1 he region of strong hoxc-4 expression in the prevertebral anlagen (i.e., pv7-14).

2  the eye, the lens of the eye, otic vesicle, prevertebral bodies (notochord), and endocardial cushion

3 the aged mouse paravertebral SCG than in the prevertebral celiac/superior mesenteric (C/SMG) sympathe

4  sympathetic neuroaxonal dystrophy occurs in prevertebral celiac/superior mesenteric ganglia (CG/SMG)

5 the rat SNS with higher transcript levels in prevertebral (e.g., celiac-superior mesenteric and major

6  mesenteric ganglion (S.m.g.), a sympathetic prevertebral ganglion, is an integrating center for gast

7 tion was paid to the juncture of the central prevertebral, perivascular, and extraperitoneal regions,

8   The separation of preganglionic axons into prevertebral, rostral paravertebral or caudal paraverteb

9     Alterations of the normal cervicocranial prevertebral soft-tissue contour due to hemorrhage into

10 al CT prompted by an abnormal cervicocranial prevertebral soft-tissue contour has yielded a 16% posit

11 istended with air, the normal cervicocranial prevertebral soft-tissue contour is congruent with the a

12 bscesses were the most common lesions in the prevertebral space.

13  and dendrites ("neuritic dystrophy") in the prevertebral superior mesenteric and celiac ganglia (SMG

14 tonomic nerve terminals, particularly in the prevertebral superior mesenteric ganglia (SMG) and celia

15 ucibly develop NAD in nerve terminals in the prevertebral sympathetic ganglia and the distal portions

16                                              Prevertebral sympathetic ganglia develop markedly enlarg

17 pheral innervation of both paravertebral and prevertebral sympathetic ganglia targets in vivo indepen

18 rk of diabetic autonomic neuropathy in mouse prevertebral sympathetic ganglia, were increased 14.8-fo

19                               Projections to prevertebral targets are established by development of t