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1 at are perturbed by single-gene mutations in primary immune deficiency.
2 fer several advantages for the correction of primary immune deficiencies.
3 he enrolled persons was 9 years, and 99% had primary immune deficiencies.
4 understanding of the molecular basis of many primary immune deficiencies.
5 leukocyte adhesion deficiency are the major primary immune deficiencies affecting phagocytic blood c
7 se for cell-based therapies for cancer, HIV, primary immune deficiencies, and autoimmune diseases, bu
9 ommon variable immune deficiency (CVID) is a primary immune deficiency characterized by low levels of
11 h American centers studying therapy for rare primary immune deficiency diseases (PIDs), including sev
15 ciency (CVID), the most frequent symptomatic primary immune deficiency in humans, is a heterogeneous
16 ophil homeostasis using a zebrafish model of primary immune deficiency induced by the human inhibitor
17 we summarize the recent advances in treating primary immune deficiency (PID) disorders by stem cell t
20 al missense variants in a gene implicated in primary immune deficiency, PLCG2, and a negative regulat
21 s of donors that is used to prevent or treat primary immune deficiency, several infectious diseases,
22 tion in a range of new, genetically defined, primary immune deficiency states; and (iii) experimental
23 ts cause loss of gene function, resulting in primary immune deficiencies such as severe combined immu
24 icacy of the ongoing gene therapy trials for primary immune deficiencies, the first reports of new tr
31 ur review of the literature regarding PML in primary immune deficiencies we found 26 cases, only 54%
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