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1 using a drop of blood obtained from a finger prick.
5 ion in an unsupervised manner, based on skin prick and sIgE tests taken throughout childhood and adol
6 sensations of irritation, stinging, burning, pricking, and cooling using visual analog scales (VAS).
7 ks (89.3%, P = 0.03) or prepared from finger-prick blood and stored at ambient temperature for 2 week
8 ident adult (>/=16 y) was asked for a finger-prick blood sample, which was used to estimate HIV preva
9 ent assay) analyses were performed on finger-prick blood samples from a population-based survey in 3
14 ed venous blood and a fifth card from finger-prick blood were prepared from 103 HIV patients with a m
17 c tests, but these typically evaluate finger-prick capillary blood samples ( approximately 5 mul) and
18 to examine the influence of the location of pricking in the apple on prick-to-prick skin prick test
19 ngle probe was assessed in an in vivo needle prick model to mimic sequelae of traumatic brain injury.
21 ve (SAW) biochips, to detect HIV in a finger prick of blood within 10 seconds (sample-in-result-out).
22 utomation for plasma extraction (from finger-prick of blood), metering and aliquoting into separate r
23 nd other environmental toxins using a finger prick of blood, thereby providing new insights into thei
27 double sensation: an initial Adelta-related pricking pain is followed by a C-related prolonged burni
28 ng a simple microcentrifugation step, finger-prick PoC testing was a quick and accurate approach for
29 ) survey, we measured the prevalence of skin prick positivity to a panel of allergens, and geometric
34 cessfully to the determination of DBS finger-prick samples from 47 paediatric patients and results co
35 off-label protocol using whole blood finger-prick samples tested with and without a simple three min
38 d at age 12 months: food sensitization (skin prick test >/= 2 mm) and allergy (oral food challenge) t
44 ze in last year, atopy assessed both by skin prick test (SPT) and by the measurement of allergen-spec
47 d the highest AUC (0.79), comparable to skin prick test (SPT) and sIgE to soy extract (0.76 and 0.77,
49 ht to determine the association between skin prick test (SPT) and specific IgE (sIgE) to egg proteins
50 nonallergic (n = 25) children underwent skin prick test (SPT) and specific IgE (sIgE) to peanut and i
51 e, were all negative.The results of the skin prick test (SPT) for Citrus unshiu and specific IgE test
52 c sensitization was determined based on skin prick test (SPT) of five mites, three molds, and nine ot
54 ws' milk-specific IgE antibodies (IgE), skin prick test (SPT) reactivity and double-blind, placebo-co
58 n to egg, milk, or both with a positive skin prick test (SPT) response to the trigger food and/or (2)
59 , milk allergy, or both with a positive skin prick test (SPT) response to the trigger food and/or (2)
60 years of age and develop thresholds for skin prick test (SPT) results and specific IgE (sIgE) levels
61 peanut allergy, and the implications of skin prick test (SPT) screening before peanut introduction.
64 ile atopic dermatitis and preceding egg skin prick test (SPT) sensitization, we found a strong and si
66 276 one-year-old children who underwent skin prick test (SPT) to 4 food allergens and those with dete
67 A total of 433 patients with positive skin prick test (SPT) to birch pollen were analyzed regarding
69 a, egg allergy, or both but 0-mm peanut skin prick test (SPT) wheal responses (n = 542); group III, p
70 Q-5D) health questionnaire, spirometry, skin prick test (SPT), exhaled nitric oxide (FeNO), smell tes
71 Patients with CMA and/or RA underwent skin prick test (SPT), intracutaneous test (ICT), and, when r
75 ic IgE was 10.1% (95% CI: 9.4-10.8) and skin prick test 2.7% (95% CI: 2.4-3.0), food challenge positi
78 AID in history were tested first with a skin prick test and if negative challenged with the culprit N
85 d for selected cases where the history, skin prick test and/or specific IgE are not definitive for th
86 itive ELISA results correlated with the skin prick test areas with the whole body and the setae extra
87 In two patients who showed positive skin prick test but negative for challenge test, titer of spe
95 Inclusion criteria included a positive skin prick test of 6 mm or more (wheal diameter, above the ne
96 med challenge test in 41 cases with positive prick test of Glupearl 19S(R), a major allergic HWP foun
97 ding detection of milk-specific IgE (by skin prick test or serum assay), diagnostic elimination diet,
98 ion, milk-specific IgE levels, and milk skin prick test performed at enrollment, 6 months, 12 months,
99 ed to measure geographical variation in skin prick test positivity and assess whether it was explaine
100 raphical variation in the prevalence of skin prick test positivity in Europe is unlikely to be explai
101 tted for allergic sensitization (either skin prick test positivity or serum-specific IgE >/= 0.35 kU/
104 ociations of NVAS and atopy (defined as skin prick test reaction of >/=3 mm) were analysed using bino
107 ctions to peanut were reported in 1.5%, skin prick test reactivity in 2.0%, and IgE sensitization (>/
109 es demonstrated that exercise increases skin prick test reactivity to and bioavailability of the alle
110 tal IgE, grass pollen-specific IgE, and skin prick test reactivity to grass pollen were all reduced c
111 dren with eczema, wheeze, or a positive skin prick test response before ending exclusive breast-feedi
112 (n = 38) was defined as specific IgE or skin prick test response positivity to Aspergillus fumigatus,
115 POIT was associated with reduced peanut skin prick test responses and peanut-specific IgE levels and
118 ble by using routinely available peanut skin prick test responses or specific IgE levels, but this si
120 total IgE levels, specific IgE levels, skin prick test responses to common aeroallergens, and IgG4 a
121 ith allergic disease) but with negative skin prick test responses to common allergens at randomizatio
122 history of ragweed allergy and positive skin prick test responses to ragweed were randomized and rece
124 ure and sensitization (as determined by skin prick test responses) was analyzed in more than 1000 ref
125 eanut- and Ara h 2-specific IgE levels, skin prick test responses, basophil activation, and TH2 cytok
126 but have peanut-specific IgE, positive skin prick test responses, or both represents a significant d
128 om sample of participants with negative skin prick test results attended a hospital-based food challe
130 tivenoms and cetuximab induced positive skin prick test results in patients with sIgE to alpha-gal.
131 llergy was positive in only 28% and positive prick test results were present in 55% of the 49 VKC-lik
132 Based on available clinical data and skin prick test results, 922 (73%) patients would have been i
133 er IgG4 values (P = .001) and lower egg skin prick test scores (P = .0002) over time and a lower medi
134 g M+ participants tracked the following skin prick test sensitization statuses: M+P+C- > M+P+C+ > M+P
138 culture for varicella-zoster virus, and skin prick test to common food and animal allergens were nond
139 nts aged 18 to 65 years with a positive skin prick test to Dactylis glomerata pollen were exposed to
140 al allergic rhinitis (SAR) and positive skin prick test to grass and olive pollens and evaluate how k
141 action after peanut ingestion, positive skin prick test to peanuts, and positive by double-blind plac
147 3 kUA /l (7.2-120.2), and median peanut skin prick test wheal 11.3 mm (6.5-18)]; four experienced no
152 atopy (grass, house dust mite, and cat skin prick test) and atopic vs. non-atopic asthma at the age
155 y outcomes were desensitization, peanut skin prick test, and specific IgE and specific IgG4 measureme
157 -demographic questionnaire, spirometry, skin prick test, and specific IgE to aeroallergens were done
162 using 6 predictors: sex, age, history, skin prick test, peanut specific immunoglobulin E (sIgE), and
165 o initially had negative results on the skin-prick test, the prevalence of peanut allergy at 60 month
166 which was determined with the use of a skin-prick test--one consisting of participants with no measu
169 allergens: OR = 1.81, 95% CI 0.80-4.24; skin prick test/4+ allergens: OR = 2.27, 95% CI 1.34-3.95).
170 of "atopic eczema," "any positive SPT [skin-prick test]," "sensitization to egg," and "sensitization
173 blot and IgE-ELISA were complemented by Skin Prick Testing (SPT) and mediator release assay to determ
175 eta lactam testing with 17% undertaking skin prick testing (SPT) only, 77% SPT followed by intra-derm
176 y fever, eczema, food allergy, positive skin prick testing (SPT), or elevated allergen-specific serum
179 vention (structured allergy history and skin prick testing and appropriate advice on allergy avoidanc
180 tervention (structured allergy history, skin prick testing and appropriate allergy avoidance advice)
182 ined as one or more positive results on skin prick testing and clinically relevant symptoms of rhinit
185 aking a structured allergy history with skin prick testing and tailored advice on allergy avoidance r
190 levant sensitizations are elucidated by skin prick testing or by the determination of specific IgE in
191 of 5276 one-year-old infants underwent skin prick testing to peanut, egg, sesame, and cow's milk or
194 s pollen allergic individuals underwent skin prick testing with allergen alone, allergen plus Bet-APE
195 cted to topical cowhage provocation and skin prick testing with histamine and assessed for difference
197 Opishorchis felineus and specific IgE, skin prick testing, and atopic symptoms in Western Siberia, w
198 interviewer-administered questionnaire, skin prick testing, and measurement of lung function from the
199 ar in children positive and negative on skin prick testing, and were not appreciably altered by the e
202 -bronchodilator spirometry (n = 1,389), skin prick testing, lung volumes, and diffusing capacity meas
203 or scorings of symptoms and medication, skin prick testing, total IgE, specific IgE, and Der p 1-spec
213 Conjunctival provocation tests (CPT), skin prick tests (SPT), BAT, and sIgE determination including
215 inst common allergens was determined by skin prick tests (SPT); specific immunoglobulin E (sIgE) tite
217 hey answered a questionnaire, underwent skin prick tests (SPTs) for common aeroallergens, and provide
218 invited to a parental questionnaire and skin prick tests (SPTs) to ten airborne allergens, and 2148 (
219 eactions was obtained, and standardized skin prick tests (SPTs) using finely ground tree-nut solution
220 10-fold dilutions of milk protein, and skin prick tests (SPTs) were performed to commercial milk ext
222 al work-up included a detailed history, skin prick tests (SPTs) with IVIP, and basophil activation te
223 lowing outcomes at age 2 years: eczema, skin prick tests (SPTs), increased allergen-specific IgE leve
224 h mollusc tolerance) were studied using skin prick tests (SPTs), specific IgEs (sIgEs) and SDS-PAGE i
226 lergic subjects (group 4) by performing skin prick tests and APTs with rBet v 1 and hypoallergenic rB
230 aking a structured allergy history with skin prick tests and giving tailored advice on allergy avoida
233 Associations with allergic diseases, skin prick tests and lung function assessed at 12 and 18 year
234 Clinic evaluation, which consisted of skin prick tests and OFC where eligible, was undertaken if st
237 d 5276 infants (74% participation) with skin prick tests and sensitized infants underwent food challe
240 ites was diagnosed longitudinally using skin prick tests and specific IgE measurements at (1/2), 1(1/
241 , because of the limited sensitivity of skin prick tests and specific IgE tests to meat extracts.
244 d consent; evidencing of an allergen by skin prick tests and/or serum-specific IgE dosages; being abl
245 (5-17 years old) with asthma underwent skin prick tests at baseline and had clinical data collected
246 itization (FAS) was identified by using skin prick tests conducted between 1 and 18 years of age to a
249 etermine whether C+ assayed by means of skin prick tests influenced AR symptom severity in controlled
250 o hundred eighty-one children had valid skin prick tests performed, and 14% (39/281) were atopic.
254 on between 10 loci and specific IgE and skin prick tests to individual allergens and poly-sensitizati
259 ood-specific serum IgE measurements and skin prick tests were performed before initiating the diet.
260 E inhibition, ImmunoCAP inhibition, and skin prick tests were performed using samples from selected p
264 , we studied the possibility to perform skin prick tests with cetuximab, which carries the alpha-gal
267 es with standardized doses of rMal d 1, skin prick tests with recombinant allergens, and measurements
269 c immunoglobulin E-antibodies in serum, skin prick tests, and double-blind, placebo-controlled food c
271 e of testing (specific IgE measurement, skin prick tests, and oral food challenges), and the timing a
272 allergic sensitization were measured by skin prick tests, and physician-diagnosed inhalant and food a
275 unction samples, we performed histamine skin prick tests, investigated the contribution of STAT3 to a
290 ny offering blood tests obtained from finger prick (Theranos) and 2 major clinical testing services t
296 a "sample-to-answer" diagnosis from a finger-prick volume of human blood, within 45 min, with minimal
297 At Day 85, 6 weeks after the last dose, skin prick wheal responses to allergen were suppressed by > 9
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