ライフサイエンスコーパス: primary lesion

1 flammation to sites that are remote from the primary lesion.

2 perception, suggesting that transport is the primary lesion.

3 [CI], 81.8% to 98.0%) achieved cure of their primary lesion.

4 anoma in the CNS without evidence of another primary lesion.

5 tant Staphylococcus aureus isolated from the primary lesion.

6 release of ATP in the region surrounding the primary lesion.

7 ence of disease at the site of the regressed primary lesion.

8 in viral load and delayed resolution of the primary lesion.

9 derived from the nucleolytic processing of a primary lesion.

10 ases cluster on a branch separate from their primary lesion.

11 d oligodendrocyte and RGC death far from the primary lesion.

12 ximately 15% of patients with a thin (<1 mm) primary lesion.

13 dine photodimers (CPD) are the most abundant primary lesion.

14 d of symptoms into areas not affected by the primary lesion.

15 ell line that originated from the autologous primary lesion.

16 s almost as effective in identification of a primary lesion.

17 c niches uncoupled from lymphangiogenesis at primary lesions.

18 ch metastatic deposits compared with ovarian primary lesions.

19 osis interval, 6.0+/-4.4 months) and 8 human primary lesions.

20 mor purity is sufficient to characterize the primary lesions.

21 NET lesions, especially for bone and unknown primary lesions.

22 xpressed in human coronary restenotic versus primary lesions.

23 nized control group (pVAX-FI-Mock) developed primary lesions.

24 e aberrant overgrowth in tissues surrounding primary lesions.

25 s higher in liver metastatic lesions than in primary lesions.

26 on and corresponding nuclear localization in primary lesions.

27 have a high (2-3%/year) incidence of second primary lesions.

28 he basis of the normal spread pattern of the primary lesion, 58 abnormal unexpected foci of hypermeta

29 Of these, 213 underwent resection of the primary lesion and evaluation of the draining lymph node

30 2 different sequencing methods, both in the primary lesion and in 1 metastasis.

31 ancer demonstrated significant uptake in the primary lesion and in the metastases.

32 e mutation being found in all four tumors (a primary lesion and three different metastases) from the

33 noma have microscopically thin (< or = 1 mm) primary lesions and are cured with excision.

34 ized with either gD or gHt-gL showed reduced primary lesions and exhibited no secondary zosteriform l

35 larly associated with regions of invasion of primary lesions and metastases.

36 Posterior fossa primary lesions and metastatic disease at diagnosis were

37 spreads through the skin, resulting in large primary lesions and satellite lesions, and infects inter

38 showed that it is strand-specific repair of primary lesions and site-specific selection of the resul

39 h more effective in reducing the severity of primary lesions and the number of satellite lesions than

40 procedure, Breslow depth, ulceration of the primary lesion, and disease stage.

41 variation in isolated cases (especially when primary lesions are located on the trunk or the head and

42 Mice with nonhealing primary lesions are nonetheless resistant to reinfection

43 s the additional advantage of evaluating the primary lesions as well as metastases, it could be cost-

44 e spinal cord was crushed at T8, producing a primary lesion at the site of the trauma and a secondary

45 Uptake in the primary lesions at 4 representative time points (5, 17,

46 We conclude that the primary lesion caused by this cationic peptide is not gr

47 ata collected included patient demographics, primary lesion characteristics, operative details, and r

48 uantitative image analysis revealed that the primary lesion decreased dramatically in size and cavita

49 2) were commonly differentially expressed in primary lesions, each significantly more often in squamo

50 Twenty-three patients with CMM (primary lesions > 1.5 mm thick) scheduled for lymph node

51 Sixty-seven (69%) of 97 with assessable primary lesions had a clinical CR at the primary tumor s

52 h muscle cells (SMCs), and its expression in primary lesions has been proposed to be predictive of re

53 ses are responsible for amplification of the primary lesion in CNS trauma and stroke.

54 The primary lesion in FAA.K1B was identified as a 95% reduct

55 hematopoietic cells and suggesting that the primary lesion in Foxo1(KR/KR) mice occurs in the vessel

56 Primary lesions in affected birds were inclusion body he

57 or 5,6-dihydroxy-5,6-dihydropyrimidines, are primary lesions in DNA induced by reactive oxygen specie

58 FDG uptake of the primary lesions in patients with a new diagnosis of adva

59 Irradiated primary lesions included craniopharyngioma, pituitary ad

60 , whereas the remaining 6 patients had their primary lesions intact.

61 No primary lesion is identified in 10% to 20% of patients p

62 ograde oligodendrocyte stress, away from the primary lesion, is an important factor after ischemic op

63 prominent when analyzing those patients with primary lesions larger than 2 cm.

64 t microvascular endothelial apoptosis is the primary lesion leading to stem cell dysfunction.

65 Interestingly, all patients with IMSLNs had primary lesions located inferior to the breasts.

66 In group 2, primary lesions located medially had a higher rate of IM

67 taneous melanoma returned 1158 patients with primary lesion < or = 1 mm thick and who received their

68 ntinel was 7.3%, 19.7%, 33.2%, and 39.7% for primary lesions </=1.0, 1.01-2.0, 2.01-4.0, and >4.0 mm,

69 Patients with recurrence had deeper primary lesions (mean thickness, 2.7 vs 1.8 mm; P < .01)

70 atment with anti-IL-17 decreased the size of primary lesions, numbers of satellite lesions, and viral

71 We found that TNFR1-/- mice developed larger primary lesions, numerous satellite lesions, and higher

72 dds of finding TprK variants in disseminated primary lesions (odds ratio [OR] = 3.3 [95% confidence i

73 pproach, we have conclusively shown that the primary lesion of Al is apoplastic.

74 esent and functional in GNU embryos, and the primary lesion of the gnu mutation has therefore remaine

75 howed a decrease in the diameter of 6 and 12 primary lesions of the 13 and 14 patients examined, resp

76 that recapitulates the pathology seen in the primary lesions of the Pten mutant prostate model.

77 Neuropathic pain is caused by a primary lesion or dysfunction of the nervous system and

78 Primary lesions progressed in severity, satellite lesion

79 oma patients who are diagnosed with multiple primary lesions remains controversial.

80 t the time of imaging, 20 patients had their primary lesions resected, whereas the remaining 6 patien

81 esis of IL-12 p40 in ICSBP-/- animals is the primary lesion responsible for the loss in resistance to

82 By univariate analysis, nodal status of the primary lesion, short disease-free interval before detec

83 te marker of neuronal injury not only at the primary lesion site but also in the antero- and retrogra

84 Variables examined included sex, stage of primary lesion, size of liver lesion(s), number of lesio

85 lified in the recurrent tumor but not in the primary lesion, suggesting this locus can be involved in

86 re identified either simultaneously with the primary lesion (synchronous) or after a period of time (

87 eters were computed: maximum diameter of the primary lesion (T), of the largest lymph node (N), and o

88 We tested the hypothesis that primary lesions that develop restenosis after coronary a

89 ncreased in coronary atherectomy tissue from primary lesions that develop restenosis, suggesting a po

90 ore macrophages and smooth muscle cells than primary lesions that do not develop restenosis.

91 albumin (OVA) sensitization exhibited larger primary lesions that were erosive, more satellite lesion

92 melanoma can be divided into therapy of the primary lesion, treatment of the lymph nodes, treatment

93 etastasis, lymph nodes, bone metastasis, and primary lesion was 95%, 95%, 90%, and 93% for (68)Ga-DOT

94 The scan was considered false-positive if no primary lesion was found corresponding to the (68)Ga-DOT

95 The primary lesion was localized to the stria vascularis, wh

96 ximum standardized uptake value (SUV) of the primary lesion was measured in each patient.

97 ivity for detection of pathologically proven primary lesions was 100%, 62.5% and 87.5% with FDG-PET,

98 The mean SUV value of primary lesions was 9.4 (range 2.0-20.7, n = 16), with o

99 p16 staining of the primary lesions was correlated with genetic analysis inc

100 Clinical improvement of primary lesions was seen in all patients; improvement wi

101 and restenosis following angioplasty of the primary lesion, we fed apolipoprotein E-deficient mice a

102 Two unknown primary lesions were identified solely by (68)Ga-DOTATAT

103 Primary lesions were more often on the extremities (40%)

104 Primary lesions were more variable in tracer accumulatio

105 ere afforded only partial protection in that primary lesions were observed in most animals, although

106 s after infection were associated with fewer primary lesions, while strong tuberculin reactions at 33

107 tities only 4% more, the identification of a primary lesion with SRS obviates for the most part the u

108 and 1361A melanoma cell lines (derived from primary lesions with metastases), respectively.