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1 rimary CNS lymphoma (PCNSL) is an aggressive primary brain tumor.
2 orme (GBM) is the most common and aggressive primary brain tumor.
3 toma (GBM) is the most common and aggressive primary brain tumor.
4 mon, aggressive and highly malignant type of primary brain tumor.
5 strocytic tumors are the most common form of primary brain tumor.
6 (GBM), the highest grade and most aggressive primary brain tumor.
7 ma multiforme is the most aggressive type of primary brain tumor.
8 Astrocytic tumor is the most prevalent primary brain tumor.
9 Glioblastoma multiforme (GBM) is a deadly primary brain tumor.
10 ovide insight into disease causation of this primary brain tumor.
11 ation, as well as in patients with untreated primary brain tumors.
12 erapeutic opportunities to effectively treat primary brain tumors.
13 terface, were estimated for 33 patients with primary brain tumors.
14 Astrocytomas are the most common primary brain tumors.
15 , Parkinson's disease, stroke, epilepsy, and primary brain tumors.
16 gent with a cytotoxic agent in patients with primary brain tumors.
17 entify new potential therapeutic targets for primary brain tumors.
18 genesis of oligodendrogliomas, a subtype of primary brain tumors.
19 g development, in response to injury, and in primary brain tumors.
20 m occurs in up to one-third of patients with primary brain tumors.
21 vel insights into the pathogenesis of VTE in primary brain tumors.
22 that accounts for the majority of malignant primary brain tumors.
23 astoma multiforme (GBM), the most aggressive primary brain tumors.
24 gene transfer throughout highly disseminated primary brain tumors.
25 ers to aid diagnosis and characterization of primary brain tumors.
26 understood comorbidity for individuals with primary brain tumors.
27 e the motility-promoting role of brevican in primary brain tumors.
28 nitoring and targeting this pathway in these primary brain tumors.
30 married, non-Hispanic white sample, 66% had primary brain tumors, 27% had brain metastases, and 8% u
32 oblastomas are the most prevalent and lethal primary brain tumor and are comprised of hierarchies wit
33 ma (GBM) is the most prevalent and malignant primary brain tumor and contains self-renewing, tumorige
37 ma multiforme (GBM) is the most common adult primary brain tumor and is comprised of a heterogeneous
39 mas (GBMs) are the most common and malignant primary brain tumors and are aggressively treated with s
40 nt gliomas are the most frequently occurring primary brain tumors and are resistant to conventional t
41 tumors and whole-brain irradiation, 19 with primary brain tumors and chemotherapy only, 52 with prim
42 m of brain cancer, is responsible for 23% of primary brain tumors and has extremely poor outcome.
43 lpha(v)beta(3) is frequently up-regulated in primary brain tumors and metastatic lesions of brain hom
44 ictive of outcome in children with recurrent primary brain tumors and should be evaluated as a progno
45 hanced JNK activation is frequently found in primary brain tumors and that this activation contribute
47 ed MR imaging examinations (55 patients with primary brain tumors and whole-brain irradiation, 19 wit
48 cts grouped for urgent referral, 2 confirmed primary brain tumors (and a possible but unconfirmed thi
49 lid tumors, 50% (six of 12) of patients with primary brain tumors, and 0% (zero of nine) of patients
53 Malignant gliomas, the most common type of primary brain tumor, are a spectrum of tumors of varying
54 malignant glioma, the most lethal and common primary brain tumor, are undergoing a dramatic evolution
56 ally, examination of frozen sections from 10 primary brain tumor biopsies by immunohistochemistry rev
57 -regulate or maintain EGFR overexpression in primary brain tumors by directly interacting with the re
58 nhanced our understanding of the genetics of primary brain tumors by uncovering several novel driver
62 rme (GBM) are aggressive and uniformly fatal primary brain tumors characterized by their diffuse inva
64 transformed mouse embryo fibroblasts and for primary brain tumor development in a rat model of neuroc
65 clude 298 children under age 20 years with a primary brain tumor diagnosed from 1984 to 1991 and 298
66 These studies demonstrate that melanomas and primary brain tumors express common MAAs and could be ex
68 ic gliomas, the most common and malignant of primary brain tumors, frequently contain activating muta
73 Glioblastoma multiforme (GBM) is a fatal primary brain tumor harboring myriad genetic and epigene
79 ients with glioma, the most common malignant primary brain tumor in adults, examination of cell-free
80 toma multiforme, the most aggressive form of primary brain tumor in adults, is nearly universally fat
81 ioblastoma is the most common and aggressive primary brain tumor in adults, with a poor prognosis bec
87 oblastoma (GBM) is the most common and fatal primary brain tumor in humans, and it is essential that
93 ress has been made in this regard for common primary brain tumors in adults, especially diffuse gliom
98 ancer survivors at risk of developing second primary brain tumors, in particular survivors of childho
100 NRP/B expression was also observed in human primary brain tumors including glioblastoma multiformae
101 n institutional phase II trial for localized primary brain tumors, including craniopharyngioma, epend
102 er factor (HGF/SF) and Met staining in human primary brain tumors increases with the grade of maligna
103 In conclusion, high podoplanin expression in primary brain tumors induces platelet aggregation, corre
104 of cognitive deficits in adult patients with primary brain tumors is the tumor itself and more signif
106 Glioblastoma, the most malignant type of primary brain tumor, is one of the solid cancers where c
108 analysis of 300 meningiomas, the most common primary brain tumors, leading to the discovery of mutati
112 he nucleus of normal brain cells, whereas in primary brain tumors NRP/B was almost exclusively contai
114 ts use as a diagnostic marker to distinguish primary brain tumors of similar histology but different
119 been used in the evaluation of patients with primary brain tumors (PBTs), but so far series have repo
121 was performed on 27 children with recurrent primary brain tumors referred to our institution for inv
123 multiforme (GBM), the most common malignant primary brain tumor, represents a significant disease bu
125 analysis demonstrated that 86% (18 of 21) of primary brain tumors showed evidence of JNK activation b
130 This article reviews the classification of primary brain tumors, the histologic changes associated
131 ials using novel agents for the treatment of primary brain tumors, there have been few advances that
132 has been shown to be frequently activated in primary brain tumors, to enhance several tumorigenic phe
133 els in individual cells in sections from 167 primary brain tumors treated with BCNU were quantitated
134 e glycoprotein, increases the risk of VTE in primary brain tumors via its ability to induce platelet
136 he expression of NRP/B mutants isolated from primary brain tumors was found in the cytoplasm, and the
137 ion of these gene-inactivation mechanisms in primary brain tumors, we integrated RLGS-based methylati
140 e (MR) images in a cohort of patients with a primary brain tumor who had not received linear gadolini
141 ltiforme (GBM) is the most common and lethal primary brain tumor with median survival of only 12 to 1
142 ifome is the most common and most aggressive primary brain tumor with no current curative therapy.
145 f dying as a result of the transmission of a primary brain tumor with the risks of dying if not trans
147 brain tumors and chemotherapy only, 52 with primary brain tumors without any treatment, and 18 with
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