In preterm infants, delayed cord clamping for at least 30 s

139 Preterm infants demonstrated growth deficit early in pos

140 of 401 stools from 84 longitudinally sampled preterm infants demonstrates that meropenem, cefotaxime

141 a separate behavioral control confirmed that preterm infants detect pattern violations at the same ra

142 able disease died), facilities for screening preterm infants displaying high risk features may be ess

143 ds given in the first months of life to very preterm infants does not appear to confer any long-term

144 d vaccine had a reduced risk of delivering a preterm infant during times of high influenza virus circ

145 of intraventricular hemorrhage in extremely preterm infants during the first postnatal days.

146 of a randomized clinical trial of extremely preterm infants, early low-dose hydrocortisone was not a

147 Comparison of screened and not screened preterm infants enrolled in the EPIPAGE 2 national prosp

148 Approximately 1 in 5 preterm infants examined had IOHs, generally unilateral.

149 Preterm infants exhibit different microbiome colonizatio

150 ficits in this ability may be the reason why preterm infants experience altered developmental traject

151 popolysaccharide, or tracheal aspirates from preterm infants exposed to chorioamnionitis.

152 ion was disrupted in saccular stage lungs of preterm infants exposed to systemic inflammation.

153 In preterm infants, exposure to inflammation increases the

154 Preterm infants fed fortified human milk (HM) grow more

155 Following oxygen exposure, preterm infants frequently develop chronic lung disease

156 chronic causes of preterm birth may protect preterm infants from adverse sequelae.

157 Sixty preterm infants (gestation <32 weeks and weight <1500 g

158 Sixty preterm infants (gestation <32 weeks and weight <1500 g

159 mized, noninferiority trial, we assigned 564 preterm infants (gestational age, >/=28 weeks 0 days) wi

160 , double-blind, placebo-controlled trial, 94 preterm infants (gestational age, >/=32 + 0 and </=36 +

161 for noninvasive respiratory support of very preterm infants (gestational age, <32 weeks) after extub

162 effects in the immediate postnatal period in preterm infants (gestational age, <33 weeks); however, f

163 nical trial found that very low-birth weight preterm infants given bovine lactoferrin had a significa

164 ently implicated in nosocomial infection and preterm infant gut colonization.

165 Development of the preterm infant gut microbiota is emerging as a critical

166 ted with nosocomial infections, dominate the preterm infant gut microbiota.

167 Compared with term infants, preterm infants had a blunted cortisol response to physi

168 Preterm infants had a mean (standard deviation) gestatio

169 At birth, preterm infants had a significant decrease in %5mC at DM

170 By term-corrected age, preterm infants had decreased %5mC at both DMR2 (beta=-2

171 At 37-42 weeks PMA, preterm infants had larger vCD and vCDR than term infant

172 Preterm infants had lower values for the global measures

173 Whereas preterm infants had typical neural responses to presente

174 Preterm infants have a higher fat mass (FM) percentage a

175 We hypothesised that preterm infants have altered 5mC at the linked different

176 The safest ranges of oxygen saturation in preterm infants have been the subject of debate.

177 Preterm infants have immature respiratory control and re

178 Clinical studies in term and preterm infants have reported the neuroprotective effect

179 he antibiotics most commonly administered to preterm infants, have non-uniform effects on species ric

180 y outcomes of a randomized clinical trial of preterm infants, high-dose erythropoietin treatment with

181 den of infectious morbidity and mortality in preterm infants in low-income or middle-income countries

182 population-based follow-up study (Extremely Preterm Infants in Sweden Study [EXPRESS]) was conducted

183 January 15, 2014, of the national Extremely Preterm Infants in Sweden Study, including preterm child

184 icator of neurodevelopmental health for very preterm infants in the intensive care nursery.

185 inadequate to meet the requirements of very preterm infants; in addition, intraindividual and interi

186 splasia (BPD), a devastating lung disease in preterm infants, includes inflammation, the mechanisms o

187 ation, or both during the neonatal period in preterm infants is associated with adverse outcomes, inc

188 er early hydrocortisone therapy in extremely preterm infants is associated with neurodevelopmental im

189 spital variation in outcomes among extremely preterm infants is largely unexplained and may reflect d

190 sed to prevent chronic lung disease (CLD) in preterm infants is optimal: noninvasive continuous posit

191 o drop 100-fold, the estrogen replacement in preterm infants is physiological.

192 eferred timing of umbilical-cord clamping in preterm infants is unclear.

193 xygen saturation (SpO2) target for extremely preterm infants is unknown.

194 cause of neurodevelopmental disabilities in preterm infants, is characterized by reduced oligodendro

195 Intraventricular hemorrhage (IVH) in preterm infants leads to cerebral inflammation, reduced

196 e function in the growing population of very preterm infants (less than 32 weeks' gestation) with neo

197 We found that in preterm infants, low serum APN concentrations positively

198 The population included extremely preterm infants (<27 weeks' gestation) born in Sweden be

199 Assisted ventilation for extremely preterm infants (<28 weeks of gestation) has become less

200 rtical cartography methods to a cohort of 52 preterm infants (<31 weeks gestation, mild or no injury

201 Preterm infants (<32 weeks' gestation) previously treate

202 lymphocytes from HCA-positive and -negative preterm infants matched for gestational age, sex, race,

203 Extremely preterm infants may experience intermittent hypoxemia or

204 ocosahexaenoic acid (DHA) supplementation of preterm infants may improve outcomes in visual processin

205 plementation in total parental nutrition for preterm infants may suppress ROP.

206 In preterm infants (n = 44), periventricular leukomalacia w

207 Women delivering moderate/late preterm infants (n = 477) and their infants/placentae (n

208 A prospective cohort of extremely preterm infants (N = 86) and healthy term controls (N =

209 , and were significantly impaired only among preterm infants, not in term infants and young children.

210 ted autopsy materials from human fetuses and preterm infants of 16-35 gestational weeks (gw).

211 ssociation study (GWAS) included 174 Finnish preterm infants of gestational age 24-30 weeks.

212 Both measures were lower in preterm infants of lower birth weight standard deviation

213 Preterm infants often require surfactant administration

214 primary approach to mild to moderate RDS in preterm infants older than 28 weeks' GA.

215 Transfaunation of microbiota from HBM-fed preterm infants or a newly identified and cultured Propi

216 tating lower respiratory tract infections in preterm infants or when other serious health problems ar

217 e interval {CI}, 1.3-8.4], P = .015 for late preterm infants; OR, 7.3 [95% CI, 2.7-20.9], P < .001 fo

218 an adequate knowledge of the development of preterm infants' oral feeding skills so as to optimize t

219 ifferences in motor milestone achievement in preterm infants.Our results suggest that differences in

220 Among 448 preterm infants randomized (mean gestational age, 29.0 [

221 hout Bronchopulmonary Dysplasia in Extremely Preterm Infants) randomized clinical trial conducted bet

222 Ex-preterm infants, regardless of caffeine status, are at r

223 Thinner RNFL in very preterm infants relative to term-born infants may relate

224 er administration of inhaled nitric oxide to preterm infants requiring positive pressure respiratory

225 Preterm infants' risk of RSV hospitalization was similar

226 oid macular edema observed on SD OCT in very preterm infants screened for ROP is associated with poor

227 ational population-based cohort of extremely preterm infants, screening echocardiography before day 3

228 commended that nutritional management of the preterm infant should aim to achieve body composition th

229 Preterm infants should be extubated to noninvasive respi

230 udies seeking immunomodulatory therapies for preterm infants should consider gender as a critical var

231 Extremely preterm infants should receive colostrum, a natural lact

232 Preterm infants show abnormal structural and functional

233 The age at which moderate-preterm infants showed RSV hospitalization risk similar

234 In extremely preterm infants, targeting oxygen saturations of 85% to

235 create body composition reference curves for preterm infants that approximate the body composition of

236 te matter injury (NWMI) is a lesion found in preterm infants that can lead to cerebral palsy.

237 a sensitive marker of brain injury in human preterm infants that predicts poor neurodevelopmental ou

238 In hospitalized preterm infants, the etiology of both early- and late-on

239 Among extremely preterm infants, the incidence of bronchopulmonary dyspl

240 and late-preterm (hereafter, "moderate/late preterm") infants, the largest cohort of preterm infants

241 ctice Guidelines for the Nutritional Care of Preterm Infants: The Pre-B Project" is the first phase i

242 In very preterm infants, thinner papillomacular bundle RNFL corr

243 ence charts for total FM and FFM at birth in preterm infants to assist in following AAP guidelines.

244 , noninferiority trial, we assigned 303 very preterm infants to receive treatment with either high-fl

245 In human preterm infants, tracheal aspirate Clec9a expression pos

246 One hundred thirty-one preterm infants undergoing retinopathy of prematurity (R

247 ermediate progenitors were abundant in human preterm infants until 28 gw.

248 andards should be used for the assessment of preterm infants until 64 weeks' postmenstrual age, after

249 m (SNP)-based genotypes from a cohort of 272 preterm infants, using Sparse Reduced Rank Regression (s

250 th the use of current oximeters in extremely preterm infants was associated with an increased risk of

251 The prevalence of exclusive breastfeeding in preterm infants was lower than in term infants at 4 mo p

252 Breast milk from mothers of preterm infants was monitored in three lactation phases

253 condary outcome, white matter disease of the preterm infant, was semiquantitatively assessed from MRI

254 the creation of postnatal growth charts for preterm infants, was conducted.

255 elial cells (HUVECs) obtained from extremely preterm infants were associated with risk for BPD or dea

256 d with decreased risk of an FFM deficit when preterm infants were compared with reference values for

257 A total of 128 preterm infants were included during the study period.

258 Whereas preterm infants were previously at high risk for destruc

259 g (MRI) scans clinically obtained in 26 very preterm infants were scored for global structural abnorm

260 gnancies (78%) resulted in a live birth, two preterm infants were stillborn, and four pregnancies res

261 d controlled trial, 12 hospitalized tube-fed preterm infants were their own control group in comparin

262 ins in utero for a significant proportion of preterm infants, which focuses attention on the developm

263 Preterm infants who develop neurodevelopmental impairmen

264 at compared early INSURE with NCPAP alone in preterm infants who had never been intubated before the

265 clusions and Relevance: Of the 441 extremely preterm infants who had received active perinatal care,

266 historical high mortality rate of extremely preterm infants who now survive and develop BPD, we hypo

267 Among very preterm infants who received prophylactic early high-dos

268 irth in infants with chorioamnionitis and in preterm infants who subsequently went on to develop lung

269 Among extremely preterm infants who survived to 36 weeks' postmenstrual

270 Triangle Park, NC) imaging was obtained from preterm infants who were being screened for ROP and grad

271 Preterm infants who were exposed to maternal smoking had

272 on (particularly for proteins and lipids) in preterm infants who were fed their mothers' own milk eit

273 clinical trials or observational studies of preterm infants who were given surfactant for respirator

274 ge of term birth, vCD and vCDR are larger in preterm infants who were screened for ROP than in term i

275 /kg/d did not further enhance growth of very preterm infants with a median birth weight of 1200 g, wh

276 atent ductus arteriosus (PDA) ligation among preterm infants with adverse neonatal outcomes and neuro

277 The differences between preterm infants with and those without CSVT were analyze

278 In 23 preterm infants with Bayley Scales of Infant Development

279 Preterm infants with birth weight (BW) </=1250 g.

280 Prospectively enrolled preterm infants with birthweights 500-1,250 g underwent

281 (PH) is associated with poor outcomes among preterm infants with bronchopulmonary dysplasia (BPD), b

282 Among the 1513 preterm infants with data available to determine exposur

283 In a cohort of 90 preterm infants with detailed placental histology and ne

284 Currently no treatments exist for preterm infants with diffuse white matter injury (DWMI)

285 ore diverse and different from that of older preterm infants with established chronic lung disease (b

286 with GA 29-32 weeks without ROP, 13/59 (22%) preterm infants with GA </= 28 weeks without ROP and 14/

287 In this cross-sectional study, 239 former preterm infants with gestational age (GA) </= 32 weeks a

288 med a prospective, longitudinal study of 587 preterm infants with gestational age less than 34 weeks

289 tinal structure differences were observed in preterm infants with no treated ROP compared to term inf

290 Early preterm infants with previous treated ROP had decreased

291 the primary means of respiratory support for preterm infants with respiratory distress has not been p

292 factant in the pathogenesis and treatment of preterm infants with respiratory distress syndrome.

293 When used as primary support for preterm infants with respiratory distress, high-flow the

294 1beta:IL1ra ratio in tracheal aspirates from preterm infants with respiratory failure is predictive o

295 se the observed increase in the incidence of preterm infants with treatment-demanding ROP during a re

296 , 4/264 (2%) full-term infants, 15/125 (12%) preterm-infants with GA 29-32 weeks without ROP, 13/59 (

297 raemia, inflammation, and cerebral injury in preterm infants, with an emphasis on the underlying biol

298 ent of infants diagnosed with BPD and 34% of preterm infants without BPD had no clinical signs of lat

299 less symmetric (F = 6.91; p < 0.015) than in preterm infants without intraventricular hemorrhage.

300 This retrospective cohort study of preterm infants younger than 28 weeks gestational age bo