ライフサイエンスコーパス: preterm labor

1 o adverse effects of betaAR agonists used in preterm labor.

2 een implicated in the onset of both term and preterm labor.

3 ials to determine their efficacy in delaying preterm labor.

4 e treatment of hormone-sensitive cancers and preterm labor.

5 ected subclinical IAI in a human cohort with preterm labor.

6 ed risk of earlier preterm birth, PPROM, and preterm labor.

7 ying nitric oxide-donors in the treatment of preterm labor.

8 isolated from the amniotic fluid of women in preterm labor.

9 ly isolated from amniotic fluids of women in preterm labor.

10 nitiation of full-term labor and LPS-induced preterm labor.

11 ensitivity and positive predictive value for preterm labor.

12 lective COX inhibitors for the management of preterm labor.

13 e premature cervical changes associated with preterm labor.

14 n assessments of their symptoms and signs of preterm labor.

15 ly used therapeutically for the treatment of preterm labor.

16 contractile responsiveness, and the onset of preterm labor.

17 us and placenta during PGN+poly(I:C)-induced preterm labor.

18 anscription factor Hes1 were assessed during preterm labor.

19 us and placenta during PGN+poly(I:C)-induced preterm labor.

20 in utero infections, and the pathogenesis of preterm labor.

21 ations associated with fetal surgery such as preterm labor.

22 are growing in popularity but often lead to preterm labor.

23 ng the fetal period, as they are to women in preterm labor.

24 macrophages and did not independently induce preterm labor.

25 fetal membranes during PGN+poly(I:C)-induced preterm labor.

26 ction of amniotic fluid, chorioamnionitis or preterm labor.

27 ntributes to susceptibility for experiencing preterm labor.

28 ck pain, uterine cramps, fetal distress, and preterm labor.

29 plicated or involved in infection-associated preterm labor.

30 ant mother to bacterial products and promote preterm labor.

31 B1 and ZEB2 in two different mouse models of preterm labor.

32 ve strongly linked bacterial infections with preterm labor.

33 ed the development of improved therapies for preterm labor.

34 preterm delivery resulting from spontaneous preterm labor (10.2 percent vs. 3.5 percent; relative ri

35 ike polarization during spontaneous term and preterm labor; 2) anti-inflammatory (M2)-like macrophage

36 ach delivery was classified by presentation: preterm labor (40%), prelabor premature rupture of membr

37 eceptor gamma (PPARgamma) during spontaneous preterm labor; 5) decidual macrophages from women who un

38 common in the second trimester (55.3%), and preterm labor (52.3%) and abnormal fetal heart rate moni

39 roach for the control of infection-triggered preterm labor, a condition for which there is no effecti

40 Spontaneous preterm labor, a syndrome caused by multiple pathologic

41 s increased for infants delivered because of preterm labor (adjusted odds ratio (OR) = 2.3, 95% confi

42 a higher risk than those who delivered after preterm labor (adjusted odds ratio = 1.11, 95 percent co

43 sia (adjusted OR, 1.59 [95% CI, 1.54-1.63]), preterm labor (adjusted OR, 1.54 [95% CI, 1.50-1.57]), a

44 ith a glucocorticoid (dexamethasone, used in preterm labor), an organophosphate pesticide (chlorpyrif

45 s should remain focused on the prevention of preterm labor and BPD, novel research aimed at promoting

46 ) has been implicated in the pathogenesis of preterm labor and clinical chorioamnionitis at term.

47 w transplant are likely to be accompanied by preterm labor and delivery of LBW or VLBW babies who do

48 um can identify women at risk for subsequent preterm labor and delivery.

49 Chorioamnionitis is associated with preterm labor and fetal inflammatory response syndrome (

50 th U. parvum infection in pregnancy, such as preterm labor and fetal sequelae.

51 ed with the birth of small babies (from both preterm labor and growth restriction), but so is failure

52 Preterm labor and infections are the leading causes of n

53 ed risk of adverse fetal outcomes, including preterm labor and intrauterine death.

54 A correlation between the onset of preterm labor and myometrial activation of the inflammat

55 inflammation, and may contribute to onset of preterm labor and other adverse perinatal outcomes.

56 bsequent effects on the better management of preterm labor and other inflammatory conditions.

57 ys a significant role in the pathogenesis of preterm labor and premature rupture of fetal membranes.

58 um bile acids and complicated by spontaneous preterm labor and stillbirth.

59 rm delivery without IAI, and in 0 of 11 with preterm labor and term delivery without infection (P<.00

60 quirement for infection/inflammation-induced preterm labor and that AP-1 activation is sufficient to

61 mmatory (M1) polarization during spontaneous preterm labor and that PPARgamma activation via rosiglit

62 macrophages are associated with spontaneous preterm labor and that PPARgamma activation via RSG can

63 e doubling of diparesis risk associated with preterm labor and with preterm premature rupture of feta

64 taneous preterm birth presenting as PPROM or preterm labor, and it suggests that bleeding is less pre

65 f pregnancy, including spontaneous abortion, preterm labor, and low amniotic fluid volume at the time

66 rean delivery, length of stay, preeclampsia, preterm labor, and stillbirth.

67 ty in amniotic fluid, increased incidence of preterm labor, and with decreased incidence of respirato

68 ophages from women who underwent spontaneous preterm labor; and 7) treatment with RSG reduces the rat

69 gestive heart failure (CHF), length of stay, preterm labor, anemia complicating pregnancy, placental

70 d odds ratio [aOR], 3.18; 95% CI, 1.27-7.98; preterm labor: aOR, 2.18; 95% CI, 1.06-4.48), intrapartu

71 Clinical GBS isolates obtained from women in preterm labor are hyperhemolytic and some are associated

72 men at 13 North American centers who were in preterm labor at <30 weeks' gestation in a double-blind,

73 ernal stress and terbutaline (used to arrest preterm labor), autism risk factors in humans, on measur

74 2008 to February 2010, women with threatened preterm labor between 26 weeks (plus 0 days) and 32 week

75 sues is associated with noninfection-related preterm labor/birth.

76 ture preventative strategies for spontaneous preterm labor/birth.

77 matory response that accompanies spontaneous preterm labor/birth; however, their role is poorly under

78 ffect of MCPT4 against GBS dissemination and preterm labor can be attributed in part to MCPT4-mediate

79 Preterm labor caused by uterine contractions is a major

80 arly-life factors, including maternal fever, preterm labor, cesarean delivery, and antibiotic or acid

81 ophages from women who underwent spontaneous preterm labor display plasticity by M1<-->M2 polarizatio

82 sted odds ratio, 24.29; 95% CI, 7.48-78.81), preterm labor during current pregnancy (adjusted odds ra

83 for such outcomes are cervical incompetence, preterm labor during current pregnancy, vaginitis or vul

84 ates for other problems, such as spontaneous preterm labor, fetuses small for gestational age, congen

85 d antenatal corticosteroids for mothers with preterm labor from 24 to 34 weeks' gestational age, but

86 The prevention and the treatment of preterm labor have been long-standing challenges.

87 actors such as bacterial vaginosis or during preterm labor have not consistently reduced the preterm

88 mal models of infection/inflammation-induced preterm labor; however, inconsistencies in maternal and

89 of a PTB (HR: 0.84; 95% CI: 0.73, 0.95) and preterm labor (HR: 0.80; 95% CI: 0.69, 0.94).

90 sitive women also had a higher prevalence of preterm labor in a prior pregnancy (20.7%) than did the

91 causes spontaneous abortion, stillbirth, and preterm labor in humans and serves as a model for placen

92 the role of a2V during inflammation-induced preterm labor in mice and its relationship to the regula

93 nd physiological mechanisms of both term and preterm labor in primates.

94 2 pregnant women with known risk factors for preterm labor (including 844 women who were pregnant wit

95 tronidazole did not reduce the occurrence of preterm labor, intraamniotic or postpartum infections, n

96 molecular mechanisms involved in UTI-induced preterm labor involving bacterial adhesins.

97 We suggest that preterm labor is a polymicrobial disease, which requires

98 Although a link between infection and preterm labor is widely accepted, surprisingly little is

99 o infection and are strongly associated with preterm labor, little is known about how human fetal imm

100 cidence of low birth weight (<2.5 kg) and of preterm labor (<37 completed weeks) occurs in associatio

101 In threatened preterm labor, maintenance tocolysis with nifedipine, af

102 nistration of magnesium sulphate to women in preterm labor may aid in primary prevention of cerebral

103 IL-1 in amniotic fluid in preterm labor may promote lung maturation and thus be pa

104 een isolated from amniotic fluid of women in preterm labor, mechanisms of in utero infection remain u

105 via Rho/ROCK signaling, thus contributing to preterm labor-mediated preterm birth.

106 In patients with threatened preterm labor, nifedipine-maintained tocolysis did not r

107 To identify the role of Notch signaling in preterm labor, Notch receptors (Notch1-4), its ligands (

108 A subsequent preterm labor occurred in 17 U. urealyticum-positive wom

109 se of labor triggers and hence contribute to preterm labor onset.

110 birth (ie, preterm birth that occurred after preterm labor or membrane rupture, without other complic

111 ch is elevated in pregnancies complicated by preterm labor or preeclampsia, triggers an inflammatory

112 ng cause of pregnancy complications, such as preterm labor or preeclampsia.

113 ntaneous," which was preceded by spontaneous preterm labor or preterm premature rupture of membranes,

114 , defined as preterm preceded by spontaneous preterm labor or preterm premature rupture of the membra

115 s increased for infants delivered because of preterm labor (OR = 2.7, 95% CI: 1.2, 5.7) and intrauter

116 the placenta to cause spontaneous abortion, preterm labor, or significant disease in the surviving n

117 in progesterone production and a subsequent preterm labor phenotype.

118 cental microbe recovery, was associated with preterm labor, prelabor premature rupture of membranes,

119 and PTBs and its clinical presentations (ie, preterm labor, premature rupture of membranes, and medic

120 es into 2 groups: intrauterine inflammation (preterm labor, preterm membrane rupture, placental abrup

121 cularly associated with infection-associated preterm labor (PTL) in both women and mouse models.

122 occurs in various diseases, but its role in preterm labor (PTL) is unknown.

123 on of uterine inflammatory pathways leads to preterm labor (PTL), associated with high rates of neona

124 f the following: gestational age < 37 weeks, preterm labor (PTL), or premature rupture of membranes (

125 and inflammation cause 30-40% of spontaneous preterm labor (PTL), which precedes PTB.

126 injured women were also at increased risk of preterm labor (relative risk = 7.9, 95% confidence inter

127 he causes of pregnancy complications such as preterm labor requires greater insight into how the uter

128 ng is activated during PGN+poly(I:C)-induced preterm labor, resulting in upregulation of pro-inflamma

129 ers was associated with preterm birth due to preterm labor (RR = 3.6, 95% CI: 1.9, 6.8).

130 disease in pregnant women, which can lead to preterm labor, stillbirth, or severe neonatal disease.

131 of oxidative stress on membranes at term or preterm labor, term not in labor samples in an organ exp

132 fection is a recognized cause of spontaneous preterm labor, the noninfection-related etiologies are p

133 taste receptors as targets for tocolytics in preterm labor therapy.

134 ulphate (MgSO4) are administered to women in preterm labor to reduce neurologic morbidity.

135 that the double hit of PGN+poly(I:C) induces preterm labor via reduction of a2V expression and simult

136 mice injected with the PR antagonist RU486, preterm labor was associated with increased miR-200a, de

137 an amount of cervical dilatation at the time preterm labor was diagnosed (1.8 cm, 1.5 cm, and 1.4 cm,

138 points included cervical status at the time preterm labor was diagnosed and birth weight.

139 Preterm labor was initiated on gestation day 14.5 by int

140 Solving the mystery of preterm labor, which compromises the health of future ge

141 nditions, including cancer-related cachexia, preterm labor with delivery, and osteoporosis.

142 ors may provide effective treatment to delay preterm labor with fewer adverse effects on fetal or neo

143 ster (n = 39), third trimester (n = 40), and preterm labor with intact (n = 131, 85 negative IAI and

144 events were preterm delivery (360 [35.4%]), preterm labor without preterm delivery (269 [26.4%]), an