ライフサイエンスコーパス: primary sensory

1 This ATP excites primary sensory afferent fibers and also stimulates neig

2 mocortical cells receive sensory signals via primary sensory afferents and cortical signals via corti

3 associated with peripheral sensitization of primary sensory afferents and the development of inflamm

4 Primary sensory afferents and their neighboring host-def

5 ion of the presynaptic NMDAR activity in the primary sensory afferents is an effective approach to at

6 Primary sensory afferents of the dorsal root ganglion (D

7 nsmission at the central synapses of crushed primary sensory afferents through a mechanism that can b

8 hough these channels are highly expressed in primary sensory afferents, accumulating evidence indicat

9 ed with enhanced release of Substance P from primary sensory afferents.

10 using conventional seed-based analyses in 3 primary sensory and 3 association networks as normal you

11 rneuron dendrite remodeling is common across primary sensory and higher-level cortices.

12 The cilia on these cells combine features of primary sensory and motile cilia, but how this cilia sub

13 ccur regularly during REM sleep, but only in primary sensory and motor areas and mostly in layer 4, t

14 vibrissa leads to a rapid depolarization of primary sensory and motor areas that subsequently spread

15 Networks derived using primary sensory and motor cortex seeds were already well

16 In contrast, "unimodal" regions such as the primary sensory and motor cortices show a much more sing

17 In contrast, primary sensory and motor cortices showed effector-indep

18 long the cortical processing hierarchy, from primary sensory and motor to frontal cortices.

19 d allow signals to flow reciprocally between primary sensory and parietal cortex.

20 and posterior cingulate cortex, and several primary sensory areas (all r > 0.58; P < 0.05, corrected

21 cal layout consists of few modality-specific primary sensory areas and a multitude of higher order on

22 in both children with and without ADHD, with primary sensory areas attaining peak cortical thickness

23 studied structures, which was greater in the primary sensory areas during the encoding (Wilcoxon rank

24 ovide evidence for bottom-up processing from primary sensory areas into higher association areas duri

25 on of neuronal and non-neuronal cells in the primary sensory areas of the neocortex of a South Americ

26 multisensory integration emerges already in primary sensory areas or is deferred to higher-order ass

27 upport the view that intermingled neurons in primary sensory areas send specific stimulus features to

28 Cortical-feedback projections to primary sensory areas terminate most heavily in layer 1

29 the transcription factor Ctip1 functions in primary sensory areas to repress motor and activate sens

30 r to the pyramidal network commonly found in primary sensory areas, consisting of accommodating pyram

31 ent development should be revisited even for primary sensory areas, in that the connectivity basis fo

32 ral mechanisms of cross-modal integration in primary sensory areas, such as the primary visual cortex

33 om multiple senses, even at the level of the primary sensory areas.

34 y serve a general modulatory function across primary sensory areas.

35 minar circuitry similar to those observed in primary sensory areas.

36 ed cortical association areas but extends to primary sensory areas.

37 imited to a spatial window of integration in primary sensory areas.

38 higher order association areas and to spare primary sensory areas.

39 of topographic maps, which are so helpful in primary sensory areas.

40 fat was considered to be tasteless, and its primary sensory attribute was believed to be its texture

41 alibut larvae at the time of hatching may be primary sensory cells or interneurons representing the f

42 Many primary sensory cilia exhibit unique architectures that

43 Most studies of BBS have focused on primary, sensory cilia.

44 uid movement over epithelial surfaces, while primary (sensory) cilia play roles in cellular signallin

45 se the possibility that basic dysfunction in primary sensory circuitry may illustrate mechanisms impo

46 Primary sensory circuits encode both weak and intense st

47 Norepinephrine released within primary sensory circuits from locus coeruleus afferent f

48 The gain of signaling in primary sensory circuits is matched to the stimulus inte

49 m rod and cone photoreceptors, acting as the primary sensory conduit mediating non-image-forming resp

50 tterns of plaques in the vibrissae-receptive primary sensory cortex (barrel cortex), in which the cor

51 eorganization of the body part somatotopy in primary sensory cortex (S1 complex, hereafter S1) [1, 2]

52 the anterior cingulate cortex (ACC) and the primary sensory cortex (S1) in rats with inflammatory pa

53 normalized coordinates, broadcast throughout primary sensory cortex and provides strong modulation of

54 irst to reveal distinct interactions between primary sensory cortex and rIFC in humans and suggest th

55 e unique or overlapping representations in a primary sensory cortex and whether learning can modulate

56 Recent evidence suggests that neurons in primary sensory cortex arrange into competitive groups,

57 al connectivity between thalamus and lateral primary sensory cortex but reduced connectivity between

58 study and past measurements, which show that primary sensory cortex codes the whisking envelope as a

59 We conclude that the primary sensory cortex exerts a significant influence on

60 iated with increased activations in the left primary sensory cortex face area due to median nerve sti

61 cortex leg area during handgrip and the left primary sensory cortex face area during median nerve sti

62 dies have demonstrated the importance of the primary sensory cortex for the detection, discrimination

63 -sensory influences on neuronal responses in primary sensory cortex has been observed previously usin

64 Neurons in primary sensory cortex have diverse response properties,

65 clusion, SDs are inducible preferentially in primary sensory cortex in mice and most likely in humans

66 ultiple cubic millimeter regions of vibrissa primary sensory cortex in mouse.

67 robe, to image tissue oxygenation in the rat primary sensory cortex in response to sensory stimulatio

68 The primary sensory cortex is positioned at a confluence of

69 Such expectancy-driven modulation of primary sensory cortex may affect perceptions of externa

70 to the more likely stimulus modality and the primary sensory cortex may participate in the redistribu

71 anges in neuronal representation as early as primary sensory cortex mediate the perceptual advantage

72 the periphery may shape the organization of primary sensory cortex of other modalities as well.

73 The subgranular layers (layers 5 and 6) of primary sensory cortex provide corticofugal output to th

74 Layer (L)2 is a major output of primary sensory cortex that exhibits very sparse spiking

75 ive learning reconfigures neural circuits in primary sensory cortex to "learn" associative attributes

76 l responses, not only in their corresponding primary sensory cortex, but in other primary sensory cor

77 dulation in supragranular pyramidal cells in primary sensory cortex.

78 n of activity across neuronal populations in primary sensory cortex.

79 euronal representation, even at the level of primary sensory cortex.

80 epresentation of specific sensory content in primary sensory cortex.

81 sufficient to generate attractor dynamics in primary sensory cortex.

82 nd overlapping thalamic representations than primary sensory cortex.

83 memory is stored via synaptic plasticity in primary sensory cortex.

84 hroughout thalamic, limbic, and particularly primary sensory cortical areas in addition to known head

85 ome receptive field properties of neurons in primary sensory cortical areas.

86 rge degree of variability in body weight and primary sensory cortical field size, as defined by myelo

87 tion in brain size, cortical sheet size, and primary sensory cortical field sizes in the adult short-

88 tical plasticity extends beyond reshaping of primary sensory cortical fields to respecification of th

89 from the well-known patterns identified for primary sensory cortical regions.

90 l-[(3)H]glutamate-labeled NMDA receptors in primary sensory cortical regions.

91 l linkage of structure and function within a primary, sensory cortical area.

92 tical targets of these layers from the three primary sensory cortices (somatosensory, auditory, and v

93 that an anatomical trade-off exists between primary sensory cortices and anterior prefrontal cortex

94 elated impairment in the interaction between primary sensory cortices and subcortical regions suggest

95 odal interactions are commonplace across the primary sensory cortices and that some of the underlying

96 s with ASDs displayed stronger activation in primary sensory cortices and the amygdala (P < .05, corr

97 gh deficits in sensory function suggest that primary sensory cortices are affected by aging, our unde

98 Whether primary sensory cortices are essentially multisensory or

99 Response properties in primary sensory cortices are highly dependent on behavio

100 ing body of literature has demonstrated that primary sensory cortices are not exclusively unimodal, b

101 Altogether, the data show that primary sensory cortices can encode for cues predicting

102 Grouped primary sensory cortices defined network inputs, display

103 The primary sensory cortices display topographic organizatio

104 arietal cortices, and less activation in the primary sensory cortices during rest and sensory stimula

105 ther these laminar variations differ between primary sensory cortices has never been systematically a

106 The primary sensory cortices have been shown in recent years

107 In contrast, primary sensory cortices have few NFTs, even in late-sta

108 oscillations and direct connectivity between primary sensory cortices in visual-somatosensory interac

109 he feature-selective spatial organization of primary sensory cortices remains controversial.

110 nt studies show sustained neural activity in primary sensory cortices that can represent the timing o

111 e functional connectome and transitions from primary sensory cortices to higher-order brain systems.

112 g hierarchy; claustrum neurons projecting to primary sensory cortices were scant and restricted in di

113 high levels of catecholamines strengthen the primary sensory cortices, amygdala and striatum, rapidly

114 We found that, for all three primary sensory cortices, double-labeled cells were extr

115 ed almost exclusively through studies of the primary sensory cortices, for which principles of lamina

116 Sensory areas, especially primary sensory cortices, have long been held to be invo

117 e topographical representation seen in other primary sensory cortices, making it difficult to test th

118 In primary sensory cortices, thalamocortical (TC) inputs ca

119 In primary sensory cortices, there are two main sources of

120 network connectivity profiles downstream of primary sensory cortices, to investigate neural reorgani

121 These results indicate that primary sensory cortices, traditionally regarded as unis

122 the degree of clustering of plaques between primary sensory cortices, we found that the degree of pl

123 roanatomical precursors are predominantly in primary sensory cortices.

124 ponding primary sensory cortex, but in other primary sensory cortices.

125 mic systems that project to middle layers of primary sensory cortices.

126 o changes were found for connectivity within primary sensory cortices.

127 rhemispheric functional connectivity between primary sensory cortices.

128 me direct connections may exist even between primary sensory cortices.

129 rmation engages brain regions outside of the primary sensory cortices.

130 the cerebral cortex is well described in the primary sensory cortices.

131 A primary sensory difference was observed among wine sampl

132 eity in threshold sensitivity among the 5-30 primary sensory fibers innervating a single inner hair c

133 early sensory regions, in addition to their primary sensory functions, may be actively involved in p

134 Hence, we systematically studied primary sensory ganglia in rat to determine if the perip

135 V1 is primarily restricted to nociceptors in primary sensory ganglia, with minimal expression in a fe

136 cortex and some parts of the amygdala: 1) a primary sensory input population (intercalated pallium);

137 luences olfactory processing as early as the primary sensory input to the brain by modulating norepin

138 dLGN; however, their direct influence on the primary sensory input, namely retinogeniculate afferents

139 cal neurons receiving direct inputs from the primary sensory (lemniscal) pathway show the greatest de

140 direction of the Earth's magnetic field, the primary sensory mechanism behind this remarkable feat is

141 orrelation, with highest correlations across primary sensory-motor cortices (0.758, SD=0.152), signif

142 e suggest differences in coordination across primary sensory-motor cortices versus higher-order assoc

143 ivity between homotopic regions, contrasting primary sensory-motor cortices, unimodal association are

144 lations both may be utilized to localize the primary sensory-motor hand area in pre-surgical evaluati

145 is anchored by, at one end, regions serving primary sensory/motor functions and at the other end, tr

146 l surface-and are precisely equidistant-from primary sensory/motor morphological landmarks.

147 an alter functional properties of neurons in primary sensory neocortex but it is poorly understood ho

148 t selective synchronization between rIFC and primary sensory neocortex occurs in these frequency band

149 7-14 Hz) and beta (15-29 Hz) oscillations in primary sensory neocortical areas are enhanced in the re

150 tch sensation results from the excitation of primary sensory nerve endings in the skin, but the under

151 stained electrical activation of nociceptive primary sensory nerve fibres.

152 process of sensory transduction in cutaneous primary sensory nerve terminals, which converts thermal

153 ed increased functional connectivity between primary sensory networks and subcortical networks (thala

154 connectivity using seeds from two comparison primary sensory networks: visual and auditory networks.

155 In vitro primary sensory neuron culture systems were subjected to

156 ing molecules responsible for this change in primary sensory neuron excitability are still not well d

157 athways are segregated immediately after the primary sensory neuron in the chemotaxis circuit, and se

158 These data indicate that the primary sensory neuron population and its projections ma

159 Combining primary sensory neuron-specific GCaMP3 imaging with a tr

160 despite both forms occurring within the AWC primary sensory neuron.

161 cranial nerves (gV, gVII, gIV and gX) and in primary sensory neurons (i.e., Rohon-Beard neurons) in t

162 nctional interactions in a sub-population of primary sensory neurons (PSN).

163 They are expressed by primary sensory neurons and by glial cells in the centra

164 nish the resting membrane potential of mouse primary sensory neurons and cause cold-resistant hyperex

165 The lipid sensitizes TRPV1 ion channels in primary sensory neurons and causes increased frequency o

166 thesis of nonstructural proteins like KOR in primary sensory neurons and demonstrated a mechanism of

167 tion channel V1 (TRPV1) is also expressed in primary sensory neurons and detects painful stimuli such

168 unsaturated bonds, activate TRPA1 to excite primary sensory neurons and enteroendocrine cells.

169 ts high voltage-activated Ca(2+) channels in primary sensory neurons and excitatory synaptic transmis

170 alysis, metabolomics, and calcium imaging of primary sensory neurons and find no evidence of ligands

171 ted by hypochlorite and hydrogen peroxide in primary sensory neurons and heterologous cells.

172 Cbln2 expression tends to be more common in primary sensory neurons and in second-order sensory regi

173 avbeta(3) subunit augments HVACC activity in primary sensory neurons and nociceptive input to dorsal

174 c proteins that are exclusively expressed in primary sensory neurons and provoke pain in humans.

175 tor) that decreases glucose-induced death of primary sensory neurons and reverses numerous clinical i

176 t the molecules that mediate chronic itch in primary sensory neurons and skin.

177 suggest that DOR-KOR heteromers exist in rat primary sensory neurons and that KOR antagonists can act

178 xtensive localization of the EP3 receptor in primary sensory neurons and the spinal cord.

179 e receptor 7 (TLR7) was expressed in C-fiber primary sensory neurons and was important for inducing i

180 Dissociated primary sensory neurons are commonly used to study growt

181 isms mediating histamine-independent itch in primary sensory neurons are largely unknown.

182 Changes in primary sensory neurons are likely to contribute to the

183 Many primary sensory neurons are polymodal, responding to mul

184 This is what nociceptors do--these primary sensory neurons are specialized to detect intens

185 SDHN following the activation of nociceptive primary sensory neurons by burn injury, capsaicin applic

186 in HEK293 cells, Xenopus laevis oocytes, and primary sensory neurons by measuring Ca(2+) signals.

187 Using these methods, primary sensory neurons can be transfected with an effic

188 from the peripheral and central terminals of primary sensory neurons can critically contribute to noc

189 otection against glucose-induced toxicity of primary sensory neurons compared to 2.

190 lation in injured dorsal root ganglion (DRG) primary sensory neurons consistent with an early phase o

191 How primary sensory neurons contribute to persistent pain re

192 Primary sensory neurons convey information from the exte

193 The peripheral terminals of primary sensory neurons detect histamine and non-histami

194 ctoderm and neural folds in the region where primary sensory neurons develop.

195 sts that substance P (SP) is up-regulated in primary sensory neurons following axotomy and that this

196 rat trigeminal ganglion, the location of the primary sensory neurons for face sensation, specific sil

197 Primary sensory neurons form the interface between world

198 plitude of CXCL12-induced Ca(2+) response in primary sensory neurons from CCD mice was significantly

199 rent in prephenotype dorsal root ganglia and primary sensory neurons from dt mice, suggesting they ar

200 Adult Wistar rat primary sensory neurons grown at physiological pH 7.4, t

201 However, the function of ERK2 in primary sensory neurons has not been directly tested.

202 Viral tracing of the circuits engaged by primary sensory neurons has, however, been hampered by t

203 In cultured primary sensory neurons IL-31 triggered Ca(2+) release a

204 at induction of eIF2alpha phosphorylation in primary sensory neurons in a chronic inflammation pain m

205 TLR3 is expressed mainly by small-sized primary sensory neurons in dorsal root ganglions (DRGs)

206 lly to transcriptional repression of MORs in primary sensory neurons in neuropathic pain.

207 roid hormones, targeted TRPA1 in peptidergic primary sensory neurons in rodent and human cells expres

208 Primary sensory neurons in the DRG play an essential rol

209 al mechanical origin of tactile information, primary sensory neurons in the trigeminal ganglion (Vg)

210 have demonstrated that anandamide can excite primary sensory neurons in vitro via transient receptor

211 that the satellite glial cells that surround primary sensory neurons located in sensory ganglia of th

212 report highly effective gene transfer to the primary sensory neurons of the dorsal root ganglia (DRGs

213 Since primary sensory neurons of the dorsal root ganglion show

214 ature olfactory sensory neurons (mOSNs), the primary sensory neurons of the olfactory epithelium.

215 expressed on olfactory sensory neurons, the primary sensory neurons of the olfactory epithelium.

216 terminals of olfactory sensory neurons (the primary sensory neurons of the olfactory system, which p

217 l molecular detector of cold temperatures in primary sensory neurons of the somatosensory system.

218 s of these results under the assumption that primary sensory neurons of the trigeminal ganglion are s

219 Our findings suggest that MyD88 in primary sensory neurons plays an active role in regulati

220 in controlling the intrinsic excitability of primary sensory neurons possibly via Ca(2+)-activated SK

221 When the axons of primary sensory neurons project into the embryonic mamma

222 Thus, the primary sensory neurons responsive to beta-alanine are l

223 A recent study in primary sensory neurons shows that electrical activity--

224 causative factor may be that only 50-60% of primary sensory neurons succeed in regenerating axons af

225 associated with inflammation and trauma, but primary sensory neurons that convey the sensation of acu

226 Thus, PKD1, PKD2, and PKD3 are expressed in primary sensory neurons that mediate neurogenic inflamma

227 dorants by olfactory sensory neurons (OSNs), primary sensory neurons that physically contact odor mol

228 ve protease-activated receptor 2 (PAR(2)) on primary sensory neurons to induce neurogenic inflammatio

229 t acts as a coincidence detector that allows primary sensory neurons to integrate information from ne

230 The principle by which unmyelinated primary sensory neurons transducing thermal, itch and pa

231 effects were mediated by the transduction of primary sensory neurons via transport of FIV vectors fro

232 e and the frequency of transport in axons of primary sensory neurons were both reduced.

233 ive ligand-gated channel highly expressed in primary sensory neurons where it mediates nociception.

234 olfactory abilities likely originates in the primary sensory neurons, and suggest that hormonal modul

235 In cultured rodent primary sensory neurons, BoNT-A decreased the proportion

236 , wheat germ agglutinin (WGA), is induced in primary sensory neurons, but only after transection of t

237 ct connections between the motor neurons and primary sensory neurons, indicating that further study w

238 axonal injury, concentrations of BH4 rose in primary sensory neurons, owing to upregulation of GCH1.

239 sfer is a feature shared by other classes of primary sensory neurons, permitting the identification a

240 f WNT/frizzled/beta-catenin signaling in the primary sensory neurons, the spinal dorsal horn neurons,

241 espite prominent expression of NMDARs in DRG primary sensory neurons, the unique role of peripheral N

242 in sodium channel subunit Nav1.8-expressing primary sensory neurons, to examine the unique role of n

243 vents might also impact central processes of primary sensory neurons, triggering in nociceptors a hyp

244 C(50) = 0.1 nm) and TRPA1 (IC(50) = 2 nm) in primary sensory neurons, whereas RvE1 and RvD1 selective

245 nnections between cochlear sensory cells and primary sensory neurons, without loss of the sensory cel

246 ervous system, including the spinal cord and primary sensory neurons.

247 Kv7.2 activity increases the excitability of primary sensory neurons.

248 nduction and maintenance of sensitization of primary sensory neurons.

249 led receptor expressed by a subpopulation of primary sensory neurons.

250 pression and function of Cavbeta subunits in primary sensory neurons.

251 s activates the D1-like dopamine receptor on primary sensory neurons.

252 nd murine TRPM8 channels, including those on primary sensory neurons.

253 riety of receptors and channels expressed by primary sensory neurons.

254 d its ligand ephrinB2 in the dorsal horn and primary sensory neurons.

255 voltage-gated calcium and sodium currents in primary sensory neurons.

256 ally expressed in small-diameter nociceptive primary sensory neurons.

257 iciency in coding that has not been found in primary sensory neurons.

258 nt receptor potential vanilloid I (TRPV1) in primary sensory neurons.

259 spinal synapses made by axotomized group IA primary sensory neurons.

260 us particles in axons following infection of primary sensory neurons.

261 uired for efficient transcription of TrkA in primary sensory neurons.

262 irect synaptic input from both pain and itch primary sensory neurons.

263 erve injury on the regenerative state of the primary sensory neurons.

264 between neurotropic herpesviruses and murine primary sensory neurons.

265 el Nav1.8 is expressed almost exclusively in primary sensory neurons.

266 r 1 TGR5 (encoded by GPBAR1) is expressed by primary sensory neurons; its activation induces neuronal

267 of selectively blocking the excitability of primary sensory nociceptor (pain-sensing) neurons by int

268 Primary sensory nuclei of the thalamus process and relay

269 Cilia of olfactory sensory neurons are the primary sensory organelles for olfaction.

270 ostsynaptic responsiveness highlight how the primary sensory organs have been optimized and can be mo

271 with sensitization within and outside of the primary sensory pathway.

272 izations of AstA- and TK-positive neurons in primary sensory processing centers and higher order inte

273 e N100 or P200 components, indicating intact primary sensory processing.

274 The primary sensory receptive field maps of the olfactory sy

275 The inner hair cells (IHCs) are the primary sensory receptors adapted for rapid auditory sig

276 Inner hair cells (IHCs), the primary sensory receptors of the mammalian cochlea, fire

277 aring and adult inner hair cells (IHCs), the primary sensory receptors of the mammalian cochlea, is m

278 association areas, but was also seen in the primary sensory region investigated.

279 and that remodeling rates are similar across primary sensory regions of different modalities, but may

280 It is generally held that non-primary sensory regions of the brain have a strong impac

281 ined significantly more non-neurons than the primary sensory regions.

282 t emotional learning of odor cues alters the primary sensory representation within the nose and brain

283 orms not only the information content of the primary sensory representation, but also its underlying

284 and suggest that synchrony between rIFC and primary sensory representations plays a role in the inhi

285 This linkage of amygdalar and LC output to primary sensory signaling may have implications for affe

286 These results suggest that primary sensory structures develop through the concurren

287 surfaces of the frontal lobe, extending into primary sensory, superior parietal, and anterior superio

288 d by inner hair cells (IHCs), which form the primary sensory synapse.

289 Whether experience can alter the strength of primary sensory synapses remains mostly unknown.

290 ity of prey species that use vision as their primary sensory system and suppressed the activity of sp

291 The primary sensory system requires the integrated function

292 via their long-range feedback projections to primary sensory thalamic nuclei.

293 ensory discrimination is believed to require primary sensory thalamus and cortex for early stimulus i

294 Here we report that neurons in the primary sensory thalamus of the mouse vibrissal system (

295 ew example of cross-modal integration in the primary sensory thalamus.

296 t to anterograde transsynaptic transfer from primary sensory to spinal target neurons, and can deline

297 AWC-sensed odors by acting downstream of the primary sensory transduction.

298 lasting transduction of the vast majority of primary sensory vagal neurons without transduction of pa

299 chanical signals so generated constitute the primary sensory variables upon which these animals base

300 alities, but may differ in magnitude between primary sensory versus higher cortical areas.