ライフサイエンスコーパス: primary mesenchyme

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1 The rescue includes expression of the primary mesenchyme cell (PMC) differentiation program, e

2 In the sea urchin, the primary mesenchyme cell (PMC) GRN controls the developme

3 The micromere-primary mesenchyme cell (PMC) GRN drives the development

4 Here, we characterize FoxN2/3 in the primary mesenchyme cell (PMC) GRN state.

5 ude failure of spiculogenesis and of correct primary mesenchyme cell (pmc) patterning in the postgast

6 dramatic cell adhesion changes contribute to primary mesenchyme cell ingression movements and to cell

7 s in a profound but reversible inhibition of primary mesenchyme cell organization and endoderm morpho

8 ires a conversation between the skeletogenic primary mesenchyme cells (PMCs) and the overlying patter

9 Primary mesenchyme cells (PMCs) at the vegetal pole of t

10 adhesion assays demonstrated previously that primary mesenchyme cells (PMCs) change their affinity fo

11 lular compartment surrounding the calcifying primary mesenchyme cells (PMCs) conforms to the surround

12 oviding guidance and differentiation cues to primary mesenchyme cells (PMCs) during sea urchin gastru

13 In the sea urchin embryo, primary mesenchyme cells (PMCs) fuse to form syncytial f

14 he calcium carbonate skeleton is secreted by primary mesenchyme cells (PMCs) in response to largely u

15 Sea urchin primary mesenchyme cells (PMCs) ingress into the blastoc

16 The primary mesenchyme cells (PMCs) of the sea urchin embryo

17 The primary mesenchyme cells (PMCs) of the sea urchin embryo

18 The migratory primary mesenchyme cells (PMCs) of the sea urchin embryo

19 Uniquely, the primary mesenchyme cells (PMCs) that construct the endos

20 genitors of the skeletogenic mesenchyme, the primary mesenchyme cells (PMCs), are important both for

21 (KD) disrupts the patterning and function of primary mesenchyme cells (PMCs), which form the embryoni

22 n embryogenesis, the skeleton is produced by primary mesenchyme cells (PMCs).

23 formation of parallel oral skeletal rods by primary mesenchyme cells (PMCs).

24 the sea urchin embryo is synthesized by the primary mesenchyme cells (PMCs).

25 widely all over the embryo, including in the primary mesenchyme cells and in the surface epithelial c

26 at although most proteases in homogenates of primary mesenchyme cells are not sensitive to the inhibi

27 process begins by ingression of prospective primary mesenchyme cells into the blastocoel, after whic

28 Ingression of primary mesenchyme cells is accompanied by the rapid los

29 undance detectable in the medium of cultured primary mesenchyme cells is inhibited by BB-94.

30 quired for correct spatial expression in the primary mesenchyme cells of an indirect-developing host.

31 The primary mesenchyme cells of the sea urchin embryo constr

32 lectron microscopic examination of inhibited primary mesenchyme cells shows an accumulation of charac

33 hese inhibitors, BB-94, shows that fusion of primary mesenchyme cells still occurs in the presence of

34 essation of massive Ca ion transport via the primary mesenchyme cells to the spicule.

35 ectoderm influences both the arrangement of primary mesenchyme cells within the blastocoel and the s

36 ies the specification of skeletogenic cells (primary mesenchyme cells, or PMCs) has recently been elu

37 ecification of embryonic skeletogenic cells (primary mesenchyme cells, PMCs) has recently been elucid

38 oteins includes many candidate regulators of primary mesenchyme morphogenesis, including PMC-specific

39 n the effects of serum on expression of some primary mesenchyme-specific proteins in micromere cultur

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